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        <title>Cancer Causes and Control via MedWorm.com</title>
        <description>MedWorm.com provides a medical RSS filtering service. Over 6000 RSS medical sources are combined and output via different filters. This feed contains the latest items from the 'Cancer Causes and Control' source.</description>
        <link><![CDATA[http://www.medworm.com/rss/search.php?qu=Cancer+Causes+and+Control&t=Cancer+Causes+and+Control&s=Search&f=source]]></link>
        <lastBuildDate>Sun, 21 Mar 2010 13:51:57 +0100</lastBuildDate>
        <item>
            <title>A case–control study of tobacco use and other non-occupational risk factors for lymphoma subtypes defined by t(14; 18) translocations and bcl-2 expression</title>
            <link>http://www.medworm.com/index.php?rid=3371345&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fv121433v6385wr17%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;The association between t(14; 18)-negative NHL and cigarette smoking was unexpected given previous evidence of associations between smoking and follicular
 lymphoma (which is largely t(14; 18)-positive). Future studies characterizing additional molecular characteristics of t(14; 18)-negative NHL may help determine whether the association with smoking may have been causal versus an artifact of chance
 or bias.
 
 
 
 
	Content Type Journal ArticleCategory Brief reportDOI 10.1007/s10552-010-9531-8Authors
		Cindy M. Chang, National Cancer Institute Infections and Immunoepidemiology Branch Rockville MD USAJane C. Schroeder, University of North Carolina at Chapel Hill Department of Epidemiology, School of Public Health Chapel Hill NC USAAndrew F. Olshan, University of Nor...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3371345</comments>
            <pubDate>Mon, 15 Mar 2010 17:57:58 +0100</pubDate>
            <guid isPermaLink="false">3371345</guid>        </item>
        <item>
            <title>Intake of fiber and nuts during adolescence and incidence of proliferative benign breast disease</title>
            <link>http://www.medworm.com/index.php?rid=3367020&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fq0265x5rl8904x41%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;These findings support the hypothesis that dietary intake of fiber and nuts during adolescence influences subsequent risk
 of breast disease and may suggest a viable means for breast cancer prevention.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9532-7Authors
		Xuefen Su, Brigham and Women’s Hospital and Harvard Medical School Channing Laboratory, Department of Medicine 181 Longwood Avenue, 3rd Floor Boston MA 02115 USARulla M. Tamimi, Brigham and Women’s Hospital and Harvard Medical School Channing Laboratory, Department of Medicine 181 Longwood Avenue, 3rd Floor Boston MA 02115 USALaura C. Collins, Harvard Medical School and Beth Israel Deaconess Medical Center Department of Pathology Boston MA USAHeather J. Baer, Harvard ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3367020</comments>
            <pubDate>Sun, 14 Mar 2010 11:24:59 +0100</pubDate>
            <guid isPermaLink="false">3367020</guid>        </item>
        <item>
            <title>History of diabetes mellitus and the risk of prostate cancer: the Ohsaki Cohort Study</title>
            <link>http://www.medworm.com/index.php?rid=3367021&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fg432n3128w121l64%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;This population-based prospective cohort study indicates that a history of diabetes mellitus is associated with an increased
 incidence of advanced prostate cancer.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9530-9Authors
		Qiang Li, Tohoku University Graduate School of Medicine Division of Epidemiology, Department of Public Health and Forensic Medicine 2-1 Seiryo-Machi, Aoba-Ku Sendai, Miyagi 980-8575 JapanShinichi Kuriyama, Tohoku University Graduate School of Medicine Division of Epidemiology, Department of Public Health and Forensic Medicine 2-1 Seiryo-Machi, Aoba-Ku Sendai, Miyagi 980-8575 JapanMasako Kakizaki, Tohoku University Graduate School of Medicine Division of Epidemiology, Department of Public Health and Forensic M...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3367021</comments>
            <pubDate>Sun, 14 Mar 2010 11:24:58 +0100</pubDate>
            <guid isPermaLink="false">3367021</guid>        </item>
        <item>
            <title>Pregnancy characteristics and maternal breast cancer risk: a review of the epidemiologic literature</title>
            <link>http://www.medworm.com/index.php?rid=3360384&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ff5678778430011l4%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;The short- and long-term effects of pregnancy on breast cancer risk are well documented. Insight into potential biological
 mechanisms for these associations may be gained by studying breast cancer risk and pregnancy characteristics (e.g., preeclampsia,
 twining), which may reflect hormone levels during pregnancy. To date, no review has synthesized the published literature for
 pregnancy characteristics and maternal breast cancer using systematic search methods. We conducted a systematic search to
 identify all published studies. Using PUBMED (to 31 July 2009), 42 relevant articles were identified. Several studies suggest
 that multiple births may be associated with a lowered breast cancer risk of about 10–30%, but results were inconsistent across
 18 studies. The maj...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3360384</comments>
            <pubDate>Thu, 11 Mar 2010 18:21:54 +0100</pubDate>
            <guid isPermaLink="false">3360384</guid>        </item>
        <item>
            <title>Risk of non-Hodgkin lymphoma and nitrate and nitrite from the diet in Connecticut women</title>
            <link>http://www.medworm.com/index.php?rid=3355668&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F41352090k5475030%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;It has been estimated that 65,980 individuals were diagnosed with non-Hodgkin lymphoma (NHL) and 19,500 died from NHL in the
 United States in 2009. Although established risk factors such as immunodeficiency and viral infections may be responsible
 for a portion of the cases, the majority of NHL cases remain unexplained. Dietary nitrate and nitrite intake are exposures
 of particular interest for NHL risk as they are precursors in the endogenous formation of N-nitroso compounds, which cause
 lymphomas in animal studies. We investigated NHL risk overall and by histologic type in relation to dietary nitrate and nitrite
 intake in a population-based case–control study of 1,304 women in Connecticut. Nitrate and nitrite intake were assessed using
 a 120-item food frequency...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3355668</comments>
            <pubDate>Wed, 10 Mar 2010 13:22:28 +0100</pubDate>
            <guid isPermaLink="false">3355668</guid>        </item>
        <item>
            <title>Head injury, diagnostic X-rays, and risk of medulloblastoma and primitive neuroectodermal tumor: a Children’s Oncology Group study</title>
            <link>http://www.medworm.com/index.php?rid=3355667&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fc88q135381278672%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Head injury and exposure to diagnostic head X-rays were not associated with medulloblastoma/PNET in this study. Future studies
 should investigate all imaging procedures with ionizing radiation exposure including computed tomography scans and utilize
 radiation dose estimations.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9529-2Authors
		Saira Khan, Drexel University School of Public Health Department of Epidemiology and Biostatistics Philadelphia PA USAAlison A. Evans, Drexel University School of Public Health Department of Epidemiology and Biostatistics Philadelphia PA USALucy Rorke-Adams, University of Pennsylvania School of Medicine Departments of Pathology, Neurology, and Pediatrics Philadelphia PA USAManuela A. Orjuela, Co...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3355667</comments>
            <pubDate>Wed, 10 Mar 2010 13:22:28 +0100</pubDate>
            <guid isPermaLink="false">3355667</guid>        </item>
        <item>
            <title>Estimation of cancer incidence and mortality attributable to smoking in China</title>
            <link>http://www.medworm.com/index.php?rid=3355666&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Flk23577230528237%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Tobacco smoking is responsible for one-third of the total cancer deaths among men. Involuntary smoking is an important individual
 risk factor for lung cancer among non-smoking women. There is a need to continue and strengthen tobacco-control programs and
 initiatives to reduce smoking-related cancer burden in China.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9523-8Authors
		Jian-Bing Wang, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences &amp; School of Basic Medicine, Peking Union Medical College Department of Epidemiology and Statistics Beijing People’s Republic of ChinaYong Jiang, Cancer Institute, Chinese Academy of Medical Sciences Department of Cancer Epidemiology 17 South, Panjiayuan Lane 100021 Bei...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3355666</comments>
            <pubDate>Wed, 10 Mar 2010 13:22:28 +0100</pubDate>
            <guid isPermaLink="false">3355666</guid>        </item>
        <item>
            <title>Cancer risk in DES daughters</title>
            <link>http://www.medworm.com/index.php?rid=3355669&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fb40q244p37185482%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Except for an elevated risk of CCA, persisting at older ages, and an increased risk of melanoma at young ages, we found no
 increased risk of cancer. Longer follow-up is warranted to examine cancer risk at ages when cancer occurs more frequently.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9526-5Authors
		Janneke Verloop, Netherlands Cancer Institute Department of Epidemiology Plesmanlaan 121 1066 CX Amsterdam The NetherlandsFlora E. van Leeuwen, Netherlands Cancer Institute Department of Epidemiology Plesmanlaan 121 1066 CX Amsterdam The NetherlandsTheo J. M. Helmerhorst, Erasmus MC, University Hospital Rotterdam Department of Obstetrics and Gynaecology Rotterdam The NetherlandsHester H. van Boven, Netherlands Cancer Institute ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3355669</comments>
            <pubDate>Wed, 10 Mar 2010 13:22:24 +0100</pubDate>
            <guid isPermaLink="false">3355669</guid>        </item>
        <item>
            <title>Poverty and childhood cancer incidence in the United States</title>
            <link>http://www.medworm.com/index.php?rid=3336458&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F77404857n7241875%2F</link>
            <description>This study examined socioeconomic differentials in cancer incidence rates during 2000–2005 among children aged 0–19 in the
 United States. The data on childhood cancers, which were classified by the International Classification of Childhood Cancer,
 Third Edition (ICCC-3), were obtained from the Surveillance, Epidemiology, and End Results program. The socioeconomic status
 of residential area at diagnosis was estimated by county-level poverty rate in Census 2000, i.e., percentage of persons in
 the county living below the national poverty thresholds. Counties were categorized as low-, medium-, and high-poverty areas
 when the poverty rates were &amp;lt;10, 10–19.99, and 20% or higher, respectively. The results showed that medium- and high-poverty
 counties had lower age-adjusted incidenc...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3336458</comments>
            <pubDate>Wed, 03 Mar 2010 08:03:45 +0100</pubDate>
            <guid isPermaLink="false">3336458</guid>        </item>
        <item>
            <title>Dietary patterns and risk of advanced prostate cancer: a principal component analysis in Uruguay</title>
            <link>http://www.medworm.com/index.php?rid=3336459&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F72p8h30l77337715%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;The Western and traditional patterns could partially explain the high incidence of advanced prostate cancer in Uruguay, a main producer of beef in the
 World.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9527-4Authors
		Eduardo De Stefani, Hospital de Clínicas Grupo de Epidemiología, Departamento de Anatomía Patológica, Facultad de Medicina Avenida Brasil 3080 dep 402 11300 Montevideo UruguayAlvaro L. Ronco, IUCLAEH Departamento de Epidemiología y Métodos Científicos, Facultad de Medicina Maldonado UruguayHugo Deneo-Pellegrini, Hospital de Clínicas Grupo de Epidemiología, Departamento de Anatomía Patológica, Facultad de Medicina Avenida Brasil 3080 dep 402 11300 Montevideo UruguayPaolo Boffetta, International Agency for...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3336459</comments>
            <pubDate>Wed, 03 Mar 2010 08:03:42 +0100</pubDate>
            <guid isPermaLink="false">3336459</guid>        </item>
        <item>
            <title>Incidence rates of exocrine and endocrine pancreatic cancers in the United States</title>
            <link>http://www.medworm.com/index.php?rid=3312753&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fh71014375u0h5274%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Descriptive studies of pancreatic cancer incidence have been sparse particularly in terms of tumor histology and stage. The
 purpose of this study was to examine the incidence rate trends of exocrine and endocrine pancreatic cancers by demographic
 and tumor characteristics using data from the Surveillance, Epidemiology, and End Results (SEER) program from 1977 to 2005.
 During this period, the incidence of exocrine pancreatic cancer generally decreased whereas the incidence of endocrine pancreatic
 cancer increased. This difference in trends by histology was evident across age, gender, and racial groups. It was also evident
 among different racial/ethnic groups using data from 1992 to 2005. Variation in trends was observed by stage. The incidence
 of exocrine cancers d...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3312753</comments>
            <pubDate>Thu, 25 Feb 2010 06:56:27 +0100</pubDate>
            <guid isPermaLink="false">3312753</guid>        </item>
        <item>
            <title>Australian women’s awareness of breast cancer symptoms and responses to potential symptoms</title>
            <link>http://www.medworm.com/index.php?rid=3303678&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fu1615r28284m6q30%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Health promotion efforts need to continue to aim at increasing community understanding of potential breast cancer symptoms
 and encouraging women to act on potential symptoms by seeking medical advice.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9522-9Authors
		Sandra C. Jones, University of Wollongong Centre for Health Initiatives Wollongong NSW 2522 AustraliaParri Gregory, University of Wollongong Centre for Health Initiatives Wollongong NSW 2522 AustraliaCaroline Nehill, National Breast and Ovarian Cancer Centre Surry Hills NSW 2010 AustraliaLance Barrie, University of Wollongong Centre for Health Initiatives Wollongong NSW 2522 AustraliaKaren Luxford, National Breast and Ovarian Cancer Centre Surry Hills NSW 2010 AustraliaAn...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3303678</comments>
            <pubDate>Tue, 23 Feb 2010 07:51:42 +0100</pubDate>
            <guid isPermaLink="false">3303678</guid>        </item>
        <item>
            <title>Childhood leukaemia and parental occupational exposure to pesticides: a systematic review and meta-analysis</title>
            <link>http://www.medworm.com/index.php?rid=3295576&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fr12324224703pk60%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;The strongest evidence of an increased risk of childhood leukaemia comes from studies with maternal occupational exposure
 to pesticides. The associations with paternal exposure were weaker and less consistent. These results add to the evidence
 leading to recommend minimizing parental occupational exposure to pesticides. Our findings also support the need to rely more
 on studies that clearly stipulate exposure to pesticides rather than those that assume pesticide exposure because of farm/agriculture
 employment.
 
 
 
 
	Content Type Journal ArticleCategory Review articleDOI 10.1007/s10552-010-9516-7Authors
		Geneviève Van Maele-Fabry, Université catholique de Louvain Louvain Center for Toxicology and Applied Pharmacology (LTAP) Avenue E. Mounier 53.02 1200 Bruss...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3295576</comments>
            <pubDate>Sun, 21 Feb 2010 06:55:59 +0100</pubDate>
            <guid isPermaLink="false">3295576</guid>        </item>
        <item>
            <title>Non-cancer mortality among long-term survivors of adult cancer in Korea: national cancer registry study</title>
            <link>http://www.medworm.com/index.php?rid=3289101&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fv72253438200550q%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Appropriate medical attention for long-term adult cancer survivors, especially younger survivors, is warranted to prevent
 premature deaths from non-cancer causes.
 
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9521-xAuthors
		Dong Wook Shin, National Cancer Center National Cancer Control Institute (NCCI) and Hospital 323 Ilsanro, Ilsan dong-gu Goyang-si Gyeonggi-do 411-769 KoreaEunmi Ahn, National Cancer Center National Cancer Control Institute (NCCI) and Hospital 323 Ilsanro, Ilsan dong-gu Goyang-si Gyeonggi-do 411-769 KoreaHyerim Kim, National Cancer Center National Cancer Control Institute (NCCI) and Hospital 323 Ilsanro, Ilsan dong-gu Goyang-si Gyeonggi-do 411-769 KoreaSohee Park, National Cancer Center National Cancer Control...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3289101</comments>
            <pubDate>Thu, 18 Feb 2010 17:56:22 +0100</pubDate>
            <guid isPermaLink="false">3289101</guid>        </item>
        <item>
            <title>Body size and risk of prostate cancer in Jamaican men</title>
            <link>http://www.medworm.com/index.php?rid=3277121&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F26581qh0203v06uv%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;We investigated the associations between body size and risk of prostate cancer in a hospital-based case–control study in Jamaica.
 Height, weight, waist, and hip circumference were measured at enrollment, and data collected on medical and lifestyle factors
 for newly diagnosed cases (n&amp;nbsp;=&amp;nbsp;243) and controls (n&amp;nbsp;=&amp;nbsp;275). Compared with men in the normal range of waist-hip ratio (WHR), men with WHR&amp;nbsp;≥0.95 were at greater risk of total
 prostate cancer (OR,1.72; CI, 1.01–3.00) and high-grade cancer (OR, 2.02; CI, 1.03–3.96). With additional control for BMI,
 the association with WHR remained significant for total prostate cancer (OR, 1.90; CI, 1.01–3.53) and high-grade disease (OR,
 2.94; CI, 1.34–6.38). There was no association between waist...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3277121</comments>
            <pubDate>Mon, 15 Feb 2010 18:02:16 +0100</pubDate>
            <guid isPermaLink="false">3277121</guid>        </item>
        <item>
            <title>Alcohol and risk of breast cancer in Mexican women</title>
            <link>http://www.medworm.com/index.php?rid=3273544&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fr1mr13776v2mr487%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Our findings support evidence that any alcohol intake increases risk of breast cancer. Insufficient intake of folate may further
 elevate risk for developing breast cancer among women who consume alcohol.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9513-xAuthors
		Jeannette M. Beasley, Group Health Research Institute 1730 Minor Avenue Suite 1600 Seattle WA 98101 USAGloria D. Coronado, Fred Hutchinson Cancer Research Center Cancer Prevention Program 1100 Fairview Ave. N M3-B232 Seattle WA 98109-1024 USAJennifer Livaudais, Fred Hutchinson Cancer Research Center Cancer Prevention Program 1100 Fairview Ave. N M3-B232 Seattle WA 98109-1024 USAAngélica Angeles-Llerenas, Centro de Investigación en Salud Poblacional National Institut...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3273544</comments>
            <pubDate>Sat, 13 Feb 2010 06:49:26 +0100</pubDate>
            <guid isPermaLink="false">3273544</guid>        </item>
        <item>
            <title>Occupational exposure to terbufos and the incidence of cancer in the Agricultural Health Study</title>
            <link>http://www.medworm.com/index.php?rid=3273545&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F95x600318hj27425%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;We found suggestive associations between occupational terbufos use and several cancer sites. However, cautious interpretation
 of these results is warranted by the lack of existing experimental and epidemiologic evidence to support carcinogenic effects
 of terbufos.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9514-9Authors
		Matthew R. Bonner, University at Buffalo Department of Social and Preventive Medicine, School of Public Health and Health Professions 270 Farber Hall Buffalo NY USABrent A. Williams, University at Buffalo Department of Social and Preventive Medicine, School of Public Health and Health Professions 270 Farber Hall Buffalo NY USAJennifer A. Rusiecki, Uniformed Services University of the Health Sciences Department ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3273545</comments>
            <pubDate>Sat, 13 Feb 2010 06:49:25 +0100</pubDate>
            <guid isPermaLink="false">3273545</guid>        </item>
        <item>
            <title>Diabetes and risk of incident cancer: a large population-based cohort study in Israel</title>
            <link>http://www.medworm.com/index.php?rid=3270407&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ft7j50024w605282p%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Type 2 diabetes mellitus has been associated with an increased risk of a variety of cancers in observational studies, but
 few have reported the relationship between diabetes and cancer risk in men and women separately. The main goal of this retrospective
 cohort study was to evaluate the sex-specific risk of incident overall and site-specific cancer among people with DM compared
 with those without, who had no reported history of cancer at the start of the follow-up in January 2000. During an average
 of 8&amp;nbsp;years of follow-up (SD&amp;nbsp;=&amp;nbsp;2.5), we documented 1,639 and 7,945 incident cases of cancer among 16,721 people with DM and
 83,874 free of DM, respectively. In women, DM was associated with an adjusted hazard ratio of 1.96 (95% CI: 1.53–2.50) and
 1.41 (9...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3270407</comments>
            <pubDate>Thu, 11 Feb 2010 11:51:37 +0100</pubDate>
            <guid isPermaLink="false">3270407</guid>        </item>
        <item>
            <title>Genetic variation in sex-steroid receptors and synthesizing enzymes and colorectal cancer risk in women</title>
            <link>http://www.medworm.com/index.php?rid=3270408&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fr63h775810t27178%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Our data offer insufficient support for an association between variation in ESR1, ESR2, PGR, CYP19A1, and HSD17B2 and risk
 for developing colorectal cancer.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9518-5Authors
		Jennifer Lin, Brigham and Women’s Hospital and Harvard Medical School Division of Preventive Medicine, Department of Medicine Boston MA USARobert Y. L. Zee, Brigham and Women’s Hospital and Harvard Medical School Division of Preventive Medicine, Department of Medicine Boston MA USAKuang-Yu Liu, Brigham and Women’s Hospital and Harvard Medical School Department of Anesthesiology, Perioperative and Pain Medicine Boston MA USAShumin M. Zhang, Brigham and Women’s Hospital and Harvard Medical School Division of Pre...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3270408</comments>
            <pubDate>Thu, 11 Feb 2010 11:51:35 +0100</pubDate>
            <guid isPermaLink="false">3270408</guid>        </item>
        <item>
            <title>Intra-individual variation in serum C-reactive protein over 4 years: an implication for epidemiologic studies</title>
            <link>http://www.medworm.com/index.php?rid=3247908&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fu2082860u2458362%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Investigators planning to measure hsCRP only once should design adequately sized studies to preserve inferences for hypothesized
 modest to moderate RRs.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9511-zAuthors
		Elizabeth A. Platz, Johns Hopkins Bloomberg School of Public Health Department of Epidemiology 615 N. Wolfe St., Rm E6132 Baltimore MD 21205 USASiobhan Sutcliffe, Washington University School of Medicine Alvin J. Siteman Cancer Center and the Department of Surgery St. Louis MO USAAngelo M. De Marzo, Johns Hopkins Medical Institutions James Buchanan Brady Urological Institute Baltimore MD USACharles G. Drake, Johns Hopkins Medical Institutions Sidney Kimmel Comprehensive Cancer Center Baltimore MD USANader Rifai, Children...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3247908</comments>
            <pubDate>Thu, 04 Feb 2010 18:13:21 +0100</pubDate>
            <guid isPermaLink="false">3247908</guid>        </item>
        <item>
            <title>Reproductive factors and risk of contralateral breast cancer by BRCA1 and BRCA2 mutation status: results from the WECARE study</title>
            <link>http://www.medworm.com/index.php?rid=3240906&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fk8440nq578414434%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;For two reproductive factors previously shown to be associated with CBC risk, we observed similar associations for BRCA1/2 carriers. This suggests that reproductive variables that affect CBC risk may have similar effects in mutation carriers and
 non-carriers.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9510-0Authors
		Jenny N. Poynter, University of Southern California Department of Preventive Medicine Los Angeles CA USABryan Langholz, University of Southern California Department of Preventive Medicine Los Angeles CA USAJoan Largent, University of California Department of Epidemiology Irvine CA USALene Mellemkjær, Danish Cancer Society Institute of Cancer Epidemiology Copenhagen DenmarkLeslie Bernstein, City of Hope National Medi...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3240906</comments>
            <pubDate>Wed, 03 Feb 2010 17:57:36 +0100</pubDate>
            <guid isPermaLink="false">3240906</guid>        </item>
        <item>
            <title>Spatial Clustering of Myelodysplastic Syndromes (MDS) in the Seattle-Puget Sound Region of Washington State</title>
            <link>http://www.medworm.com/index.php?rid=3209573&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fk06023545x458448%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Interpretation is limited because our data are based on the residential location of the MDS case only at the time of diagnosis.
 Nevertheless, inclusion of identified cluster regions in future population-based research and investigation of individual-level
 exposures could shed light on environmental risk factors for MDS.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9509-6Authors
		Michelle E. Ross, University of Washington Department of Biostatistics Seattle WA USAJon Wakefield, University of Washington Department of Biostatistics Seattle WA USAScott Davis, Fred Hutchinson Cancer Research Center Division of Public Health Sciences 1100 Fairview Avenue N, M4-B874 Seattle WA 98109-1024 USAAnneclaire J. De Roos, Fred Hutchinson Cancer ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3209573</comments>
            <pubDate>Mon, 25 Jan 2010 17:00:09 +0100</pubDate>
            <guid isPermaLink="false">3209573</guid>        </item>
        <item>
            <title>Longitudinal associations of blood markers of insulin and glucose metabolism and cancer mortality in the third National Health and Nutrition Examination Survey</title>
            <link>http://www.medworm.com/index.php?rid=3205712&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ft03815kxtv114634%2F</link>
            <description>In conclusion, hyperglycemia and insulin resistance may be ‘high-risk’ conditions for cancer mortality. Managing these conditions
 may be effective cancer control tools.
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9492-yAuthors
		Niyati Parekh, New York University Department of Nutrition, Food Studies, and Public Health 35 W4th Street, Room 1077F New York NY 10012 USAYong Lin, University of Medicine and Dentistry of New Jersey (UMDNJ) The Cancer Institute of New Jersey 195 Little Albany Street, Room #5536 New Brunswick NJ 08901 USARichard B. Hayes, New York University, Cancer Institute New York NY 10016 USAJeanine B. Albu, Columbia University College of Physicians and Surgeons, St. Luke’s Roosevelt Hospital Center New York NY 10019 USAGrace L. Lu-Yao,...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3205712</comments>
            <pubDate>Fri, 22 Jan 2010 10:12:03 +0100</pubDate>
            <guid isPermaLink="false">3205712</guid>        </item>
        <item>
            <title>Genetic variation in chromosomal translocation breakpoint and immune function genes and risk of non-Hodgkin lymphoma</title>
            <link>http://www.medworm.com/index.php?rid=3194975&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fel5l86484g80653m%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;We found no support for a role of the studied variants in BCL2, CCND1, or MYC in risk of NHL or subtypes, but we provide further evidence of putative susceptibility loci in TNF and IL10 for specific NHL subtypes.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9504-yAuthors
		Pia Fernberg, Karolinska Institutet Department of Medical Epidemiology and Biostatistics P.O. Box 281 171 77 Stockholm SwedenEllen T. Chang, Northern California Cancer Center Fremont CA USAKristina Duvefelt, Karolinska Institutet Department of Biosciences at Novum, Clinical Research Centre Stockholm SwedenHenrik Hjalgrim, Statens Serum Institut Department of Epidemiology Research Copenhagen DenmarkSandra Eloranta, Karolinska Institutet Department of Medical Epide...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3194975</comments>
            <pubDate>Tue, 19 Jan 2010 17:45:02 +0100</pubDate>
            <guid isPermaLink="false">3194975</guid>        </item>
        <item>
            <title>A bupropion smoking cessation clinical trial for cancer patients</title>
            <link>http://www.medworm.com/index.php?rid=3194977&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F56591v3t65570266%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;For patients with depression symptoms, bupropion increases abstinence rates, lowers withdrawal, and increases quality of life.
 However, abstinence rates among patients with depression symptoms were low vs. patients without depression symptoms, who exhibited
 similar abstinence rates when treated with bupropion or transdermal nicotine and counseling alone. These results can guide
 future smoking cessation intervention studies with cancer patients.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9507-8Authors
		Robert A. Schnoll, University of Pennsylvania Department of Psychiatry 3535 Market Street, 4th Floor Philadelphia PA 19104 USAElisa Martinez, Fox Chase Cancer Center 333 Cottman Avenue Philadelphia PA 19111 USAKristina L. Tatum,...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3194977</comments>
            <pubDate>Tue, 19 Jan 2010 17:45:00 +0100</pubDate>
            <guid isPermaLink="false">3194977</guid>        </item>
        <item>
            <title>Human papillomavirus is not associated with colorectal cancer in a large international study</title>
            <link>http://www.medworm.com/index.php?rid=3194976&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fd331424v24187337%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;We conclude that HPV types associated with malignant transformation do not meaningfully contribute to adenocarcinoma of the
 colon.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9502-0Authors
		Michele C. Gornick, University of Michigan Department of Human Genetics, School of Medicine 1524 BSRB, 109 Zina Pitcher Ann Arbor MI 48109-2200 USAXavier Castellsague, IDIBELL-Institut Català d’Oncologia (ICO), CIBER-ESP, L’Hospitalet de Llobregat Barcelona SpainGloria Sanchez, University of Antioquia Medellin ColumbiaThomas J. Giordano, University of Michigan Department of Pathology, School of Medicine Ann Arbor MI USAMichelle Vinco, University of Michigan Department of Pathology, School of Medicine Ann Arbor MI USAJoel K. Greenson, Uni...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3194976</comments>
            <pubDate>Tue, 19 Jan 2010 17:45:00 +0100</pubDate>
            <guid isPermaLink="false">3194976</guid>        </item>
        <item>
            <title>Early-life or lifetime sun exposure, sun reaction, and the risk of squamous cell carcinoma in an Asian population</title>
            <link>http://www.medworm.com/index.php?rid=3190428&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ff477667083p73700%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Lifetime sun exposure was more related to SCC risk in women, while early-age sun exposure was more relevant to men’s SCC risk.
 This may be attributable to different lifestyle between men and women.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9505-xAuthors
		Yen-Ching Chen, National Taiwan University Research Center for Genes, Environment, and Human Health, and Institute of Preventive Medicine, College of Public Health Taipei Taiwan, ROCDavid C. Christiani, Harvard School of Public Health Environmental and Occupational Medicine and Epidemiology Program, Department of Environmental Health Boston MA USAHuey-Jen Jenny Su, National Cheng-Kung University Department of Environmental and Occupational Health, Medical College Tainan Taiw...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3190428</comments>
            <pubDate>Mon, 18 Jan 2010 18:48:39 +0100</pubDate>
            <guid isPermaLink="false">3190428</guid>        </item>
        <item>
            <title>Maternal hormones during early pregnancy: a cross-sectional study</title>
            <link>http://www.medworm.com/index.php?rid=3190429&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fn346336488u86461%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Our analyses indicated that potentially modifiable maternal characteristics (maternal weight and smoking) influence first-trimester
 pregnancy maternal hormone concentrations.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9500-2Authors
		Tianhui Chen, German Cancer Research Center Division of Cancer Epidemiology In Neuenheimer Feld 280 Heidelberg 69120 GermanyEva Lundin, University of Umeå Department of Medical Biosciences Umeå SwedenKjell Grankvist, University of Umeå Department of Medical Biosciences Umeå SwedenAnne Zeleniuch-Jacquotte, New York University School of Medicine Department of Environmental Medicine New York USAMarianne Wulff, University of Umeå Department of Clinical Sciences Umeå SwedenYelena Afanasyeva, New Yo...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3190429</comments>
            <pubDate>Mon, 18 Jan 2010 18:48:37 +0100</pubDate>
            <guid isPermaLink="false">3190429</guid>        </item>
        <item>
            <title>Risk factors for lung cancer: a case–control study in Hong Kong women</title>
            <link>http://www.medworm.com/index.php?rid=3190432&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fd55432052p512215%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;To identify etiological connections of lung cancer in Chinese women in Hong Kong, who are among the highest in lung cancer
 incidence and mortality, we conducted a case–control study, in which 279 female lung cancer cases and 322 controls were selected
 and frequency matched. A variety of information, including dietary habits, occupational history, smoking, domestic environmental
 exposures, and family history of cancer was collected, and their associations with lung cancer were analyzed with logistic
 analysis approach. In addition to positive associations with exposures to cooking emissions and to radon at home, smoking
 and family cancer history, we observed that increasing consumption of meat was linked to a higher risk, whereas consumptions
 of vegetables had a s...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3190432</comments>
            <pubDate>Mon, 18 Jan 2010 18:48:36 +0100</pubDate>
            <guid isPermaLink="false">3190432</guid>        </item>
        <item>
            <title>Moderate physical activity and breast cancer risk: the effect of menopausal status</title>
            <link>http://www.medworm.com/index.php?rid=3190431&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fm71418mq95937103%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Strategies need to be identified that will engage women in physical activity programs.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9487-8Authors
		Angélica Ángeles-Llerenas, Centro de Investigaciones en Salud Poblacional Instituto Nacional de Salud Pública Avenida Universidad 655, Col. Santa María Ahuacatitlán 62508 Cuernavaca Morelos MéxicoCarolina Ortega-Olvera, Centro de Investigaciones en Salud Poblacional Instituto Nacional de Salud Pública Avenida Universidad 655, Col. Santa María Ahuacatitlán 62508 Cuernavaca Morelos MéxicoEdelmiro Pérez-Rodríguez, Hospital Universitario Monterrey Nuevo León MéxicoJesús Pablo Esparza-Cano, IMSS Hospital No. 23 de Ginecología Monterrey Nuevo León MéxicoEduardo Lazcano-Ponce...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3190431</comments>
            <pubDate>Mon, 18 Jan 2010 18:48:36 +0100</pubDate>
            <guid isPermaLink="false">3190431</guid>        </item>
        <item>
            <title>A multi-center prospective cohort study of benign breast disease and risk of subsequent breast cancer</title>
            <link>http://www.medworm.com/index.php?rid=3190430&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fw3v222106gu016n6%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Our results indicate that, compared to women with normal pathology/non-proliferative disease, women with proliferative disease
 without atypia have a modestly increased risk of breast cancer, whereas women with atypical hyperplasia have a substantially
 increased risk.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9508-7Authors
		Geoffrey C. Kabat, Albert Einstein College of Medicine Department of Epidemiology and Population Health Bronx NY 10461 USAJoan G. Jones, New York–Presbyterian Hospital New York NY 10032 USANeal Olson, Kaiser Permanente Northwest Center for Health Research Portland OR USAAbdissa Negassa, Albert Einstein College of Medicine Department of Epidemiology and Population Health Bronx NY 10461 USACatherine Duggan, ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3190430</comments>
            <pubDate>Mon, 18 Jan 2010 18:48:36 +0100</pubDate>
            <guid isPermaLink="false">3190430</guid>        </item>
        <item>
            <title>Lifetime exposure to arsenic in drinking water and bladder cancer: a population-based case–control study in Michigan, USA</title>
            <link>http://www.medworm.com/index.php?rid=3190433&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fv4193577680823h1%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;We did not find persuasive evidence of an association between low-level arsenic exposure and bladder cancer. Selecting the
 appropriate exposure metric needs to be thoughtfully considered when investigating risk from low-level arsenic exposure.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9503-zAuthors
		Jaymie R. Meliker, Stony Brook University Medical Center Graduate Program in Public Health, Department of Preventive Medicine HSC L3, Rm 071 Stony Brook NY 11794-8338 USAMelissa J. Slotnick, University of Michigan Department of Environmental Health Sciences, School of Public Health Ann Arbor MI USAGillian A. AvRuskin, BioMedware, Inc. Ann Arbor MI USADavid Schottenfeld, University of Michigan Department of Epidemiology, School of P...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3190433</comments>
            <pubDate>Mon, 18 Jan 2010 18:48:34 +0100</pubDate>
            <guid isPermaLink="false">3190433</guid>        </item>
        <item>
            <title>Defining research priorities for pancreatic cancer in Australia: results of a consensus development process</title>
            <link>http://www.medworm.com/index.php?rid=3177056&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F82280r5g505g82v2%2F</link>
            <description>Discussion&amp;nbsp;&amp;nbsp;This prioritisation exercise provided a much needed “road map” for research prioritisation in PC and served as a checklist
 to researchers applying for PC research grants to confirm how their research can contribute towards accelerating progress
 in PC research in Australia.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-010-9501-1Authors
		Monica C. Robotin, Cancer Council NSW 153 Dowling St Woolloomooloo, Sydney NSW 2011 AustraliaSandra C. Jones, University of Wollongong Centre for Health Initiatives Wollongong NSW AustraliaAndrew V. Biankin, Garvan Institute of Medical Research Cancer Research Program Sydney NSW AustraliaLouise Waters, University of Wollongong Centre for Health Initiatives Wollongong NSW AustraliaDon Iverson, Unive...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3177056</comments>
            <pubDate>Wed, 13 Jan 2010 17:54:25 +0100</pubDate>
            <guid isPermaLink="false">3177056</guid>        </item>
        <item>
            <title>Insulin resistance-related biomarker clustering and subclinical inflammation as predictors of cancer mortality during 21.5 years of follow-up</title>
            <link>http://www.medworm.com/index.php?rid=3177058&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fp5nj3j111r7u3518%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Despite insulin concentrations or derived indices of insulin resistance failing to predict cancer mortality, insulin resistance-related
 biomarker clustering was highly predictive and predicted independently of simple measures of subclinical inflammation.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9499-4Authors
		Wann J. Loh, Imperial College London Endocrinology and Metabolic Medicine St Mary’s Campus, Mint Wing, Praed Street London W2 1PG UKBernard V. North, Imperial College London Department of Epidemiology and Public Health London UKDesmond G. Johnston, Imperial College London Endocrinology and Metabolic Medicine St Mary’s Campus, Mint Wing, Praed Street London W2 1PG UKIan F. Godsland, Imperial College London Endocrinolo...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3177058</comments>
            <pubDate>Wed, 13 Jan 2010 17:54:24 +0100</pubDate>
            <guid isPermaLink="false">3177058</guid>        </item>
        <item>
            <title>Statin drugs, serum cholesterol, and prostate-specific antigen in the National Health and Nutrition Examination Survey 2001–2004</title>
            <link>http://www.medworm.com/index.php?rid=3177057&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fy8465600802722lx%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Statin users and men with lower cholesterol may have lower PSA. If so, the probability of detecting asymptomatic prostate
 cancer might be lower at present, but these cases might be more likely to be diagnosed at an advanced stage in the future.
 Thus, PSA-associated bias is unlikely to explain the inverse association of statins with advanced prostate cancer.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9494-9Authors
		Alison M. Mondul, Johns Hopkins Bloomberg School of Public Health Department of Epidemiology Baltimore MD USAElizabeth Selvin, Johns Hopkins Bloomberg School of Public Health Department of Epidemiology Baltimore MD USAAngelo M. De Marzo, Johns Hopkins University School of Medicine Department of Pathology Baltimore MD ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3177057</comments>
            <pubDate>Wed, 13 Jan 2010 17:54:24 +0100</pubDate>
            <guid isPermaLink="false">3177057</guid>        </item>
        <item>
            <title>Toenail selenium status and DNA repair capacity among female BRCA1 mutation carriers</title>
            <link>http://www.medworm.com/index.php?rid=3168586&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ft2735150684476r2%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Selenium is an important cofactor of various antioxidant enzymes and has been shown to enhance DNA repair in normal human
 fibroblasts. Oral selenium supplementation has also been shown to decrease the number of chromosome breaks in BRCA1 mutation carriers. Because the predisposition to cancer among BRCA1 mutation carriers may be linked to high rates of DNA damage and chromosome breakage, we evaluated the association between
 toenail selenium concentrations and three measures of DNA repair capacity (the single-cell alkaline gel electrophoresis (comet)
 assay, the micronucleus test, and the enumeration of γ-H2AX nuclear foci) in female BRCA1 mutation carriers and in non-carriers. Toenail selenium levels were inversely associated with levels of chromosomal damage
 follow...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3168586</comments>
            <pubDate>Fri, 08 Jan 2010 21:40:28 +0100</pubDate>
            <guid isPermaLink="false">3168586</guid>        </item>
        <item>
            <title>Ensuring long-term sustainability of existing cohorts remains the highest priority to inform cancer prevention and control</title>
            <link>http://www.medworm.com/index.php?rid=3168587&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fj2853r1n12722565%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;The case for continued follow-up of existing cohorts arises from the key attributes of cohorts that are already meeting the
 goals proposed by Potter for the creation of a new cohort. These attributes include the basic nature of ongoing cohorts in
 that they are, by design, hypothesis-driven and must adapt to emerging technologies over time. Importantly, cohort investigators
 must identify and address gaps in knowledge that will inform public health strategies and clinical practices. Above all, cohorts
 must capitalize on their unique features to address public health priorities and inform our prevention strategies. Continued
 follow-up adds substantial return on investment to guide cancer prevention.
 
	Content Type Journal ArticleCategory EditorialDOI 10.1007/s10552-0...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3168587</comments>
            <pubDate>Fri, 08 Jan 2010 21:40:27 +0100</pubDate>
            <guid isPermaLink="false">3168587</guid>        </item>
        <item>
            <title>Birth weight, breast cancer susceptibility loci, and breast cancer risk</title>
            <link>http://www.medworm.com/index.php?rid=3154301&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F34567g83v1tm5104%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Although our findings require confirmation, we found suggestive evidence that genetic susceptibility modifies the positive
 association of birth weight with breast cancer.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9496-7Authors
		Rulla M. Tamimi, Harvard School of Public Health Department of Epidemiology 677 Huntington Avenue Boston MA 02115 USAPagona Lagiou, Harvard School of Public Health Department of Epidemiology 677 Huntington Avenue Boston MA 02115 USAKamila Czene, Karolinska Institutet Department of Medical Epidemiology and Biostatistics Box 281 171 77 Stockholm SwedenJianjun Liu, Genome Institute of Singapore Population Genetics Singapore 138672 SingaporeAnders Ekbom, Karolinska Institutet/Karolinska University Hospital ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3154301</comments>
            <pubDate>Wed, 06 Jan 2010 18:06:44 +0100</pubDate>
            <guid isPermaLink="false">3154301</guid>        </item>
        <item>
            <title>Parkinson’s disease and cancer risk: a systematic review and meta-analysis</title>
            <link>http://www.medworm.com/index.php?rid=3154302&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fr771q44585830038%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Studies on cancer risk among patients with PD collectively show significantly reduced cancer risk ratios. Further research
 to explain the biological mechanisms, particularly for the association with non-smoking-related cancers, appears warranted.
 
 
 
	Content Type Journal ArticleCategory Original PaperDOI 10.1007/s10552-009-9497-6Authors
		Archna Bajaj, Brigham and Women’s Hospital and Harvard Medical School Channing Laboratory, Department of Medicine 181 Longwood Avenue Boston MA 02115 USAJane A. Driver, Brigham and Women’s Hospital Division of Aging, Department of Medicine Boston MA USAEva S. Schernhammer, Brigham and Women’s Hospital and Harvard Medical School Channing Laboratory, Department of Medicine 181 Longwood Avenue Boston MA 02115 USA
	

	
		Journ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3154302</comments>
            <pubDate>Wed, 06 Jan 2010 18:06:41 +0100</pubDate>
            <guid isPermaLink="false">3154302</guid>        </item>
        <item>
            <title>Prospective study of physical activity and risk of primary adenocarcinomas of the oesophagus and stomach in the EPIC (European Prospective Investigation into Cancer and nutrition) cohort</title>
            <link>http://www.medworm.com/index.php?rid=3154303&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F5n272700m7008225%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Overall and distal (non-cardia) gastric tumours were inversely associated with time spent on cycling and sports and a total
 PA index. No association was found for any type of PA and risk of cardia cancers of the stomach.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9493-xAuthors
		José María Huerta, CIBER Epidemiología y Salud Pública (CIBERESP) Barcelona SpainCarmen Navarro, CIBER Epidemiología y Salud Pública (CIBERESP) Barcelona SpainMaría-Dolores Chirlaque, CIBER Epidemiología y Salud Pública (CIBERESP) Barcelona SpainMaría-José Tormo, CIBER Epidemiología y Salud Pública (CIBERESP) Barcelona SpainKaren Steindorf, German Cancer Research Center Unit of Environmental Epidemiology Heidelberg GermanyGenevieve Buckland,...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3154303</comments>
            <pubDate>Tue, 05 Jan 2010 17:01:24 +0100</pubDate>
            <guid isPermaLink="false">3154303</guid>        </item>
        <item>
            <title>Oral health and risk for head and neck squamous cell carcinoma: the Carolina Head and Neck Cancer Study</title>
            <link>http://www.medworm.com/index.php?rid=3154304&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F951kx2117666627m%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;These data provide support for a possible modest association of periodontal disease, as measured by self-reported tooth loss
 indicators, but not tooth loss per se, with SCCHN risk.
 
 
 
	Content Type Journal ArticleCategory Original PaperDOI 10.1007/s10552-009-9486-9Authors
		Kimon Divaris, University of North Carolina Department of Epidemiology, CB#7435, Gillings School of Global Public Health Chapel Hill NC 27599-7435 USAAndrew F. Olshan, University of North Carolina Department of Epidemiology, CB#7435, Gillings School of Global Public Health Chapel Hill NC 27599-7435 USAJoanna Smith, University of North Carolina Department of Epidemiology, CB#7435, Gillings School of Global Public Health Chapel Hill NC 27599-7435 USAMary E. Bell, University of North Carolina Dep...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3154304</comments>
            <pubDate>Mon, 04 Jan 2010 18:22:13 +0100</pubDate>
            <guid isPermaLink="false">3154304</guid>        </item>
        <item>
            <title>Weight change patterns among breast cancer survivors: results from the Shanghai breast cancer survival study</title>
            <link>http://www.medworm.com/index.php?rid=3134341&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ferp6x578337m9758%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;In a population-based cohort study of 5,014 women with stage 0–III breast cancer, we evaluated weight change patterns from
 diagnosis to 6, 18, and 36&amp;nbsp;months post-diagnosis. Patients were recruited to the study approximately 6&amp;nbsp;months after cancer
 diagnosis between 2002 and 2006 and followed through 36&amp;nbsp;months post-diagnosis. The medians of weight change from diagnosis
 to 6, 18, and 36&amp;nbsp;months post-diagnosis were 1.0, 2.0, and 1.0&amp;nbsp;kg, respectively. Approximately, 26% of survivors gained ≥5%
 of their at-diagnosis body weight during the first 6&amp;nbsp;months after diagnosis, while 37% and 33% of women gained the same percentage
 of weight at 18 and 36&amp;nbsp;months post-diagnosis. More weight gain was observed among women who had a more advanced d...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3134341</comments>
            <pubDate>Wed, 30 Dec 2009 16:46:10 +0100</pubDate>
            <guid isPermaLink="false">3134341</guid>        </item>
        <item>
            <title>Timeliness and follow-up patterns of cervical cancer detection in a cohort of medically underserved California women</title>
            <link>http://www.medworm.com/index.php?rid=3134342&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fw70377wgt12p3769%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Patient’s demographic characteristics determine the odds of being scheduled for final diagnosis and treatment as well as timeliness
 of follow-up from screening to final diagnosis. Findings suggest that the dual goal of reducing health disparities and cost-effective
 detection and treatment of precancerous disease to prevent cervical cancers cannot be achieved without consideration of racial/ethnic
 differences and needs.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9473-1Authors
		Farzaneh Tabnak, Division of Chronic Diseases and Injury Control, California Department of Public Health Cancer Detection Section Sacramento CA USAHans-Georg Müller, University of California Department of Statistics Davis CA USAJane-Ling Wang, Univers...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3134342</comments>
            <pubDate>Wed, 30 Dec 2009 16:46:07 +0100</pubDate>
            <guid isPermaLink="false">3134342</guid>        </item>
        <item>
            <title>Minerals and vitamins and the risk of bladder cancer: results from the New Hampshire Study</title>
            <link>http://www.medworm.com/index.php?rid=3134343&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fm20n74hh471k4934%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Our findings suggest further investigation of the effect of vitamin E, carotenoids, vitamin D, thiamin, and niacin on bladder
 cancer risk may be warranted. Future studies should focus on high risk groups such as heavy smokers and older individuals.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9490-0Authors
		Maree T. Brinkman, The Cancer Council Victoria Cancer Epidemiology Centre 1 Rathdowne Street Carlton VIC 3053 AustraliaMargaret R. Karagas, Dartmouth Medical School and Norris-Cotton Cancer Centre Section of Biostatistics and Epidemiology, Department of Community and Family Medicine Lebanon NH USAMichael Scott Zens, Dartmouth Medical School and Norris-Cotton Cancer Centre Section of Biostatistics and Epidemiology, Department of...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3134343</comments>
            <pubDate>Wed, 30 Dec 2009 16:46:06 +0100</pubDate>
            <guid isPermaLink="false">3134343</guid>        </item>
        <item>
            <title>Genes involved with folate uptake and distribution and their association with colorectal cancer risk</title>
            <link>http://www.medworm.com/index.php?rid=3125087&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fg845h0778w8k1114%2F</link>
            <description>In conclusion, we found no significant evidence that genetic variants
 in FOLR1, GGH, FPGS and SLC19A1 are associated with the risk of colorectal cancer.
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9489-6Authors
		Jane C. Figueiredo, University of Southern California Department of Preventive Medicine, Keck School of Medicine 1450 Biggy Street Room 1509J Los Angeles CA 90033 USAA. Joan Levine, University of Southern California Department of Preventive Medicine, Keck School of Medicine 1450 Biggy Street Room 1509J Los Angeles CA 90033 USAWon H. Lee, University of Southern California Department of Preventive Medicine, Keck School of Medicine 1450 Biggy Street Room 1509J Los Angeles CA 90033 USADavid V. Conti, University of Southern California Department of Pre...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3125087</comments>
            <pubDate>Sat, 26 Dec 2009 16:39:08 +0100</pubDate>
            <guid isPermaLink="false">3125087</guid>        </item>
        <item>
            <title>Cancer incidence in the vicinity of Finnish nuclear power plants: an emphasis on childhood leukemia</title>
            <link>http://www.medworm.com/index.php?rid=3125086&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fqp5446740wn53012%2F</link>
            <description>The objective of this paper was to study cancer incidence, especially leukemia in children (&amp;lt;15&amp;nbsp;years), in the vicinity of
 Finnish nuclear power plants (NPPs). We used three different approaches: ecological analysis at municipality level, residential
 cohorts defined from census data, and case–control analysis with individual residential histories. The standardized incidence
 ratio of childhood leukemia for the seven municipalities in the vicinity of NPPs was 1.0 (95% CI 0.6, 1.6) compared to the
 rest of Finland. The two cohorts defined by censuses of 1980 and 1990 gave rate ratios of 1.0 (95% CI 0.3, 2.6) and 0.9 (95%
 CI 0.2, 2.7), respectively, for childhood leukemia in the population residing within 15&amp;nbsp;km from the NPPs compared to the 15–50&amp;nbsp;km
 zone. The case–...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3125086</comments>
            <pubDate>Sat, 26 Dec 2009 16:39:08 +0100</pubDate>
            <guid isPermaLink="false">3125086</guid>        </item>
        <item>
            <title>Racial differences in the incidence of breast cancer subtypes defined by combined histologic grade and hormone receptor status</title>
            <link>http://www.medworm.com/index.php?rid=3105972&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F135k66673v2h2412%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Breast cancer encompasses several distinct clinical entities of very different characteristics and behaviors, a fact which
 likely contributes to the higher breast cancer mortality in African-Americans (AA) despite the higher incidence in European-Americans
 (EA). We are interested in how incidence variability in cancer subtypes defined by combined estrogen receptor (ER) and grade
 contributes to racial mortality disparities. As an initial step, we compared age-specific and age-adjusted incidence rates
 for each ER/Grade subtype in South Carolina (SC—a southern state) with Ohio (a northern mid-western state), using state registry
 data for 1996–2004. Each ER/Grade subtype had a distinct incidence pattern and rate, with three striking racial/geographic
 differences. ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3105972</comments>
            <pubDate>Fri, 18 Dec 2009 16:11:46 +0100</pubDate>
            <guid isPermaLink="false">3105972</guid>        </item>
        <item>
            <title>The impact of delayed blood centrifuging, choice of collection tube, and type of assay on 25-hydroxyvitamin D concentrations</title>
            <link>http://www.medworm.com/index.php?rid=3105973&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F0026815nw70x005j%2F</link>
            <description>We examined the potential impact of delayed
 blood centrifuging, choice of collection tube, and type of assay on 25(OH)D concentrations. Blood samples from 20 healthy
 volunteers underwent alternative laboratory procedures: four centrifuging times (2, 24, 72, and 96&amp;nbsp;h after blood draw); three
 types of collection tubes (red top serum tube, two different plasma anticoagulant tubes containing heparin or EDTA); and two
 types of assays (DiaSorin radioimmunoassay [RIA] and chemiluminescence immunoassay [CLIA/LIAISON®]). Log-transformed 25(OH)D concentrations were analyzed using the generalized estimating equations (GEE) linear regression
 models. We found no difference in 25(OH)D concentrations by centrifuging times or type of assay. There was some indication
 of a difference in 25(OH)D ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3105973</comments>
            <pubDate>Fri, 18 Dec 2009 07:12:22 +0100</pubDate>
            <guid isPermaLink="false">3105973</guid>        </item>
        <item>
            <title>Endotoxin exposure and lung cancer risk: a systematic review and meta-analysis of the published literature on agriculture and cotton textile workers</title>
            <link>http://www.medworm.com/index.php?rid=3085897&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fd5g7822152373k32%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Despite several limitations, this meta-analysis based on high-quality studies adds weight to the hypothesis that occupational
 exposure to endotoxin in cotton textile production and agriculture is protective against lung cancer.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9483-zAuthors
		Virissa Lenters, Utrecht University Institute for Risk Assessment Sciences, Division Environmental Epidemiology P.O. Box 80178 3508 TD Utrecht The NetherlandsIoannis Basinas, Aarhus University Department of Environmental and Occupational Medicine, Institute of Public Health Aarhus DenmarkLaura Beane-Freeman, National Institutes of Health Occupational and Environmental Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cance...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3085897</comments>
            <pubDate>Sat, 12 Dec 2009 14:18:51 +0100</pubDate>
            <guid isPermaLink="false">3085897</guid>        </item>
        <item>
            <title>Determinants of serum 25 hydroxyvitamin D levels in a nationwide cohort of blacks and non-Hispanic whites</title>
            <link>http://www.medworm.com/index.php?rid=3085899&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fk342h80q1824g478%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Combining races with a term for race and using variables with measured UV radiation capture the variance in s25(OH)D levels
 better than analyzing races separately.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9481-1Authors
		Jacqueline Chan, Loma Linda University Department of Epidemiology and Biostatistics, School of Public Health 24785 Stewart St Loma Linda CA 92350 USAKaren Jaceldo-Siegl, Loma Linda University Department of Epidemiology and Biostatistics, School of Public Health 24785 Stewart St Loma Linda CA 92350 USAGary E. Fraser, Loma Linda University Department of Epidemiology and Biostatistics, School of Public Health 24785 Stewart St Loma Linda CA 92350 USA
	

	
		Journal Cancer Causes and ControlOnline ISSN 1573-7225Prin...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3085899</comments>
            <pubDate>Thu, 10 Dec 2009 15:12:27 +0100</pubDate>
            <guid isPermaLink="false">3085899</guid>        </item>
        <item>
            <title>One-carbon metabolism and CpG island methylator phenotype status in incident colorectal cancer: a nested case–referent study</title>
            <link>http://www.medworm.com/index.php?rid=3085898&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fup0412t562521180%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Though limited by low power, these findings suggest the possibility of different roles for one-carbon metabolism in different
 pathways of colorectal tumorigenesis.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9484-yAuthors
		Bethany Van Guelpen, Umeå University Department of Medical Biosciences, Pathology Building 6M, 2nd Floor 90185 Umeå SwedenAnna M. Dahlin, Umeå University Department of Medical Biosciences, Pathology Building 6M, 2nd Floor 90185 Umeå SwedenJohan Hultdin, Umeå University Department of Medical Biosciences, Clinical Chemistry Umeå SwedenVincy Eklöf, Umeå University Department of Medical Biosciences, Pathology Building 6M, 2nd Floor 90185 Umeå SwedenIngegerd Johansson, Umeå University Department of Odontol...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3085898</comments>
            <pubDate>Thu, 10 Dec 2009 15:12:27 +0100</pubDate>
            <guid isPermaLink="false">3085898</guid>        </item>
        <item>
            <title>Proanthocyanidins and the risk of colorectal cancer in Italy</title>
            <link>http://www.medworm.com/index.php?rid=3085901&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F27j3821p0t685281%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Proanthocyanidins are a group of polymers of flavanols. Animal and in vitro studies suggest they decrease cancer risk, particularly
 of colorectal cancer. We used data from an Italian case–control study to investigate whether proanthocyanidins are related
 to colorectal cancer risk. Cases were 1,953 patients with incident, histologically confirmed colorectal cancer (1,225 colon
 cancers, 728 rectal cancers). Controls were 4,154 patients admitted for acute, non-neoplastic conditions. A reproducible and
 valid food frequency questionnaire was used. We estimated odds ratios (ORs) through multiple logistic regression models, including
 terms for potential confounding factors, and energy intake. A trend of decreasing risk with increasing intake of proanthocyanidins
 emerge...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3085901</comments>
            <pubDate>Thu, 10 Dec 2009 15:12:24 +0100</pubDate>
            <guid isPermaLink="false">3085901</guid>        </item>
        <item>
            <title>Health and lifestyle behaviors among persons at risk of Lynch syndrome</title>
            <link>http://www.medworm.com/index.php?rid=3085900&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fj211735517072578%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;We identified several health behaviors for potential intervention, including smoking, alcohol use, and diet. Genetic counseling
 offers a promising avenue for education and risk behavior reduction in persons at increased risk for cancer due to a familial
 or genetic predisposition, and a teachable moment to introduce lifestyle modifications.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9482-0Authors
		Allison M. Burton, The University of Texas M. D. Anderson Cancer Center Department of Behavioral Science-Unit 1330 P.O. Box 301439 Houston TX 77230-1439 USASusan K. Peterson, The University of Texas M. D. Anderson Cancer Center Department of Behavioral Science-Unit 1330 P.O. Box 301439 Houston TX 77230-1439 USASalma K. Marani, The Uni...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3085900</comments>
            <pubDate>Thu, 10 Dec 2009 15:12:24 +0100</pubDate>
            <guid isPermaLink="false">3085900</guid>        </item>
        <item>
            <title>Recreational physical activity and risk of epithelial ovarian cancer</title>
            <link>http://www.medworm.com/index.php?rid=3064158&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F54t572378368v0wn%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Our findings are compatible with an overall reduction in risk of invasive epithelial ovarian cancer associated with recreational
 activity but suggest that this association may differ in women with different histologic types of disease. Inconsistent findings
 across studies that have considered histologic type indicate that this issue is not yet resolved.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9479-8Authors
		Mary Anne Rossing, Fred Hutchinson Cancer Research Center Program in Epidemiology P.O. Box 19024 Seattle WA 98108-1024 USAKara L. Cushing-Haugen, Fred Hutchinson Cancer Research Center Program in Epidemiology P.O. Box 19024 Seattle WA 98108-1024 USAKristine G. Wicklund, Fred Hutchinson Cancer Research Center Program in E...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3064158</comments>
            <pubDate>Tue, 01 Dec 2009 21:02:38 +0100</pubDate>
            <guid isPermaLink="false">3064158</guid>        </item>
        <item>
            <title>Association between cervical dysplasia and human papillomavirus in HIV seropositive women from Johannesburg South Africa</title>
            <link>http://www.medworm.com/index.php?rid=3053744&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F21j8451l5445l081%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;The burden of HPV infection and HSIL was high and correlated with HIV-induced immunosuppression. HPV 16 was the most common
 type in HSIL and increased in prevalence with greater immune suppression. Prophylactic HPV 16 vaccination could prevent approximately
 40% of HSIL cases. Strengthening screening programs is imperative in this population.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9475-zAuthors
		Cynthia Firnhaber, University of Witwatersrand Department of Medicine, Clinical HIV Research Unit Johannesburg South AfricaHoa Van Le, University of North Carolina Gillings School of Global Public Health Department of Epidemiology Campus Box 7435 Chapel Hill NC 27599 USAAudrey Pettifor, University of North Carolina Gillings School of...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3053744</comments>
            <pubDate>Tue, 01 Dec 2009 08:32:31 +0100</pubDate>
            <guid isPermaLink="false">3053744</guid>        </item>
        <item>
            <title>Blood pressure and risk of prostate cancer: cohort Norway (CONOR)</title>
            <link>http://www.medworm.com/index.php?rid=3053745&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fh2323m41158k5111%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Raised blood pressure was associated with an increased risk of prostate cancer, particularly advanced cancers at diagnosis.
 Understanding the mechanisms underlying these findings may provide biological insights into prostate carcinogenesis. Even
 if the association was causal, our data suggest that raised blood pressure would account for only 3% of prostate cancers,
 so the public health impact of this association may be limited.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9477-xAuthors
		Richard M. Martin, University of Bristol Department of Social Medicine Bristol BS8 2PS UKLars Vatten, University of Bristol Department of Social Medicine Bristol BS8 2PS UKDavid Gunnell, University of Bristol Department of Social Medicine Bristo...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3053745</comments>
            <pubDate>Tue, 01 Dec 2009 08:32:30 +0100</pubDate>
            <guid isPermaLink="false">3053745</guid>        </item>
        <item>
            <title>Dietary habits and risk of pancreatic cancer: an Italian case–control study</title>
            <link>http://www.medworm.com/index.php?rid=3040796&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ff5t78g05j0617043%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;The present study supports an inverse association between fruits and vegetables and pancreatic cancer risk, and it confirms
 a direct relation with meat. The increased risk for table sugar suggests that insulin resistance may play a role in pancreatic
 carcinogenesis.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9480-2Authors
		Jerry Polesel, IRCCS Unità di Epidemiologia e Biostatistica, Centro di Riferimento Oncologico Via Franco Gallini, 2 33081 Aviano Pordenone ItalyRenato Talamini, IRCCS Unità di Epidemiologia e Biostatistica, Centro di Riferimento Oncologico Via Franco Gallini, 2 33081 Aviano Pordenone ItalyEva Negri, Istituto di Ricerche Farmacologiche “Mario Negri” Milan ItalyCristina Bosetti, Istituto di Ricerche Farm...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3040796</comments>
            <pubDate>Sun, 29 Nov 2009 09:32:24 +0100</pubDate>
            <guid isPermaLink="false">3040796</guid>        </item>
        <item>
            <title>Predictors of endoscopic colorectal cancer screening over time in 11 states</title>
            <link>http://www.medworm.com/index.php?rid=3040797&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F345301r18wr424j2%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Screening for CRC in our Medicare-insured sample was less than optimal, and reasons varied considerably across states. Negative
 managed care spillovers were observed, demonstrating that policy interventions to improve screening rates should reflect local
 market conditions as well as population diversity.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9476-yAuthors
		Lee Mobley, RTI International 3040 Cornwallis Road, Research Triangle Park Durham NC 27709-2194 USATzy-Mey Kuo, RTI International 3040 Cornwallis Road, Research Triangle Park Durham NC 27709-2194 USAMatthew Urato, RTI International 3040 Cornwallis Road, Research Triangle Park Durham NC 27709-2194 USAJohn Boos, RTI International 3040 Cornwallis Road, Research Triangle Pa...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3040797</comments>
            <pubDate>Sat, 28 Nov 2009 07:05:00 +0100</pubDate>
            <guid isPermaLink="false">3040797</guid>        </item>
        <item>
            <title>Assessing the acceptability of self-sampling for HPV among Haitian immigrant women: CBPR in action</title>
            <link>http://www.medworm.com/index.php?rid=3040799&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fu421033u7482h08n%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;When paired with CHWs, who are of Haitian descent and well respected in Little Haiti, self-sampling is a highly acceptable
 method of cervical screening for Haitian women in this ethnic enclave. This approach addresses critical access barriers, including
 poverty, language difficulties, and sociocultural concerns about modesty, that may similarly affect Pap smear utilization
 among other immigrant or medically underserved population sub-groups. Coupled with generally positive reviews of the device,
 the low rate of insufficient specimens for testing suggests that this device is promising for use in non-clinical settings.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9474-0Authors
		Lindley Barbee, University of Washington School of ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3040799</comments>
            <pubDate>Fri, 27 Nov 2009 07:05:48 +0100</pubDate>
            <guid isPermaLink="false">3040799</guid>        </item>
        <item>
            <title>A case–control study of reproductive factors, female hormone use, and risk of pancreatic cancer</title>
            <link>http://www.medworm.com/index.php?rid=3040800&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fh44633661822724n%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Findings from several previous studies that have assessed the relation of reproductive factors and female hormone use to the
 risk of pancreatic cancer are inconclusive. The authors examined the association between reproductive factors and the use
 of oral contraceptives and postmenopausal hormone therapy to the risk of pancreatic cancer among 284 patients with pancreatic
 cancer and 1,096 controls using data from the hospital-based Case–Control Surveillance Study. Older age at first pregnancy
 and long-duration oral contraceptive use were associated with an increased risk of pancreatic cancer: the odds ratio was 2.0
 (95% CI: 1.1–3.3) for first birth at age 30 or older compared with before age 20 (p for trend&amp;nbsp;=&amp;nbsp;0.042) and 2.0 (95% CI: 1.0–4.0) for ten o...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3040800</comments>
            <pubDate>Thu, 26 Nov 2009 08:02:35 +0100</pubDate>
            <guid isPermaLink="false">3040800</guid>        </item>
        <item>
            <title>Race moderates the relationship between obesity and colorectal cancer screening in women</title>
            <link>http://www.medworm.com/index.php?rid=3040801&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F910g2244x6812477%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Obesity is associated with lower CRC screening rates in white, but not African-American women.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9469-xAuthors
		Lucia A. Leone, University of North Carolina at Chapel Hill Lineberger Comprehensive Cancer Center 1700 Martin Luther King Jr. Blvd., CB#7294 Chapel Hill NC 27599-7294 USAMarci K. Campbell, University of North Carolina at Chapel Hill Lineberger Comprehensive Cancer Center 1700 Martin Luther King Jr. Blvd., CB#7294 Chapel Hill NC 27599-7294 USAJessie A. Satia, University of North Carolina at Chapel Hill Lineberger Comprehensive Cancer Center 1700 Martin Luther King Jr. Blvd., CB#7294 Chapel Hill NC 27599-7294 USAJ. Michael Bowling, University of North Carolina at Chapel Hill Depa...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3040801</comments>
            <pubDate>Thu, 26 Nov 2009 08:02:34 +0100</pubDate>
            <guid isPermaLink="false">3040801</guid>        </item>
        <item>
            <title>Human papillomavirus infection in women with and without cervical cancer in Nepal</title>
            <link>http://www.medworm.com/index.php?rid=3031672&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fn6n2783h77230021%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Nepal has an intermediate burden of HPV infection, lower than many areas in India and China. Approximately 80% of cervical
 cancer in Nepal is theoretically preventable by HPV16/18 vaccines. In the meantime, screen-and-treat approaches should be
 encouraged to overcome difficulties that were encountered to recall women with screening-positive findings.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9467-zAuthors
		Ang Tshering Lama Sherpa, Kist Medical College Lalitpur NepalGary M. Clifford, International Agency for Research on Cancer 150 cours Albert Thomas 69372 Lyon cedex 08 FranceSalvatore Vaccarella, International Agency for Research on Cancer 150 cours Albert Thomas 69372 Lyon cedex 08 FranceSadhina Shrestha, B.P. Koirala Memor...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3031672</comments>
            <pubDate>Mon, 23 Nov 2009 17:09:32 +0100</pubDate>
            <guid isPermaLink="false">3031672</guid>        </item>
        <item>
            <title>Germline polymorphisms in the one-carbon metabolism pathway and DNA methylation in colorectal cancer</title>
            <link>http://www.medworm.com/index.php?rid=3019976&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fl7q0p078mh810160%2F</link>
            <description>In this study, we evaluated the relationship between
 single nucleotide polymorphisms (SNPs) in the one-carbon metabolism pathway and DNA methylation status in colorectal cancer.
 Utilizing 182 colorectal cancers cases in two prospective cohort studies, we determined the CpG island methylator phenotype
 (CIMP) status on eight CIMP-specific promoters and measured LINE-1 methylation level that correlates well with genome-wide
 DNA methylation level. We genotyped 23 nonsynonymous SNPs in the one-carbon metabolism genes using buffy coat DNA. Most of
 the 23 SNPs in the one-carbon metabolism pathway were not significantly associated with CIMP-high status (≥6/8 methylated
 promoters). However, the MTHFR 429 Ala/Ala variant (rs1801131) and the TCN2 259 Arg/Arg variant (rs1801198) were associate...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3019976</comments>
            <pubDate>Sat, 21 Nov 2009 08:34:23 +0100</pubDate>
            <guid isPermaLink="false">3019976</guid>        </item>
        <item>
            <title>A comprehensive analysis of common genetic variation in MUC1, MUC5AC, MUC6 genes and risk of stomach cancer</title>
            <link>http://www.medworm.com/index.php?rid=3016043&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fk235q558rh36572t%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Overall, our data provide evidence that some common variations in MUC1 and MUC5AC genes contribute to an elevated risk of stomach cancer. Further studies are needed to confirm these novel findings.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9463-3Authors
		Yanbin Jia, Karolinska Institutet Department of Medical Epidemiology and Biostatistics Box 281 17177 Stockholm SwedenChristina Persson, Karolinska Institutet Department of Medical Epidemiology and Biostatistics Box 281 17177 Stockholm SwedenLifang Hou, National Cancer Institute Division of Cancer Epidemiology and Genetics Bethesda MD USAZongli Zheng, Karolinska Institutet Department of Medical Epidemiology and Biostatistics Box 281 17177 Stockholm SwedenMeredith Yeager, Nationa...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3016043</comments>
            <pubDate>Wed, 18 Nov 2009 19:22:28 +0100</pubDate>
            <guid isPermaLink="false">3016043</guid>        </item>
        <item>
            <title>Fruits and vegetables consumption and the risk of histological subtypes of lung cancer in the European Prospective Investigation into Cancer and Nutrition (EPIC)</title>
            <link>http://www.medworm.com/index.php?rid=3016044&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fb12l3032206n5021%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;We observed inverse associations between the consumption of vegetables and fruits and risk of lung cancer without a clear
 effect on specific histological subtypes of lung cancer. In current smokers, consumption of vegetables and fruits may reduce
 lung cancer risk, in particular the risk of squamous cell carcinomas.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9468-yAuthors
		F. L. Büchner, National Institute of Public Health and the Environment (RIVM) P.O. Box 1 3720 BA Bilthoven The NetherlandsH. B. Bueno-de-Mesquita, National Institute of Public Health and the Environment (RIVM) P.O. Box 1 3720 BA Bilthoven The NetherlandsJ. Linseisen, Deutsches Krebsforschungszentrum Division of Clinical Epidemiology Heidelberg GermanyH. C. Bo...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3016044</comments>
            <pubDate>Wed, 18 Nov 2009 19:22:27 +0100</pubDate>
            <guid isPermaLink="false">3016044</guid>        </item>
        <item>
            <title>Inverse association between serum 25(OH) vitamin D levels and non-melanoma skin cancer in elderly men</title>
            <link>http://www.medworm.com/index.php?rid=3009593&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fg31246612103q047%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;To determine the relationship between 25(OH) vitamin D levels and non-melanoma skin cancer (NMSC), we performed a nested case–control
 study in ambulatory, elderly men enrolled in the Osteoporotic Fractures in Men (MrOS) Study. Health habit and medical history,
 including self-reported history of NMSC were recorded and 25(OH)D levels were measured on serum collected at baseline from
 a random sample of Caucasian MrOS subjects. Mean age (73&amp;nbsp;±&amp;nbsp;5), BMI, daily vitamin D and calcium intake were similar in the
 men with (n&amp;nbsp;=&amp;nbsp;178) and without NMSC (n&amp;nbsp;=&amp;nbsp;930), but higher levels of 25(OH)D were associated with a decreased risk of having a history of NMSC (P
 
 trend
 &amp;nbsp;=&amp;nbsp;0.04). Men in the highest quintile of 25(OH)D (&amp;gt;30&amp;nbsp;ng/mL) ha...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3009593</comments>
            <pubDate>Wed, 18 Nov 2009 04:43:50 +0100</pubDate>
            <guid isPermaLink="false">3009593</guid>        </item>
        <item>
            <title>Prospective studies of dietary alpha-linolenic acid intake and prostate cancer risk: a meta-analysis</title>
            <link>http://www.medworm.com/index.php?rid=3009594&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F8473470111631443%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Individual-based studies that investigated the relation between dietary alpha-linolenic acid (ALA) intake and prostate cancer
 risk have shown inconsistent results. We carried out a meta-analysis of prospective studies to examine this association. We
 systematically searched studies published up to December 2008. Log relative risks (RRs) were weighted by the inverse of their
 variances to obtain a pooled estimate with its 95% confidence interval (CI). We identified five prospective studies that met
 our inclusion criteria and reported risk estimates by categories of ALA intake. Comparing the highest to the lowest ALA intake
 category, the pooled RR was 0.97 (95% CI:0.86–1.10) but the association was heterogeneous. Using the reported numbers of cases
 and non-cases in ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3009594</comments>
            <pubDate>Wed, 18 Nov 2009 04:43:47 +0100</pubDate>
            <guid isPermaLink="false">3009594</guid>        </item>
        <item>
            <title>Breastfeeding reduces breast cancer risk: a case–control study in Tunisia</title>
            <link>http://www.medworm.com/index.php?rid=3009595&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F0l45254882045v33%2F</link>
            <description>In this report, we examined the relationship between mother’s breastfeeding history and her risk of breast cancer, in a case–control
 study in Tunisia between 2006 and 2009. About 400 breast cancer cases and 400 controls were included. Cases and controls were
 interviewed using a standardized structured questionnaire to obtain information on breastfeeding and other risk factors. Mean
 duration of breastfeeding per child was significantly associated with a reduced risk of breast cancer for women who breastfed
 for &amp;gt;24&amp;nbsp;months per child. The OR was 0.46 (95% CI, 0.28–0.76) when compared those who breastfed for &amp;lt;6&amp;nbsp;months. The test for
 trend was significant (p&amp;nbsp;=&amp;nbsp;0.01). A significantly reduced risk of breast cancer was found for those whose lifetime duration of b...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3009595</comments>
            <pubDate>Wed, 18 Nov 2009 04:43:46 +0100</pubDate>
            <guid isPermaLink="false">3009595</guid>        </item>
        <item>
            <title>Vitamin D association with estradiol and progesterone in young women</title>
            <link>http://www.medworm.com/index.php?rid=2996723&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fu873351275gj78nx%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Higher levels of vitamin D may reduce progesterone and estradiol, providing a potential mechanism for reduction in breast
 cancer risk from increased vitamin D exposure in young women.
 
 
 
	Content Type Journal ArticleCategory Brief reportDOI 10.1007/s10552-009-9466-0Authors
		Julia A. Knight, Mount Sinai Hospital Prosserman Centre for Health Research, Samuel Lunenfeld Research Institute 60 Murray Street, Room 5-237 Box 18 Toronto ON M5T 3L9 CanadaJody Wong, Mount Sinai Hospital Prosserman Centre for Health Research, Samuel Lunenfeld Research Institute 60 Murray Street, Room 5-237 Box 18 Toronto ON M5T 3L9 CanadaKristina M. Blackmore, Mount Sinai Hospital Prosserman Centre for Health Research, Samuel Lunenfeld Research Institute 60 Murray Street, Room 5-237 Box 18 ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2996723</comments>
            <pubDate>Sat, 14 Nov 2009 20:51:56 +0100</pubDate>
            <guid isPermaLink="false">2996723</guid>        </item>
        <item>
            <title>Active, but not passive cigarette smoking was inversely associated with mammographic density</title>
            <link>http://www.medworm.com/index.php?rid=2996724&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F90t75x15vk61q8m1%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Our findings support the hypothesis that tobacco smoke exerts an antiestrogenic effect on breast tissue, but counters the
 known increased risk of breast cancer with smoking prior to first full-term birth. Thus, our data suggest that the antiestrogenic
 but not the carcinogenic effects of smoking may be reflected by breast density.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9462-4Authors
		Lesley M. Butler, Colorado State University Department of Environmental and Radiological Health Sciences 1681 Campus Delivery Fort Collins CO 80523-1681 USAEllen B. Gold, University of California Department of Public Health Sciences Davis CA USAShannon M. Conroy, University of California Department of Public Health Sciences Davis CA USACarolyn ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2996724</comments>
            <pubDate>Fri, 13 Nov 2009 18:16:18 +0100</pubDate>
            <guid isPermaLink="false">2996724</guid>        </item>
        <item>
            <title>Genetic variation in DNA repair genes and prostate cancer risk: results from a population-based study</title>
            <link>http://www.medworm.com/index.php?rid=2982433&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fv31640462873364h%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Overall, this study found no associations between prostate cancer and the SNPs in DNA repair genes. Given the complexity of
 this pathway and its crucial role in maintenance of genomic stability, a pathway-based analysis of all 150 genes in DNA repair
 pathways, as well as exploration of gene–environment interactions may be warranted.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9461-5Authors
		Ilir Agalliu, Albert Einstein College of Medicine Department of Epidemiology and Population Health Bronx NY USAErika M. Kwon, National Institutes of Health Cancer Genetics Branch, National Human Genome Research Institute Bethesda MD USAClaudia A. Salinas, Fred Hutchinson Cancer Research Center Division of Public Health Sciences 1100 Fairvie...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2982433</comments>
            <pubDate>Mon, 09 Nov 2009 19:01:19 +0100</pubDate>
            <guid isPermaLink="false">2982433</guid>        </item>
        <item>
            <title>Breast-feeding the last born child and risk of ovarian cancer</title>
            <link>http://www.medworm.com/index.php?rid=2982434&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F48038h1457384252%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Conflicting reports regarding the relationship between breast-feeding and ovarian cancer risk suggest a possible influence
 of patterns of breast-feeding. We used logistic regression to examine breast-feeding in a large population of parous women
 who participated in a case–control study of ovarian cancer in New Hampshire and MA, USA. Risk of ovarian cancer was reduced
 in parous women who ever breast-fed (OR: 0.75; 95% CI: 0.62, 0.92), but evidence was limited for an influence of duration
 of breast-feeding and the number of children breast-fed. Compared to never breast-feeding, inverse associations were seen
 for breast-feeding all children (OR: 0.72; 95% CI: 0.58, 0.91) and for breast-feeding some children when the last born child
 was breast-fed (OR: 0.58; 95% CI:...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2982434</comments>
            <pubDate>Mon, 09 Nov 2009 19:01:18 +0100</pubDate>
            <guid isPermaLink="false">2982434</guid>        </item>
        <item>
            <title>Estimates of overdiagnosis of invasive breast cancer associated with screening mammography</title>
            <link>http://www.medworm.com/index.php?rid=2971467&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F89735jnxl44w2236%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Overdiagnosis of invasive breast cancer attributable to mammography screening appears to be substantial. Our estimates are
 similar to recent estimates from other screening programmes. Overdiagnosis merits greater attention in research and in clinical
 and public health policy making.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9459-zAuthors
		Stephen Morrell, University of Sydney School of Public Health Sydney NSW 2006 AustraliaAlexandra Barratt, University of Sydney School of Public Health Sydney NSW 2006 AustraliaLes Irwig, University of Sydney School of Public Health Sydney NSW 2006 AustraliaKirsten Howard, University of Sydney School of Public Health Sydney NSW 2006 AustraliaCorné Biesheuvel, University of Sydney Screening an...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2971467</comments>
            <pubDate>Thu, 05 Nov 2009 18:40:52 +0100</pubDate>
            <guid isPermaLink="false">2971467</guid>        </item>
        <item>
            <title>Depression and cancer risk: 24 years of follow-up of the Baltimore Epidemiologic Catchment Area sample</title>
            <link>http://www.medworm.com/index.php?rid=3097715&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fa538366905124381%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Results reveal a specificity to the association between depression and hormonally mediated cancers, which provides support
 to hypotheses about a common biological pathway between depression and cancer. Further research can build on observational
 studies to examine the mechanisms through which our emotions affect our health.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9449-1Authors
		Alden L. Gross, Johns Hopkins Bloomberg School of Public Health Department of Mental Health 798 Hampton House, 624 N. Broadway Baltimore MD 21205 USAJoseph J. Gallo, Johns Hopkins Bloomberg School of Public Health Department of Mental Health 624 N. Broadway Baltimore MD 21205 USAWilliam W. Eaton, Johns Hopkins Bloomberg School of Public Health Depart...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3097715</comments>
            <pubDate>Tue, 03 Nov 2009 11:01:42 +0100</pubDate>
            <guid isPermaLink="false">3097715</guid>        </item>
        <item>
            <title>Depression and cancer risk: 24 years of follow-up of the Baltimore Epidemiologic Catchment Area sample</title>
            <link>http://www.medworm.com/index.php?rid=2960346&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fa538366905124381%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Results reveal a specificity to the association between depression and hormonally mediated cancers, which provides support
 to hypotheses about a common biological pathway between depression and cancer. Further research can build on observational
 studies to examine the mechanisms through which our emotions affect our health.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9449-1Authors
		Alden L. Gross, Johns Hopkins Bloomberg School of Public Health Department of Mental Health 798 Hampton House, 624 N. Broadway Baltimore MD 21205 USAJoseph J. Gallo, Johns Hopkins Bloomberg School of Public Health Department of Mental Health 624 N. Broadway Baltimore MD 21205 USAWilliam W. Eaton, Johns Hopkins Bloomberg School of Public Health Depart...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2960346</comments>
            <pubDate>Tue, 03 Nov 2009 11:01:42 +0100</pubDate>
            <guid isPermaLink="false">2960346</guid>        </item>
        <item>
            <title>Objectively measured physical activity and sedentary time of breast cancer survivors, and associations with adiposity: findings from NHANES (2003–2006)</title>
            <link>http://www.medworm.com/index.php?rid=2960347&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fe7657ru074tvnx8t%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;This is the first study to describe the objectively assessed physical activity and sedentary time of breast cancer survivors.
 Increasing moderate-to-vigorous and light intensity physical activity, and decreasing sedentary time, may assist with weight
 management and improve other metabolic health outcomes for breast cancer survivors.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9460-6Authors
		Brigid M. Lynch, The University of Queensland Cancer Prevention Research Centre, School of Population Health Herston Road Brisbane QLD 4006 AustraliaDavid W. Dunstan, The University of Queensland Cancer Prevention Research Centre, School of Population Health Herston Road Brisbane QLD 4006 AustraliaGenevieve N. Healy, The University of Queens...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2960347</comments>
            <pubDate>Mon, 02 Nov 2009 18:37:00 +0100</pubDate>
            <guid isPermaLink="false">2960347</guid>        </item>
        <item>
            <title>Increasing trend of the incidence of esophageal squamous cell carcinoma, but not adenocarcinoma, in Taiwan</title>
            <link>http://www.medworm.com/index.php?rid=2944903&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F68110n0v3n714336%2F</link>
            <description>We examined temporal trends in the incidence of esophageal
 squamous cell carcinoma (ESCC) and esophageal adenocarcinoma (EA) in Taiwan. Patients with esophageal cancer were identified
 from the Taiwan Cancer Registry between 1979 and 2003. Age-standardized incidences of ESCC and EA were calculated based on
 the national census and world standard population. Trends in incidence rates were estimated by calculating the annual percentage
 change (APC). The age-standardized incidence of ESCC increased progressively (from 1979–1983 to 1999–2003: 2.63 to 4.37 per
 100,000-year), with an APC higher in male (3.27%, P&amp;nbsp;&amp;lt;&amp;nbsp;0.0001) than that in female (1.23%, P&amp;nbsp;=&amp;nbsp;0.03). Though the crude incidence of EA progressively increased in both gender (male: 0.28–0.49 per 100,000-year...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2944903</comments>
            <pubDate>Thu, 29 Oct 2009 07:14:47 +0100</pubDate>
            <guid isPermaLink="false">2944903</guid>        </item>
        <item>
            <title>Association of maternal and intrauterine characteristics with age at menarche in a multiethnic population in Hawaii</title>
            <link>http://www.medworm.com/index.php?rid=2940783&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fn418177366141271%2F</link>
            <description>This study seeks to further elucidate the mother–daughter hormonal relationship and its effects on daughter’s breast cancer
 risk through the association with early age at menarche. Four hundred and thirty-eight healthy girls, age 9–18 and of White,
 Asian, and/or Polynesian race/ethnicity, were recruited from an HMO on Oahu, Hawaii. Anthropometric measures were taken at
 a clinic visit, and family background questionnaires were completed. Cox proportional hazards regression was used to test
 the association of maternal and intrauterine hormone-related exposures with age at menarche. Weight and gestational age at
 birth and maternal pregnancy-induced nausea were not associated with age at menarche. Each year older of the mother’s age
 at menarche was associated with a 21% reduced r...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2940783</comments>
            <pubDate>Tue, 27 Oct 2009 18:24:48 +0100</pubDate>
            <guid isPermaLink="false">2940783</guid>        </item>
        <item>
            <title>Serum calcium levels are elevated among women with untreated postmenopausal breast cancer</title>
            <link>http://www.medworm.com/index.php?rid=2928351&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F0v8302641u165872%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;The distribution of serum calcium levels among postmenopausal women with incident breast cancer was shifted significantly
 toward the right. These findings are consistent with an effect of early breast tumors on calcium homeostasis. However, the
 lack of association between serum calcium levels and tumor size or stage supports the hypothesis that subclinical hyperparathyroidism
 may increase the risk for breast cancer.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9456-2Authors
		Erica Martin, University of North Dakota School of Medicine and Health Sciences Grand Forks ND USAMegan Miller, University of North Dakota School of Medicine and Health Sciences Grand Forks ND USALacey Krebsbach, University of North Dakota School of Medicine...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2928351</comments>
            <pubDate>Fri, 23 Oct 2009 19:01:09 +0100</pubDate>
            <guid isPermaLink="false">2928351</guid>        </item>
        <item>
            <title>Citrus fruit and cancer risk in a network of case–control studies</title>
            <link>http://www.medworm.com/index.php?rid=2928352&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fp1115171433034r7%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Our findings indicate that citrus fruit has a protective role against cancers of the digestive and upper respiratory tract.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9454-4Authors
		Roberto Foschi, Istituto di Ricerche Farmacologiche “Mario Negri” Via Giuseppe La Masa 19 20156 Milan ItalyClaudio Pelucchi, Istituto di Ricerche Farmacologiche “Mario Negri” Via Giuseppe La Masa 19 20156 Milan ItalyLuigino Dal Maso, Centro di Riferimento Oncologico S.O.C. di Epidemiologia e Biostatistica Aviano PN ItalyMarta Rossi, Istituto di Ricerche Farmacologiche “Mario Negri” Via Giuseppe La Masa 19 20156 Milan ItalyFabio Levi, Centre Hospitalier Universitaire Vaudois et Université de Lausanne Unité d’Épidémiologie du Cancer e...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2928352</comments>
            <pubDate>Fri, 23 Oct 2009 19:01:07 +0100</pubDate>
            <guid isPermaLink="false">2928352</guid>        </item>
        <item>
            <title>Intentional weight loss and risk of lymphohematopoietic cancers</title>
            <link>http://www.medworm.com/index.php?rid=2924202&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F4342x1h4256m1533%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Further assessment of intentional weight loss as a possible risk factor for lymphomas may provide insight into the etiology
 of these cancers.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9453-5Authors
		Anneclaire J. De Roos, Fred Hutchinson Cancer Research Center Epidemiology Program, Division of Public Health Sciences 1100 Fairview Avenue N, M4-B874 Seattle WA 98109-1024 USACornelia M. Ulrich, Fred Hutchinson Cancer Research Center Cancer Prevention Program, Division of Public Health Sciences 1100 Fairview Avenue N, M4-B874 Seattle WA 98109-1024 USARoberta M. Ray, Fred Hutchinson Cancer Research Center Cancer Prevention Program, Division of Public Health Sciences 1100 Fairview Avenue N, M4-B874 Seattle WA 98109-1024 USAYasmin Mos...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2924202</comments>
            <pubDate>Thu, 22 Oct 2009 19:00:09 +0100</pubDate>
            <guid isPermaLink="false">2924202</guid>        </item>
        <item>
            <title>Determinants of inequalities in cervical cancer stage at diagnosis and survival in New Zealand</title>
            <link>http://www.medworm.com/index.php?rid=2921361&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fr7t464557163j61g%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;These results indicate that later stage at diagnosis is the main determinant of Māori women’s higher mortality from cervical
 cancer. Improving cervical screening among Māori women would reduce stage at diagnosis and therefore ethnic inequalities in
 mortality.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9451-7Authors
		Patricia Priest, University of Otago Department of Preventive and Social Medicine, Dunedin School of Medicine P.O. Box 913 Dunedin 9054 New ZealandLynn Sadler, National Women’s Health, Auckland District Health Board Auckland New ZealandPeter Sykes, University of Otago Department of Obstetrics and Gynaecology Christchurch New ZealandRoger Marshall, University of Auckland Section of Epidemiology and Biostatisti...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2921361</comments>
            <pubDate>Wed, 21 Oct 2009 22:53:27 +0100</pubDate>
            <guid isPermaLink="false">2921361</guid>        </item>
        <item>
            <title>Mortality from malignant diseases—update of the Baltimore union poultry cohort</title>
            <link>http://www.medworm.com/index.php?rid=2921362&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F9555p338r8643127%2F</link>
            <description>In this report, cancer mortality was updated to the year 2003 for 2,580 of the 2,639 subjects who worked exclusively in poultry plants. Mortality in poultry workers was compared with that in the US general population through the estimation
 of proportional mortality and standardized mortality ratios separately for each race/sex group and for the whole cohort. Compared
 to the US general population, an excess of cancers of the buccal and nasal cavities and pharynx (base of the tongue, palate
 and other unspecified mouth, tonsil and oropharynx, nasal cavity/middle ear/accessory sinus), esophagus, recto-sigmoid/rectum/anus,
 liver and intrabiliary system, myelofibrosis, lymphoid leukemia and multiple myeloma was observed in particular subgroups
 or in the entire poultry cohort. We hypothesize...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2921362</comments>
            <pubDate>Wed, 21 Oct 2009 22:53:24 +0100</pubDate>
            <guid isPermaLink="false">2921362</guid>        </item>
        <item>
            <title>trans-Fatty acid consumption and its association with distal colorectal cancer in the North Carolina Colon Cancer Study II</title>
            <link>http://www.medworm.com/index.php?rid=2917497&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F314r13k587371043%2F</link>
            <description>In conclusion, high consumption of trans-fatty acids was positively associated with distal colorectal cancer among Whites.
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9447-3Authors
		Lisa C. Vinikoor, University of North Carolina at Chapel Hill Department of Epidemiology, School of Public Health Chapel Hill NC USARobert C. Millikan, University of North Carolina at Chapel Hill Department of Epidemiology, School of Public Health Chapel Hill NC USAJessie A. Satia, University of North Carolina at Chapel Hill Department of Epidemiology, School of Public Health Chapel Hill NC USAJane C. Schroeder, University of North Carolina at Chapel Hill Department of Epidemiology, School of Public Health Chapel Hill NC USAChristopher F. Martin, University of North Carolina at...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2917497</comments>
            <pubDate>Wed, 21 Oct 2009 03:26:07 +0100</pubDate>
            <guid isPermaLink="false">2917497</guid>        </item>
        <item>
            <title>Family history of cancer and malignant germ cell tumors in children: A report from the Children’s Oncology Group</title>
            <link>http://www.medworm.com/index.php?rid=2917498&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Ftu4176pn32285hx8%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Family history of testicular cancer is an established risk factor for adult testicular germ cell tumors (GCT). We evaluated
 the association between family history of cancer and pediatric GCT in a Children’s Oncology Group case–control study that
 included 274 GCT cases (195 female and 79 male) diagnosed &amp;lt;age 15&amp;nbsp;years and 418 controls frequency matched to cases on sex
 and age. Family history data were collected through telephone interviews with biological mothers and fathers and unconditional
 logistic regression was used to evaluate associations with GCT adjusting for potential confounders. A family history of cancer
 with onset &amp;lt;age 40&amp;nbsp;years was associated with a reduced risk of GCT among female cases (Odds Ratio (OR)&amp;nbsp;=&amp;nbsp;0.50, 95% Confide...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2917498</comments>
            <pubDate>Wed, 21 Oct 2009 03:25:58 +0100</pubDate>
            <guid isPermaLink="false">2917498</guid>        </item>
        <item>
            <title>Plasma enterolactone and risk of colon and rectal cancer in a case–cohort study of Danish men and women</title>
            <link>http://www.medworm.com/index.php?rid=2917499&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fp0w81650345m8432%2F</link>
            <description>In conclusion, with higher enterolactone levels, we found lower risk of colon cancer among women and
 higher risk of rectal cancer among men.
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9445-5Authors
		Nina Føns Johnsen, Danish Cancer Society Institute of Cancer Epidemiology Strandboulevarden 49 2100 Copenhagen Ø DenmarkAnja Olsen, Danish Cancer Society Institute of Cancer Epidemiology Strandboulevarden 49 2100 Copenhagen Ø DenmarkBirthe Lykke Riegels Thomsen, Danish Cancer Society Institute of Cancer Epidemiology Strandboulevarden 49 2100 Copenhagen Ø DenmarkJane Christensen, Danish Cancer Society Institute of Cancer Epidemiology Strandboulevarden 49 2100 Copenhagen Ø DenmarkRikke Egeberg, Danish Cancer Society Institute of Cancer Epidemiology Strandb...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2917499</comments>
            <pubDate>Wed, 21 Oct 2009 02:26:41 +0100</pubDate>
            <guid isPermaLink="false">2917499</guid>        </item>
        <item>
            <title>Screening mammography intervals among postmenopausal hormone therapy users and nonusers</title>
            <link>http://www.medworm.com/index.php?rid=2917500&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fn1208p268lw11805%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Our findings indicate that screening mammography intervals are similar whether or not women are using HT. Thus, reduced utilization
 of screening mammography is unlikely to account for the decrease in breast cancer incidence seen soon after the WHI report.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9444-6Authors
		Tracy Onega, Dartmouth Medical School Department of Community and Family Medicine Hanover NH USATodd MacKenzie, Dartmouth Hitchcock Medical Center Norris Cotton Cancer Center Lebanon NH USAJulia Weiss, Dartmouth Medical School Department of Community and Family Medicine Hanover NH USAMartha Goodrich, Dartmouth Medical School Department of Community and Family Medicine Hanover NH USALinda Titus-Ernstoff, Dartmouth Medica...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2917500</comments>
            <pubDate>Wed, 21 Oct 2009 02:26:35 +0100</pubDate>
            <guid isPermaLink="false">2917500</guid>        </item>
        <item>
            <title>Atrophic gastritis and the risk of incident colorectal cancer</title>
            <link>http://www.medworm.com/index.php?rid=2905930&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F5352g5413207n006%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Atrophic gastritis is not associated with an increased risk of colorectal cancer among male smokers.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9446-4Authors
		Adeyinka O. Laiyemo, National Cancer Institute (NCI), National Institutes of Health (NIH) Cancer Prevention Fellowship Program, Office of Preventive Oncology Bethesda MD USAFarin Kamangar, National Cancer Institute (NCI), National Institutes of Health (NIH) Nutritional Epidemiology Branch, Division of Cancer Epidemiology and Genetics Bethesda MD USAPamela M. Marcus, National Cancer Institute (NCI), National Institutes of Health (NIH) Biometry Research Group, Division of Cancer Prevention Bethesda MD USAPhilip R. Taylor, National Cancer Institute (NCI), National Institutes o...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2905930</comments>
            <pubDate>Fri, 16 Oct 2009 18:07:11 +0100</pubDate>
            <guid isPermaLink="false">2905930</guid>        </item>
        <item>
            <title>Interaction between organochlorines and the AHR gene, and risk of non-Hodgkin lymphoma</title>
            <link>http://www.medworm.com/index.php?rid=2883858&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fj3801521m68547t1%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Results suggest that the AHR gene may play a role in determining the risk of NHL with exposure to organochlorines, and highlight
 the importance of understanding gene-environment interactions.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9429-5Authors
		Carmen H. Ng, BC Cancer Agency 675 W. 10th Avenue Vancouver BC V5Z 1L3 CanadaRozmin Janoo-Gilani, BC Cancer Agency 675 W. 10th Avenue Vancouver BC V5Z 1L3 CanadaPayal Sipahimalani, BC Cancer Agency 675 W. 10th Avenue Vancouver BC V5Z 1L3 CanadaRichard P. Gallagher, BC Cancer Agency 675 W. 10th Avenue Vancouver BC V5Z 1L3 CanadaRandy D. Gascoyne, BC Cancer Agency 675 W. 10th Avenue Vancouver BC V5Z 1L3 CanadaJoseph M. Connors, BC Cancer Agency 675 W. 10th Avenue Vancouver BC V5Z...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2883858</comments>
            <pubDate>Sat, 10 Oct 2009 07:24:59 +0100</pubDate>
            <guid isPermaLink="false">2883858</guid>        </item>
        <item>
            <title>Reproductive factors, exogenous female hormone use and breast cancer risk in Japanese: the Miyagi Cohort Study</title>
            <link>http://www.medworm.com/index.php?rid=2880505&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fy521875027564740%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;The incidence of breast cancer among Japanese women is substantially increasing. This population-based prospective cohort
 study in Japan evaluated the associations of reproductive factors and exogenous female hormone use with breast cancer risk,
 both overall and separately among premenopausal and postmenopausal women. A total of 24,064 women aged 40–64 were followed
 from 1990 to 2003. During 309,424 person-years of follow-up, 285 breast cancer cases were documented. In overall evaluation,
 nulliparity was significantly associated with an increased risk of breast cancer. There was a significant decrease in risk
 with increasing parity number among parous women (trend P&amp;nbsp;=&amp;nbsp;0.008). No association was observed between age at menarche or age at first birth and ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2880505</comments>
            <pubDate>Fri, 09 Oct 2009 06:23:32 +0100</pubDate>
            <guid isPermaLink="false">2880505</guid>        </item>
        <item>
            <title>Medical history, body size, and cigarette smoking in relation to fatal prostate cancer</title>
            <link>http://www.medworm.com/index.php?rid=2880504&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F0286r24780184gju%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;The observed increase in risk associated with recent cigarette smoking is consistent with the findings of several other studies.
 However, in contrast with some reports, we observed no connection between fatal prostate cancer and some prior health conditions
 or measures of body size.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9441-9Authors
		Sheila Weinmann, Kaiser Permanente Northwest Center for Health Research 3800 N Interstate Avenue Portland OR USAJean A. Shapiro, Centers for Disease Control and Prevention Division of Cancer Prevention and Control Atlanta GA USABenjamin A. Rybicki, Henry Ford Health System Josephine Ford Cancer Center Detroit MI USAShelley M. Enger, Kaiser Permanente, Southern California Region Department of...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2880504</comments>
            <pubDate>Fri, 09 Oct 2009 06:23:32 +0100</pubDate>
            <guid isPermaLink="false">2880504</guid>        </item>
        <item>
            <title>Genetic variation in N-acetyltransferases 1 and 2, cigarette smoking, and risk of non-Hodgkin lymphoma</title>
            <link>http://www.medworm.com/index.php?rid=2873324&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fx6w411h642110374%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Cigarette smoke contains many carcinogens that are metabolically activated through xenobiotic metabolism by phase I and II
 enzymes, including N-acetyltransferases 1 and 2 (NAT1 and NAT2). We investigated non-Hodgkin lymphoma risk in general and by subtype in relation to NAT1 and NAT2 genotypes and cigarette smoking in a population-based case–control study in Connecticut. Of the 535 controls, 53.1% reported
 ever smoking, and of the 461 cases, 55.7% reported ever smoking. We found a two-fold increased risk of T-cell lymphoma among
 those possessing the NAT1*10 genotype compared to those with other NAT1 genotypes; including an OR of 2.0 (95% CI: 1.0–2.4) for those heterozygous or homozygous for NAT1*10 genotypes. Rapid acetylator NAT2 phenotype increased the risk of ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2873324</comments>
            <pubDate>Wed, 07 Oct 2009 07:03:52 +0100</pubDate>
            <guid isPermaLink="false">2873324</guid>        </item>
        <item>
            <title>Association between plasma total cholesterol concentration and incident prostate cancer in the CLUE II cohort</title>
            <link>http://www.medworm.com/index.php?rid=2870111&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fx03w4668k8400gj6%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Statin drugs appear to protect against advanced and possibly high-grade prostate cancer, perhaps through cholesterol-lowering.
 Thus, we evaluated the association between plasma cholesterol and prostate cancer. We conducted a prospective study in the
 CLUE II cohort of Washington County, MD. Included were 6,816 male county residents aged 35+&amp;nbsp;years old who did not have a cancer
 diagnosis at baseline in 1989. Plasma cholesterol, measured enzymatically at baseline, was categorized by clinical cutpoints.
 Cox proportional hazards regression was used to estimate multivariable-adjusted hazard ratios (HR) and 95% confidence intervals
 (CI) for total (n&amp;nbsp;=&amp;nbsp;438) and high-grade (Gleason sum ≥7, n&amp;nbsp;=&amp;nbsp;137) prostate cancer. Compared to men with high cholest...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2870111</comments>
            <pubDate>Tue, 06 Oct 2009 12:48:40 +0100</pubDate>
            <guid isPermaLink="false">2870111</guid>        </item>
        <item>
            <title>The impact of changes in hormone therapy on breast cancer incidence in the US population</title>
            <link>http://www.medworm.com/index.php?rid=2853183&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F310k3322n82h0808%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Changes in reported breast cancer incidence may be partially or largely explained by changes in HT use in the US population.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9437-5Authors
		Nathan J. Coombs, Great Western Hospital Swindon UKKathleen A. Cronin, National Cancer Institute Bethesda MD USARichard J. Taylor, University of Queensland School of Population Health Brisbane AustraliaAndrew N. Freedman, National Cancer Institute Bethesda MD USAJohn Boyages, NSW Breast Cancer Institute Westmead (Sydney) Australia
	

	
		Journal Cancer Causes and ControlOnline ISSN 1573-7225Print ISSN 0957-5243 (Source: Cancer Causes and Control)</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2853183</comments>
            <pubDate>Wed, 30 Sep 2009 18:28:53 +0100</pubDate>
            <guid isPermaLink="false">2853183</guid>        </item>
        <item>
            <title>Beliefs about the causes of breast and colorectal cancer among women in the general population</title>
            <link>http://www.medworm.com/index.php?rid=2847979&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fq46837627n664077%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Both genetic and environmental causes for breast and colorectal cancer are endorsed by unaffected women. Misconceptions about
 the causes of these cancers are important targets for public education and risk communication efforts.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9439-3Authors
		Catharine Wang, Boston University School of Public Health Department of Community Health Sciences 801 Massachusetts Avenue, 4th Floor Boston MA 02118 USASuzanne M. Miller, Fox Chase Cancer Center Philadelphia PA USABrian L. Egleston, Fox Chase Cancer Center Philadelphia PA USAJennifer L. Hay, Memorial Sloan Kettering Cancer Center New York NY USADavid S. Weinberg, Fox Chase Cancer Center Philadelphia PA USA
	

	
		Journal Cancer Causes and Contro...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2847979</comments>
            <pubDate>Tue, 29 Sep 2009 04:07:34 +0100</pubDate>
            <guid isPermaLink="false">2847979</guid>        </item>
        <item>
            <title>Leukocyte telomere length in a population-based case–control study of ovarian cancer: a pilot study</title>
            <link>http://www.medworm.com/index.php?rid=2847980&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F6111x2j61064v681%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;This study shows that short leukocyte telomeres are associated with serous ovarian adenocarcinoma. These findings should be
 confirmed in large, prospective studies.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9436-6Authors
		Lisa Mirabello, National Cancer Institute, National Institutes of Health, Department of Health and Human Services Division of Cancer Epidemiology and Genetics Bethesda MD 20892 USAMontserrat Garcia-Closas, National Cancer Institute, National Institutes of Health, Department of Health and Human Services Division of Cancer Epidemiology and Genetics Bethesda MD 20892 USARichard Cawthon, University of Utah Department of Human Genetics Salt Lake City UT 84132 USAJolanta Lissowska, The M. Sklodowska-Curie Memorial ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2847980</comments>
            <pubDate>Tue, 29 Sep 2009 04:01:02 +0100</pubDate>
            <guid isPermaLink="false">2847980</guid>        </item>
        <item>
            <title>Frequently cited sources in cancer news coverage: a content analysis examining the relationship between cancer news content and source citation</title>
            <link>http://www.medworm.com/index.php?rid=2847981&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fg10151gp14565xh0%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;The media are a frequent and sometimes sole source of cancer information for many people. News coverage of cancer can be influential
 to cancer-related practices such as prevention or detection behaviors, and sources cited by journalists may be influential
 in shaping this coverage. A content analysis (n&amp;nbsp;=&amp;nbsp;3,656 stories) revealed that the most frequently cited sources in cancer news articles—research institutions and medical
 journals—receive disproportionately more attention compared to the National Cancer Institute (NCI), the American Cancer Society
 (ACS), and pharmaceutical companies. Research institutions were cited twice as frequently as medical journals, and more than
 three times as frequently as pharmaceutical companies. Most clinical trial storie...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2847981</comments>
            <pubDate>Tue, 29 Sep 2009 01:11:28 +0100</pubDate>
            <guid isPermaLink="false">2847981</guid>        </item>
        <item>
            <title>Association of IGF1 and IGFBP3 polymorphisms with colorectal polyps and colorectal cancer risk</title>
            <link>http://www.medworm.com/index.php?rid=2847982&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F7278717154361156%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Our results suggest that the SNP rs6214 of IGF1 could have an impact on developing colorectal cancer and colorectal polyps
 with villous elements.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9438-4Authors
		Elisabeth Feik, Medical University of Vienna Department of Medicine I, Institute of Cancer Research Borschkegasse 8a 1090 Vienna AustriaAndreas Baierl, University of Vienna Department of Statistics and Decision Support Systems Vienna AustriaBarbara Hieger, Medical University of Vienna Department of Medicine I, Institute of Cancer Research Borschkegasse 8a 1090 Vienna AustriaGerhard Führlinger, Medical University of Vienna Department of Medicine I, Institute of Cancer Research Borschkegasse 8a 1090 Vienna AustriaAstrid Pentz, Me...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2847982</comments>
            <pubDate>Tue, 29 Sep 2009 01:11:27 +0100</pubDate>
            <guid isPermaLink="false">2847982</guid>        </item>
        <item>
            <title>Breastfeeding and risk of epithelial ovarian cancer</title>
            <link>http://www.medworm.com/index.php?rid=2839862&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fvp10751175151298%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;A long total duration of breastfeeding appears to be associated with a substantial reduction in the overall risk of ovarian
 cancer, independent of the decrease in risk due to childbirth, but this may vary according to histological subtype.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9440-xAuthors
		Susan J. Jordan, The University of Queensland School of Population Health Public Health Building, Herston Rd Herston, Brisbane 4006 AustraliaVictor Siskind, The Queensland Institute of Medical Research Genetics and Population Health Division Brisbane AustraliaAdèle C Green, The Queensland Institute of Medical Research Genetics and Population Health Division Brisbane AustraliaDavid C. Whiteman, The Queensland Institute of Medical Resea...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2839862</comments>
            <pubDate>Thu, 24 Sep 2009 16:48:11 +0100</pubDate>
            <guid isPermaLink="false">2839862</guid>        </item>
        <item>
            <title>Low cancer incidence rates in Ohio Amish</title>
            <link>http://www.medworm.com/index.php?rid=2835085&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F5ggkuh8338jl728g%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Cancer incidence is low in the Ohio Amish. These data strongly support reduction of cancer incidence by tobacco abstinence
 but cannot be explained solely on this basis. Understanding these contributions may help to identify additional important
 factors to target to reduce cancer among the non-Amish.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9435-7Authors
		Judith A. Westman, The Ohio State University Division of Human Genetics Columbus OH USAAmy K. Ferketich, The Ohio State University Division of Epidemiology 320 West 10th Avenue Columbus OH 43210 USARoss M. Kauffman, Indiana University School of Nursing Indianapolis IN USASteven N. MacEachern, The Ohio State University Department of Statistics Columbus OH USAJ. R. Wilkins, The...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2835085</comments>
            <pubDate>Thu, 24 Sep 2009 16:48:11 +0100</pubDate>
            <guid isPermaLink="false">2835085</guid>        </item>
        <item>
            <title>Race, healthcare access and physician trust among prostate cancer patients</title>
            <link>http://www.medworm.com/index.php?rid=2835087&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F533584587237123q%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Both race and access are important in explaining overall lower levels and greater variability in physician trust among black
 prostate cancer patients. Access barriers among black patients may spill over to the clinical encounter in the form of less
 physician trust, potentially contributing to racial disparities in treatment received and subsequent outcomes. Policy efforts
 to address the racial disparities in prostate cancer should prioritize improving healthcare access among minority groups.
 
 
 
	Content Type Journal ArticleCategory Original PaperDOI 10.1007/s10552-009-9431-yAuthors
		Young Kyung Do, University of North Carolina at Chapel Hill Gillings School of Global Public Health Department of Health Policy and Management 1102F McGavran-Greenberg Hall Chapel ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2835087</comments>
            <pubDate>Thu, 24 Sep 2009 14:36:47 +0100</pubDate>
            <guid isPermaLink="false">2835087</guid>        </item>
        <item>
            <title>The association between fruit and vegetable consumption and HPV viral load in high-risk HPV-positive women with cervical intraepithelial neoplasia</title>
            <link>http://www.medworm.com/index.php?rid=2835086&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F5084615h04400133%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;We evaluated the relationship between the dietary intake of vegetables and fruits, and the risk of cervical intraepithelial
 neoplasia (CIN) and determined whether these associations were modified by human papillomavirus (HPV) viral load. We enrolled
 1,096 women aged 18–65 to participate in a HPV cohort study from March 2006 up to present. For this analysis, we included
 328 HPV-positive women (166 controls, 90 CIN I and 72 CIN II/III). The multivariate odds ratios (ORs) and corresponding 95%
 confidence intervals (CIs) were estimated by multinomial logistic methods. After controlling for potential confounders, we
 found that a higher HPV viral load was associated with an increased risk of CIN I (OR&amp;nbsp;=&amp;nbsp;2.68, 95% CI, 1.19–6.04) and CIN II/III
 (OR&amp;nbsp;=&amp;nb...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2835086</comments>
            <pubDate>Thu, 24 Sep 2009 14:36:47 +0100</pubDate>
            <guid isPermaLink="false">2835086</guid>        </item>
        <item>
            <title>Metabolic syndrome components and colorectal adenoma in the CLUE II cohort</title>
            <link>http://www.medworm.com/index.php?rid=2833115&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fvt41t64081w6p226%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Our findings do not support that components of the metabolic syndrome influence risk of colorectal adenomas, except possibly
 for severe diabetes mellitus warranting medical treatment.
 
 
 
	Content Type Journal ArticleCategory Original PaperDOI 10.1007/s10552-009-9428-6Authors
		Konstantinos K. Tsilidis, Johns Hopkins Bloomberg School of Public Health Department of Epidemiology 615 N. Wolfe St., Rm E6132 Baltimore MD 21205 USAFrederick L. Brancati, Johns Hopkins Bloomberg School of Public Health Department of Epidemiology 615 N. Wolfe St., Rm E6132 Baltimore MD 21205 USAMichael N. Pollak, Jewish General Hospital and McGill University Departments of Medicine and Oncology Montreal QC CanadaNader Rifai, Children’s Hospital and Harvard Medical School Department of Lab...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2833115</comments>
            <pubDate>Wed, 23 Sep 2009 05:50:19 +0100</pubDate>
            <guid isPermaLink="false">2833115</guid>        </item>
        <item>
            <title>Promoter methylation and the detection of breast cancer</title>
            <link>http://www.medworm.com/index.php?rid=2810673&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fe1n5148435m55tm9%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Mammographic screening has been shown to reduce breast cancer mortality in women over the age of 50&amp;nbsp;years, and to a lesser
 extent in younger women. The sensitivity of mammography, however, is reduced in some groups of women. There remains a need
 for a minimally invasive, cost-effective procedure that could be used along side mammography to improve screening sensitivity.
 Silencing of tumor suppressor genes through promoter hypermethylation is known to be a frequent and early event in carcinogenesis.
 Further, changes in methylation patterns observed in tumors are also detectable in the circulation of women with breast cancer.
 This makes these alterations candidate markers for early tumor detection. In this paper, we review the current literature
 on promoter hyp...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2810673</comments>
            <pubDate>Fri, 18 Sep 2009 16:26:38 +0100</pubDate>
            <guid isPermaLink="false">2810673</guid>        </item>
        <item>
            <title>Green tea consumption and the risk of liver cancer in Japan: the Ohsaki Cohort study</title>
            <link>http://www.medworm.com/index.php?rid=2810674&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fw771l14p8k526wm1%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Green tea consumption is associated with a reduced risk of liver cancer incidence.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9388-xAuthors
		Akane Ui, Tohoku University Graduate School of Medicine Division of Epidemiology, Department of Public Health and Forensic Medicine 2-1, Seiryo-machi, Aoba-ku Sendai, Miyagi 980-8575 JapanShinichi Kuriyama, Tohoku University Graduate School of Medicine Division of Epidemiology, Department of Public Health and Forensic Medicine 2-1, Seiryo-machi, Aoba-ku Sendai, Miyagi 980-8575 JapanMasako Kakizaki, Tohoku University Graduate School of Medicine Division of Epidemiology, Department of Public Health and Forensic Medicine 2-1, Seiryo-machi, Aoba-ku Sendai, Miyagi 980-8575 JapanToshimasa Sone, T...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2810674</comments>
            <pubDate>Fri, 18 Sep 2009 16:26:37 +0100</pubDate>
            <guid isPermaLink="false">2810674</guid>        </item>
        <item>
            <title>Association of common polymorphisms in IL10, and in other genes related to inflammatory response and obesity with colorectal cancer</title>
            <link>http://www.medworm.com/index.php?rid=2810676&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F5q7711434m224355%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Our study suggests that polymorphisms in IL10, and also possibly in CRP and other genes related to immune response or obesity may be associated with colorectal cancer.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9427-7Authors
		Konstantinos K. Tsilidis, Johns Hopkins Bloomberg School of Public Health Department of Epidemiology 615 N. Wolfe St., Rm E6132 Baltimore MD 21205 USAKathy J. Helzlsouer, Johns Hopkins Bloomberg School of Public Health Department of Epidemiology 615 N. Wolfe St., Rm E6132 Baltimore MD 21205 USAMichael W. Smith, SAIC-Frederick, Inc., NCI-Frederick Genetics and Genomics, Advanced Technology Program Frederick MD USAVictoriya Grinberg, SAIC-Frederick, Inc., NCI-Frederick Laboratory of Molecular Technology, Ad...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2810676</comments>
            <pubDate>Thu, 17 Sep 2009 12:31:42 +0100</pubDate>
            <guid isPermaLink="false">2810676</guid>        </item>
        <item>
            <title>The effects of polymorphisms in methylenetetrahydrofolate reductase (MTHFR), methionine synthase (MTR), and methionine synthase reductase (MTRR) on the risk of cervical intraepithelial neoplasia and cervical cancer in Korean women</title>
            <link>http://www.medworm.com/index.php?rid=2810675&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fd380h34305138u25%2F</link>
            <description>In conclusion, there was no significant association between most SNPs in MTHFR, MTR, or MTRR and the risk of CIN and cervical cancer in Korean women. In addition, there was no significant association of MTHFR haplotypes with risk of CIN2/3 and cervical cancer.
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9430-zAuthors
		Seo-Yun Tong, Kyung Hee University Department of Obstetrics and Gynecology, East-West Neo Medical Center #149 Sangil-Dong, Gangdong-Gu Seoul 134-890 KoreaJong-Min Lee, Kyung Hee University Department of Obstetrics and Gynecology, East-West Neo Medical Center #149 Sangil-Dong, Gangdong-Gu Seoul 134-890 KoreaEun-Seop Song, Inha University School of Medicine Department of Obstetrics and Gynecology Incheon KoreaKwang-Beom Lee, Gachon University o...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2810675</comments>
            <pubDate>Thu, 17 Sep 2009 12:31:42 +0100</pubDate>
            <guid isPermaLink="false">2810675</guid>        </item>
        <item>
            <title>Family history of cancer and risk of breast cancer in the Black Women’s Health Study</title>
            <link>http://www.medworm.com/index.php?rid=2810677&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fc16485670x708426%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;These findings indicate a strong familial relationship for breast cancer in African American women. The associations with
 family history of colon cancer and leukemia warrant further investigation.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9425-9Authors
		Julie R. Palmer, Boston University Slone Epidemiology Center 1010 Commonwealth Avenue Boston MA 02215 USADeborah A. Boggs, Boston University Slone Epidemiology Center 1010 Commonwealth Avenue Boston MA 02215 USALucile L. Adams-Campbell, Georgetown University Lombardi Comprehensive Cancer Center Washington DC USALynn Rosenberg, Boston University Slone Epidemiology Center 1010 Commonwealth Avenue Boston MA 02215 USA
	

	
		Journal Cancer Causes and ControlOnline ISSN 1573-7225Pri...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2810677</comments>
            <pubDate>Thu, 17 Sep 2009 12:31:41 +0100</pubDate>
            <guid isPermaLink="false">2810677</guid>        </item>
        <item>
            <title>Pancreatitis and pancreatic cancer in two large pooled case–control studies</title>
            <link>http://www.medworm.com/index.php?rid=2810678&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fu0h0g2v0126n4h79%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;A short temporal history of pancreatitis was highly associated with pancreatic cancer, suggesting that pancreatitis may be
 an early manifestation of pancreatic cancer in some individuals. Pancreatic cancer should be considered in the differential
 diagnosis of individuals with an episode of pancreatitis.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9424-xAuthors
		Paige M. Bracci, University of California Department of Epidemiology and Biostatistics, School of Medicine 3333 California St. Suite 280 San Francisco CA 94143 USAFurong Wang, University of California Department of Epidemiology and Biostatistics, School of Medicine 3333 California St. Suite 280 San Francisco CA 94143 USAManal M. Hassan, The University of Texas M.D. Anders...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2810678</comments>
            <pubDate>Thu, 17 Sep 2009 12:31:40 +0100</pubDate>
            <guid isPermaLink="false">2810678</guid>        </item>
        <item>
            <title>Incomplete pregnancy and risk of ovarian cancer: results from two Australian case–control studies and systematic review</title>
            <link>http://www.medworm.com/index.php?rid=2810679&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fkh528342366m55v6%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Although full-term pregnancies reduce the risk of ovarian cancer, it has not been conclusively established whether incomplete
 pregnancies also influence risk. We investigated the relationship between a history of incomplete pregnancy and incident epithelial
 ovarian cancer among over 4,500 women who participated in two large Australian population-based case–control studies in 1990–1993
 and 2002–2005. They provided responses to detailed questions about their reproductive histories and other personal factors.
 Summary odds ratios (OR) and confidence intervals (CI) derived for each study using the same covariates were aggregated. We
 found no significant associations between the number of incomplete pregnancies and ovarian cancer, for parous (OR&amp;nbsp;=&amp;nbsp;0.98,
 ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2810679</comments>
            <pubDate>Thu, 17 Sep 2009 12:31:39 +0100</pubDate>
            <guid isPermaLink="false">2810679</guid>        </item>
        <item>
            <title>Marine n-3 polyunsaturated fatty acids in adipose tissue and breast cancer risk: a case–cohort study from Denmark</title>
            <link>http://www.medworm.com/index.php?rid=2742413&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F8770678675629584%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;This study does not indicate any association between the content of total or individual marine n-3 PUFA in adipose tissue
 and development of BC.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9423-yAuthors
		Petra M. Witt, Aarhus University Hospital Department of Oncology, Aalborg Hospital Hobrovej 18-22 9100 Aalborg DenmarkJeppe H. Christensen, Aarhus University Hospital Department of Nephrology, Center for Cardiovascular Research, Aalborg Hospital Aalborg DenmarkErik B. Schmidt, Aarhus University Hospital Department of Cardiology, Center for Cardiovascular Research, Aalborg Hospital Aalborg DenmarkClaus Dethlefsen, Aarhus University Hospital Department of Cardiology, Center for Cardiovascular Research, Aalborg Hospital Aalborg Denm...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2742413</comments>
            <pubDate>Thu, 27 Aug 2009 15:52:12 +0100</pubDate>
            <guid isPermaLink="false">2742413</guid>        </item>
        <item>
            <title>Human papillomavirus type-distribution in cervical cancer in China: the importance of HPV 16 and 18</title>
            <link>http://www.medworm.com/index.php?rid=2731386&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fx42m2n578q471521%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Prophylactic vaccination against HPV 16 and 18 has the potential for effective prevention of high-grade precancer (cervical
 intraepithelial neoplasia [CIN)] 2/3) and ICC caused by these viruses (globally 50 and 70%, respectively) when employed in
 women prior to starting sexual activity. To provide data for decisions on HPV vaccination in China, we determined HPV type-distribution
 in ICC and CIN 2/3 from women of different regions within China. A multicenter study was conducted by randomized sampling
 of paraffin blocks of 664 ICC (630 squamous cell carcinoma [SCC]; 34 adenocarcinoma [ADC]), 569 CIN 2/3 cases from seven regions
 of China. Histological diagnosis was confirmed in 1,233 cases by consensus review. HPV DNA was detected using the SPF10 LiPA25
 version 1 ass...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2731386</comments>
            <pubDate>Mon, 24 Aug 2009 17:38:37 +0100</pubDate>
            <guid isPermaLink="false">2731386</guid>        </item>
        <item>
            <title>Impact of a positive family history on diagnosis, management, and survival of breast cancer: different effects across socio-economic groups</title>
            <link>http://www.medworm.com/index.php?rid=2723610&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F5v0v41253ht28382%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;A positive FH of breast cancer may reduce SES differences in access to screening and optimal treatment. However, even with
 better access to early detection and optimal treatment, women of low SES have higher risks of death from their disease than
 those of high SES.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9420-1Authors
		H. M. Verkooijen, Geneva University Geneva Cancer Registry Geneva SwitzerlandE. Rapiti, Geneva University Geneva Cancer Registry Geneva SwitzerlandG. Fioretta, Geneva University Geneva Cancer Registry Geneva SwitzerlandV. Vinh-Hung, University Hospital Brussels Oncology Center Brussels BelgiumJ. Keller, Geneva University Geneva Cancer Registry Geneva SwitzerlandS. Benhamou, INSERM, U946, Fondation Jean Dausset...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2723610</comments>
            <pubDate>Fri, 21 Aug 2009 17:10:00 +0100</pubDate>
            <guid isPermaLink="false">2723610</guid>        </item>
        <item>
            <title>Genetic variants in the cell cycle control pathways contribute to early onset colorectal cancer in Lynch syndrome</title>
            <link>http://www.medworm.com/index.php?rid=2717353&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F4413155206243335%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Polymorphisms in these cell cycle-related genes work together to modify the age at the onset of CRC in patients with Lynch
 syndrome. These studies provide an important part of the foundation for development of a model for stratifying age of onset
 risk among those with Lynch syndrome.
 
 
 
	Content Type Journal ArticleCategory Brief reportDOI 10.1007/s10552-009-9416-xAuthors
		Jinyun Chen, The University of Texas M. D. Anderson Cancer Center Department of Epidemiology Unit 1365, 1155 Pressler Boulevard Houston TX 77030 USACarol J. Etzel, The University of Texas M. D. Anderson Cancer Center Department of Epidemiology Unit 1365, 1155 Pressler Boulevard Houston TX 77030 USAChristopher I. Amos, The University of Texas M. D. Anderson Cancer Center Department of Epidemio...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2717353</comments>
            <pubDate>Wed, 19 Aug 2009 08:19:38 +0100</pubDate>
            <guid isPermaLink="false">2717353</guid>        </item>
        <item>
            <title>A community effort to reduce the black/white breast cancer mortality disparity in Chicago</title>
            <link>http://www.medworm.com/index.php?rid=2713736&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fml68g07421212791%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;There are significant access barriers to high quality mammography and treatment services that could be contributing to the
 mortality differences in Chicago. A metropolitan wide taskforce has been established to address the disparity.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9419-7Authors
		David Ansell, Rush University Medical Center 544 Academic Facility 600 South Paulina Avenue Chicago IL 60612 USAPaula Grabler, Northwestern Memorial Hospital Chicago IL USASteven Whitman, Sinai Urban Health Institute Chicago IL USACarol Ferrans, University of Illinois at Chicago Chicago IL USAJacqueline Burgess-Bishop, American Cancer Society Chicago IL USALinda Rae Murray, Cook County Department of Public Health Chicago IL USARuta Rao, Rush ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2713736</comments>
            <pubDate>Mon, 17 Aug 2009 19:01:14 +0100</pubDate>
            <guid isPermaLink="false">2713736</guid>        </item>
        <item>
            <title>Factors related to second cancer screening practice in disease-free cervical cancer survivors</title>
            <link>http://www.medworm.com/index.php?rid=2713735&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F567625081l5p8462%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Lack of a recommendation for second cancer screening from health care providers and the misperception of second cancer risk
 might have negative impacts on the breast cancer screening behaviors in cervical cancer survivors.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9421-0Authors
		Sang Min Park, National Cancer Center Division of Cancer Control 809 Madu-dong, Ilsan-gu Goyang-si Gyeonggi-do 411-769 KoreaChong Taik Park, Pochon CHA University Department of Obstetrics and Gynecology, Kangnam Cha Hospital, College of Medicine Seoul KoreaSang Yoon Park, National Cancer Center Center for Uterine Cancer Goyang Gyeonggi KoreaDuk-Soo Bae, Sungkyunkwan University School of Medicine Department of Obstetrics and Gynecology, Samsung Medical C...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2713735</comments>
            <pubDate>Mon, 17 Aug 2009 19:01:14 +0100</pubDate>
            <guid isPermaLink="false">2713735</guid>        </item>
        <item>
            <title>Human papillomavirus infection in honduran women with normal cytology</title>
            <link>http://www.medworm.com/index.php?rid=2706068&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fp256681212w812q5%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;In the population studied, there was a broad diversity of HPV infections, with high-risk types being the most common types
 detected. The establishment of a well-characterized population with regard to the community prevalence of type-specific HPV
 infection will provide a valuable baseline for monitoring population effectiveness of an HPV vaccine.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9414-zAuthors
		N. Tábora, Universidad Nacional Autónoma de Honduras (UNAH) Department of Microbiology Tegucigalpa HondurasJ. M. J. E. Bakkers, Radboud University Nijmegen Medical Centre Department of Medical Microbiology Nijmegen The NetherlandsW. G. V. Quint, DDL Diagnostic Laboratory Voorburg The NetherlandsL. F. A. G. Massuger, Radboud U...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2706068</comments>
            <pubDate>Fri, 14 Aug 2009 16:54:28 +0100</pubDate>
            <guid isPermaLink="false">2706068</guid>        </item>
        <item>
            <title>Meat intake, meat mutagens and risk of lung cancer in Uruguayan men</title>
            <link>http://www.medworm.com/index.php?rid=2706069&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F7u82548715tnw313%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;This study suggests that red and processed meat and meat mutagens may play a role in the etiology of lung cancer.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9411-2Authors
		Eduardo De Stefani, Hospital de Clínicas Grupo de Epidemiología, Departamento de Anatomía Patológica, Facultad de Medicina Avenida Brasil 3080 department 402 11300 Montevideo UruguayPaolo Boffetta, International Agency for Research on Cancer Lyon FranceHugo Deneo-Pellegrini, Hospital de Clínicas Grupo de Epidemiología, Departamento de Anatomía Patológica, Facultad de Medicina Avenida Brasil 3080 department 402 11300 Montevideo UruguayAlvaro L. Ronco, Centro Latinoamericano de Economía Humana (CLAEH) Departamento de Epidemiología y Métodos Científic...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2706069</comments>
            <pubDate>Fri, 14 Aug 2009 16:54:26 +0100</pubDate>
            <guid isPermaLink="false">2706069</guid>        </item>
        <item>
            <title>Ambient air pollution and brain cancer mortality</title>
            <link>http://www.medworm.com/index.php?rid=2706070&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fx32n682n21686785%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;The findings do not provide evidence of increased risk of brain cancer mortality due to air pollutants.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9412-1Authors
		Roberta McKean-Cowdin, University of Southern California Keck School of Medicine, Norris Comprehensive Cancer Center Department of Preventive Medicine 1441 Eastlake Avenue MS/44 Room 4419 Los Angeles CA 90089-9175 USAEugenia E. Calle, American Cancer Society, National Home Office 1599 Clifton Road, NE Atlanta GA 30329-4251 USAJohn M. Peters, University of Southern California Keck School of Medicine, Norris Comprehensive Cancer Center Department of Preventive Medicine 1441 Eastlake Avenue MS/44 Room 4419 Los Angeles CA 90089-9175 USAJane Henley, American Cancer Society, N...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2706070</comments>
            <pubDate>Fri, 14 Aug 2009 16:54:25 +0100</pubDate>
            <guid isPermaLink="false">2706070</guid>        </item>
        <item>
            <title>Is the incidence of invasive vulvar cancer increasing in the United States?</title>
            <link>http://www.medworm.com/index.php?rid=2700733&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F144lur82655m8017%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Incidence rates of invasive vulvar cancer have increased in the United States during the last three decades. The reasons for
 this increase are unknown.
 
 
 
	Content Type Journal ArticleCategory Brief reportDOI 10.1007/s10552-009-9418-8Authors
		Clara Bodelon, University of Washington Department of Epidemiology, School of Public Health Box 357236 1959 NE Pacific Street Seattle WA 98195-7236 USAMargaret M. Madeleine, University of Washington Department of Epidemiology, School of Public Health Box 357236 1959 NE Pacific Street Seattle WA 98195-7236 USALynda F. Voigt, University of Washington Department of Epidemiology, School of Public Health Box 357236 1959 NE Pacific Street Seattle WA 98195-7236 USANoel S. Weiss, University of Washington Department of Epidemiology,...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2700733</comments>
            <pubDate>Wed, 12 Aug 2009 22:29:10 +0100</pubDate>
            <guid isPermaLink="false">2700733</guid>        </item>
        <item>
            <title>Polymorphisms in arsenic metabolism genes, urinary arsenic methylation profile and cancer</title>
            <link>http://www.medworm.com/index.php?rid=2700734&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fh2446t031m813672%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Arsenic-metabolism-related genes can regulate the arsenic methylation process and may influence susceptibility to cancer.
 We evaluated the roles of arsenic metabolism genes on urinary arsenic profiles of repeated measurement with 15-year follow-up
 (1988–2004) through general linear model (GLM) and assessed the effect of the changed extent of urinary arsenic profiles on
 cancer risk. Questionnaire information and blood samples and two urines (1988 and 2004) were collected from 208 subjects in
 an arseniasis hyperendemic area in Taiwan. Profiles for concentrations of urinary arsenic were determined using HPLC-HG-AAS.
 The relative proportion of each arsenic species was calculated by dividing the concentration of each arsenic species by the
 total arsenic concentration...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2700734</comments>
            <pubDate>Wed, 12 Aug 2009 15:58:50 +0100</pubDate>
            <guid isPermaLink="false">2700734</guid>        </item>
        <item>
            <title>Trends in adult leukemia incidence and survival in Denmark, 1943–2003</title>
            <link>http://www.medworm.com/index.php?rid=2697092&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fl465502n78u3h1q0%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;The etiology of leukemia is largely unknown. Ecological data indicating trends in incidence and survival can provide information
 about changes in risk factors, can reflect underlying changes in diagnostic classification, and can measure therapeutic advances.
 From the records of the Danish Cancer Registry with registration starting from 1943, we calculated age-specific, period-specific,
 and age-standardized (world standard) incidence rates of chronic lymphoid leukemia (CLL), acute lymphoid leukemia (ALL), chronic
 myeloid leukemia (CML), and acute myeloid leukemia (AML) for persons above the age of 18. Kaplan–Meier survival curves and
 median survival times were calculated. Between 1943 and 2003, there were 26,036 cases of leukemia reported. The age-specific
 incide...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2697092</comments>
            <pubDate>Tue, 11 Aug 2009 18:43:10 +0100</pubDate>
            <guid isPermaLink="false">2697092</guid>        </item>
        <item>
            <title>Microsatellite instability and survival in rectal cancer</title>
            <link>http://www.medworm.com/index.php?rid=2697093&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F4650085p316v1mt7%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;The genetic and epigenetic characteristics of MSI-H rectal cancers suggest that they are enriched for Lynch-associated tumors;
 adverse prognosis associated with MSI-H in these tumors may reflect the relatively high frequency of Lynch-associated cancers
 and/or the effect of radiation or chemotherapy on Lynch-associated rectal cancers or MSI tumors in general.
 
 
 
	Content Type Journal ArticleCategory Brief reportDOI 10.1007/s10552-009-9410-3Authors
		Wade S. Samowitz, University of Utah Health Sciences Center Department of Pathology Salt Lake City UT 84132 USAKaren Curtin, University of Utah Health Sciences Center Department of Internal Medicine Salt Lake City UT 84132 USARoger K. Wolff, University of Utah Health Sciences Center Department of Internal Medicine Salt...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2697093</comments>
            <pubDate>Tue, 11 Aug 2009 02:10:55 +0100</pubDate>
            <guid isPermaLink="false">2697093</guid>        </item>
        <item>
            <title>Correlates of sexually transmitted infection histories in a cohort of American male health professionals</title>
            <link>http://www.medworm.com/index.php?rid=2679727&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fv29465n01l0642q2%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;These findings provide useful information on potential confounders for epidemiologic investigations of STIs and chronic diseases,
 and interesting new hypotheses for STI prevention (e.g., STI counseling before vasectomy).
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9409-9Authors
		Siobhan Sutcliffe, Washington University School of Medicine Alvin J. Siteman Cancer Center and the Department of Surgery St. Louis MO USAIchiro Kawachi, Harvard School of Public Health Department of Society, Human Development and Health Boston MA USAJohn F. Alderete, Washington State University School of Molecular Biosciences, Fulmer Hall Pullman WA USACharlotte A. Gaydos, Johns Hopkins Medical Institutions Division of Infectious Diseases, Department of ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2679727</comments>
            <pubDate>Tue, 04 Aug 2009 20:03:06 +0100</pubDate>
            <guid isPermaLink="false">2679727</guid>        </item>
        <item>
            <title>Metformin use and prostate cancer in Caucasian men: results from a population-based case–control study</title>
            <link>http://www.medworm.com/index.php?rid=2668795&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F524142489791x527%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Metformin use was associated with a borderline significant decrease in the relative risk of PCa in Caucasians. Further study
 into this relationship is needed to confirm the association and determine the underlying pathways involved.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9407-yAuthors
		Jonathan L. Wright, University of Washington School of Medicine Department of Urology Seattle WA USAJanet L. Stanford, Fred Hutchinson Cancer Research Center Division of Public Health Sciences 1100 Fairview Avenue North, M4-B874 Seattle WA 98109-1024 USA
	

	
		Journal Cancer Causes and ControlOnline ISSN 1573-7225Print ISSN 0957-5243 (Source: Cancer Causes and Control)</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2668795</comments>
            <pubDate>Mon, 03 Aug 2009 17:52:59 +0100</pubDate>
            <guid isPermaLink="false">2668795</guid>        </item>
        <item>
            <title>Menstrual and reproductive factors and pancreatic cancer in the SEARCH program of the IARC</title>
            <link>http://www.medworm.com/index.php?rid=2668796&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fc7l2041367212232%2F</link>
            <description>In conclusion, earlier age at menarche
 may be weakly associated with pancreatic cancer, but it seems unlikely that menstrual and reproductive factors play more than
 only a minor role in pancreatic cancer. Additional analyses in large prospective study populations and in pooled studies may
 help to clarify remaining inconsistencies.
 
	Content Type Journal ArticleCategory Brief reportDOI 10.1007/s10552-009-9408-xAuthors
		Eric J. Duell, Institut Català d’Oncologia (ICO), Catalan Institute of Oncology Unit of Nutrition, Environment and Cancer, Cancer Epidemiology Research Programme Avda Gran Via 199-203, 08907 L’Hospitalet de Llobregat Barcelona SpainPatrick Maisonneuve, European Institute of Oncology Milan ItalyPeter A. Baghurst, Women’s and Children’s Hospital Public Health Rese...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2668796</comments>
            <pubDate>Mon, 03 Aug 2009 17:52:58 +0100</pubDate>
            <guid isPermaLink="false">2668796</guid>        </item>
        <item>
            <title>Legume intake and the risk of cancer: a multisite case–control study in Uruguay</title>
            <link>http://www.medworm.com/index.php?rid=2668797&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fuv330j7213j67127%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Higher intake of legumes was associated with a decreased risk of several cancers including those of the upper aerodigestive
 tract, stomach, colorectum, and kidney, but not lung, breast, prostate or bladder. Further investigations of these associations
 in prospective cohort studies are warranted.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9406-zAuthors
		Dagfinn Aune, University of Oslo Department of Biostatistics, Institute of Basic Medical Sciences P.O. Box 1122 0317 Oslo NorwayEduardo De Stefani, Universidad de la República Grupo de Epidemiología, Departamento de Anatomía Patológica, Hospital de Clínicas, Facultad de Medicina Montevideo UruguayAlvaro Ronco, Centro Latinoamericano de Economıa Humana (CLAEH) Departamento d...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2668797</comments>
            <pubDate>Mon, 03 Aug 2009 17:52:55 +0100</pubDate>
            <guid isPermaLink="false">2668797</guid>        </item>
        <item>
            <title>Total visual blindness is protective against breast cancer</title>
            <link>http://www.medworm.com/index.php?rid=2664762&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fb03452w702x87786%2F</link>
            <description>Abstract
 Objective&amp;nbsp;&amp;nbsp;Observational data, though sparse and based on small studies with limited ability to control for known breast cancer risk
 factors, support a lower risk of breast cancer in blind women compared to sighted women. Mechanisms influenced by ocular light
 perception, such as melatonin or circadian synchronization, are thought to account for this lower risk.
 
 
 
 Methods&amp;nbsp;&amp;nbsp;To evaluate whether blind women with no perception of light (NPL) have a lower prevalence of breast cancer compared to blind
 women with light perception (LP), we surveyed a cohort of 1,392 blind women living in North America (66 breast cancer cases).
 
 
 
 Results&amp;nbsp;&amp;nbsp;In multivariate-logistic regression models controlling for breast cancer risk factors, women with NPL had a si...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2664762</comments>
            <pubDate>Fri, 31 Jul 2009 19:44:49 +0100</pubDate>
            <guid isPermaLink="false">2664762</guid>        </item>
        <item>
            <title>Effects of Helicobacter pylori infection and smoking on gastric cancer incidence in China: a population-level analysis of trends and projections</title>
            <link>http://www.medworm.com/index.php?rid=2661485&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fkl7g001j1914m874%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;The decline in gastric cancer incidence has been slower than in developed countries and will be offset by population growth
 and aging. Public health interventions should be implemented to reduce the total number of cases.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9397-9Authors
		Jennifer M. Yeh, Harvard School of Public Health Center for Health Decision Science 718 Huntington Avenue, 2nd Floor Boston MA 02115 USASue J. Goldie, Harvard School of Public Health Center for Health Decision Science 718 Huntington Avenue, 2nd Floor Boston MA 02115 USAKaren M. Kuntz, University of Minnesota Division of Health Policy and Management, School of Public Health Minneapolis MN USAMajid Ezzati, Harvard School of Public Health Department of Glo...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2661485</comments>
            <pubDate>Thu, 30 Jul 2009 07:48:38 +0100</pubDate>
            <guid isPermaLink="false">2661485</guid>        </item>
        <item>
            <title>Hypothesis: neoplasms in myotonic dystrophy</title>
            <link>http://www.medworm.com/index.php?rid=2661484&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fk8pr5377l2118k73%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Tumorigenesis is a multi-step process due to an accumulation of genetic mutations in multiple genes in diverse pathways which
 ultimately lead to loss of control over cell growth. It is well known that inheritance of rare germline mutations in genes
 involved in tumorigenesis pathways confer high lifetime risk of neoplasia in affected individuals. Furthermore, a substantial
 number of multiple malformation syndromes include cancer susceptibility in their phenotype. Studies of the mechanisms underlying
 these inherited syndromes have added to the understanding of both normal development and the pathophysiology of carcinogenesis.
 Myotonic dystrophy (DM) represents a group of autosomal dominant, multisystemic diseases that share the clinical features
 of myotonia, muscle ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2661484</comments>
            <pubDate>Thu, 30 Jul 2009 07:48:38 +0100</pubDate>
            <guid isPermaLink="false">2661484</guid>        </item>
        <item>
            <title>Perinatal markers of estrogen exposure and risk of testicular cancer: follow-up of 1,333,873 Danish males born between 1950 and 2002</title>
            <link>http://www.medworm.com/index.php?rid=2650900&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fe85gr4k4206x4580%2F</link>
            <description>Abstract
 Objective&amp;nbsp;&amp;nbsp;To examine whether indicators of an increased prenatal estrogen exposure correlate with risk of testicular cancer.
 
 
 
 Methods&amp;nbsp;&amp;nbsp;This nationwide follow-up study was conducted by linking data on prenatal exposures obtained from medical and birth records
 for information on incident testicular cancer cases identified from the Danish Cancer Registry. The study population included
 all boys born between 1950 and 2002 and alive in April 1968 or later with restriction to sons of mothers born after 1935 and
 alive in April 1968.
 
 
 
 Results&amp;nbsp;&amp;nbsp;A total of 2,151 incident testicular cancer cases were identified in 29&amp;nbsp;million person-years at risk. Men born with a high
 birth weight (&amp;gt;4,150&amp;nbsp;g) had an increased risk of testicular cancer...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2650900</comments>
            <pubDate>Mon, 27 Jul 2009 21:02:40 +0100</pubDate>
            <guid isPermaLink="false">2650900</guid>        </item>
        <item>
            <title>International variation in the incidence of adult primary malignant neoplasms of the brain and central nervous system</title>
            <link>http://www.medworm.com/index.php?rid=2642115&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F6tu51755810532m6%2F</link>
            <description>We examined international variation in adult brain cancer incidence in 69 populations from a subset of cancer registries included
 in Cancer Incidence in Five Continents, Volume IX that met stringent quality standards. We grouped these populations into World Areas. Age-standardized incidence rates varied
 by World Area similarly for men and women and were highest in northern America non-Hispanic whites and Australians, followed
 closely behind by Europeans, then South Americans and northern America Hispanic whites. Rates among south-central Asians and
 northern America blacks were half to three-fifths the rates among northern America non-Hispanic whites. Rates among eastern
 and southeastern Asians and northern America Asians and Pacific Islanders were one-third to two-fifths the rates amo...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2642115</comments>
            <pubDate>Sat, 25 Jul 2009 14:44:32 +0100</pubDate>
            <guid isPermaLink="false">2642115</guid>        </item>
        <item>
            <title>Modeling the transition of lung cancer from early to advanced stage</title>
            <link>http://www.medworm.com/index.php?rid=2642116&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Frhq15x638w83370u%2F</link>
            <description>We present a stochastic parametric model of the natural history of lung cancer that predicts the primary tumor volume at the
 moment the disease transits from early to advanced stage. Our model also produces estimates for the probability of symptomatic
 detection as a function of tumor volume and clinical stage. We estimate model parameters by likelihood maximization using
 data from the Mayo Lung Project (MLP), which was a clinical trial that evaluated screening for lung cancer in the 1970s. Mayo
 Lung Project cancer cases reported in Stage III or greater, according to the 1979 AJCC staging for lung cancer, were considered
 advanced stage. Our estimator distinguishes between the cases detected because of clinical symptoms and cases detected by
 screening. For nonsmall cell lung cancer cas...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2642116</comments>
            <pubDate>Sat, 25 Jul 2009 04:33:37 +0100</pubDate>
            <guid isPermaLink="false">2642116</guid>        </item>
        <item>
            <title>Indirect method to estimate specific Hispanic group cancer rates</title>
            <link>http://www.medworm.com/index.php?rid=2631600&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F97432658x2146421%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;The approach yielded reasonable and useful information to explore etiologic differences among the populations, as well as
 to develop relevant cancer control interventions. However, direct identification of specific Hispanic ethnicity in medical
 records and annual Census estimates of these populations would be preferable if they ever became available.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9398-8Authors
		Holly L. Howe, North American Association of Central Cancer Registries, Inc. Springfield IL USAAndrew Lake, Information Management Services, Inc. Silver Spring MD USAMaria J. Schymura, New York State Department of Health Albany NY USABrenda K. Edwards, National Cancer Institute Bethesda MD USA
	

	
		Journal Cancer Causes an...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2631600</comments>
            <pubDate>Tue, 21 Jul 2009 00:04:55 +0100</pubDate>
            <guid isPermaLink="false">2631600</guid>        </item>
        <item>
            <title>Maternal smoking during pregnancy and childhood cancer in New South Wales: a record linkage investigation</title>
            <link>http://www.medworm.com/index.php?rid=2631599&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F182515300w156570%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Following linkage between the NSW Central Cancer Registry (CCR) and the NSW Midwives Data Collection, an investigation of
 the association between maternal smoking during pregnancy and the risk of childhood cancer in their offspring was undertaken.
 Children born in NSW between 1994 and 2005, inclusive of 1,045,966 babies, were matched to 948 cancer cases in the CCR. After
 adjustment for maternal age, gestational age, baby’s gender, birth weight, remoteness index, socioeconomic disadvantage and
 maternal health factors, no association (OR&amp;nbsp;=&amp;nbsp;0.96, 95% CI 0.81–1.15, p&amp;nbsp;=&amp;nbsp;0.68) was found with childhood cancer between mothers who smoked (81/100,000) and those who did not smoke during pregnancy
 (99/100, 000). Maternal smoking was, however, significan...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2631599</comments>
            <pubDate>Tue, 21 Jul 2009 00:04:55 +0100</pubDate>
            <guid isPermaLink="false">2631599</guid>        </item>
        <item>
            <title>Genetic polymorphisms of glutathione S-transferase genes GSTP1, GSTM1, and GSTT1 and risk of esophageal and gastric cardia cancers</title>
            <link>http://www.medworm.com/index.php?rid=2631601&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fgg7374806u611632%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Glutathione S-transferase (GST) enzymes are known to metabolize tobacco-related carcinogens. Previous studies on the association
 of functional polymorphisms of GST genes with esophageal squamous cell carcinoma have yielded conflicting but overall null
 results. A few studies of esophageal adenocarcinoma were likewise conflicting, but the scarcity of data is striking. We aimed
 to study associations of the GSTM1 and GSTT1 null deletion polymorphisms as well as the GSTP1 Ile105Val polymorphism with risks for esophageal and gastric cardia cancers. DNA was prepared from 96 and 79 cases of esophageal
 adenocarcinoma and squamous cell carcinoma, respectively, 126 cardia cancer cases, and 471 population-based controls. Pyrosequencing
 typed the GSTP1 Ile105Val polymorphism, w...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2631601</comments>
            <pubDate>Tue, 21 Jul 2009 00:04:54 +0100</pubDate>
            <guid isPermaLink="false">2631601</guid>        </item>
        <item>
            <title>Jasmine tea consumption and upper gastrointestinal cancer in China</title>
            <link>http://www.medworm.com/index.php?rid=2603538&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F26k02k74l70j2736%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Overall, we found no evidence for a protective effect of tea in esophageal or gastric cancer. Further studies of the potential
 effects of thermal damage, tea quality, and water quality on UGI cancers are suggested.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9394-zAuthors
		Ying Gao, National Cancer Institute Division of Cancer Epidemiology and Genetics Bethesda MD USANan Hu, National Cancer Institute Division of Cancer Epidemiology and Genetics Bethesda MD USAXiaoYou Han, Shanxi Cancer Hospital Taiyuan Shanxi People’s Republic of ChinaCarol Giffen, Information Management Services, Inc. Silver Spring MD USATi Ding, Shanxi Cancer Hospital Taiyuan Shanxi People’s Republic of ChinaAlisa M. Goldstein, National Cancer Institute Div...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2603538</comments>
            <pubDate>Tue, 14 Jul 2009 10:05:32 +0100</pubDate>
            <guid isPermaLink="false">2603538</guid>        </item>
        <item>
            <title>Coffee and black tea consumption and risk of breast cancer by estrogen and progesterone receptor status in a Swedish cohort</title>
            <link>http://www.medworm.com/index.php?rid=2599329&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fmj566x23647766p8%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Findings from this prospective study do not support a role of coffee consumption in the development of breast cancer but suggest
 that black tea consumption may be positively associated with risk of ER+/PR+ tumors.
 
 
 
	Content Type Journal ArticleCategory Brief reportDOI 10.1007/s10552-009-9396-xAuthors
		Susanna C. Larsson, The National Institute of Environmental Medicine, Karolinska Institute Division of Nutritional Epidemiology Box 210 171 77 Stockholm SwedenLeif Bergkvist, Central Hospital Department of Surgery and Centre for Clinical Research Vasteras SwedenAlicja Wolk, The National Institute of Environmental Medicine, Karolinska Institute Division of Nutritional Epidemiology Box 210 171 77 Stockholm Sweden
	

	
		Journal Cancer Causes and ControlOnline ISSN 1...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2599329</comments>
            <pubDate>Mon, 13 Jul 2009 06:13:11 +0100</pubDate>
            <guid isPermaLink="false">2599329</guid>        </item>
        <item>
            <title>Cancer risk factors associated with insulin-like growth factor (IGF)-I and IGF-binding protein-3 levels in healthy women: effect modification by menopausal status</title>
            <link>http://www.medworm.com/index.php?rid=2599330&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fl61072u34v222280%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Insulin-like growth factor-I (IGF-I) and IGF-binding protein-3 (IGFBP-3) appear to influence breast cancer risk and are hypothesized
 to mediate the effects of several cancer risk factors that depend on menopausal status. We investigated associations among
 cancer risk factors and plasma IGF-I and IGFBP-3 in 882 healthy control women (426 premenopausal, 456 postmenopausal) from
 two population-based breast cancer case–control studies. Interactions with menopausal status were statistically tested. We
 observed associations with non-modifiable (age, benign breast disease) and modifiable factors [body mass index (BMI), physical
 activity, smoking habits, alcohol consumption]. Furthermore, we demonstrated statistical interactions with menopausal status.
 Premenopausal IGF...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2599330</comments>
            <pubDate>Mon, 13 Jul 2009 06:13:09 +0100</pubDate>
            <guid isPermaLink="false">2599330</guid>        </item>
        <item>
            <title>Screening pharmaceuticals for possible carcinogenic effects: initial positive results for drugs not previously screened</title>
            <link>http://www.medworm.com/index.php?rid=2589412&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fy000545317265223%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;These preliminary findings suggest that further studies are indicated regarding sulindac, hyoscyamine, nortriptyline, oxazepam,
 fluoxetine, paroxetine, hydrochlorothiazide, and nifedipine.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9375-2Authors
		Gary D. Friedman, Kaiser Permanente Medical Care Program Division of Research 2000 Broadway Oakland CA 94612 USANatalia Udaltsova, Kaiser Permanente Medical Care Program Division of Research 2000 Broadway Oakland CA 94612 USAJames Chan, Kaiser Permanente Medical Care Program Pharmacy Outcomes Research Group 1800 Harrison Oakland CA USACharles P. Quesenberry, Kaiser Permanente Medical Care Program Division of Research 2000 Broadway Oakland CA 94612 USALaurel A. Habel, Kaiser Permanente ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2589412</comments>
            <pubDate>Tue, 07 Jul 2009 15:11:49 +0100</pubDate>
            <guid isPermaLink="false">2589412</guid>        </item>
        <item>
            <title>Dairy intake and 1,25-dihydroxyvitamin D levels in men at high risk for prostate cancer</title>
            <link>http://www.medworm.com/index.php?rid=2589414&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fe267405k6615h0v8%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Our findings, in a sample including a large proportion of black participants, do not confirm previous findings showing an
 inverse association between calcium intake and 1,25(OH)2D levels. As such, they suggest that future work should explore other mechanisms by which dairy foods and calcium might increase
 prostate cancer risk.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9389-9Authors
		Marilyn Tseng, California Polytechnic State University Department of Kinesiology 1 Grand Avenue San Luis Obispo CA 93407 USAVeda Giri, Fox Chase Cancer Center Division of Cancer Prevention and Control 333 Cottman Avenue Philadelphia PA 19111 USADeborah Watkins-Bruner, University of Pennsylvania School of Nursing Pennsylvania PA USAEdward Giovannucc...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2589414</comments>
            <pubDate>Mon, 06 Jul 2009 20:25:12 +0100</pubDate>
            <guid isPermaLink="false">2589414</guid>        </item>
        <item>
            <title>Lay interpersonal sources for health information related to beliefs about the modifiability of cancer risk</title>
            <link>http://www.medworm.com/index.php?rid=2589413&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fe7648p572208tx8m%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;These findings showed that lay interpersonal health information sources are associated with beliefs about the modifiability
 of colon and skin cancer risk. Future research is needed to investigate whether and how such information sources might influence
 decisions about engaging in preventive behaviors.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9392-1Authors
		Beth M. Ford, Harvard School of Public Health Department of Society, Human Development, and Health Boston MA USAKimberly A. Kaphingst, National Human Genome Research Institute Social and Behavioral Research Branch Building 31, Room B1B37E, 31 Center Drive, MSC 2073 Bethesda MD 20892 USA
	

	
		Journal Cancer Causes and ControlOnline ISSN 1573-7225Print ISSN 0957-5243 (Sourc...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2589413</comments>
            <pubDate>Mon, 06 Jul 2009 20:25:12 +0100</pubDate>
            <guid isPermaLink="false">2589413</guid>        </item>
        <item>
            <title>Cigarette smoking and risk of prostate cancer among Singapore Chinese</title>
            <link>http://www.medworm.com/index.php?rid=2589415&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fm74863474404567l%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Prospective epidemiologic studies conducted in Western populations support an association between current smoking and aggressive
 subtypes of prostate cancer. In Singapore, where prostate-specific antigen is not used for population-wide screening, prostate
 cancer incidence has tripled within the past two decades. Using Cox regression methods, we examined the relationship between
 smoking and prostate cancer established between 1993 and 1998 in a cohort of 27,293 Singapore Chinese men. As of December
 2006, 250 incident prostate cancer cases were diagnosed. In our cohort, 42.2% reported never smoking cigarettes, 15.7% quit
 over 5&amp;nbsp;years ago (long-term former), 5.7% quit within the past 5&amp;nbsp;years (recent former), and 36.4% were current smokers. From
 multivariabl...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2589415</comments>
            <pubDate>Mon, 06 Jul 2009 20:02:56 +0100</pubDate>
            <guid isPermaLink="false">2589415</guid>        </item>
        <item>
            <title>Smoking and risk of glioma: a meta-analysis</title>
            <link>http://www.medworm.com/index.php?rid=2564134&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fb4u21j27234658q8%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Overall, results of pooling of all studies suggested that smoking is not associated with risk of glioma. However, the small
 but significant increased risk seen for cohort studies remains to be clarified.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9386-zAuthors
		Lori Mandelzweig, Chaim Sheba Medical Center Cancer and Radiation Epidemiology Unit, Gertner Institute of Epidemiology and Health Policy Research Tel Hashomer 52621 IsraelIlya Novikov, Chaim Sheba Medical Center Biostatistics Unit, Gertner Institute of Epidemiology and Health Policy Research Tel Hashomer 52621 IsraelSiegal Sadetzki, Chaim Sheba Medical Center Cancer and Radiation Epidemiology Unit, Gertner Institute of Epidemiology and Health Policy Research Tel Hashomer ...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2564134</comments>
            <pubDate>Wed, 01 Jul 2009 06:22:01 +0100</pubDate>
            <guid isPermaLink="false">2564134</guid>        </item>
        <item>
            <title>Polymorphisms in glutathione S-transferase genes increase risk of prostate cancer biochemical recurrence differentially by ethnicity and disease severity</title>
            <link>http://www.medworm.com/index.php?rid=2564135&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fa14767827162mj24%2F</link>
            <description>Conclusion&amp;nbsp;&amp;nbsp;Our results suggest GSTs may hold promise as therapeutic targets in more advanced prostate cancers, particularly, in African-Americans.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9385-0Authors
		Nora L. Nock, Case Western Reserve University Department of Epidemiology and Biostatistics Cleveland OH USACathryn Bock, Wayne State University Karmanos Cancer Institute Detroit MI USAChristine Neslund-Dudas, Henry Ford Health System Department of Biostatistics and Epidemiology 1 Ford Place, 3E Detroit MI 48202 USAJennifer Beebe-Dimmer, Wayne State University Karmanos Cancer Institute Detroit MI USAAndrew Rundle, Columbia University Department of Environmental Health Sciences New York NY USADeliang Tang, Columbia University Department of E...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2564135</comments>
            <pubDate>Wed, 01 Jul 2009 06:22:00 +0100</pubDate>
            <guid isPermaLink="false">2564135</guid>        </item>
        <item>
            <title>Bladder cancer and reproductive factors among women in Spain</title>
            <link>http://www.medworm.com/index.php?rid=2564136&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fr72756832q7471j5%2F</link>
            <description>Abstract&amp;nbsp;&amp;nbsp;Hormonal factors, possibly related to reproductive characteristics, may play a role in the risk of bladder cancer among women.
 To study this, we investigated the effects of reproductive factors on female bladder cancer risk. Information on reproductive
 and other risk factors was gathered in personal interviews from 152 female cases and 166 matched controls from 18 hospitals
 in five regions of Spain during 1998–2001. Logistic regression was used to estimate the association between bladder cancer
 and reproductive factors, including ever-parous status, age at first live birth, age at last live birth, age at menarche,
 age at menopause, menopausal status, and duration of menstruation. After adjustment for age, smoking, and high-risk occupation,
 ever-parous women were...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2564136</comments>
            <pubDate>Wed, 01 Jul 2009 06:21:59 +0100</pubDate>
            <guid isPermaLink="false">2564136</guid>        </item>
        <item>
            <title>Timing of births and endometrial cancer risk in Swedish women</title>
            <link>http://www.medworm.com/index.php?rid=2564137&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fy2848nl2072t040m%2F</link>
            <description>We examined the relationship between endometrial cancer risk and reproductive characteristics in
 a population-based cohort of 2,674,465 Swedish women, 20–72&amp;nbsp;years of age. During follow-up from 1973 to 2004, 7,386 endometrial
 cancers were observed. Compared to uniparous women, nulliparous women had a significantly elevated endometrial cancer risk
 (hazard ratio [HR]&amp;nbsp;=&amp;nbsp;1.32, 95% confidence interval [CI], 1.22–1.42). Endometrial cancer risk decreased with increasing parity;
 compared to uniparous women, women with ≥4 births had a HR&amp;nbsp;=&amp;nbsp;0.66 (95% CI, 0.59–0.74); p-trend&amp;nbsp;&amp;lt;&amp;nbsp;0.001. Among multiparous women, we observed no relationship of risk with age at first birth after adjustment for
 other reproductive factors. While we initially observed a decrea...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2564137</comments>
            <pubDate>Tue, 30 Jun 2009 15:52:11 +0100</pubDate>
            <guid isPermaLink="false">2564137</guid>        </item>
        <item>
            <title>Dietary change among breast and colorectal cancer survivors and cancer-free women in the Norwegian Women and Cancer cohort study</title>
            <link>http://www.medworm.com/index.php?rid=2564138&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F5711183g174t6082%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Cancer survivors showed little change toward cancer-preventive guidelines, although more advanced stage and being more than
 2.4&amp;nbsp;years post diagnosis was associated with greater change in diet and smoking behaviors.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9390-3Authors
		Guri Skeie, University of Tromsø Institute of Community Medicine 9037 Tromsø NorwayAnette Hjartåker, Cancer Registry of Norway Oslo NorwayTonje Braaten, University of Tromsø Institute of Community Medicine 9037 Tromsø NorwayEiliv Lund, University of Tromsø Institute of Community Medicine 9037 Tromsø Norway
	

	
		Journal Cancer Causes and ControlOnline ISSN 1573-7225Print ISSN 0957-5243 (Source: Cancer Causes and Control)</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2564138</comments>
            <pubDate>Tue, 30 Jun 2009 15:52:05 +0100</pubDate>
            <guid isPermaLink="false">2564138</guid>        </item>
        <item>
            <title>Smoking attenuates the negative association between carotenoids consumption and colorectal cancer risk</title>
            <link>http://www.medworm.com/index.php?rid=2557404&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2Fk275w15v68749048%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Consumption of most dietary carotenoids was found to be strongly associated with reduced risk of CRC. However, smoking significantly
 attenuated or reversed this observed protective effect on CRC occurrence. Smokers should be advised that smoking also hampers
 the potential health promoting effects of high fruit and vegetable consumption.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9354-7Authors
		Y. Chaiter, Technion, Israel Institute of Technology Department of Community Medicine and Epidemiology, CHS National Cancer Control Center, Carmel Medical Center and B. Rappaport Faculty of Medicine 7 Michal St 34362 Haifa IsraelS. B. Gruber, University of Michigan Schools of Medicine and Public Health Department of Internal Medicine Ann...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2557404</comments>
            <pubDate>Mon, 29 Jun 2009 08:16:24 +0100</pubDate>
            <guid isPermaLink="false">2557404</guid>        </item>
        <item>
            <title>Influence of tumor stage, symptoms, and time of blood draw on serum concentrations of organochlorine compounds in exocrine pancreatic cancer</title>
            <link>http://www.medworm.com/index.php?rid=2557405&amp;cid=s_35914_6_f&amp;fid=35914&amp;url=http%3A%2F%2Fwww.springerlink.com%2Fcontent%2F31706hu75g3jh84t%2F</link>
            <description>Conclusions&amp;nbsp;&amp;nbsp;Restriction or adjustment by stage and timing of blood draw may be insufficient to prevent biases associated with cancer progression.
 Symptoms may enable investigators to assess disease-induced changes in lipophilic exposure biomarkers.
 
 
 
	Content Type Journal ArticleCategory Original paperDOI 10.1007/s10552-009-9383-2Authors
		Miquel Porta, Institut Municipal d’Investigació Mèdica (IMIM)-Hospital del Mar, Universitat Autònoma de Barcelona Carrer del Dr. Aiguader 88 E-08003 Barcelona Catalonia SpainJosé Pumarega, Institut Municipal d’Investigació Mèdica (IMIM)-Hospital del Mar, Universitat Autònoma de Barcelona Carrer del Dr. Aiguader 88 E-08003 Barcelona Catalonia SpainTomàs López, Institut Municipal d’Investigació Mèdica (IMIM)-Hospital del Ma...</description>
            <author>Cancer Causes and Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2557405</comments>
            <pubDate>Mon, 29 Jun 2009 08:16:17 +0100</pubDate>
            <guid isPermaLink="false">2557405</guid>        </item>
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