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        <title>American Journal of Nephrology via MedWorm.com</title>
        <description>MedWorm.com provides a medical RSS filtering service. Over 6000 RSS medical sources are combined and output via different filters. This feed contains the latest items from the 'American Journal of Nephrology' source.</description>
        <link><![CDATA[http://www.medworm.com/rss/search.php?qu=American+Journal+of+Nephrology&t=American+Journal+of+Nephrology&s=Search&f=source]]></link>
        <lastBuildDate>Sat, 13 Mar 2010 15:41:28 +0100</lastBuildDate>
        <item>
            <title>Diabetic kidney disease: act now or pay later.</title>
            <link>http://www.medworm.com/index.php?rid=3351256&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20213919%26dopt%3DAbstract</link>
            <description>Authors: Atkins RC, Zimmet P
    
    PMID: 20213919 [PubMed - in process] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3351256</comments>
            <pubDate>Thu, 11 Mar 2010 11:32:03 +0100</pubDate>
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            <title>Mesangial Cells Initiate Compensatory Tubular Cell Hypertrophy.</title>
            <link>http://www.medworm.com/index.php?rid=3284084&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20160436%26dopt%3DAbstract</link>
            <description>Authors: Sinuani I, Beberashvili I, Averbukh Z, Cohn M, Gitelman I, Weissgarten J
    Unilateral nephrectomy results in compensatory renal growth, in which both the size and the functional capacity of the remaining kidney are increased. The functional adaptation to the removal of the contralateral kidney consists mostly of an increase in the glomerular filtration rate of the remaining kidney, and hypertrophy of cells comprising the nephron, mainly of the proximal tubular cells. Although the phenomenon of single kidney hypertrophy has been known for the past thousand years and despite intensive research over the past century, the mechanism of this process still remains unclear. The present article reviews the role of mesangial cells in compensatory renal hypertrophy.
    PMID: 20160436 [Pub...</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3284084</comments>
            <pubDate>Wed, 17 Feb 2010 00:00:00 +0100</pubDate>
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        <item>
            <title>Curcumin Inhibits Transforming Growth Factor-beta Activity via Inhibition of Smad Signaling in HK-2 Cells.</title>
            <link>http://www.medworm.com/index.php?rid=3284083&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20160437%26dopt%3DAbstract</link>
            <description>Conclusions: Curcumin blocks the profibrotic actions of TGF-beta on HK-2 cells through the down-regulation of the Smad signaling pathway, and curcumin may have some similar effect as serine/threonine protein phosphatases. Our findings suggest curcumin as a potential candidate for treatment of tubulointerstitial fibrosis.
    PMID: 20160437 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3284083</comments>
            <pubDate>Wed, 17 Feb 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3284083</guid>        </item>
        <item>
            <title>Mesangial Cells Initiate Compensatory Tubular Cell Hypertrophy</title>
            <link>http://www.medworm.com/index.php?rid=3281514&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D287229</link>
            <description>Am J Nephrol 2010;31:326331 (DOI:10.1159/000287229) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3281514</comments>
            <pubDate>Tue, 16 Feb 2010 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3281514</guid>        </item>
        <item>
            <title>Curcumin Inhibits Transforming Growth Factor-ß Activity via Inhibition of Smad Signaling in HK-2 Cells</title>
            <link>http://www.medworm.com/index.php?rid=3281513&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D287230</link>
            <description>Am J Nephrol 2010;31:332341 (DOI:10.1159/000287230) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3281513</comments>
            <pubDate>Tue, 16 Feb 2010 23:00:00 +0100</pubDate>
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        <item>
            <title>Urinary Angiotensinogen Accurately Reflects Intrarenal Renin-Angiotensin System Activity.</title>
            <link>http://www.medworm.com/index.php?rid=3284085&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20160435%26dopt%3DAbstract</link>
            <description>Conclusion: These data indicate that UAGT are increased in chronic glomerulonephritis patients and treatment with RAS blockers suppressed UAGT. The efficacy of RAS blockade to reduce the intrarenal RAS activity can be confirmed by measurement of UAGT in chronic glomerulonephritis patients.
    PMID: 20160435 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3284085</comments>
            <pubDate>Mon, 15 Feb 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3284085</guid>        </item>
        <item>
            <title>Effects of Interleukin-6 on Depression Risk in Dialysis Patients.</title>
            <link>http://www.medworm.com/index.php?rid=3284086&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20160434%26dopt%3DAbstract</link>
            <description>Conclusions: IL-6 may play a role in the pathogenesis of depression in patients with ESKD.
    PMID: 20160434 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3284086</comments>
            <pubDate>Sat, 13 Feb 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3284086</guid>        </item>
        <item>
            <title>Novel Markers of Left Ventricular Hypertrophy in Uremia.</title>
            <link>http://www.medworm.com/index.php?rid=3244148&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20130393%26dopt%3DAbstract</link>
            <description>Conclusions: These data suggest left ventricular remodeling consists of cardiomyocyte hypertrophy and interstitial cell proliferation, but not cardiomyocyte proliferation. p27 and cyclin D2 may play important roles in the development of LVH. In addition, phosphorus can be an independent risk factor for the development of LVH.
    PMID: 20130393 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3244148</comments>
            <pubDate>Sat, 30 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3244148</guid>        </item>
        <item>
            <title>Contrasting Effects of Steroids and Mizoribine on Macrophage Activation and Glomerular Lesions in Rat Thy-1 Mesangial Proliferative Glomerulonephritis.</title>
            <link>http://www.medworm.com/index.php?rid=3223716&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20110667%26dopt%3DAbstract</link>
            <description>Conclusions: Glucocorticoid induced alternative macrophage activation failed to ameliorate rat mesangial proliferative glomerulonephritis, whereas MZR suppression of this disease model was attributed, in part, to inhibition of M1-like pro-inflammatory macrophage activation.
    PMID: 20110667 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3223716</comments>
            <pubDate>Wed, 27 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3223716</guid>        </item>
        <item>
            <title>Chronic Sildenafil Treatment Corrects Endothelial Dysfunction and Improves Hypertension.</title>
            <link>http://www.medworm.com/index.php?rid=3223715&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20110668%26dopt%3DAbstract</link>
            <description>Conclusions: These results demonstrate beneficial effects of long-term PDE-5 inhibition in SHR and suggest that its use as an adjunct therapy in essential hypertension should be investigated.
    PMID: 20110668 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3223715</comments>
            <pubDate>Wed, 27 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3223715</guid>        </item>
        <item>
            <title>Immobilized Kidney 28-kDa Endostatin-Related (KES28kDa) Fragment Promotes Endothelial Cell Survival.</title>
            <link>http://www.medworm.com/index.php?rid=3223718&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20110665%26dopt%3DAbstract</link>
            <description>Conclusion: These data demonstrate the local synthesis of a 28-kDa ES-related fragment following AKI and suggest its role in endothelium survival.
    PMID: 20110665 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3223718</comments>
            <pubDate>Mon, 25 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3223718</guid>        </item>
        <item>
            <title>Nebivolol Attenuates Maladaptive Proximal Tubule Remodeling in Transgenic Rats.</title>
            <link>http://www.medworm.com/index.php?rid=3223717&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20110666%26dopt%3DAbstract</link>
            <description>Conclusion: These observations support the notion that nebivolol may improve PTC reabsorption of albumin and other glomerular filtered small molecular weight proteins in association with the attenuation of oxidative stress, tubulointerstitial injury and fibrosis in this rat model of metabolic kidney disease.
    PMID: 20110666 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3223717</comments>
            <pubDate>Mon, 25 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3223717</guid>        </item>
        <item>
            <title>Forging ahead with lessons from the past.</title>
            <link>http://www.medworm.com/index.php?rid=3200706&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20093805%26dopt%3DAbstract</link>
            <description>Authors: Bakris GL
    
    PMID: 20093805 [PubMed - in process] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3200706</comments>
            <pubDate>Sat, 23 Jan 2010 17:10:03 +0100</pubDate>
            <guid isPermaLink="false">3200706</guid>        </item>
        <item>
            <title>Leukocyte Toll-Like Receptor Expression in End-Stage Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=3200707&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20090311%26dopt%3DAbstract</link>
            <description>Conclusions: Monocyte TLR2 and TLR4 and neutrophil TLR4 expressions and TLR4 activity are increased hemodialysis patients, representing another dimension of ESRD-associated inflammation.
    PMID: 20090311 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3200707</comments>
            <pubDate>Fri, 15 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3200707</guid>        </item>
        <item>
            <title>New-Onset Diabetes after Hemodialysis Initiation: Impact on Survival.</title>
            <link>http://www.medworm.com/index.php?rid=3176626&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20068288%26dopt%3DAbstract</link>
            <description>Conclusion: The USRDS showed a high incidence of NODAD, associated with significantly higher mortality compared to those who did not develop NODAD. The mechanism of NODAD needs to be explored further in experimental and clinical studies.
    PMID: 20068288 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3176626</comments>
            <pubDate>Wed, 13 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3176626</guid>        </item>
        <item>
            <title>Involvement of Alpha-Klotho and Fibroblast Growth Factor Receptor in the Development of Secondary Hyperparathyroidism.</title>
            <link>http://www.medworm.com/index.php?rid=3176627&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20068287%26dopt%3DAbstract</link>
            <description>Conclusion: Decreased expression of alpha-Klotho and FGFR1c in parallel with CaR expression and parathyroid cell growth may be involved in the pathogenesis of SHPT.
    PMID: 20068287 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3176627</comments>
            <pubDate>Wed, 06 Jan 2010 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3176627</guid>        </item>
        <item>
            <title>Predictors of Carotid Artery Intima-Media Thickness in Chronic Kidney Disease and Kidney Transplant Patients without Overt Cardiovascular Disease.</title>
            <link>http://www.medworm.com/index.php?rid=3176629&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20068285%26dopt%3DAbstract</link>
            <description>Conclusion: Our data does not exclude IMT as a predictor of mortality in CKD, but suggests that other etiologies than atherosclerosis may be more important in determining IMT levels in the population with CKD.
    PMID: 20068285 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3176629</comments>
            <pubDate>Thu, 31 Dec 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3176629</guid>        </item>
        <item>
            <title>Effect of Chronic Kidney Disease and Comorbid Conditions on Health Care Costs: A 10-Year Observational Study in a General Population.</title>
            <link>http://www.medworm.com/index.php?rid=3176628&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20068286%26dopt%3DAbstract</link>
            <description>Conclusions: The direct effect of CKD on costs is modified by comorbid conditions. Therefore, early treatment of CKD and its precipitous factors may save future health care costs.
    PMID: 20068286 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3176628</comments>
            <pubDate>Thu, 31 Dec 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3176628</guid>        </item>
        <item>
            <title>Use of Body Surface Area for Assessing Extracellular Fluid Volume and Glomerular Filtration Rate in Obesity.</title>
            <link>http://www.medworm.com/index.php?rid=3119214&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20029177%26dopt%3DAbstract</link>
            <description>Conclusions: For subjects of similar BSA, a high BMI decreases ECV (the 'obesity effect'). Subjects with high BMI generally have high BSA, which tends to increase ECV/BSA because of the disproportionate relation between ECV and BSA (the 'BSA effect'). These opposing effects serendipitously and erroneously create the impression that BSA is suitable for scaling ECV (and by implication, GFR) in obesity.
    PMID: 20029177 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3119214</comments>
            <pubDate>Tue, 22 Dec 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3119214</guid>        </item>
        <item>
            <title>Mechanisms and Control of Protein Translation in the Kidney.</title>
            <link>http://www.medworm.com/index.php?rid=3119216&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20029175%26dopt%3DAbstract</link>
            <description>Authors: Schaeffer V, Abrass CK
    Translational control of protein synthesis is critical for cell division, homeostasis and survival. Recent data indicate that dysregulation of protein synthesis that leads to either increased or decreased expression of specific proteins contributes to the manifestations of various kidney diseases. Most of the control of protein synthesis occurs in the first or initiation phase, which is also the most complicated. Following the initiation phase is the elongation phase where the peptide chain is formed. RNA transcripts are released from ribosomes after the termination phase. Transcripts can be translated in a cap-dependent or cap-independent manner. The mTOR (mammalian target of rapamycin) cascade regulates translation of most cap-dependent transcripts at ...</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3119216</comments>
            <pubDate>Mon, 21 Dec 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3119216</guid>        </item>
        <item>
            <title>The Association of African Ancestry and Elevated Creatinine in the Coronary Artery Risk Development in Young Adults (CARDIA) Study.</title>
            <link>http://www.medworm.com/index.php?rid=3119215&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20029176%26dopt%3DAbstract</link>
            <description>Authors: Peralta CA, Risch N, Lin F, Shlipak MG, Reiner A, Ziv E, Tang H, Siscovick D, Bibbins-Domingo K
    Whether genetic factors account for differences in early kidney disease among blacks in a young healthy population is not well known. We evaluated the association of self-reported race and genetic African ancestry with elevated creatinine (&amp;gt;/=1.3 mg/dl for men, &amp;gt;/=1.1 mg/dl for women) among 3,113 black and white participants in the Coronary Artery Risk Development in Young Adults (CARDIA) study, ages 38-50 years. We estimated individual African ancestry using 42 ancestry informative markers. Blacks were more likely to have elevated creatinine than whites, and this effect was more pronounced in men: adjusted odds ratio (AOR) for black versus white men = 7.03, 4.15-11.91; AOR fo...</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3119215</comments>
            <pubDate>Mon, 21 Dec 2009 00:00:00 +0100</pubDate>
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        <item>
            <title>N-Acetylcysteine Amide Protects Renal Proximal Tubular Epithelial Cells against Iohexol-Induced Apoptosis by Blocking p38 MAPK and iNOS Signaling.</title>
            <link>http://www.medworm.com/index.php?rid=3102363&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20016144%26dopt%3DAbstract</link>
            <description>Conclusions: This study demonstrates that apoptosis occurs during CIN. Apoptosis is associated with activations of p38 MAPK and iNOS. Pretreatment with the antioxidant NACA could prevent IH-induced cell death by blocking the p38 MAPK/iNOS signaling pathway.
    PMID: 20016144 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3102363</comments>
            <pubDate>Thu, 17 Dec 2009 00:00:00 +0100</pubDate>
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        <item>
            <title>N-Acetylcysteine Amide Protects Renal Proximal Tubular Epithelial Cells against Iohexol-Induced Apoptosis by Blocking p38 MAPK and iNOS Signaling</title>
            <link>http://www.medworm.com/index.php?rid=3098455&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D268161</link>
            <description>Am J Nephrol 2010;31:178-188 (DOI:10.1159/000268161) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3098455</comments>
            <pubDate>Wed, 16 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3098455</guid>        </item>
        <item>
            <title>Alterations in the Renin-Angiotensin System in a Rat Model of Human Preeclampsia.</title>
            <link>http://www.medworm.com/index.php?rid=3102364&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20016143%26dopt%3DAbstract</link>
            <description>Conclusions: (1) The peripheral RAS is downregulated, and the uteroplacental RAS is upregulated in this rat model of preeclampsia; (2) MBG is involved in the causation of these alterations, and (3) RBG prevents these changes.
    PMID: 20016143 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3102364</comments>
            <pubDate>Wed, 16 Dec 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3102364</guid>        </item>
        <item>
            <title>Contents Vol. 30, 2009</title>
            <link>http://www.medworm.com/index.php?rid=3091055&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D268236</link>
            <description>Am J Nephrol 2009;30:I-VI (DOI:10.1159/000268236) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3091055</comments>
            <pubDate>Mon, 14 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3091055</guid>        </item>
        <item>
            <title>Consultants for the American Journal of Nephrology 2009</title>
            <link>http://www.medworm.com/index.php?rid=3098456&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D268111</link>
            <description>Am J Nephrol 2009;30:564 (DOI:10.1159/000268111) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3098456</comments>
            <pubDate>Sun, 13 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3098456</guid>        </item>
        <item>
            <title>Consultatns for the American Journal of Nephrology 2009</title>
            <link>http://www.medworm.com/index.php?rid=3091058&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D268111</link>
            <description>Am J Nephrol 2009;30:564 (DOI:10.1159/000268111) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3091058</comments>
            <pubDate>Sun, 13 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3091058</guid>        </item>
        <item>
            <title>Author Index Vol. 30, 2009</title>
            <link>http://www.medworm.com/index.php?rid=3091057&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D268112</link>
            <description>Am J Nephrol 2009;30:566-568 (DOI:10.1159/000268112) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3091057</comments>
            <pubDate>Sun, 13 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3091057</guid>        </item>
        <item>
            <title>Subject Index Vol. 30, 2009</title>
            <link>http://www.medworm.com/index.php?rid=3091056&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D268113</link>
            <description>Am J Nephrol 2009;30:569-570 (DOI:10.1159/000268113) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3091056</comments>
            <pubDate>Sun, 13 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3091056</guid>        </item>
        <item>
            <title>Differential Effects of Paricalcitol and Calcitriol on Intestinal Calcium Absorption in Hemodialysis Patients.</title>
            <link>http://www.medworm.com/index.php?rid=3102365&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20016142%26dopt%3DAbstract</link>
            <description>Conclusion: Overall, paricalcitol-treated patients absorbed approximately 14% less Ca compared with calcitriol-treated patients with similar effects on PTH. In hemodialysis patients, paricalcitol may provide a benefit by lowering the Ca available for removal by dialysis and/or for deposit in bone or soft tissues.
    PMID: 20016142 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3102365</comments>
            <pubDate>Fri, 11 Dec 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3102365</guid>        </item>
        <item>
            <title>Differential Effects of Paricalcitol and Calcitriol on Intestinal Calcium Absorption in Hemodialysis Patients</title>
            <link>http://www.medworm.com/index.php?rid=3081063&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D266204</link>
            <description>Am J Nephrol 2010;31:165-170 (DOI:10.1159/000266204) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3081063</comments>
            <pubDate>Thu, 10 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3081063</guid>        </item>
        <item>
            <title>An Oral Sorbent, AST-120, Increases Klotho Expression and Inhibits Cell Senescence in the Kidney of Uremic Rats.</title>
            <link>http://www.medworm.com/index.php?rid=3054557&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19955715%26dopt%3DAbstract</link>
            <description>Conclusions: AST-120 increased Klotho expression, and inhibited cell senescence in the kidney of CRF rats, probably by alleviating indoxyl sulfate overload on the kidney.
    PMID: 19955715 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3054557</comments>
            <pubDate>Thu, 03 Dec 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3054557</guid>        </item>
        <item>
            <title>An Oral Sorbent, AST-120, Increases Klotho Expression and Inhibits Cell Senescence in the Kidney of Uremic Rats</title>
            <link>http://www.medworm.com/index.php?rid=3054492&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D264634</link>
            <description>Am J Nephrol 2010;31:160-164 (DOI:10.1159/000264634) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3054492</comments>
            <pubDate>Wed, 02 Dec 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3054492</guid>        </item>
        <item>
            <title>Changes of Renal AT(1)/AT(2) Receptors and Structures in Ovine Fetuses following Exposure to Long-Term Hypoxia.</title>
            <link>http://www.medworm.com/index.php?rid=3014635&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19923800%26dopt%3DAbstract</link>
            <description>Conclusion: The results demonstrate an adverse effect of chronic hypoxia on renal AT(1)R and AT(2)R expression and functions in the fetus, suggesting a possible role of fetal hypoxia in the programming of renal diseases in fetal origins.
    PMID: 19923800 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3014635</comments>
            <pubDate>Wed, 18 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3014635</guid>        </item>
        <item>
            <title>Measurement Error in Estimated GFR Slopes across Transplant Chronic Kidney Disease Stages.</title>
            <link>http://www.medworm.com/index.php?rid=3014634&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19923801%26dopt%3DAbstract</link>
            <description>Conclusions: Transplant GFR slope prediction is affected by the degree of renal dysfunction. Errors in slope prediction are much higher in those with better function and thus add another limitation for eGFR use in longitudinal studies on progressive graft dysfunction.
    PMID: 19923801 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3014634</comments>
            <pubDate>Wed, 18 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3014634</guid>        </item>
        <item>
            <title>Prospective Evaluation of Waist Circumference and Visceral Adipose Tissue in Patients with Chronic Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=3014645&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19923795%26dopt%3DAbstract</link>
            <description>Conclusion: Anthropometric measurement of WC is poorly correlated with changes in VAT measured by CT in nondialysis-dependent CKD patients. Therefore, caution should be taken when using WC as a surrogate marker of VAT changes in this population.
    PMID: 19923795 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3014645</comments>
            <pubDate>Tue, 17 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3014645</guid>        </item>
        <item>
            <title>Anti-Glycation and Anti-Albuminuric Effects of GLY-230 in Human Diabetes.</title>
            <link>http://www.medworm.com/index.php?rid=3014644&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19923796%26dopt%3DAbstract</link>
            <description>Conclusions: This first-in-diabetic man study indicates that GLY-230 lowers glycated albumin and that this decrease is associated with a reduction in urine albumin excretion in patients with preexisting microalbuminuria. These data encourage further evaluation of GLY-230 in diabetic renal dysfunction.
    PMID: 19923796 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3014644</comments>
            <pubDate>Tue, 17 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3014644</guid>        </item>
        <item>
            <title>The Modifier Protein of Glyceraldehyde-3-Phosphate Dehydrogenase Reduces Free Radical-Mediated Renal Damage in a Rat Model of Acute Kidney Injury.</title>
            <link>http://www.medworm.com/index.php?rid=3014643&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19923797%26dopt%3DAbstract</link>
            <description>Conclusion: Using a gentamicin model of AKI, MP was able to reduce free radical production in kidney tissue and in the circulation, thus preventing oxidant injury and minimizing damage in renal epithelial cells.
    PMID: 19923797 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3014643</comments>
            <pubDate>Tue, 17 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3014643</guid>        </item>
        <item>
            <title>JNK/ATF2 Pathway Is Involved in Iodinated Contrast Media-Induced Apoptosis.</title>
            <link>http://www.medworm.com/index.php?rid=3014642&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19923798%26dopt%3DAbstract</link>
            <description>Conclusions: Differential activation of ATF2 by different contrast media provides a new insight into the mechanism and prevention of contrast-induced nephropathy.
    PMID: 19923798 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3014642</comments>
            <pubDate>Tue, 17 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3014642</guid>        </item>
        <item>
            <title>Role of Cellular Cholesterol in Pharmacologic Preconditioning with Cyclosporine in Experimental Kidney Transplantation.</title>
            <link>http://www.medworm.com/index.php?rid=3014636&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19923799%26dopt%3DAbstract</link>
            <description>Conclusion: CsA preconditioning leads to better outcomes in kidney transplantation and is associated with up-regulation of renal FC content. The latter may then contribute to acquired cytoresistance, possibly by stabilizing the plasma membrane. Thus, use of statins around the time of transplantation may need to be evaluated until further studies are conducted to determine the clinical relevance of this observation.
    PMID: 19923799 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3014636</comments>
            <pubDate>Tue, 17 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">3014636</guid>        </item>
        <item>
            <title>Prospective Evaluation of Waist Circumference and Visceral Adipose Tissue in Patients with Chronic Kidney Disease</title>
            <link>http://www.medworm.com/index.php?rid=3001888&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D259896</link>
            <description>Am J Nephrol 2010;31:104-109 (DOI:10.1159/000259896) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3001888</comments>
            <pubDate>Mon, 16 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3001888</guid>        </item>
        <item>
            <title>Anti-Glycation and Anti-Albuminuric Effects of GLY-230 in Human Diabetes</title>
            <link>http://www.medworm.com/index.php?rid=3001887&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D259897</link>
            <description>Am J Nephrol 2010;31:110-116 (DOI:10.1159/000259897) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3001887</comments>
            <pubDate>Mon, 16 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3001887</guid>        </item>
        <item>
            <title>The Modifier Protein of Glyceraldehyde-3-Phosphate Dehydrogenase Reduces Free Radical-Mediated Renal Damage in a Rat Model of Acute Kidney Injury</title>
            <link>http://www.medworm.com/index.php?rid=3001886&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D259898</link>
            <description>Am J Nephrol 2010;31:117-124 (DOI:10.1159/000259898) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3001886</comments>
            <pubDate>Mon, 16 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3001886</guid>        </item>
        <item>
            <title>JNK/ATF2 Pathway Is Involved in Iodinated Contrast Media-Induced Apoptosis</title>
            <link>http://www.medworm.com/index.php?rid=3001885&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D259899</link>
            <description>Am J Nephrol 2010;31:125-133 (DOI:10.1159/000259899) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3001885</comments>
            <pubDate>Mon, 16 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3001885</guid>        </item>
        <item>
            <title>Role of Cellular Cholesterol in Pharmacologic Preconditioning with Cyclosporine in Experimental Kidney Transplantation</title>
            <link>http://www.medworm.com/index.php?rid=3001884&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D259900</link>
            <description>Am J Nephrol 2010;31:134-140 (DOI:10.1159/000259900) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3001884</comments>
            <pubDate>Mon, 16 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3001884</guid>        </item>
        <item>
            <title>CYP3A4 and CYP3A5 Polymorphisms and Blood Pressure Response to Amlodipine among African-American Men and Women with Early Hypertensive Renal Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2989963&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19907160%26dopt%3DAbstract</link>
            <description>Conclusions: Our findings suggest that blood pressure response to amlodipine among high-risk African-Americans appears to be determined by CYP3A4 genotypes, and sex specificity may be an important consideration. Clinical applications of CYP3A4 genotype testing for individualized treatment regimens warrant further study.
    PMID: 19907160 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2989963</comments>
            <pubDate>Thu, 12 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2989963</guid>        </item>
        <item>
            <title>Age- and Gender-Dependent Correlations between Body Composition and Chronic Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2989965&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19907158%26dopt%3DAbstract</link>
            <description>Conclusions: The associations between BMI, body composition and CKD differed according to age and gender stratifications.
    PMID: 19907158 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2989965</comments>
            <pubDate>Wed, 11 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2989965</guid>        </item>
        <item>
            <title>The Death of John Paul Jones and Resurrection as 'Father of the US Navy'</title>
            <link>http://www.medworm.com/index.php?rid=2989964&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19907159%26dopt%3DAbstract</link>
            <description>Authors: Weir MR, Bogle LL, Mackowiak PA
    John Paul Jones, 'father of the US Navy', died under mysterious circumstances on July 18, 1792. Although his remains were resurrected and autopsied 113 years later, the cause of his death has yet to be explained fully. In this detailed review of his clinical history and autopsy findings, we conclude, as have others, that Jones died of kidney failure of undermined etiology. Although 'interstitial nephritis' was diagnosed by the prosectors who performed his autopsy, we believe glomerulopathy (possibly IgA nephropathy) was more likely responsible for his kidney failure and that his death was due to a cardiac arrhythmia or some other complication of uremia.
    PMID: 19907159 [PubMed - as supplied by publisher] (Source: American Journal of Nephrolog...</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2989964</comments>
            <pubDate>Wed, 11 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2989964</guid>        </item>
        <item>
            <title>The Death of John Paul Jones and Resurrection as Father of the US Navy</title>
            <link>http://www.medworm.com/index.php?rid=3121508&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D258661</link>
            <description>Am J Nephrol 2010;31:90-94 (DOI:10.1159/000258661) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3121508</comments>
            <pubDate>Tue, 10 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3121508</guid>        </item>
        <item>
            <title>Age- and Gender-Dependent Correlations between Body Composition and Chronic Kidney Disease</title>
            <link>http://www.medworm.com/index.php?rid=2983000&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D258660</link>
            <description>Am J Nephrol 2010;31:83-89 (DOI:10.1159/000258660) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2983000</comments>
            <pubDate>Tue, 10 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2983000</guid>        </item>
        <item>
            <title>The Death of John Paul Jones and Resurrection as 'Father of the US Navy'</title>
            <link>http://www.medworm.com/index.php?rid=2982999&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D258661</link>
            <description>Am J Nephrol 2010;31:90-94 (DOI:10.1159/000258661) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2982999</comments>
            <pubDate>Tue, 10 Nov 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Retraction Statement.</title>
            <link>http://www.medworm.com/index.php?rid=2971871&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19893299%26dopt%3DAbstract</link>
            <description>Authors: Bakris GL
    
    PMID: 19893299 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2971871</comments>
            <pubDate>Thu, 05 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2971871</guid>        </item>
        <item>
            <title>Aldosterone Promotes Fibronectin Synthesis in Rat Mesangial Cells via ERK1/2-Stimulated Na-H(+) Exchanger Isoform 1.</title>
            <link>http://www.medworm.com/index.php?rid=2971870&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19893300%26dopt%3DAbstract</link>
            <description>Conclusion: The present study suggests that NHE1 may play an important role in aldosterone-mediated fibronectin accumulation in rat mesangial cells via the ERK1/2 pathway.
    PMID: 19893300 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2971870</comments>
            <pubDate>Thu, 05 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2971870</guid>        </item>
        <item>
            <title>Aldosterone Promotes Fibronectin Synthesis in Rat Mesangial Cells via ERK1/2-Stimulated Na + -H +  Exchanger Isoform 1</title>
            <link>http://www.medworm.com/index.php?rid=2965336&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D256665</link>
            <description>Am J Nephrol 2010;31:75-82 (DOI:10.1159/000256665) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2965336</comments>
            <pubDate>Wed, 04 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2965336</guid>        </item>
        <item>
            <title>Retraction Statement</title>
            <link>http://www.medworm.com/index.php?rid=2965335&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D257391</link>
            <description>Am J Nephrol 2009;30:563 (DOI:10.1159/000257391) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2965335</comments>
            <pubDate>Wed, 04 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2965335</guid>        </item>
        <item>
            <title>Estimating Glomerular Filtration Rate Using the New CKD-EPI Equation and Other Equations in Patients with Autosomal Dominant Polycystic Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2967792&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19887788%26dopt%3DAbstract</link>
            <description>Conclusion: For estimating GFR in ADPKD patients the MDRD equation with cystatin C incorporated had the best performance. The CKD-EPI or the Cockcroft-Gault equations showed better performance compared to the 4-variable MDRD equation.
    PMID: 19887788 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2967792</comments>
            <pubDate>Wed, 04 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2967792</guid>        </item>
        <item>
            <title>Impact of Low Glucose Degradation Product Bicarbonate/Lactate-Buffered Dialysis Solution on the Epithelial-Mesenchymal Transition of Peritoneum.</title>
            <link>http://www.medworm.com/index.php?rid=2967791&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19887789%26dopt%3DAbstract</link>
            <description>Conclusion: GDPs found in PDF induce EMT of HPMCs, which is associated with peritoneal fibrosis and vascularization. Conversely, L-GDP PDF reduces EMT and peritoneal fibrosis.
    PMID: 19887789 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2967791</comments>
            <pubDate>Wed, 04 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2967791</guid>        </item>
        <item>
            <title>TGF-beta1-Induced Epithelial-to-Mesenchymal Transition and Therapeutic Intervention in Diabetic Nephropathy.</title>
            <link>http://www.medworm.com/index.php?rid=2967790&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19887790%26dopt%3DAbstract</link>
            <description>Conclusion: TGF-beta-induced EMT is a key contributor to fibrotic scar formation as seen in DN, and novel routes for future therapeutic intervention are discussed.
    PMID: 19887790 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2967790</comments>
            <pubDate>Wed, 04 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2967790</guid>        </item>
        <item>
            <title>Estimating Glomerular Filtration Rate Using the New CKD-EPI Equation and Other Equations in Patients with Autosomal Dominant Polycystic Kidney Disease</title>
            <link>http://www.medworm.com/index.php?rid=2956784&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D256657</link>
            <description>Am J Nephrol 2010;31:53-57 (DOI:10.1159/000256657) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2956784</comments>
            <pubDate>Tue, 03 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2956784</guid>        </item>
        <item>
            <title>Impact of Low Glucose Degradation Product Bicarbonate/Lactate-Buffered Dialysis Solution on the Epithelial-Mesenchymal Transition of Peritoneum</title>
            <link>http://www.medworm.com/index.php?rid=2956783&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D256658</link>
            <description>Am J Nephrol 2010;31:58-67 (DOI:10.1159/000256658) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2956783</comments>
            <pubDate>Tue, 03 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2956783</guid>        </item>
        <item>
            <title>TGF-b1-Induced Epithelial-to-Mesenchymal Transition and Therapeutic Intervention in Diabetic Nephropathy</title>
            <link>http://www.medworm.com/index.php?rid=2956782&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D256659</link>
            <description>Am J Nephrol 2010;31:68-74 (DOI:10.1159/000256659) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2956782</comments>
            <pubDate>Tue, 03 Nov 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2956782</guid>        </item>
        <item>
            <title>Analysis of the Urine Proteome of Human Contrast-Induced Kidney Injury Using Two-Dimensional Fluorescence Differential Gel Electrophoresis/Matrix-Assisted Laser Desorption Time-of-Flight Mass Spectrometry/Liquid Chromatography Mass Spectrometry.</title>
            <link>http://www.medworm.com/index.php?rid=2967793&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19887787%26dopt%3DAbstract</link>
            <description>Conclusion: The studies suggested that MBL may be associated with contrast-induced acute kidney injury. It leads to an attempt to define a new pathogenesis and a novel biomarker for CIN.
    PMID: 19887787 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2967793</comments>
            <pubDate>Mon, 02 Nov 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2967793</guid>        </item>
        <item>
            <title>Tissue Expression of TRPC3 and TRPC6 in Hypertensive Munich Wistar Frömter Rats Showing Proteinuria.</title>
            <link>http://www.medworm.com/index.php?rid=2967794&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19887786%26dopt%3DAbstract</link>
            <description>Conclusion: Altered TRPC expression pattern in kidney cortex is associated with kidney damage in MWF rats showing hypertension and albuminuria.
    PMID: 19887786 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2967794</comments>
            <pubDate>Fri, 30 Oct 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2967794</guid>        </item>
        <item>
            <title>Tissue Expression of TRPC3 and TRPC6 in Hypertensive Munich Wistar Frömter Rats Showing Proteinuria</title>
            <link>http://www.medworm.com/index.php?rid=2945533&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D254060</link>
            <description>Am J Nephrol 2010;31:36-44 (DOI:10.1159/000254060) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2945533</comments>
            <pubDate>Thu, 29 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2945533</guid>        </item>
        <item>
            <title>Phosphoproteomic Study of Human Tubular Epithelial Cell in Response to Transforming Growth Factor-Beta-1-Induced Epithelial-to-Mesenchymal Transition.</title>
            <link>http://www.medworm.com/index.php?rid=2940392&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19864886%26dopt%3DAbstract</link>
            <description>Conclusion: We demonstrate a TGF-beta1-mediated post-transcriptional regulation of EMT in tubular epithelial cells. Phosphorylation of moesin and HSP90alpha might play a role in TGF-beta-induced EMT.
    PMID: 19864886 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2940392</comments>
            <pubDate>Wed, 28 Oct 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2940392</guid>        </item>
        <item>
            <title>Phosphoproteomic Study of Human Tubular Epithelial Cell in Response to Transforming Growth Factor-Beta-1-Induced Epithelial-to-Mesenchymal Transition</title>
            <link>http://www.medworm.com/index.php?rid=2936716&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D253865</link>
            <description>Am J Nephrol 2010;31:24-35 (DOI:10.1159/000253865) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2936716</comments>
            <pubDate>Tue, 27 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2936716</guid>        </item>
        <item>
            <title>Adaptation and Validation of the Kidney Disease Quality of Life-Short Form Questionnaire in the Greek Language.</title>
            <link>http://www.medworm.com/index.php?rid=2940394&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19864884%26dopt%3DAbstract</link>
            <description>Conclusions: The present study shows that the Greek version of the KDQOL-SF questionnaire has high validity, reproducibility and internal consistency. Production of validated translations of the KDQOL-SF questionnaire in various languages will help promote health-related quality of life of end-stage renal disease patients all over the world.
    PMID: 19864884 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2940394</comments>
            <pubDate>Mon, 26 Oct 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2940394</guid>        </item>
        <item>
            <title>Physical Activity Is a Determinant of Circadian Blood Pressure Variation in Chronic Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2940393&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19864885%26dopt%3DAbstract</link>
            <description>Conclusion: Physical activity is an independent determinant of circadian variation in BP. We speculate that among patients with CKD, a sedentary lifestyle, rather than non-dipping, mediates increased cardiovascular risk.
    PMID: 19864885 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2940393</comments>
            <pubDate>Mon, 26 Oct 2009 00:00:00 +0100</pubDate>
            <guid isPermaLink="false">2940393</guid>        </item>
        <item>
            <title>Renal Protection by Ischemic Preconditioning Is Associated with p50/p50 Homodimers.</title>
            <link>http://www.medworm.com/index.php?rid=2923722&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19851061%26dopt%3DAbstract</link>
            <description>Conclusion: In conclusion, our investigation suggests that: (1) the general activity of NF-kappaB is attenuated in IPC kidneys, and (2) the remaining activation of NF-kappaB is dominated with p50/p50 homodimer. Both of these effects might subsequently downregulate IL-18 expression, as well as provide renal protective effects of IPC. Though we focused on the phenomenological observations in this study, the detailed mechanism is still to be determined.
    PMID: 19851061 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2923722</comments>
            <pubDate>Thu, 22 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2923722</guid>        </item>
        <item>
            <title>Circadian Blood Pressure Classification Scheme and the Health of Patients with Chronic Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2916772&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19844085%26dopt%3DAbstract</link>
            <description>Conclusions: Among patients with CKD, circadian BP profile described by either dipper-based or phase-based classification is related to the level of physical activity and the severity of kidney damage. The circadian BP profile is related to overall health and nutritional intake only when using the phase-based classification. The value of these classification schemes to profile circadian BP will require longitudinal studies.
    PMID: 19844085 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2916772</comments>
            <pubDate>Tue, 20 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2916772</guid>        </item>
        <item>
            <title>Prognostic Value of Circadian Blood Pressure Variation in Chronic Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2916771&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19844086%26dopt%3DAbstract</link>
            <description>Conclusion: Among elderly veterans with or without CKD, circadian variation in BP is associated with mortality, but not ESRD. However, after accounting for common clinical risk factors, this association of circadian BP variation with mortality or ESRD is abolished.
    PMID: 19844086 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2916771</comments>
            <pubDate>Tue, 20 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2916771</guid>        </item>
        <item>
            <title>Sex and Age Result in Differential Regulation of the Renal Thiazide-Sensitive NaCl Cotransporter and the Epithelial Sodium Channel in Angiotensin II-Infused Mice.</title>
            <link>http://www.medworm.com/index.php?rid=2916770&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19844087%26dopt%3DAbstract</link>
            <description>Conclusions: Increased sensitivity to Ang II in aging and male mice may involve overactivity of NCC.
    PMID: 19844087 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2916770</comments>
            <pubDate>Tue, 20 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2916770</guid>        </item>
        <item>
            <title>Circadian Blood Pressure Classification Scheme and the Health of Patients with Chronic Kidney Disease</title>
            <link>http://www.medworm.com/index.php?rid=2914231&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D252774</link>
            <description>Am J Nephrol 2009;30:536-546 (DOI:10.1159/000252774) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2914231</comments>
            <pubDate>Tue, 20 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2914231</guid>        </item>
        <item>
            <title>Prognostic Value of Circadian Blood Pressure Variation in Chronic Kidney Disease</title>
            <link>http://www.medworm.com/index.php?rid=2914230&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D252775</link>
            <description>Am J Nephrol 2009;30:547-553 (DOI:10.1159/000252775) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2914230</comments>
            <pubDate>Tue, 20 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2914230</guid>        </item>
        <item>
            <title>Sex and Age Result in Differential Regulation of the Renal Thiazide-Sensitive NaCl Cotransporter and the Epithelial Sodium Channel in Angiotensin II-Infused Mice</title>
            <link>http://www.medworm.com/index.php?rid=2914229&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D252776</link>
            <description>Am J Nephrol 2009;30:554-562 (DOI:10.1159/000252776) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2914229</comments>
            <pubDate>Tue, 20 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2914229</guid>        </item>
        <item>
            <title>Prevalence and Cardiovascular Features of Japanese Hemodialysis Patients with Fabry Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2916773&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19844084%26dopt%3DAbstract</link>
            <description>Conclusions: Measurement of the alpha-galactosidase A activity and the results of a genetic analysis indicated that the prevalence of FD in our hemodialysis patients was 0.29% (0.16% in men and 0.5% in women). Furthermore, comprehensive examination detected cardiovascular abnormalities in Japanese hemodialysis patients with FD.
    PMID: 19844084 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2916773</comments>
            <pubDate>Thu, 15 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2916773</guid>        </item>
        <item>
            <title>Protective Effect of Tonapofylline (BG9928), an Adenosine A(1) Receptor Antagonist, against Cisplatin-Induced Acute Kidney Injury in Rats.</title>
            <link>http://www.medworm.com/index.php?rid=2899994&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19828940%26dopt%3DAbstract</link>
            <description>Conclusion: These data support the involvement of A1Rs in CIS-induced AKI in rats. Tonapofylline may be useful in the clinical setting for the prevention of kidney failure induced by nephrotoxic agents such as CIS.
    PMID: 19828940 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2899994</comments>
            <pubDate>Mon, 12 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2899994</guid>        </item>
        <item>
            <title>Mutagenicity of Immunosuppressive Medications among Renal Transplant Recipients.</title>
            <link>http://www.medworm.com/index.php?rid=2881378&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19816013%26dopt%3DAbstract</link>
            <description>Conclusion: In patients with renal transplantation, immunosuppressive therapy intensifies the genotoxic damage of the preceding chronic renal failure.
    PMID: 19816013 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2881378</comments>
            <pubDate>Sun, 04 Oct 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2881378</guid>        </item>
        <item>
            <title>Genome-Wide Analysis of Histone H3 Lysine 4 Trimethylation in Peripheral Blood Mononuclear Cells of Minimal Change Nephrotic Syndrome Patients.</title>
            <link>http://www.medworm.com/index.php?rid=2857977&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19797895%26dopt%3DAbstract</link>
            <description>Conclusion: Our studies indicate that there are significant alterations of H3K4me3 in MCNS patients. These significant H3K4me3 candidates may help to explain the immunological disturbance involved in MCNS patients.
    PMID: 19797895 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2857977</comments>
            <pubDate>Wed, 30 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2857977</guid>        </item>
        <item>
            <title>Genome-Wide Analysis of Histone H3 Lysine 4 Trimethylation in Peripheral Blood Mononuclear Cells of Minimal Change Nephrotic Syndrome Patients</title>
            <link>http://www.medworm.com/index.php?rid=2853797&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D243811</link>
            <description>Am J Nephrol 2009;30:505-513 (DOI:10.1159/000243811) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2853797</comments>
            <pubDate>Wed, 30 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2853797</guid>        </item>
        <item>
            <title>Effect of Community Characteristics on Familial Clustering of End-Stage Renal Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2857978&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19797894%26dopt%3DAbstract</link>
            <description>Conclusions: Although select community measures of lower socioeconomic status may contribute to the familial clustering of ESRD, non-socioeconomic factors, potentially inherited, appear to be more important contributors to familial aggregation of the common forms of ESRD.
    PMID: 19797894 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2857978</comments>
            <pubDate>Tue, 29 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2857978</guid>        </item>
        <item>
            <title>Effect of Community Characteristics on Familial Clustering of End-Stage Renal Disease</title>
            <link>http://www.medworm.com/index.php?rid=2844619&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D243716</link>
            <description>Am J Nephrol 2009;30:499-504 (DOI:10.1159/000243716) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2844619</comments>
            <pubDate>Tue, 29 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2844619</guid>        </item>
        <item>
            <title>Hemoglobin Level Variability: Anemia Management among Variability Groups.</title>
            <link>http://www.medworm.com/index.php?rid=2844713&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19786739%26dopt%3DAbstract</link>
            <description>Conclusions: Anemia management protocols describing coordinated administration of ESAs and iron may help to increase the number of patients achieving target hemoglobin levels.
    PMID: 19786739 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2844713</comments>
            <pubDate>Mon, 28 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2844713</guid>        </item>
        <item>
            <title>Hemoglobin Level Variability: Anemia Management among Variability Groups</title>
            <link>http://www.medworm.com/index.php?rid=2844620&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D243565</link>
            <description>Am J Nephrol 2009;30:491-498 (DOI:10.1159/000243565) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2844620</comments>
            <pubDate>Mon, 28 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2844620</guid>        </item>
        <item>
            <title>Modeling of Human Anti-GBM Antibody-alpha3(IV)NC1 Interactions Predicts Antigenic Cross-Linking through Contact of Both Heavy Chains with Repeating Epitopes on alpha3(IV)NC1.</title>
            <link>http://www.medworm.com/index.php?rid=2844715&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19786737%26dopt%3DAbstract</link>
            <description>Conclusions: The results suggest that the repetitive nature and relatedness of the alpha3(IV)NC1 antigenic epitopes facilitate cross-linking of pathogenic Ab, in vivo, by allowing both IgG Fab to bind to the basement membrane. This most likely accounts for the high-affinity Ab binding we and others observed among human anti-alpha3(IV)NC1 Ab. Based on these observations, we postulate that this interaction provides for the stability of the Ab interaction, resulting in a high-affinity interaction that serves as an ideal scaffold for optimal FcR engagement and complement activation, thereby accelerating inflammation and contributing to the rapidly progressive nature of this disease.
    PMID: 19786737 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2844715</comments>
            <pubDate>Sun, 27 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2844715</guid>        </item>
        <item>
            <title>Positive Feedback Loop between Plasminogen Activator Inhibitor-1 and Transforming Growth Factor-Beta1 during Renal Fibrosis in Diabetes.</title>
            <link>http://www.medworm.com/index.php?rid=2844714&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19786738%26dopt%3DAbstract</link>
            <description>Conclusion: Since TGF-beta1 is well known to stimulate the PAI-1 promoter, we suggest that TGF-beta1 and PAI-1 together constitute a positive feedback loop in the development of renal fibrosis in diabetes.
    PMID: 19786738 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2844714</comments>
            <pubDate>Thu, 24 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2844714</guid>        </item>
        <item>
            <title>Positive Feedback Loop between Plasminogen Activator Inhibitor-1 and Transforming Growth Factor-Beta1 during Renal Fibrosis in Diabetes</title>
            <link>http://www.medworm.com/index.php?rid=2833434&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D242477</link>
            <description>Am J Nephrol 2009;30:481-490 (DOI:10.1159/000242477) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2833434</comments>
            <pubDate>Thu, 24 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2833434</guid>        </item>
        <item>
            <title>Urine Proteome Analysis in Murine Nephrotoxic Serum Nephritis.</title>
            <link>http://www.medworm.com/index.php?rid=2834687&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19776558%26dopt%3DAbstract</link>
            <description>Conclusion: Proteomic analysis of mouse urine remains challenging due to the abundance of a limited subset of proteins. iTRAQ analysis does not circumvent these challenges, but can provide information on post-translational processing of some proteins. Hepcidin is identified as a potential urinary marker of nephritis and its role in disease pathogenesis warrants further study.
    PMID: 19776558 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2834687</comments>
            <pubDate>Wed, 23 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2834687</guid>        </item>
        <item>
            <title>Incidence, Predictors and Outcomes of Transplant Renal Artery Stenosis after Kidney Transplantation: Analysis of USRDS.</title>
            <link>http://www.medworm.com/index.php?rid=2834686&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19776559%26dopt%3DAbstract</link>
            <description>Conclusions: TRAS is an important complication that predicts adverse patient and graft outcomes. Treatment strategies for TRAS warrant prospective investigation in clinical trials.
    PMID: 19776559 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2834686</comments>
            <pubDate>Wed, 23 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2834686</guid>        </item>
        <item>
            <title>Effect of Pioglitazone on Survival and Renal Function in a Mouse Model of Polycystic Kidney Disease.</title>
            <link>http://www.medworm.com/index.php?rid=2834685&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19776560%26dopt%3DAbstract</link>
            <description>Conclusion: These findings suggest that pioglitazone may have a unique antihypertensive effect in polycystic kidney disease, and that such an effect may promote improved survival.
    PMID: 19776560 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2834685</comments>
            <pubDate>Wed, 23 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2834685</guid>        </item>
        <item>
            <title>Who Is Caring for Kidney Transplant Patients? Variation by Region, Transplant Center, and Patient Characteristics.</title>
            <link>http://www.medworm.com/index.php?rid=2804342&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19752531%26dopt%3DAbstract</link>
            <description>Conclusion: Frequency of visits to transplant centers varied by center and region; most visits were to nephrologists. Patients from minority groups were less likely to visit transplant centers and nephrologists, with possibly significant public health implications.
    PMID: 19752531 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2804342</comments>
            <pubDate>Mon, 14 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2804342</guid>        </item>
        <item>
            <title>Diverse Effects of Ischemic Pretreatments on the Long-Term Renal Damage Induced by Ischemia-Reperfusion.</title>
            <link>http://www.medworm.com/index.php?rid=2804341&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19752532%26dopt%3DAbstract</link>
            <description>Conclusion: Severe I/R injury may cause tubulointerstitial fibrosis in the long term, and different ischemic pretreatments have diverse effects on renal fibrosis.
    PMID: 19752532 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2804341</comments>
            <pubDate>Mon, 14 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2804341</guid>        </item>
        <item>
            <title>Who Is Caring for Kidney Transplant Patients? Variation by Region, Transplant Center, and Patient Characteristics</title>
            <link>http://www.medworm.com/index.php?rid=2798743&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D239220</link>
            <description>Am J Nephrol 2009;30:430-439 (DOI:10.1159/000239220) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2798743</comments>
            <pubDate>Mon, 14 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2798743</guid>        </item>
        <item>
            <title>Diverse Effects of Ischemic Pretreatments on the Long-Term Renal Damage Induced by Ischemia-Reperfusion</title>
            <link>http://www.medworm.com/index.php?rid=2798742&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D239574</link>
            <description>Am J Nephrol 2009;30:440-449 (DOI:10.1159/000239574) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2798742</comments>
            <pubDate>Mon, 14 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2798742</guid>        </item>
        <item>
            <title>Uric Acid: A Novel Risk Factor for Acute Kidney Injury in High-Risk Cardiac Surgery Patients?</title>
            <link>http://www.medworm.com/index.php?rid=2804343&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19752530%26dopt%3DAbstract</link>
            <description>Conclusion: This preliminary study suggests that uric acid may be a novel risk factor for acute kidney injury in patients undergoing high-risk cardiovascular surgery.
    PMID: 19752530 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2804343</comments>
            <pubDate>Thu, 10 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2804343</guid>        </item>
        <item>
            <title>Predictors of Hyperkalemia Risk following Hypertension Control with Aldosterone Blockade</title>
            <link>http://www.medworm.com/index.php?rid=2780373&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D237742</link>
            <description>Am J Nephrol 2009;30:418-424 (DOI:10.1159/000237742) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2780373</comments>
            <pubDate>Tue, 08 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2780373</guid>        </item>
        <item>
            <title>Predictors of Hyperkalemia Risk following Hypertension Control with Aldosterone Blockade.</title>
            <link>http://www.medworm.com/index.php?rid=2779344&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19738369%26dopt%3DAbstract</link>
            <description>Conclusion: Aldosterone antagonism is effective and safe for achieving a BP goal among people with diabetic nephropathy when added to a triple antihypertensive regimen that includes a blocker of the renin-angiotensin system and an appropriately selected and dosed diuretic. Caution is advised when using aldosterone blockade for BP control in people with advanced stage 3 nephropathy with a serum potassium of &amp;gt;4.5 mEq/l for safety reasons.
    PMID: 19738369 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2779344</comments>
            <pubDate>Tue, 08 Sep 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2779344</guid>        </item>
        <item>
            <title>Validation of Endogenous Internal Real-Time PCR Controls in Renal Tissues.</title>
            <link>http://www.medworm.com/index.php?rid=2770317&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19729889%26dopt%3DAbstract</link>
            <description>Conclusion: A combination of the top-ranked genes will provide a suitable endogenous internal control for similar studies of kidney tissues across a wide range of disease severity.
    PMID: 19729889 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2770317</comments>
            <pubDate>Mon, 31 Aug 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Validation of Endogenous Internal Real-Time PCR Controls in Renal Tissues</title>
            <link>http://www.medworm.com/index.php?rid=2754968&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D235993</link>
            <description>Am J Nephrol 2009;30:413-417 (DOI:10.1159/000235993) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2754968</comments>
            <pubDate>Mon, 31 Aug 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Antiphospholipid Antibodies and Renal Involvement.</title>
            <link>http://www.medworm.com/index.php?rid=2744902&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19713697%26dopt%3DAbstract</link>
            <description>Authors: Gigante A, Gasperini ML, Cianci R, Barbano B, Giannakakis K, Di Donato D, Fuiano G, Amoroso A
    Antiphospholipid antibodies are a heterogeneous group of autoantibodies associated with the hypercoagulable state affecting all vascular districts with thrombosis named antiphospholipid syndrome (APS). APS is an autoimmune disease with multifactorial etiology that includes cellular, molecular, genetic and pathogenic mechanisms. The APS clinical features are a combination of arterial and/or venous thrombosis, hematological events, recurrent fetal losses, neurological disorders and intra-abdominal manifestations. The renal involvement is associated with both primary and secondary APS. Clinical features include hypertension, renal artery stenosis, thrombotic microangiopathy and other his...</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
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            <pubDate>Thu, 27 Aug 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Combination of Captopril and Allopurinol Retards Fructose-Induced Metabolic Syndrome.</title>
            <link>http://www.medworm.com/index.php?rid=2723356&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19696478%26dopt%3DAbstract</link>
            <description>Conclusion: A high fructose diet can induce metabolic syndrome even in the setting of caloric restriction. Captopril and allopurinol synergistically reduce features of the metabolic syndrome, especially hypertension, insulin resistance and dyslipidemia. Combination allopurinol and ACE inhibitor therapy might provide a superior means to prevent diabetes and cardiovascular disease.
    PMID: 19696478 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2723356</comments>
            <pubDate>Thu, 20 Aug 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>KLOTHO Gene Polymorphisms G-395A and C1818T Are Associated with Low-Density Lipoprotein Cholesterol and Uric Acid in Japanese Hemodialysis Patients.</title>
            <link>http://www.medworm.com/index.php?rid=2717166&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19690404%26dopt%3DAbstract</link>
            <description>Conclusion:KLOTHO gene SNPs G-395A and C1818T are associated with low-density lipoprotein cholesterol and uric acid in HD patients.
    PMID: 19690404 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2717166</comments>
            <pubDate>Tue, 18 Aug 2009 23:00:00 +0100</pubDate>
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            <title>Interleukin-6 Is a Predictor of Mortality in Stable Hemodialysis Patients.</title>
            <link>http://www.medworm.com/index.php?rid=2717165&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19690405%26dopt%3DAbstract</link>
            <description>Conclusions: These data clearly show that plasma IL-6 is a powerful predictor of all-cause mortality in dialysis patients.
    PMID: 19690405 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2717165</comments>
            <pubDate>Tue, 18 Aug 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Effects of Nicotine Exposure on Renal Function of Normal and Hypercholesterolemic Rats.</title>
            <link>http://www.medworm.com/index.php?rid=2711020&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19684386%26dopt%3DAbstract</link>
            <description>Conclusion: Nicotine acted on the adrenergic system and nitric oxide counteracted this action in C, but the same may not be applied to HC. An impairment in renal autoregulation may explain why InCl was unchanged in HC.
    PMID: 19684386 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2711020</comments>
            <pubDate>Sun, 16 Aug 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Susceptibility to Insulin Resistance after Kidney Donation: A Pilot Observational Study.</title>
            <link>http://www.medworm.com/index.php?rid=2668004&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19648735%26dopt%3DAbstract</link>
            <description>Conclusion: Reduced GFR alone in previous kidney donors is associated with increased IR.
    PMID: 19648735 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2668004</comments>
            <pubDate>Thu, 30 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Association of (Pro)renin Receptor mRNA Expression with Angiotensin-Converting Enzyme mRNA Expression in Human Artery.</title>
            <link>http://www.medworm.com/index.php?rid=2658393&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19641301%26dopt%3DAbstract</link>
            <description>Conclusion: (Pro)renin receptor may contribute to the generation of arterial angiotensin II in kidney failure patients.
    PMID: 19641301 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2658393</comments>
            <pubDate>Wed, 29 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Announcement</title>
            <link>http://www.medworm.com/index.php?rid=2683236&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D167943</link>
            <description>Am J Nephrol 1989;9:88 (DOI:10.1159/000167943) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683236</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Announcement</title>
            <link>http://www.medworm.com/index.php?rid=2683235&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D167947</link>
            <description>Am J Nephrol 1989;9:109 (DOI:10.1159/000167947) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683235</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Quiz of the Month, Questions</title>
            <link>http://www.medworm.com/index.php?rid=2683234&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D167953</link>
            <description>Am J Nephrol 1989;9:144 (DOI:10.1159/000167953) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683234</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2683234</guid>        </item>
        <item>
            <title>Quiz of the Month, Answers</title>
            <link>http://www.medworm.com/index.php?rid=2683233&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D167960</link>
            <description>Am J Nephrol 1989;9:175-176 (DOI:10.1159/000167960) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683233</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2683233</guid>        </item>
        <item>
            <title>Medical Art</title>
            <link>http://www.medworm.com/index.php?rid=2683232&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D167973</link>
            <description>Am J Nephrol 1989;9:244 (DOI:10.1159/000167973) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683232</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2683232</guid>        </item>
        <item>
            <title>Quiz of the Month, Questions</title>
            <link>http://www.medworm.com/index.php?rid=2683231&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D167990</link>
            <description>Am J Nephrol 1989;9:343 (DOI:10.1159/000167990) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683231</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Quiz of the Month, Answers / Announcements</title>
            <link>http://www.medworm.com/index.php?rid=2683230&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D167995</link>
            <description>Am J Nephrol 1989;9:352 (DOI:10.1159/000167995) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683230</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2683230</guid>        </item>
        <item>
            <title>Quiz of the Month, Questions</title>
            <link>http://www.medworm.com/index.php?rid=2683229&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168003</link>
            <description>Am J Nephrol 1989;9:413 (DOI:10.1159/000168003) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683229</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2683229</guid>        </item>
        <item>
            <title>Quiz of the Month, Answers / Announcements</title>
            <link>http://www.medworm.com/index.php?rid=2683228&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168008</link>
            <description>Am J Nephrol 1989;9:440 (DOI:10.1159/000168008) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683228</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Quiz of the Month, Questions</title>
            <link>http://www.medworm.com/index.php?rid=2683227&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168017</link>
            <description>Am J Nephrol 1989;9:487 (DOI:10.1159/000168017) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683227</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Quiz of the Month, Answers</title>
            <link>http://www.medworm.com/index.php?rid=2683226&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168023</link>
            <description>Am J Nephrol 1989;9:516-517 (DOI:10.1159/000168023) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683226</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Author Index, Vol. 9, 1989</title>
            <link>http://www.medworm.com/index.php?rid=2683225&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168024</link>
            <description>Am J Nephrol 1989;9:518-519 (DOI:10.1159/000168024) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683225</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Subject Index, Vol. 9, 1989</title>
            <link>http://www.medworm.com/index.php?rid=2683224&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168025</link>
            <description>Am J Nephrol 1989;9:520-521 (DOI:10.1159/000168025) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683224</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Manuscript Consultants</title>
            <link>http://www.medworm.com/index.php?rid=2683223&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168026</link>
            <description>Am J Nephrol 1989;9:522 (DOI:10.1159/000168026) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683223</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Introduction</title>
            <link>http://www.medworm.com/index.php?rid=2683222&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168027</link>
            <description>Am J Nephrol 1989;9:1 (DOI:10.1159/000168027) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683222</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Title Page / Table of Contents</title>
            <link>http://www.medworm.com/index.php?rid=2683221&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168030</link>
            <description>Am J Nephrol 1989;9:I (DOI:10.1159/000168030) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683221</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Author/Subject Indexes,  Vol. 9 (suppl 1), 1989</title>
            <link>http://www.medworm.com/index.php?rid=2683220&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D168046</link>
            <description>Am J Nephrol 1989;9:91 (DOI:10.1159/000168046) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2683220</comments>
            <pubDate>Thu, 23 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Nebivolol Reduces Proteinuria and Renal NADPH Oxidase-Generated Reactive Oxygen Species in the Transgenic Ren2 Rat</title>
            <link>http://www.medworm.com/index.php?rid=2612560&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D229305</link>
            <description>Am J Nephrol 2009;30:354-360 (DOI:10.1159/000229305) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2612560</comments>
            <pubDate>Fri, 17 Jul 2009 09:31:02 +0100</pubDate>
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            <title>Chronic COX Inhibition Reduces Diabetes-Induced Hyperfiltration, Proteinuria, and Renal Pathological Markers in 36-Week B6-Ins2 Akita  Mice</title>
            <link>http://www.medworm.com/index.php?rid=2612561&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D229304</link>
            <description>Am J Nephrol 2009;30:346-353 (DOI:10.1159/000229304) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2612561</comments>
            <pubDate>Fri, 17 Jul 2009 09:30:58 +0100</pubDate>
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            <title>Chronic COX Inhibition Reduces Diabetes-Induced Hyperfiltration, Proteinuria, and Renal Pathological Markers in 36-Week B6-Ins2 Mice.</title>
            <link>http://www.medworm.com/index.php?rid=2614465&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19609076%26dopt%3DAbstract</link>
            <description>Conclusion: NSAIDs reduced diabetic change: GFR, albuminuria, p27, and fibronectin. The effects of ibuprofen are similar if not more beneficial than COX-2 inhibition by NS-398. This study has clinical relevance for diabetics prior to overt nephropathy. Future studies should reveal the effects of NSAIDs in a more severe disease environment.
    PMID: 19609076 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
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            <pubDate>Thu, 16 Jul 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Nebivolol Reduces Proteinuria and Renal NADPH Oxidase-Generated Reactive Oxygen Species in the Transgenic Ren2 Rat.</title>
            <link>http://www.medworm.com/index.php?rid=2614464&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19609077%26dopt%3DAbstract</link>
            <description>Conclusions: Our data suggest that nebivolol improves proteinuria through reductions in renal RAAS-mediated increases in NADPH oxidase/ROS and increases in bioavailable NO.
    PMID: 19609077 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2614464</comments>
            <pubDate>Thu, 16 Jul 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2614464</guid>        </item>
        <item>
            <title>Advanced Glycation End Products Suppress Neuropilin-1 Expression in Podocytes by a Reduction in Sp1-Dependent Transcriptional Activity.</title>
            <link>http://www.medworm.com/index.php?rid=2591903&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19590177%26dopt%3DAbstract</link>
            <description>Conclusions: AGE-BSA inhibits NRP1 promoter transcriptional activity in podocytes by reducing the binding ability of the Sp1 transcription factor to attach to the NRP1 promoter.
    PMID: 19590177 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2591903</comments>
            <pubDate>Thu, 02 Jul 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2591903</guid>        </item>
        <item>
            <title>Advanced Glycation End Products Suppress Neuropilin-1 Expression in Podocytes by a Reduction in Sp1-Dependent Transcriptional Activity</title>
            <link>http://www.medworm.com/index.php?rid=2565588&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D227762</link>
            <description>Am J Nephrol 2009;30:336-345 (DOI:10.1159/000227762) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2565588</comments>
            <pubDate>Thu, 02 Jul 2009 15:22:47 +0100</pubDate>
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        <item>
            <title>A New Perspective on Therapeutic Inhibition of Advanced Glycation in Diabetic Microvascular Complications: Common Downstream Endpoints Achieved Through Disparate Therapeutic Approaches?</title>
            <link>http://www.medworm.com/index.php?rid=2542242&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19556753%26dopt%3DAbstract</link>
            <description>Authors: Sourris KC, Harcourt BE, Forbes JM
    A commonality among the chemically disparate compounds that inhibit the formation and accumulation of advanced glycation end products (AGEs) or their signalling pathways is their end organ protection in experimental models of diabetes complications. Although this group of therapeutics are structurally and functionally distinct with numerous mechanisms of action, the most important factor governing their therapeutic capability is clearly their ability to alleviate the tissue burden of advanced glycation, rather than the biochemical mechanism by which this is achieved. However, it remains to be determined if it is the reduction in tissue AGE levels per se or inhibition of downstream signal pathways which is ultimately required for end organ pro...</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542242</comments>
            <pubDate>Sun, 28 Jun 2009 00:25:05 +0100</pubDate>
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        <item>
            <title>A New Perspective on Therapeutic Inhibition of Advanced Glycation in Diabetic Microvascular Complications: Common Downstream Endpoints Achieved Through Disparate Therapeutic Approaches?</title>
            <link>http://www.medworm.com/index.php?rid=2549691&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D226586</link>
            <description>Am J Nephrol 2009;30:323-335 (DOI:10.1159/000226586) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2549691</comments>
            <pubDate>Fri, 26 Jun 2009 13:19:27 +0100</pubDate>
            <guid isPermaLink="false">2549691</guid>        </item>
        <item>
            <title>Efficacy and Safety of Glucocorticoids Therapy for IgA Nephropathy: A Meta-Analysis of Randomized Controlled Trials.</title>
            <link>http://www.medworm.com/index.php?rid=2542243&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19546530%26dopt%3DAbstract</link>
            <description>Conclusions: The current cumulative evidence suggests that glucocorticoids have statistically significant effects on protecting renal function and reduction of proteinuria in patients with IgAN, but we should be careful for its gastrointestinal tract reaction. In general, glucocorticoids agents are a promising medication and should be investigated further.
    PMID: 19546530 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542243</comments>
            <pubDate>Mon, 22 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542243</guid>        </item>
        <item>
            <title>A Synergistic Association of ACE I/D and eNOS G894T Gene Variants with the Progression of Immunoglobulin A Nephropathy - A Pilot Study.</title>
            <link>http://www.medworm.com/index.php?rid=2542245&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19546528%26dopt%3DAbstract</link>
            <description>Conclusion: This study suggests that in our population with IgAN, an interaction between ACE and eNOS polymorphisms may be a prognostic factor for renal function deterioration.
    PMID: 19546528 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542245</comments>
            <pubDate>Wed, 17 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542245</guid>        </item>
        <item>
            <title>Lack of Association between Small Dense Low-Density Lipoprotein Levels and Coronary Artery Disease in Chronic Hemodialysis Patients.</title>
            <link>http://www.medworm.com/index.php?rid=2542244&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19546529%26dopt%3DAbstract</link>
            <description>Conclusion: This study showed that sd-LDL is not increased in CHD patients and is not associated with CAD in such patients.
    PMID: 19546529 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542244</comments>
            <pubDate>Wed, 17 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542244</guid>        </item>
        <item>
            <title>A Synergistic Association of ACE I/D and eNOS G894T Gene Variants with the Progression of Immunoglobulin A Nephropathy -- A Pilot Study</title>
            <link>http://www.medworm.com/index.php?rid=2492123&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D225938</link>
            <description>Am J Nephrol 2009;30:303-309 (DOI:10.1159/000225938) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2492123</comments>
            <pubDate>Wed, 17 Jun 2009 15:17:20 +0100</pubDate>
            <guid isPermaLink="false">2492123</guid>        </item>
        <item>
            <title>Lack of Association between Small Dense Low-Density Lipoprotein Levels and Coronary Artery Disease in Chronic Hemodialysis Patients</title>
            <link>http://www.medworm.com/index.php?rid=2492124&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D225939</link>
            <description>Am J Nephrol 2009;30:310-314 (DOI:10.1159/000225939) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2492124</comments>
            <pubDate>Wed, 17 Jun 2009 14:48:50 +0100</pubDate>
            <guid isPermaLink="false">2492124</guid>        </item>
        <item>
            <title>Effects of New Peritoneal Dialysis Solutions, Pyridoxamine and AT1 Receptor Blocker, on TGF-b1 and VEGF Expression in Rat Peritoneal Mesothelial Cells</title>
            <link>http://www.medworm.com/index.php?rid=2492125&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D225929</link>
            <description>Am J Nephrol 2009;30:295-302 (DOI:10.1159/000225929) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2492125</comments>
            <pubDate>Tue, 16 Jun 2009 06:44:33 +0100</pubDate>
            <guid isPermaLink="false">2492125</guid>        </item>
        <item>
            <title>Ischemic Preconditioning Attenuates Renal Ischemia-Reperfusion Injury by Inhibiting Activation of IKKb and Inflammatory Response</title>
            <link>http://www.medworm.com/index.php?rid=2492126&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D225928</link>
            <description>Am J Nephrol 2009;30:287-294 (DOI:10.1159/000225928) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2492126</comments>
            <pubDate>Tue, 16 Jun 2009 06:44:32 +0100</pubDate>
            <guid isPermaLink="false">2492126</guid>        </item>
        <item>
            <title>Ischemic Preconditioning Attenuates Renal Ischemia-Reperfusion Injury by Inhibiting Activation of IKKbeta and Inflammatory Response.</title>
            <link>http://www.medworm.com/index.php?rid=2542247&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19546526%26dopt%3DAbstract</link>
            <description>Conclusions: The evidence suggests that IKKbeta may play a role in renal I/R injury and give rise to the generation of NGAL. It appears that IPC may attenuate renal injury and the expression of NGAL following acute I/R injury. IKKbeta may offer a clinically accessible target for preventing renal injury following I/R.
    PMID: 19546526 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542247</comments>
            <pubDate>Mon, 15 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542247</guid>        </item>
        <item>
            <title>Effects of New Peritoneal Dialysis Solutions, Pyridoxamine and AT1 Receptor Blocker, on TGF-beta1 and VEGF Expression in Rat Peritoneal Mesothelial Cells.</title>
            <link>http://www.medworm.com/index.php?rid=2542246&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19546527%26dopt%3DAbstract</link>
            <description>Conclusions: Neither new pH-neutral, lactate-buffered, low-GDP, two-chamber bag PDS, nor 7.5% icodextrin PDS alone satisfactorily inhibited VEGF and TGF-beta1 overproduction in RPMC, but ARB or pyridoxamine effectively inhibited glucose-containing PDS (N 2.5%)-induced overproduction.
    PMID: 19546527 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542246</comments>
            <pubDate>Mon, 15 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542246</guid>        </item>
        <item>
            <title>D1-Like Receptor Antagonist Inhibits IL-17 Expression and Attenuates Crescent Formation in Nephrotoxic Serum Nephritis.</title>
            <link>http://www.medworm.com/index.php?rid=2542249&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19521069%26dopt%3DAbstract</link>
            <description>Conclusion: We identified treatment with a D1-like-R antagonist inhibited IL-17 expression and attenuated crescent formation in NTN mice.
    PMID: 19521069 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542249</comments>
            <pubDate>Sun, 14 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542249</guid>        </item>
        <item>
            <title>The Selective Vitamin D Receptor Activator for Albuminuria Lowering (VITAL) Study: Study Design and Baseline Characteristics.</title>
            <link>http://www.medworm.com/index.php?rid=2542248&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19521070%26dopt%3DAbstract</link>
            <description>Conclusion: This trial will be the first clinical test of the hypothesis that paricalcitol possesses pleiotropic effects and can modulate albuminuria in the setting of ACEI and/or ARB therapy. Results will have important clinical implications and are expected in November 2009.
    PMID: 19521070 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542248</comments>
            <pubDate>Sun, 14 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542248</guid>        </item>
        <item>
            <title>Immunoglobulin A Nephropathy in China: Progress and Challenges.</title>
            <link>http://www.medworm.com/index.php?rid=2542250&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19521068%26dopt%3DAbstract</link>
            <description>Conclusion: IgAN remains a great challenge to Chinese nephrologists. In-depth basic studies and multicenter clinical trials are needed in the future.
    PMID: 19521068 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542250</comments>
            <pubDate>Thu, 11 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542250</guid>        </item>
        <item>
            <title>Immunoglobulin A Nephropathy in China: Progress and Challenges</title>
            <link>http://www.medworm.com/index.php?rid=2480499&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D225563</link>
            <description>Am J Nephrol 2009;30:268-273 (DOI:10.1159/000225563) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2480499</comments>
            <pubDate>Thu, 11 Jun 2009 15:19:29 +0100</pubDate>
            <guid isPermaLink="false">2480499</guid>        </item>
        <item>
            <title>Effect of Ethnicity on the Progression of Diabetic Kidney Disease Independent of Glycemic Control.</title>
            <link>http://www.medworm.com/index.php?rid=2542251&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19494485%26dopt%3DAbstract</link>
            <description>Conclusions: Our data indicate that there were no ethnic differences in the rate of progression of DKD under equivalent glycemic control. Further research is needed to explore the mechanisms associated with higher prevalence and rapid progression of CKD in African-Americans compared to Caucasians.
    PMID: 19494485 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542251</comments>
            <pubDate>Wed, 03 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542251</guid>        </item>
        <item>
            <title>Effect of Ethnicity on the Progression of Diabetic Kidney Disease Independent of Glycemic Control</title>
            <link>http://www.medworm.com/index.php?rid=2460802&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D223527</link>
            <description>Am J Nephrol 2009;30:261-267 (DOI:10.1159/000223527) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2460802</comments>
            <pubDate>Wed, 03 Jun 2009 14:35:13 +0100</pubDate>
            <guid isPermaLink="false">2460802</guid>        </item>
        <item>
            <title>Prevention of Acute Kidney Injury by Erythropoietin in Patients Undergoing Coronary Artery Bypass Grafting: A Pilot Study</title>
            <link>http://www.medworm.com/index.php?rid=2450368&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D223229</link>
            <description>Am J Nephrol 2009;30:253-260 (DOI:10.1159/000223229) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2450368</comments>
            <pubDate>Wed, 03 Jun 2009 02:53:20 +0100</pubDate>
            <guid isPermaLink="false">2450368</guid>        </item>
        <item>
            <title>Prevention of Acute Kidney Injury by Erythropoietin in Patients Undergoing Coronary Artery Bypass Grafting: A Pilot Study.</title>
            <link>http://www.medworm.com/index.php?rid=2542252&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19494484%26dopt%3DAbstract</link>
            <description>Conclusions: In our small, pilot trial, prophylactic administration of EPO prevents AKI and improves postoperative renal function. These data are preliminary and require confirmation in a larger clinical trial.
    PMID: 19494484 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542252</comments>
            <pubDate>Mon, 01 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2542252</guid>        </item>
        <item>
            <title>High Stable Serum Adiponectin Levels Are Associated with a Better Outcome in Prevalent Dialysis Patients</title>
            <link>http://www.medworm.com/index.php?rid=2434733&amp;cid=s_36075_25_f&amp;fid=33500&amp;url=http%3A%2F%2Fcontent.karger.com%2Fproduktedb%2Fprodukte.asp%3Fdoi%3D221147</link>
            <description>Am J Nephrol 2009;30:244-252 (DOI:10.1159/000221147) (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2434733</comments>
            <pubDate>Wed, 27 May 2009 02:32:42 +0100</pubDate>
            <guid isPermaLink="false">2434733</guid>        </item>
        <item>
            <title>High Stable Serum Adiponectin Levels Are Associated with a Better Outcome in Prevalent Dialysis Patients.</title>
            <link>http://www.medworm.com/index.php?rid=2542253&amp;cid=s_36075_47_f&amp;fid=36075&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19468209%26dopt%3DAbstract</link>
            <description>Conclusions: In this population of dialysis patients, adiponectin seems to behave as a CV protective factor. Patients with high mean adiponectin levels had a better survival rate.
    PMID: 19468209 [PubMed - as supplied by publisher] (Source: American Journal of Nephrology)</description>
            <author>American Journal of Nephrology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2542253</comments>
            <pubDate>Mon, 25 May 2009 23:00:00 +0100</pubDate>
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