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        <title>Cancer Control via MedWorm.com</title>
        <description>MedWorm.com provides a medical RSS filtering service. Over 6000 RSS medical sources are combined and output via different filters. This feed contains the latest items from the 'Cancer Control' source.</description>
        <link><![CDATA[http://www.medworm.com/rss/search.php?qu=Cancer+Control&t=Cancer+Control&s=Search&f=source]]></link>
        <lastBuildDate>Wed, 08 Feb 2012 08:41:20 +0100</lastBuildDate>
        <item>
            <title>Cancer Control; +227 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5643056&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222012%252F01%252F25%252000.54%2522%255BMHDA%255D%253A%25222012%252F01%252F31%252000.30%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222012%252F01%252F25%252000.54%2522%255BEDAT%255D%29%29</link>
            <description>227 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2012/01/31PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5643056</comments>
            <pubDate>Tue, 31 Jan 2012 05:30:00 +0100</pubDate>
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            <title>Cancer Control; +514 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5625439&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222012%252F01%252F12%252009.54%2522%255BMHDA%255D%253A%25222012%252F01%252F25%252000.54%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222012%252F01%252F12%252009.54%2522%255BEDAT%255D%29%29</link>
            <description>514 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2012/01/25PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5625439</comments>
            <pubDate>Wed, 25 Jan 2012 05:54:00 +0100</pubDate>
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        <item>
            <title>Cancer Control; +232 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5579451&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222012%252F01%252F06%252006.24%2522%255BMHDA%255D%253A%25222012%252F01%252F12%252009.54%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222012%252F01%252F06%252006.24%2522%255BEDAT%255D%29%29</link>
            <description>232 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2012/01/12PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579451</comments>
            <pubDate>Thu, 12 Jan 2012 14:54:02 +0100</pubDate>
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        <item>
            <title>Cancer Control; +161 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5563291&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F12%252F30%252016.36%2522%255BMHDA%255D%253A%25222012%252F01%252F06%252006.24%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F12%252F30%252016.36%2522%255BEDAT%255D%29%29</link>
            <description>161 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2012/01/06PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5563291</comments>
            <pubDate>Fri, 06 Jan 2012 11:24:02 +0100</pubDate>
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        <item>
            <title>Cancer Control; +199 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5551735&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F12%252F24%252015.48%2522%255BMHDA%255D%253A%25222011%252F12%252F30%252016.36%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F12%252F24%252015.48%2522%255BEDAT%255D%29%29</link>
            <description>199 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/12/30PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5551735</comments>
            <pubDate>Fri, 30 Dec 2011 21:36:01 +0100</pubDate>
            <guid isPermaLink="false">5551735</guid>        </item>
        <item>
            <title>Cancer Control; +314 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5537059&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F12%252F18%252018.00%2522%255BMHDA%255D%253A%25222011%252F12%252F24%252015.48%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F12%252F18%252018.00%2522%255BEDAT%255D%29%29</link>
            <description>314 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/12/24PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5537059</comments>
            <pubDate>Sat, 24 Dec 2011 20:48:02 +0100</pubDate>
            <guid isPermaLink="false">5537059</guid>        </item>
        <item>
            <title>Cancer Control; +1087 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5514848&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F11%252F25%252011.24%2522%255BMHDA%255D%253A%25222011%252F12%252F18%252018.00%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F11%252F25%252011.24%2522%255BEDAT%255D%29%29</link>
            <description>1087 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/12/18PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5514848</comments>
            <pubDate>Sun, 18 Dec 2011 23:00:01 +0100</pubDate>
            <guid isPermaLink="false">5514848</guid>        </item>
        <item>
            <title>Cancer Control; +192 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5443505&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F11%252F19%252015.12%2522%255BMHDA%255D%253A%25222011%252F11%252F25%252011.24%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F11%252F19%252015.12%2522%255BEDAT%255D%29%29</link>
            <description>192 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/11/25PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5443505</comments>
            <pubDate>Fri, 25 Nov 2011 16:24:02 +0100</pubDate>
            <guid isPermaLink="false">5443505</guid>        </item>
        <item>
            <title>Cancer Control; +483 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5424526&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F11%252F07%252012.25%2522%255BMHDA%255D%253A%25222011%252F11%252F19%252015.12%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F11%252F07%252012.25%2522%255BEDAT%255D%29%29</link>
            <description>483 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/11/19PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5424526</comments>
            <pubDate>Sat, 19 Nov 2011 20:12:01 +0100</pubDate>
            <guid isPermaLink="false">5424526</guid>        </item>
        <item>
            <title>Cancer Control; +870 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5382359&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F10%252F15%252001.29%2522%255BMHDA%255D%253A%25222011%252F11%252F07%252012.25%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F10%252F15%252001.29%2522%255BEDAT%255D%29%29</link>
            <description>870 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/11/07PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5382359</comments>
            <pubDate>Mon, 07 Nov 2011 17:25:02 +0100</pubDate>
            <guid isPermaLink="false">5382359</guid>        </item>
        <item>
            <title>Cancer Control; +383 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5314668&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F10%252F04%252020.49%2522%255BMHDA%255D%253A%25222011%252F10%252F15%252001.29%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F10%252F04%252020.49%2522%255BEDAT%255D%29%29</link>
            <description>383 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/10/15PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5314668</comments>
            <pubDate>Sat, 15 Oct 2011 05:29:52 +0100</pubDate>
            <guid isPermaLink="false">5314668</guid>        </item>
        <item>
            <title>Cancer Control; +342 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5282787&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F09%252F29%252019.14%2522%255BMHDA%255D%253A%25222011%252F10%252F04%252020.49%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F09%252F29%252019.14%2522%255BEDAT%255D%29%29</link>
            <description>342 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/10/04PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5282787</comments>
            <pubDate>Wed, 05 Oct 2011 00:49:55 +0100</pubDate>
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        <item>
            <title>Cancer Control; +620 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5264452&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F09%252F20%252003.31%2522%255BMHDA%255D%253A%25222011%252F09%252F29%252019.14%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F09%252F20%252003.31%2522%255BEDAT%255D%29%29</link>
            <description>620 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/09/29PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5264452</comments>
            <pubDate>Thu, 29 Sep 2011 23:14:56 +0100</pubDate>
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        <item>
            <title>Cancer Control; +152 new citations</title>
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            <description>152 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/09/20PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=5234527</comments>
            <pubDate>Tue, 20 Sep 2011 07:31:52 +0100</pubDate>
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            <title>Cancer Control; +420 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5220390&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F09%252F05%252014.08%2522%255BMHDA%255D%253A%25222011%252F09%252F15%252010.55%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F09%252F05%252014.08%2522%255BEDAT%255D%29%29</link>
            <description>420 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/09/15PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=5220390</comments>
            <pubDate>Thu, 15 Sep 2011 14:55:59 +0100</pubDate>
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            <title>Cancer Control; +120 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5195091&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F09%252F01%252011.12%2522%255BMHDA%255D%253A%25222011%252F09%252F05%252014.08%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F09%252F01%252011.12%2522%255BEDAT%255D%29%29</link>
            <description>120 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/09/05PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5195091</comments>
            <pubDate>Mon, 05 Sep 2011 18:08:02 +0100</pubDate>
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            <title>Cancer Control; +245 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5179848&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F08%252F26%252011.10%2522%255BMHDA%255D%253A%25222011%252F09%252F01%252011.12%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F08%252F26%252011.10%2522%255BEDAT%255D%29%29</link>
            <description>245 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/09/01PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=5179848</comments>
            <pubDate>Thu, 01 Sep 2011 15:12:02 +0100</pubDate>
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            <title>Cancer Control; +230 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5159964&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F08%252F20%252002.54%2522%255BMHDA%255D%253A%25222011%252F08%252F26%252011.10%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F08%252F20%252002.54%2522%255BEDAT%255D%29%29</link>
            <description>230 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/08/26PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=5159964</comments>
            <pubDate>Fri, 26 Aug 2011 15:10:12 +0100</pubDate>
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            <title>Cancer Control; +425 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5142243&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F08%252F09%252014.30%2522%255BMHDA%255D%253A%25222011%252F08%252F20%252002.54%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F08%252F09%252014.30%2522%255BEDAT%255D%29%29</link>
            <description>425 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/08/20PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5142243</comments>
            <pubDate>Sat, 20 Aug 2011 06:54:02 +0100</pubDate>
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            <title>Cancer Control; +594 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5108023&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F07%252F26%252007.45%2522%255BMHDA%255D%253A%25222011%252F08%252F09%252014.30%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F07%252F26%252007.45%2522%255BEDAT%255D%29%29</link>
            <description>594 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/08/09PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5108023</comments>
            <pubDate>Tue, 09 Aug 2011 18:30:02 +0100</pubDate>
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            <title>Cancer Control; +672 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5062926&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F07%252F11%252016.15%2522%255BMHDA%255D%253A%25222011%252F07%252F26%252007.45%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F07%252F11%252016.15%2522%255BEDAT%255D%29%29</link>
            <description>672 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/07/26PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5062926</comments>
            <pubDate>Tue, 26 Jul 2011 11:45:02 +0100</pubDate>
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            <title>Cancer Control; +610 new citations</title>
            <link>http://www.medworm.com/index.php?rid=5011668&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F06%252F26%252011.00%2522%255BMHDA%255D%253A%25222011%252F07%252F11%252016.15%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F06%252F26%252011.00%2522%255BEDAT%255D%29%29</link>
            <description>610 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/07/11PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5011668</comments>
            <pubDate>Mon, 11 Jul 2011 20:15:03 +0100</pubDate>
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            <title>Cancer Control; +609 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4966205&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F06%252F12%252002.30%2522%255BMHDA%255D%253A%25222011%252F06%252F26%252011.00%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F06%252F12%252002.30%2522%255BEDAT%255D%29%29</link>
            <description>609 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/06/26PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4966205</comments>
            <pubDate>Sun, 26 Jun 2011 15:00:02 +0100</pubDate>
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            <title>Cancer Control; +570 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4922095&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F05%252F28%252022.30%2522%255BMHDA%255D%253A%25222011%252F06%252F12%252002.30%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F05%252F28%252022.30%2522%255BEDAT%255D%29%29</link>
            <description>570 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/06/12PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4922095</comments>
            <pubDate>Sun, 12 Jun 2011 06:30:02 +0100</pubDate>
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        <item>
            <title>Cancer Control; +633 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4875900&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F05%252F14%252009.45%2522%255BMHDA%255D%253A%25222011%252F05%252F28%252022.30%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F05%252F14%252009.45%2522%255BEDAT%255D%29%29</link>
            <description>633 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/05/28PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4875900</comments>
            <pubDate>Sun, 29 May 2011 02:30:05 +0100</pubDate>
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            <title>Cancer Control; +1910 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4821382&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222011%252F03%252F31%252018.45%2522%255BMHDA%255D%253A%25222011%252F05%252F14%252009.45%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222011%252F03%252F31%252018.45%2522%255BEDAT%255D%29%29</link>
            <description>1910 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/05/14PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4821382</comments>
            <pubDate>Sat, 14 May 2011 13:45:02 +0100</pubDate>
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        <item>
            <title>Cancer Control; +655 new citations</title>
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            <description>655 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/03/31PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
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            <title>Cancer Control; +672 new citations</title>
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            <description>672 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/03/17PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
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            <description>604 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/03/02PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=4540051</comments>
            <pubDate>Thu, 03 Mar 2011 04:45:02 +0100</pubDate>
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            <description>765 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/02/16PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=4483489</comments>
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            <title>Cancer Control; +514 new citations</title>
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            <description>514 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/01/31PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
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        <comments>http://www.medworm.com/rss/comments.php?id=4414962</comments>
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            <title>Cancer Control; +1453 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4353148&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F12%252F14%252009.00%2522%255BMHDA%255D%253A%25222011%252F01%252F16%252008.45%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F12%252F14%252009.00%2522%255BEDAT%255D%29%29</link>
            <description>1453 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2011/01/16PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=4353148</comments>
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            <title>Cancer Control; +536 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4255957&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F12%252F03%252003.00%2522%255BMHDA%255D%253A%25222010%252F12%252F14%252009.00%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F12%252F03%252003.00%2522%255BEDAT%255D%29%29</link>
            <description>536 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/12/14PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4255957</comments>
            <pubDate>Tue, 14 Dec 2010 14:00:06 +0100</pubDate>
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            <title>Cancer Control; +138 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4224000&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F11%252F27%252002.30%2522%255BMHDA%255D%253A%25222010%252F12%252F03%252003.00%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F11%252F27%252002.30%2522%255BEDAT%255D%29%29</link>
            <description>138 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/12/03PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4224000</comments>
            <pubDate>Fri, 03 Dec 2010 08:00:04 +0100</pubDate>
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            <title>Cancer Control; +350 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4204580&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F11%252F16%252018.35%2522%255BMHDA%255D%253A%25222010%252F11%252F27%252002.30%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F11%252F16%252018.35%2522%255BEDAT%255D%29%29</link>
            <description>350 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/11/27PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4204580</comments>
            <pubDate>Sat, 27 Nov 2010 07:30:03 +0100</pubDate>
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            <title>Cancer Control; +318 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4171937&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F11%252F07%252010.25%2522%255BMHDA%255D%253A%25222010%252F11%252F16%252018.35%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F11%252F07%252010.25%2522%255BEDAT%255D%29%29</link>
            <description>318 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/11/16PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4171937</comments>
            <pubDate>Tue, 16 Nov 2010 23:35:04 +0100</pubDate>
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            <title>Cancer Control; +310 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4140446&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F11%252F02%252018.55%2522%255BMHDA%255D%253A%25222010%252F11%252F07%252010.25%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F11%252F02%252018.55%2522%255BEDAT%255D%29%29</link>
            <description>310 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/11/07PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4140446</comments>
            <pubDate>Sun, 07 Nov 2010 15:25:04 +0100</pubDate>
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            <title>Cancer Control; +131 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4125800&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F10%252F29%252002.35%2522%255BMHDA%255D%253A%25222010%252F11%252F02%252018.55%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F10%252F29%252002.35%2522%255BEDAT%255D%29%29</link>
            <description>131 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/11/02PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4125800</comments>
            <pubDate>Tue, 02 Nov 2010 22:55:02 +0100</pubDate>
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            <title>Cancer Control; +177 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4108741&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F10%252F24%252010.05%2522%255BMHDA%255D%253A%25222010%252F10%252F29%252002.35%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F10%252F24%252010.05%2522%255BEDAT%255D%29%29</link>
            <description>177 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/10/29PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
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        <comments>http://www.medworm.com/rss/comments.php?id=4108741</comments>
            <pubDate>Fri, 29 Oct 2010 06:35:02 +0100</pubDate>
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        <item>
            <title>Cancer Control; +406 new citations</title>
            <link>http://www.medworm.com/index.php?rid=4097661&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%26debug%3Dpubmeddembup%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F10%252F15%252000.30%2522%255BMHDA%255D%253A%25222010%252F10%252F24%252010.05%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F10%252F15%252000.30%2522%255BEDAT%255D%29%29</link>
            <description>406 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/10/24PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=4097661</comments>
            <pubDate>Sun, 24 Oct 2010 14:05:03 +0100</pubDate>
            <guid isPermaLink="false">4097661</guid>        </item>
        <item>
            <title>[Biological significance of naturally occurring deuterium: the antitumor effect of deuterium depletion.]</title>
            <link>http://www.medworm.com/index.php?rid=3913334&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20739263%26dopt%3DAbstract</link>
            <description>Authors: Somlyai G, MolnÃ¡r M, Laskay G, SzabÃ³ M, BerkÃ©nyi T, Guller I, KovÃ¡cs A
    The concentration of deuterium is about 150 ppm (over 16 mmol/L) in surface water and more than 10 mmol/L in living organisms. Experiments with deuterium depleted water (30+/-5 ppm) revealed that due to D-depletion various tumorous cell lines (PC-3, human prostate, MDA, human breast, HT-29, human colon, M14, human melanoma) required longer time to multiply in vitro . DDW caused tumor regression in xenotransplanted mice (MDA and MCF-7, human breast, PC-3) and induced apoptosis in vitro and in vivo . Deuterium depleted water (25+/-5 ppm) induced complete or partial tumor regression in dogs and cats with spontaneous malignancies, it was registered as anticancer for veterinary use in 1999 (Vetera-DD...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913334</comments>
            <pubDate>Sun, 29 Aug 2010 12:32:24 +0100</pubDate>
            <guid isPermaLink="false">3913334</guid>        </item>
        <item>
            <title>Locoregional Recurrence After Sentinel Lymph Node Dissection With or Without Axillary Dissection in Patients With Sentinel Lymph Node Metastases: The American College of Surgeons Oncology Group Z0011 Randomized Trial.</title>
            <link>http://www.medworm.com/index.php?rid=3913333&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20739842%26dopt%3DAbstract</link>
            <description>CONCLUSIONS:: Despite the potential for residual axillary disease after SLND, SLND without ALND can offer excellent regional control and may be reasonable management for selected patients with early-stage breast cancer treated with breast-conserving therapy and adjuvant systemic therapy.
    PMID: 20739842 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913333</comments>
            <pubDate>Sun, 29 Aug 2010 12:32:15 +0100</pubDate>
            <guid isPermaLink="false">3913333</guid>        </item>
        <item>
            <title>Clinicopathologic features and prognostic factors in alpha-fetoprotein-producing gastric cancers: Analysis of 104 cases.</title>
            <link>http://www.medworm.com/index.php?rid=3913330&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20740583%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: AFP-positive gastric cancer had more aggressive behavior than that of AFP-negative gastric cancer. In addition to surgery, multimodal therapy should be considered. J. Surg. Oncol. 2010;102:249-255. (c) 2010 Wiley-Liss, Inc.
    PMID: 20740583 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913330</comments>
            <pubDate>Sun, 29 Aug 2010 12:31:48 +0100</pubDate>
            <guid isPermaLink="false">3913330</guid>        </item>
        <item>
            <title>Clinical pharmacology of isoflavones and its relevance for potential prevention of prostate cancer.</title>
            <link>http://www.medworm.com/index.php?rid=3913328&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20796219%26dopt%3DAbstract</link>
            <description>Authors: de Souza PL, Russell PJ, Kearsley JH, Howes LG
    Isoflavones are phytoestrogens that have pleiotropic effects in a wide variety of cancer cell lines. Many of these biological effects involve key components of signal transduction pathways within cancer cells, including prostate cancer cells. Epidemiological studies have raised the hypothesis that isoflavones may play an important role in the prevention and modulation of prostate cancer growth. Since randomized phase III trials of isoflavones in prostate cancer prevention are currently lacking, the best evidence for this concept is presently provided by case control studies. However, in vitro data are much more convincing in regard to the activity of a number of isoflavones, and have led to the development of genistein and phenoxo...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913328</comments>
            <pubDate>Sun, 29 Aug 2010 12:31:35 +0100</pubDate>
            <guid isPermaLink="false">3913328</guid>        </item>
        <item>
            <title>Return-to-work interventions integrated into cancer care: a systematic review.</title>
            <link>http://www.medworm.com/index.php?rid=3913326&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798030%26dopt%3DAbstract</link>
            <description>Conclusions Only few interventions are primarily aimed at enhancing return to work in patients with cancer and most do not fit the shared care model involving integrated cancer care. Future studies should be developed with well-structured work-directed components that should be evaluated in randomised controlled trials.
    PMID: 20798030 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913326</comments>
            <pubDate>Sun, 29 Aug 2010 12:31:21 +0100</pubDate>
            <guid isPermaLink="false">3913326</guid>        </item>
        <item>
            <title>Lgl/aPKC and Crb regulate the salvador/warts/hippo pathway.</title>
            <link>http://www.medworm.com/index.php?rid=3913321&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798605%26dopt%3DAbstract</link>
            <description>Authors: Parsons LM, Grzeschik NA, Allott ML, Richardson HE
    A key goal of developmental biology is to understand the mechanisms that coordinate organ growth. It has long been recognized that the genes that control apico-basal cell polarity also regulate tissue growth. How loss of cell polarity contributes to tissue overgrowth has been the subject of much speculation. Do loss-of-function mutations in cell polarity regulators result in secondary effects that globally deregulate cell proliferation, or do these genes specifically control growth pathways? Three recent papers have shown that the apico-basal polarity determinants Lgl/aPKC and Crb regulate tissue growth independently of their roles in cell polarity and coordinately regulate cell proliferation and cell death via the Salvador/Wa...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913321</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:37 +0100</pubDate>
            <guid isPermaLink="false">3913321</guid>        </item>
        <item>
            <title>Supporting informed decision making online in 20 minutes: an observational web-log study of a PSA test decision aid.</title>
            <link>http://www.medworm.com/index.php?rid=3913335&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20507844%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Increased usage of Prosdex leads to more informed decision making, the key aim of the UK Prostate Cancer Risk Management Programme. However, developers realistically have roughly 20 minutes to provide useful information that will support informed decision making when the patient uses a web-based interface. Future decision aids need to be developed with this limitation in mind. We recommend that web-log analysis should be an integral part of online decision aid development and analysis. TRIAL REGISTRATION: ISRCTN48473735; http://www.controlled-trials.com/ISRCTN48473735 (Archived by WebCite at http://www.webcitation.org/5pqeF89tS).
    PMID: 20507844 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913335</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:05 +0100</pubDate>
            <guid isPermaLink="false">3913335</guid>        </item>
        <item>
            <title>Risk of proximal colonic neoplasms in asymptomatic adults older than 50 years found to have distal hyperplastic polyps on routine colorectal cancer screening.</title>
            <link>http://www.medworm.com/index.php?rid=3913332&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20740111%26dopt%3DAbstract</link>
            <description>Conclusion: Routinely recommending a colonoscopy for every patient with distal HPs found only by screening flexible sigmoidoscopy is neither justified nor necessary. Nevertheless, further investigation (ie, colonoscopy) may be warranted in the aforementioned subgroups.
    PMID: 20740111 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913332</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:05 +0100</pubDate>
            <guid isPermaLink="false">3913332</guid>        </item>
        <item>
            <title>Gene Expression-Based Approaches in Differentiation of Metastases and Second Primary Tumour.</title>
            <link>http://www.medworm.com/index.php?rid=3913331&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20740207%26dopt%3DAbstract</link>
            <description>Authors: Vooder T, VÃ¤lk K, Kolde R, Roosipuu R, Vilo J, Metspalu A
    A 64-year-old male patient was diagnosed with 3 consecutive non-small cell lung carcinomas (NSCLC). In the current study, we applied whole-genome gene expression analysis to control, primary and locally recurrent cancer, and supposed metastasis samples of a single patient. According to our knowledge, there are no published papers describing the gene expression profiles of a single patient's squamous cell lung cancers. As the histology and differentiation grade of the primary cancer and the supposed metastasis differed minimally, but local recurrence was poorly differentiated, molecular profiling of the samples was carried out in order to confirm or reject the hypothesis of second primary cancer. Principal component a...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913331</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:05 +0100</pubDate>
            <guid isPermaLink="false">3913331</guid>        </item>
        <item>
            <title>Principles of memory CD8 T-cells generation in relation to protective immunity.</title>
            <link>http://www.medworm.com/index.php?rid=3913329&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20795544%26dopt%3DAbstract</link>
            <description>Authors: Zanetti M, Castiglioni P, Ingulli E
    Memory T-cell responses are of vital importance in understanding the host's response against pathogens and cancer cells and to begin establishing the correlation of protection against disease. In this review, we discuss our own data in the general context of current knowledge to sketch tentative working principles for the induction of protective T-cell responses by vaccination. We draw attention to quantitative and qualitative aspects of the initial contact with antigen, as well as to the kinetics of events leading to the generation of memory T cells thereafter. Our arguments are based on the current distinction of memory T cells into two lineages: effector memory T cells (T(EM)) and central memory T cells (T(CM)). Our provisional conclusion...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913329</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:05 +0100</pubDate>
            <guid isPermaLink="false">3913329</guid>        </item>
        <item>
            <title>Phosphoglycerate Kinase 1 Promoting Tumor Progression and Metastasis in Gastric Cancer - Detected in a Tumor Mouse Model Using Positron Emission Tomography/Magnetic Resonance Imaging.</title>
            <link>http://www.medworm.com/index.php?rid=3913324&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798498%26dopt%3DAbstract</link>
            <description>Conclusion: Consistent in-vivo findings suggest that PGK1 might be crucially involved in gastric malignancy regarding growth and metastasis, which was also underlined by novel imaging techniques. Thus, PGK1 may be exploited as a prognostic marker and/or be of potential therapeutic value preventing malignant dissemination.
    PMID: 20798498 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913324</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:04 +0100</pubDate>
            <guid isPermaLink="false">3913324</guid>        </item>
        <item>
            <title>Circadian Expression of Clock- and Tumor Suppressor Genes in Human Oral Mucosa.</title>
            <link>http://www.medworm.com/index.php?rid=3913323&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798499%26dopt%3DAbstract</link>
            <description>Conclusion: Our study revealed a proper circadian regulation profile of several clock- and tumor suppressor genes at defined points in time in the participants studied. These findings could provide important information regarding genes displaying the same expression profile in the gastrointestinal tract amounting to a physiological expression profile of healthy humans. In the future asynchronous regulations of those genes might be an additional assistant method to detect derivations distinguishing normal from malignant tissue or assessing risk factors for cancer.
    PMID: 20798499 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913323</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:04 +0100</pubDate>
            <guid isPermaLink="false">3913323</guid>        </item>
        <item>
            <title>Genes Determining the Course of Virus Persistence in the Liver: Lessons from Murine Infection with Lymphocytic Choriomeningitis Virus.</title>
            <link>http://www.medworm.com/index.php?rid=3913322&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798510%26dopt%3DAbstract</link>
            <description>Authors: Lang PA, Recher M, HÃ¤ussinger D, Lang KS
    More than 500 million people worldwide are persistently infected with either hepatitis B virus (HBV) or hepatitis C virus (HCV). Although both viruses are poorly cytopathic, persistent infection causes severe immunopathologic damage to liver tissue; histologically, such damage is characterized by fatty liver disease, liver fibrosis, and a higher likelihood of hepatocellular carcinoma. Virus-specific CD8(+) T cells play a crucial role during infection with hepatitis viruses. On the one hand, rapid activation of CD8(+) T cells can control the virus and therefore inhibit its persistence. On the other hand, once the virus persists in the liver, the chronic activation of virus-specific T cells leads to continued liver cell damage. This do...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913322</comments>
            <pubDate>Sun, 29 Aug 2010 12:30:04 +0100</pubDate>
            <guid isPermaLink="false">3913322</guid>        </item>
        <item>
            <title>Phytochemicals: cancer chemoprevention and suppression of tumor onset and metastasis.</title>
            <link>http://www.medworm.com/index.php?rid=3913318&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798979%26dopt%3DAbstract</link>
            <description>Authors: Shu L, Cheung KL, Khor TO, Chen C, Kong AN
    Carcinogenesis is a multi-step process which could be prevented by phytochemicals. Phytochemicals from dietary plants and other plant sources such as herbs are becoming increasingly important sources of anticancer drugs or compounds for cancer chemoprevention or adjuvant chemotherapy. Phytochemicals can prevent cancer initiation, promotion, and progression by exerting anti-inflammatory and anti-oxidative stress effects which are mediated by integrated Nrf2, NF-kappaB, and AP-1 signaling pathways. In addition, phytochemicals from herbal medicinal plants and/or some dietary plants developed in recent years have been shown to induce apoptosis in cancer cells and inhibition of tumor growth in vivo. In advanced tumors, a series of changes ...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913318</comments>
            <pubDate>Fri, 27 Aug 2010 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3913318</guid>        </item>
        <item>
            <title>Combined Functional Genomic and Proteomic Approaches Identify a PP2A Complex as a Negative Regulator of Hippo Signaling.</title>
            <link>http://www.medworm.com/index.php?rid=3913327&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20797625%26dopt%3DAbstract</link>
            <description>Authors: Ribeiro PS, JosuÃ© F, Wepf A, Wehr MC, Rinner O, Kelly G, Tapon N, Gstaiger M
    The Hippo (Hpo) pathway is a central determinant of tissue size in both Drosophila and higher organisms. The core of the pathway is a kinase cascade composed of an upstream kinase Hpo (MST1/2 in mammals) and a downstream kinase Warts (Wts, Lats1/2 in mammals), as well as several scaffold proteins, Sav, dRASSF, and Mats. Activation of the core kinase cassette results in phosphorylation and inactivation of the progrowth transcriptional coactivator Yki, leading to increased apoptosis and reduced tissue growth. The mechanisms that prevent inappropriate Hpo activation remain unclear, and in particular, the identity of the phosphatase that antagonizes Hpo is unknown. Using combined proteomic and RNAi scr...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913327</comments>
            <pubDate>Thu, 26 Aug 2010 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3913327</guid>        </item>
        <item>
            <title>Testicular cancer risk associated with occupational radiation exposure: a systematic literature review.</title>
            <link>http://www.medworm.com/index.php?rid=3913325&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798474%26dopt%3DAbstract</link>
            <description>Authors: Yousif L, Blettner M, Hammer GP, Zeeb H
    Testicular cancer is a rare disease, affecting mainly young men aged 15-49. There have been some recent reports that it might be associated with radiation exposure. We have systematically reviewed this topic. English-language articles published between 1990 and 2008 studying the relationship between occupational radiation exposure and testicular cancer were included. Risk of bias was assessed using a modified version of the EPHPP checklist. For ionising radiation we subdivided study populations into occupational groups. No pooled analysis was performed due to the heterogeneity of studies. Seven case-control and 30 cohort studies were included in the review. For radiation workers, one incidence study showed a significant increase and four...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913325</comments>
            <pubDate>Thu, 26 Aug 2010 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3913325</guid>        </item>
        <item>
            <title>Sphingosine-1-phosphate phosphohydrolase-1 regulates ER stress-induced autophagy.</title>
            <link>http://www.medworm.com/index.php?rid=3913320&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798685%26dopt%3DAbstract</link>
            <description>In this study, we report that depletion of sphingosine-1-phosphate phosphohydrolase-1 (SPP1), an endoplasmic reticulum (ER)-resident enzyme that specifically dephosphorylates S1P, induced autophagy. Although the mammalian target of rapamycin and class III phosphoinositide 3-kinase/Beclin-1 pathways were not involved and this autophagy was p53 independent, C/EBP homologous protein, BiP, and phospho-eucaryotic translation initiation factor-2alpha, and cleavage of procaspases 2 and 4, downstream targets of ER stress, were increased after SPP1 depletion. Autophagy was suppressed by depletion of protein kinase regulated by RNA-like ER kinase (PERK), inositol-requiring transmembrane kinase/endonuclease-1alpha, or activating transcription factor 6, three sensors of the unfolded protein response (...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913320</comments>
            <pubDate>Thu, 26 Aug 2010 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3913320</guid>        </item>
        <item>
            <title>A positive role for c-Abl in Atm and Atr activation in DNA damage response.</title>
            <link>http://www.medworm.com/index.php?rid=3913319&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20798688%26dopt%3DAbstract</link>
            <description>Authors: Wang X, Zeng L, Wang J, Chau JF, Lai KP, Jia D, Poonepalli A, Hande MP, Liu H, He G, He L, Li B
    DNA damage triggers Atm- and/or Atr-dependent signaling pathways to control cell cycle progression, apoptosis, and DNA repair. However, how Atm and Atr are activated is not fully understood. One of the downstream targets of Atm is non-receptor tyrosine kinase c-Abl, which is phosphorylated and activated by Atm. The current view is that c-Abl relays pro-apoptotic signals from Atm to p73 and p53. Here we show that c-Abl deficiency resulted in a broad spectrum of defects in cell response to genotoxic stress, including activation of Chk1 and Chk2, activation of p53, nuclear foci formation, apoptosis, and DNA repair, suggesting that c-Abl might also act upstream of the DNA damage-activat...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3913319</comments>
            <pubDate>Thu, 26 Aug 2010 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">3913319</guid>        </item>
        <item>
            <title>Cancer Control; +423 new citations</title>
            <link>http://www.medworm.com/index.php?rid=3903572&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F08%252F17%252023.27%2522%255BMHDA%255D%253A%25222010%252F08%252F26%252011.42%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F08%252F17%252023.27%2522%255BEDAT%255D%29%29</link>
            <description>423 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/08/26PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3903572</comments>
            <pubDate>Thu, 26 Aug 2010 15:42:02 +0100</pubDate>
            <guid isPermaLink="false">3903572</guid>        </item>
        <item>
            <title>Cancer Control; +177 new citations</title>
            <link>http://www.medworm.com/index.php?rid=3873501&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F08%252F12%252006.33%2522%255BMHDA%255D%253A%25222010%252F08%252F17%252023.27%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F08%252F12%252006.33%2522%255BEDAT%255D%29%29</link>
            <description>177 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/08/17PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3873501</comments>
            <pubDate>Wed, 18 Aug 2010 03:27:02 +0100</pubDate>
            <guid isPermaLink="false">3873501</guid>        </item>
        <item>
            <title>Cancer Control; +183 new citations</title>
            <link>http://www.medworm.com/index.php?rid=3858330&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F08%252F06%252011.12%2522%255BMHDA%255D%253A%25222010%252F08%252F12%252006.33%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F08%252F06%252011.12%2522%255BEDAT%255D%29%29</link>
            <description>183 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/08/12PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3858330</comments>
            <pubDate>Thu, 12 Aug 2010 10:33:08 +0100</pubDate>
            <guid isPermaLink="false">3858330</guid>        </item>
        <item>
            <title>Cancer Control; +170 new citations</title>
            <link>http://www.medworm.com/index.php?rid=3827723&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F08%252F03%252015.33%2522%255BMHDA%255D%253A%25222010%252F08%252F06%252011.12%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F08%252F03%252015.33%2522%255BEDAT%255D%29%29</link>
            <description>170 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/08/06PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3827723</comments>
            <pubDate>Fri, 06 Aug 2010 15:12:02 +0100</pubDate>
            <guid isPermaLink="false">3827723</guid>        </item>
        <item>
            <title>Cancer Control; +390 new citations</title>
            <link>http://www.medworm.com/index.php?rid=3813683&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222010%252F07%252F23%252009.42%2522%255BMHDA%255D%253A%25222010%252F08%252F03%252015.33%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222010%252F07%252F23%252009.42%2522%255BEDAT%255D%29%29</link>
            <description>390 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2010/08/03PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3813683</comments>
            <pubDate>Tue, 03 Aug 2010 19:33:02 +0100</pubDate>
            <guid isPermaLink="false">3813683</guid>        </item>
        <item>
            <title>Checkpoint bypass and cell viability.</title>
            <link>http://www.medworm.com/index.php?rid=3610797&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20505362%26dopt%3DAbstract</link>
            <description>We present evidence that the inherent survival of damaged human cells can be surprisingly independent of checkpoint control.
    PMID: 20505362 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3610797</comments>
            <pubDate>Sun, 30 May 2010 07:42:11 +0100</pubDate>
            <guid isPermaLink="false">3610797</guid>        </item>
        <item>
            <title>Therapy for minimal residual tumor disease: beta-galactosylceramide inhibits the growth of recurrent HPV16-associated neoplasms after surgery and chemotherapy.</title>
            <link>http://www.medworm.com/index.php?rid=3542957&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19739073%26dopt%3DAbstract</link>
            <description>This study was focused on the tumor-inhibitory effects of 12 carbon acyl chain beta-galactosyl ceramide (C12 beta-D-Galactosyl Ceramide; beta-GalCer(C12)) on the growth of human papillomavirus type 16 (HPV16)-associated neoplasms transplanted in syngeneic mice. Treatment of tumor-bearing mice with beta-GalCer(C12) 3-14 days after tumor cell transplantation significantly inhibited the growth of the major histocompatibility complex (MHC) Class I-positive (TC-1), as well as MHC Class I-deficient (TC-1/A9) HPV16-associated tumors. Moreover, administration of beta-GalCer(C12) after surgical removal of TC-1 tumors inhibited the growth of tumor recurrences. Similar results were obtained in the treatment of tumors after chemotherapy. beta-GalCer(C12) treatment turned out to be also synergistic wit...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542957</comments>
            <pubDate>Fri, 07 May 2010 17:10:44 +0100</pubDate>
            <guid isPermaLink="false">3542957</guid>        </item>
        <item>
            <title>CDK8 expression in 470 colorectal cancers in relation to beta-catenin activation, other molecular alterations and patient survival.</title>
            <link>http://www.medworm.com/index.php?rid=3542956&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19790197%26dopt%3DAbstract</link>
            <description>Authors: Firestein R, Shima K, Nosho K, Irahara N, Baba Y, Bojarski E, Giovannucci EL, Hahn WC, Fuchs CS, Ogino S
    Alterations in the Wnt/beta-catenin pathway define a key event in the pathogenesis of colon cancer. We have recently shown that CDK8, the gene encoding a cyclin-dependent kinase (CDK) component of the Mediator complex, acts as a colon cancer oncogene that is necessary for beta-catenin activity. Here, we tested the hypothesis that colorectal cancers with CDK8 expression have distinct clinical, prognostic and molecular attributes. Among 470 colorectal cancers identified in 2 prospective cohort studies, CDK8 expression was detected in 329 (70%) tumors by immunohistochemistry. Cox proportional hazards model and backward stepwise elimination were used to compute hazard ratio (HR...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542956</comments>
            <pubDate>Fri, 07 May 2010 17:10:41 +0100</pubDate>
            <guid isPermaLink="false">3542956</guid>        </item>
        <item>
            <title>Fetal cell microchimerism in papillary thyroid cancer: studies in peripheral blood and tissues.</title>
            <link>http://www.medworm.com/index.php?rid=3542955&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19856309%26dopt%3DAbstract</link>
            <description>In conclusion, the prevalence of FCM in peripheral blood was found to be significantly lower in patients than in healthy controls. The presence of microchimeric cells in the tumors, but not at the peripheral level, supports the hypothesis that fetal cells could reside in maternal niches and could be recruited to diseased areas, where they could differentiate to regenerate damaged tissues.
    PMID: 19856309 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542955</comments>
            <pubDate>Fri, 07 May 2010 17:10:38 +0100</pubDate>
            <guid isPermaLink="false">3542955</guid>        </item>
        <item>
            <title>Low-risk variants FGFR2, TNRC9 and LSP1 in German familial breast cancer patients.</title>
            <link>http://www.medworm.com/index.php?rid=3542954&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19856316%26dopt%3DAbstract</link>
            <description>Authors: Hemminki K, MÃ¼ller-Myhsok B, Lichtner P, Engel C, Chen B, Burwinkel B, FÃ¶rsti A, Sutter C, Wappenschmidt B, Hellebrand H, Illig T, Arnold N, Niederacher D, Dworniczak B, Deissler H, Kast K, Gadzicki D, Meitinger T, Wichmann HE, Kiechle M, Bartram CR, Schmutzler RK, Meindl A
    To validate common low-risk variants predisposing for breast cancer (BC) in a large set of BRCA1/2 negative familial or genetically enriched cases from Germany, we genotyped 1,415 cases and 1,830 healthy women by MALDI-TOF in 105 candidate SNPs. Significantly higher ORs than previously reported for heterozygous unselected cases were found for the minor allele in FGFR2 (OR = 1.43, 95% CI 1.30-1.59, p-value = 1.24 x 10(-12)) and for TNRC9 (OR = 1.33, 95% CI 1.19-1.46, p-value = 1.54 x 10(-7)). Most intr...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542954</comments>
            <pubDate>Fri, 07 May 2010 17:10:35 +0100</pubDate>
            <guid isPermaLink="false">3542954</guid>        </item>
        <item>
            <title>CHEK2 mutations and HNPCC-related colorectal cancer.</title>
            <link>http://www.medworm.com/index.php?rid=3542953&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19876921%26dopt%3DAbstract</link>
            <description>Authors: Suchy J, Cybulski C, WokoÅorczyk D, Oszurek O, GÃ³rski B, Debniak T, Jakubowska A, Gronwald J, Huzarski T, Byrski T, Dziuba I, Gogacz M, WiÅniowski R, Wandzel P, Banaszkiewicz Z, Kurzawski G, KÅadny J, Narod SA, LubiÅski J
    Recently, the 1100delC variant of cell cycle checkpoint kinase 2 (CHEK2) has been reported to confer a colorectal cancer risk in hereditary non-polyposis-colorectal cancer (HNPCC) and HNPCC-related families in the Netherlands. To investigate whether CHEK2 mutations confer increased cancer risk in HNPCC and HNPCC-related families in Poland, we genotyped 463 probands from HNPCC and HNPCC-related families, and 5,496 controls for 4 CHEK2 alleles (1100delC, IVS2+1G&amp;gt;A, del5395, I157T). All 463 probands were screened for mutations in the HNPCC-rela...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542953</comments>
            <pubDate>Fri, 07 May 2010 17:10:31 +0100</pubDate>
            <guid isPermaLink="false">3542953</guid>        </item>
        <item>
            <title>Progress and challenges in colorectal cancer screening and surveillance.</title>
            <link>http://www.medworm.com/index.php?rid=3542952&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20167216%26dopt%3DAbstract</link>
            <description>Authors: Lieberman D
    Colorectal cancer is a leading cause of cancer death throughout the world. There is evidence that screening of average-risk individuals can result in mortality reduction with early cancer detection and cancer prevention by detection and removal of cancer precursor lesions. The optimal form of screening is not clear. Fecal screening tests can be performed at home at low initial cost, but current versions lack high sensitivity for cancer precursor lesions, and tests need to be repeated at regular intervals. Adherence to repeat testing for negative tests and referral for colonoscopy for positive tests are important elements of program effectiveness. Structural examinations of the colon are more invasive and may result in detection of both early cancer and cancer precu...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542952</comments>
            <pubDate>Fri, 07 May 2010 17:10:28 +0100</pubDate>
            <guid isPermaLink="false">3542952</guid>        </item>
        <item>
            <title>Colon cancer: an update and future directions.</title>
            <link>http://www.medworm.com/index.php?rid=3542951&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20226784%26dopt%3DAbstract</link>
            <description>Authors: Yang VW, Lewis J, Wang TC, Rustgi AK
    
    PMID: 20226784 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542951</comments>
            <pubDate>Fri, 07 May 2010 17:10:26 +0100</pubDate>
            <guid isPermaLink="false">3542951</guid>        </item>
        <item>
            <title>Primary prevention of colorectal cancer.</title>
            <link>http://www.medworm.com/index.php?rid=3542950&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20420944%26dopt%3DAbstract</link>
            <description>Authors: Chan AT, Giovannucci EL
    Colorectal cancer has been strongly associated with a Western lifestyle. In the past several decades, much has been learned about the dietary, lifestyle, and medication risk factors for this malignancy. Although there is controversy about the role of specific nutritional factors, consideration of dietary pattern as a whole appears useful for formulating recommendations. For example, several studies have shown that high intake of red and processed meats, highly refined grains and starches, and sugars is related to increased risk of colorectal cancer. Replacing these factors with poultry, fish, and plant sources as the primary source of protein; unsaturated fats as the primary source of fat; and unrefined grains, legumes and fruits as the primary source o...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542950</comments>
            <pubDate>Fri, 07 May 2010 17:10:23 +0100</pubDate>
            <guid isPermaLink="false">3542950</guid>        </item>
        <item>
            <title>Hereditary and familial colon cancer.</title>
            <link>http://www.medworm.com/index.php?rid=3542949&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20420945%26dopt%3DAbstract</link>
            <description>Authors: Jasperson KW, Tuohy TM, Neklason DW, Burt RW
    Between 2% to 5% of all colon cancers arise in the setting of well-defined inherited syndromes, including Lynch syndrome, familial adenomatous polyposis, MUTYH-associated polyposis, and certain hamartomatous polyposis conditions. Each is associated with a high risk of colon cancer. In addition to the syndromes, up to one-third of colon cancers exhibit increased familial risk, likely related to inheritance. A number of less penetrant, but possibly more frequent susceptibility genes have been identified for this level of inheritance. Clarification of predisposing genes allows for accurate risk assessment and more precise screening approaches. This review examines the colon cancer syndromes, their genetics and management, and also the ...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542949</comments>
            <pubDate>Fri, 07 May 2010 17:10:20 +0100</pubDate>
            <guid isPermaLink="false">3542949</guid>        </item>
        <item>
            <title>Role of the serrated pathway in colorectal cancer pathogenesis.</title>
            <link>http://www.medworm.com/index.php?rid=3542948&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20420948%26dopt%3DAbstract</link>
            <description>Authors: Leggett B, Whitehall V
    The &quot;serrated neoplastic pathway&quot; describes the progression of serrated polyps, including sessile serrated adenomas and traditional serrated adenomas, to colorectal cancer. The recognition of this pathway during the last 15 years has led to a paradigm shift in our understanding of the molecular basis of colorectal cancer and significant changes in clinical practice. These findings are particularly relevant to prevention of interval cancers through colonoscopy surveillance programs-an important issue for colonoscopists. In the past, all serrated polyps were classified simply as hyperplastic polyps and were considered to have no malignant potential. Reappraisal of this view was largely driven by increasing recognition of the malignant potential of hyperpla...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542948</comments>
            <pubDate>Fri, 07 May 2010 17:10:17 +0100</pubDate>
            <guid isPermaLink="false">3542948</guid>        </item>
        <item>
            <title>Inflammation and colon cancer.</title>
            <link>http://www.medworm.com/index.php?rid=3542947&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20420949%26dopt%3DAbstract</link>
            <description>Authors: TerziÄ J, Grivennikov S, Karin E, Karin M
    The connection between inflammation and tumorigenesis is well-established and in the last decade has received a great deal of supporting evidence from genetic, pharmacological, and epidemiological data. Inflammatory bowel disease is an important risk factor for the development of colon cancer. Inflammation is also likely to be involved with other forms of sporadic as well as heritable colon cancer. The molecular mechanisms by which inflammation promotes cancer development are still being uncovered and could differ between colitis-associated and other forms of colorectal cancer. Recent work has elucidated the role of distinct immune cells, cytokines, and other immune mediators in virtually all steps of colon tumorigenesis, including ...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542947</comments>
            <pubDate>Fri, 07 May 2010 17:10:14 +0100</pubDate>
            <guid isPermaLink="false">3542947</guid>        </item>
        <item>
            <title>The synthesis and characterization of cellular membrane affinity chromatography columns for the study of human multidrug resistant proteins MRP1, MRP2 and human breast cancer resistant protein BCRP using membranes obtained from Spodoptera frugiperda (Sf9) insect cells.</title>
            <link>http://www.medworm.com/index.php?rid=3542946&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20441926%26dopt%3DAbstract</link>
            <description>Authors: Bhatia PA, Moaddel R, Wainer IW
    CMAC (cellular membrane affinity chromatography columns) have been developed for the study of the human multidrug transporters MRP1, MRP2 and the breast cancer resistance protein (BCRP). The columns were constructed using the immobilized artificial membrane (IAM) stationary phase and cellular membrane fragments obtained from Spodoptera frugiperda (Sf9) cells that had been stably transfected with human Mrp1, Mrp2 or Bcrp cDNA, using a baculovirus expression system. The resulting CMAC(Sf9(MRP1)), CMAC(Sf9(MRP2)) and CMAC(Sf9(BCRP)) columns and a control column produced using membrane fragments from non-transfected Sf9 cells, CMAC(Sf9), were characterized using frontal affinity chromatography using [(3)H]-etoposide as the marker ligand and etoposid...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3542946</comments>
            <pubDate>Fri, 07 May 2010 17:10:11 +0100</pubDate>
            <guid isPermaLink="false">3542946</guid>        </item>
        <item>
            <title>Oral delivery of PND-1186 FAK inhibitor decreases tumor growth and spontaneous breast to lung metastasis in pre-clinical models.</title>
            <link>http://www.medworm.com/index.php?rid=3383339&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20234193%26dopt%3DAbstract</link>
            <description>Authors: Walsh C, Tanjoni I, Uryu S, Tomar A, Nam JO, Luo H, Phillips A, Patel N, Kwok C, McMahon G, Stupack DG, Schlaepfer DD
    Tumor metastasis is a leading cause of cancer-related death. Focal adhesion kinase (FAK) is a cytoplasmic tyrosine kinase recruited to integrin-mediated matrix attachment sites where FAK activity is implicated in the control of cell survival, migration and invasion. Although genetic studies support the importance of FAK activity in promoting tumor progression, it remains unclear whether pharmacological FAK inhibition prevents tumor metastasis. Here, we show that the FAK inhibitor PND-1186 blocks FAK Tyr-397 phosphorylation in vivo and exhibits anti-tumor efficacy in orthotopic breast carcinoma mouse tumor models. PND-1186 (100 mg/kg intraperitoneal, i.p.) showe...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3383339</comments>
            <pubDate>Sat, 20 Mar 2010 00:00:08 +0100</pubDate>
            <guid isPermaLink="false">3383339</guid>        </item>
        <item>
            <title>Translational medication development for nicotine addiction.</title>
            <link>http://www.medworm.com/index.php?rid=3303487&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20001184%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Medication development for the treatment of tobacco dependence remains a scientific and public health priority.
    PMID: 20001184 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3303487</comments>
            <pubDate>Thu, 25 Feb 2010 13:22:10 +0100</pubDate>
            <guid isPermaLink="false">3303487</guid>        </item>
        <item>
            <title>From pathogenesis to prevention of castration resistant prostate cancer.</title>
            <link>http://www.medworm.com/index.php?rid=3115823&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19760632%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: CRPCa is the end-stage of a multifactorial and heterogeneous disease process. Pathogenetic factors responsible for the development of the CRPCa phenotype are detectable in the patient's PCa tissue long before the clinical onset of the disease. This approach provides opportunity for early detection and prevention by targeting pathways relevant for the individual disease process.
    PMID: 19760632 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3115823</comments>
            <pubDate>Thu, 24 Dec 2009 04:36:25 +0100</pubDate>
            <guid isPermaLink="false">3115823</guid>        </item>
        <item>
            <title>Mechanisms of T-cell inhibition: implications for cancer immunotherapy.</title>
            <link>http://www.medworm.com/index.php?rid=3115822&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20021308%26dopt%3DAbstract</link>
            <description>Authors: Mittendorf EA, Sharma P
    Cancer vaccines designed to augment effector T-cell responses have been disappointing with respect to clinical efficacy. This lack of effectiveness may be due to the fact that regulatory mechanisms, both intrinsic and extrinsic to activated T cells, play important roles in inhibiting vaccine-induced effector T-cell responses. This concept raises the possibility that blockade of these regulatory checkpoints might enhance anti-tumor immune responses. In this review, we discuss several regulatory mechanisms that act to control effector T-cell responses and identify strategies to circumvent these mechanisms in order to improve clinical responses.
    PMID: 20021308 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3115822</comments>
            <pubDate>Thu, 24 Dec 2009 04:36:22 +0100</pubDate>
            <guid isPermaLink="false">3115822</guid>        </item>
        <item>
            <title>Defective autophagy control by the p53 rheostat in cancer.</title>
            <link>http://www.medworm.com/index.php?rid=3115821&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20023384%26dopt%3DAbstract</link>
            <description>Authors: Galluzzi L, Morselli E, Kepp O, Maiuri MC, Kroemer G
    Autophagy is a finely regulated, lysosomal catabolic pathway that contributes to the turnover of long-lived proteins and to the elimination of old/damaged organelles. Autophagy exerts bona fide oncosuppressive functions by: (1) limiting chromosomal instability; (2) reducing potentially mutagenic oxidative stress; and (3) restraining intratumoral necrosis and local inflammation. Defective autophagy constitutes a hallmark of cancer cells together with: (1) provision of autonomous growth signals;, (2) insensitivity to antiproliferative stimuli; (3) disabled apoptosis; (4) limitless replication; (5) production of angiogenic factors; (6) tissue invasion with metastasis; (7) avoidance of the immune response; and (8) enhanced anabo...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3115821</comments>
            <pubDate>Thu, 24 Dec 2009 04:36:19 +0100</pubDate>
            <guid isPermaLink="false">3115821</guid>        </item>
        <item>
            <title>A role for Chk2 in DNA damage induced mitotic delays in human colorectal cancer cells.</title>
            <link>http://www.medworm.com/index.php?rid=3115820&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20023427%26dopt%3DAbstract</link>
            <description>Authors: Varmark H, Kwak S, Theurkauf WE
    Progression into mitosis in the presence of DNA damage leads to spindle checkpoint (SAC) dependent mitotic delays and cytokinesis failure. In Drosophila embryos, DNA damage does not delay mitotic entry but triggers Checkpoint kinase-2 (Chk2) kinase dependent delays in mitotic exit. It is unclear if damage associated mitotic delays in human cells result from kinase signaling or breaks in centromere DNA that disrupt kinetochore function and activate the SAC. We show that transgenic expression of Human Chk2 in a Drosophila chk2 mutant background restores damage induced mitotic delays during early embryogenesis. Parental HCT116 colorectal cancer cells that progress into mitosis following DNA damage, due to either G(2) checkpoint adaptation or G(2) c...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3115820</comments>
            <pubDate>Thu, 24 Dec 2009 04:36:16 +0100</pubDate>
            <guid isPermaLink="false">3115820</guid>        </item>
        <item>
            <title>Platelet-Derived Growth Factor BB Mediates the Tropism of Human Mesenchymal Stem Cells for Malignant Gliomas.</title>
            <link>http://www.medworm.com/index.php?rid=3115819&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20023545%26dopt%3DAbstract</link>
            <description>CONCLUSION: PDGF-BB increases the attraction of hMSCs for gliomas in vitro and in vivo, and this tropism is mediated via PDGF-beta receptors on hMSCs. These findings can be exploited for advancing hMSC treatment.
    PMID: 20023545 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3115819</comments>
            <pubDate>Thu, 24 Dec 2009 04:36:13 +0100</pubDate>
            <guid isPermaLink="false">3115819</guid>        </item>
        <item>
            <title>Regulation of invasive growth: similar epigenetic mechanisms underpin tumour progression and implantation in human pregnancy.</title>
            <link>http://www.medworm.com/index.php?rid=3115818&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20025611%26dopt%3DAbstract</link>
            <description>Authors: Perry JK, Lins RJ, Lobie PE, Mitchell MD
    Malignant and trophoblastic cells share the capacity to migrate and invade surrounding tissues; however, trophoblast invasion during implantation is tightly regulated, whereas that associated with tumour progression is not. It is likely that similar mechanisms underlie the dynamic regulation of cell invasion and migration in both cases, and that these are based on epigenetic processes. This hypothesis is supported by recent results demonstrating that expression of the intercellular adhesion molecule E-cadherin, deregulation of which is associated with increased cell motility and invasive potential in cancer, is under epigenetic control in trophoblast cell lines. Further elucidation of the epigenetic pathways shared by trophoblasts and m...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3115818</comments>
            <pubDate>Thu, 24 Dec 2009 04:36:10 +0100</pubDate>
            <guid isPermaLink="false">3115818</guid>        </item>
        <item>
            <title>Overexpression of angiotensin II type 2 receptor gene induces cell death in lung adenocarcinoma cells.</title>
            <link>http://www.medworm.com/index.php?rid=3115817&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D20026904%26dopt%3DAbstract</link>
            <description>Authors: Pickel L, Matsuzuka T, Doi C, Ayuzawa R, Maurya DK, Xie SX, Berkland C, Tamura M
    The endogenous angiotensin II (Ang II) type 2 receptor (AT(2)) has been shown to mediate apoptosis in cardiovascular tissues. Thus, the aim of this study was to explore the anti-cancer effect of AT(2) overexpression on lung adenocarcinoma cells in vitro using adenoviral (Ad), FuGENE, and nanoparticle vectors. All three gene transfection methods efficiently transfected AT(2) cDNA into lung cancer cells but caused minimal gene transfection in normal lung epithelial cells. Ad-AT(2) significantly attenuated multiple human lung cancer cell growth (A549 and H358) as compared to the control viral vector, Ad-LacZ, when cell viability was examined by direct cell count. Examination of annexin V by flow cyto...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3115817</comments>
            <pubDate>Thu, 24 Dec 2009 04:36:07 +0100</pubDate>
            <guid isPermaLink="false">3115817</guid>        </item>
        <item>
            <title>Metastasis-associated genes in oral squamous cell carcinoma and salivary adenoid cystic carcinoma: a differential DNA chip analysis between metastatic and nonmetastatic cell systems.</title>
            <link>http://www.medworm.com/index.php?rid=3067925&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19963131%26dopt%3DAbstract</link>
            <description>Authors: Maruyama S, Cheng J, Yamazaki M, Zhou XJ, Zhang ZY, He RG, Saku T
    Overall modes of differential gene expressions were analyzed between human oral/salivary carcinoma cell systems with (MK-1 and ACCM) and without (ZK-1/ZK-2 and ACC2/ACC3) metastatic potential by using micro-array analysis with cancer-associated DNA chips to determine the kinds of genes associated with metastatic behaviors. MK-1 and/or ACCM showed lower levels of gene expression in extracellular matrix-related molecules, such as collagen type IV, laminin, and adhesion molecules such as cadherin 2, but higher levels of genes which control extracellular matrix degradation, such as MMP 9, as well as cell growth and cycle, such as FGF7 and cyclin D1. Among the differentially expressed genes, similar protein expressio...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3067925</comments>
            <pubDate>Wed, 09 Dec 2009 08:40:13 +0100</pubDate>
            <guid isPermaLink="false">3067925</guid>        </item>
        <item>
            <title>TGFbeta1 T29C polymorphism and cancer risk: a meta-analysis based on 40 case-control studies.</title>
            <link>http://www.medworm.com/index.php?rid=3067924&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19963138%26dopt%3DAbstract</link>
            <description>Authors: Wei BB, Xi B, Wang R, Bai JM, Chang JK, Zhang YY, Yoneda R, Su JT, Hua LX
    Transforming growth factor-beta1 (TGFbeta1) plays a significant role in regulating cellular proliferation and apoptosis. The TGFbeta1 T29C polymorphism reportedly affects cancer risk, but pertinent studies offer conflicting results. We therefore performed a meta-analysis based on 40 studies from 32 publications, assessing the strength of the association using odds ratios with 95% confidence intervals. Overall, no evidence has indicated that individuals carrying CC or CT genotypes had significantly increased cancer risks, compared with TT genotype carriers [CC vs. TT: odds ratio (OR)=1.10, 95% confidence interval (95% CI)=1.00-1.21, P=0.06; CT vs. TT: OR=1.07, 95% CI=0.99-1.16, P=0.09). However, stratifie...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3067924</comments>
            <pubDate>Wed, 09 Dec 2009 08:40:10 +0100</pubDate>
            <guid isPermaLink="false">3067924</guid>        </item>
        <item>
            <title>Combinational treatment of gap junctional activator and tamoxifen in breast cancer cells.</title>
            <link>http://www.medworm.com/index.php?rid=3067923&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19966541%26dopt%3DAbstract</link>
            <description>This study shows for the first time that combinational treatment of tamoxifen and PQ1 (a gap junctional activator) can be used to potentiate apoptosis of T47D human breast cancer cells. Thus, a gap junctional activator, PQ1, could potentially alter either the length or dose of tamoxifen clinically used for breast cancer patients.
    PMID: 19966541 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3067923</comments>
            <pubDate>Wed, 09 Dec 2009 08:40:06 +0100</pubDate>
            <guid isPermaLink="false">3067923</guid>        </item>
        <item>
            <title>Drug ratio-dependent antagonism: a new category of multidrug resistance and strategies for its circumvention.</title>
            <link>http://www.medworm.com/index.php?rid=3052662&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19949929%26dopt%3DAbstract</link>
            <description>Authors: Harasym TO, Liboiron BD, Mayer LD
    A newly identified form of multidrug resistance (MDR) in tumor cells is presented, pertaining to the commonly encountered resistance of cancer cells to anticancer drug combinations at discrete drug:drug ratios. In vitro studies have revealed that whether anticancer drug combinations interact synergistically or antagonistically can depend on the ratio of the combined agents. Failure to control drug ratios in vivo due to uncoordinated pharmacokinetics could therefore lead to drug resistance if tumor cells are exposed to antagonistic drug ratios. Consequently, the most efficacious drug combination may not occur at the typically employed maximum tolerated doses of the combined drugs if this leads to antagonistic ratios in vivo after administration...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3052662</comments>
            <pubDate>Thu, 03 Dec 2009 19:08:11 +0100</pubDate>
            <guid isPermaLink="false">3052662</guid>        </item>
        <item>
            <title>Endotoxin exposure and inflammation markers among agricultural workers in colorado and nebraska.</title>
            <link>http://www.medworm.com/index.php?rid=3052661&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19953416%26dopt%3DAbstract</link>
            <description>This study quantified personal work-shift exposures to inhalable dust, endotoxin, and its reactive 3-hydroxy fatty acid (3-OHFA) constituents among workers in grain elevators, cattle feedlots, dairies, and on corn farms. Exposures were compared with post-work-shift nasal lavage fluid inflammation markers and respiratory symptoms. Breathing-zone personal air monitoring was performed over one work shift to quantify inhalable dust (Institute of Medicine samplers), endotoxin (recombinant factor C [rFC] assay), and 3-OHFA (gas chromatography/mass spectrometry). Post-shift nasal lavage fluids were assayed for polymorphonuclear neutrophils (PMN), myeloperoxidase (MPO), interleukin 8 (IL-8), albumin, and eosinophilic cation protein (ECP) concentrations. The geometric mean (GSD) of endotoxin exposu...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3052661</comments>
            <pubDate>Thu, 03 Dec 2009 19:08:08 +0100</pubDate>
            <guid isPermaLink="false">3052661</guid>        </item>
        <item>
            <title>Traffic air pollution and risk of death from bladder cancer in taiwan using petrol station density as a pollutant indicator.</title>
            <link>http://www.medworm.com/index.php?rid=3052660&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19953417%26dopt%3DAbstract</link>
            <description>Authors: Ho CK, Peng CY, Yang CY
    To investigate the relationship between air pollution and risk of death from bladder cancer, a matched cancer case-control study was conducted using deaths that occurred in Taiwan from 1997 through 2006. Data for all eligible bladder cancer deaths were obtained from the Bureau of Vital Statistics of the Taiwan Provincial Department of Health. The control group consisted of individuals who died from causes other than cancer or diseases associated with genitourinary problems. The controls were pair matched to the cases by gender, year of birth, and year of death. Each matched control was selected randomly from the set of possible controls for each case. Data for the number of petrol stations in study municipalities were collected from the two major petrol...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3052660</comments>
            <pubDate>Thu, 03 Dec 2009 19:08:06 +0100</pubDate>
            <guid isPermaLink="false">3052660</guid>        </item>
        <item>
            <title>Critical care issues in oncological surgery patients.</title>
            <link>http://www.medworm.com/index.php?rid=3045223&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19944277%26dopt%3DAbstract</link>
            <description>This article describes the CRS/HIPEC technique, and discusses the most common postoperative complications and critical care issues in these patients, including anastomotic leaks, intestinal perforation, abscesses, and intra-abdominal bleeding. The leading cause of mortality is sepsis leading to multiple organ failure, and such patients are at particularly higher risk due to the extensive CRS and HIPEC. The intensivist must be vigilant to ensure that source control is not overlooked. This process is a very difficult one, made even more challenging by the blunting of physiologic responses and the frequent absence of the classic acute abdomen.
    PMID: 19944277 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=3045223</comments>
            <pubDate>Tue, 01 Dec 2009 22:44:06 +0100</pubDate>
            <guid isPermaLink="false">3045223</guid>        </item>
        <item>
            <title>A glycomics approach to the discovery of potential cancer biomarkers.</title>
            <link>http://www.medworm.com/index.php?rid=2955428&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19882130%26dopt%3DAbstract</link>
            <description>Authors: An HJ, Lebrilla CB
    Glycosylation is highly sensitive to the biochemical environment and plays a key role in development and disease manifestation. Moreover, glycan biosynthesis depends on several highly competitive processes; thus, variations in the concentration of specific glycosyltransferases produce different products. For this reason, monitoring changes in glycosylation may be a more specific and sensitive approach to biomarker discovery and possibly disease diagnosis. Glycans in serum are of particular interest as approximately half of all proteins are glycosylated. We have developed the methods for profiling the glycans in human serum to identify glycan biomarker. Global release methods were used including chemical and enzymatic to access O-linked and N-linked glycans, ...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2955428</comments>
            <pubDate>Wed, 04 Nov 2009 04:14:09 +0100</pubDate>
            <guid isPermaLink="false">2955428</guid>        </item>
        <item>
            <title>Target gene discovery for novel therapeutic agents in cancer treatment.</title>
            <link>http://www.medworm.com/index.php?rid=2955427&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19882275%26dopt%3DAbstract</link>
            <description>Authors: Ammerpohl O, Tiwari S, Kalthoff H
    Target identification of novel therapeutic drugs is pivotal for the establishment of (1) new anticancer regimens, (2) to control side effects of the drugs, and (3) to identify appropriate combinations with established drugs.Here, we describe several in vitro assays applicable to characterize different characteristics of tumor cells. Furthermore, we present a protocol for establishing a reporter gene system for in vivo imaging, allowing for the study of drug effects in small animal models.
    PMID: 19882275 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2955427</comments>
            <pubDate>Wed, 04 Nov 2009 04:14:06 +0100</pubDate>
            <guid isPermaLink="false">2955427</guid>        </item>
        <item>
            <title>Weekly docetaxel in patients with platinum-refractory metastatic or recurrent squamous cell carcinoma of the head and neck.</title>
            <link>http://www.medworm.com/index.php?rid=2891280&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19381630%26dopt%3DAbstract</link>
            <description>CONCLUSION: Weekly docetaxel regimen had good clinical activity with an acceptable toxicity in patients with platinum-refractory SCCHN.
    PMID: 19381630 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2891280</comments>
            <pubDate>Wed, 14 Oct 2009 20:06:09 +0100</pubDate>
            <guid isPermaLink="false">2891280</guid>        </item>
        <item>
            <title>Expression of the proto-oncogene c-fos and the immunolocalization of c-fos, phosphorylated c-fos and estrogen receptor beta in the human testis.</title>
            <link>http://www.medworm.com/index.php?rid=2856402&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19795350%26dopt%3DAbstract</link>
            <description>Authors: Ara&amp;#xFA;jo FC, Oliveira CA, Reis AB, Del Puerto HL, Martins AS, Reis FM
    Spermatogenesis is under the control of a complex endocrine and paracrine system, including estrogen receptor (ER) signaling. In many target cells, ER promotes the transcription of c-fos and other proto-oncogenes to regulate cell growth and differentiation. Thus, in this study we evaluated the expression of the proto-oncogene c-fos and the immunolocalization of c-fos, phosphorylated c-fos and ERbeta proteins in the human testis. Testis tissue samples were obtained from 12 men undergoing orchiectomy as adjuvant treatment for prostate cancer, and were stained by immunohistochemistry for c-fos, phosphorylated c-fos and ERbeta localization. Both forms of c-fos proteins were immunoreactive, mainly in germ cell...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2856402</comments>
            <pubDate>Sat, 03 Oct 2009 14:16:11 +0100</pubDate>
            <guid isPermaLink="false">2856402</guid>        </item>
        <item>
            <title>Different spectra of therapeutic vaccine development against HPV infections.</title>
            <link>http://www.medworm.com/index.php?rid=2709589&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19684468%26dopt%3DAbstract</link>
            <description>Authors: Bolhassani A, Mohit E, Rafati S
    Human papillomaviruses (HPVs) are simple, non-enveloped, double-stranded DNA viruses and responsible for an enormous global burden of genital disease. HPV is annually associated with 500,000 new cases of cervical cancer and 250,000 cervical cancer deaths worldwide. The association between HPV infection and cervical cancer indicates that HPV serves as an ideal target for development of preventive and therapeutic vaccines. A novel approach for primary prevention of cervical cancer has become available by the discovery of efficient prophylactic HPV vaccines based on virus-like particles. Therapeutic vaccination has been limited by inadequate antigen-specific immune responses. Different therapeutic strategies have been developed including peptide im...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2709589</comments>
            <pubDate>Tue, 18 Aug 2009 15:50:06 +0100</pubDate>
            <guid isPermaLink="false">2709589</guid>        </item>
        <item>
            <title>Intratumoral injection of alpha-gal glycolipids induces a protective anti-tumor T cell response which overcomes Treg activity.</title>
            <link>http://www.medworm.com/index.php?rid=2700361&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19184002%26dopt%3DAbstract</link>
            <description>Authors: Abdel-Motal UM, Wigglesworth K, Galili U
    alpha-Gal glycolipids capable of converting tumors into endogenous vaccines, have alpha-gal epitopes (Gal alpha 1-3 Gal beta 1-4GlcNAc-R) and are extracted from rabbit RBC membranes. alpha-Gal epitopes bind anti-Gal, the most abundant natural antibody in humans constituting 1% of immunoglobulins. alpha-Gal glycolipids insert into tumor cell membranes, bind anti-Gal and activate complement. The complement cleavage peptides C5a and C3a recruit inflammatory cells and APC into the treated lesion. Anti-Gal further opsonizes the tumor cells and targets them for effective uptake by recruited APC, via Fc gamma receptors. These APC transport internalized tumor cells to draining lymph nodes, and present immunogenic tumor antigen peptides for acti...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2700361</comments>
            <pubDate>Fri, 14 Aug 2009 23:42:15 +0100</pubDate>
            <guid isPermaLink="false">2700361</guid>        </item>
        <item>
            <title>Dramatic efficacy improvement of a DC-based vaccine against AML by CD25 T cell depletion allowing the induction of a long-lasting T cell response.</title>
            <link>http://www.medworm.com/index.php?rid=2700360&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19225777%26dopt%3DAbstract</link>
            <description>In this report, C57BL6 mice were vaccinated with DC pulsed with peptides eluted (EP) from the syngeneic C1498 myelomonocytic leukemic cell line in a prophylactic setting. In this model, a natural antileukemic immunity mediated by NK cells was observed in the control unloaded DC-vaccinated group. On the other hand, we showed that the cytotoxic antileukemic immune response induced by vaccination with eluted peptides pulsed-DC (DC/EP), in vitro and in vivo, was mainly mediated by CD4(+) T cells. Treatment with anti-CD25 antibody to deplete CD4(+) CD25(+) regulatory T cells before DC-vaccination dramatically improved the antileukemic immune response induced by immunization, and allowed the development of long-lasting immune responses that were tumor protective after a re-challenge with leukemi...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2700360</comments>
            <pubDate>Fri, 14 Aug 2009 23:42:13 +0100</pubDate>
            <guid isPermaLink="false">2700360</guid>        </item>
        <item>
            <title>Anti-cancer activity and mechanistic features of a NK cell activating molecule.</title>
            <link>http://www.medworm.com/index.php?rid=2700359&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19259669%26dopt%3DAbstract</link>
            <description>In this study, we found that G&amp;#xF6;6983, a PKC inhibitor, induced a remarkable increase of NCR expression on primary NK cells, but other PKC inhibitors and NK cell stimulators such as IL-2 and PMA, did not. G&amp;#xF6;6983 increased the expression of NCR in a time- and concentration-dependent manner. Furthermore, G&amp;#xF6;6983 strongly upregulated the surface expression of death ligands FasL and TRAIL, but not cytotoxic molecules perforin and granzyme B. Unlike two other NK stimulating molecules, IL-2, and PMA, G&amp;#xF6;6983 did not induce NK cell proliferation. Up-regulation of NCRs and death ligands on NK cells by G&amp;#xF6;6983 resulted in a significant enhancement of NK cytotoxicity against various cancer cell lines. Most importantly, administration of G&amp;#xF6;6983 effectively inhibited pulmonary...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2700359</comments>
            <pubDate>Fri, 14 Aug 2009 23:42:10 +0100</pubDate>
            <guid isPermaLink="false">2700359</guid>        </item>
        <item>
            <title>Treatment of non-Hodgkin's lymphoma xenografts with the HB22.7 anti-CD22 monoclonal antibody and phosphatase inhibitors improves efficacy.</title>
            <link>http://www.medworm.com/index.php?rid=2700358&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19294384%26dopt%3DAbstract</link>
            <description>CONCLUSION: The relatively specific association of SHP-1 with CD22 suggests that CD22-specific signal augmentation by phosphatase inhibitors can improve the clinical outcome of anti-CD22 based immunotherapy.
    PMID: 19294384 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2700358</comments>
            <pubDate>Fri, 14 Aug 2009 23:42:08 +0100</pubDate>
            <guid isPermaLink="false">2700358</guid>        </item>
        <item>
            <title>Factors Interacting with HIF-1 mRNA: Novel Therapeutic Targets.</title>
            <link>http://www.medworm.com/index.php?rid=2700357&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19671045%26dopt%3DAbstract</link>
            <description>Authors: Galb&amp;#xE1;n S, Gorospe M
    The heterodimeric transcription factor HIF-1 (hypoxia-inducible factor-1) induces angiogenesis, a process that is aberrantly elevated in cancer. The HIF-1 subunit is constitutively expressed, but the levels of the HIF-1 subunit are robustly regulated, increasing under hypoxic conditions and decreasing in normoxia. These changes result from rapid alterations in the rates of HIF-1 production and degradation. While the regulation of HIF-1 degradation is understood in significant detail, much less is known about the regulation of HIF-1 biosynthesis. Here, we review recent evidence that HIF-1 production is effectively controlled by post-transcriptional mechanisms. We focus on the RNA-binding proteins (RBPs) and the non-coding RNAs that interact with the HIF...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2700357</comments>
            <pubDate>Fri, 14 Aug 2009 23:42:06 +0100</pubDate>
            <guid isPermaLink="false">2700357</guid>        </item>
        <item>
            <title>Dietary zinc supplementation and methotrexate-induced small intestinal mucositis in metallothionein-knockout and wild-type mice.</title>
            <link>http://www.medworm.com/index.php?rid=2646574&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19633421%26dopt%3DAbstract</link>
            <description>Conclusions: Dietary Zn supplementation did not ameliorate MTX-induced small bowel damage, possibly signifying a deficiency in induction of MT by Zn. However, the presence of MT was able to reduce histological damage and neutrophil infiltration caused by MTX in the gut.
    PMID: 19633421 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2646574</comments>
            <pubDate>Wed, 29 Jul 2009 08:36:05 +0100</pubDate>
            <guid isPermaLink="false">2646574</guid>        </item>
        <item>
            <title>Adenovirus-mediated suicide gene therapy under the control of Cox-2 promoter for colorectal cancer.</title>
            <link>http://www.medworm.com/index.php?rid=2563473&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19571664%26dopt%3DAbstract</link>
            <description>This study was conducted to explore the possible therapeutic approach of Cox-2 promoter-directed suicide gene therapy with herpes simplex virus thymidine kinase (HSV-tk) in combination with adenoviral technology for advanced colorectal cancer. Firstly, the activity of Cox-2 promoter was assessed by dual luciferase and enhanced green fluorescent protein reporter gene assays in colorectal cancer cell lines and normal human intestinal epithelial cell line. Then, the expression of coxsackievirus and adenovirus receptor (CAR) was detected in colorectal cancer cell lines. The Cox-2 promoter-directed HSV-tk/ganciclovir (GCV) system mediated by adenovirus (Ad-Cp-TK) was developed (Ad-CMVp-TK, Ad-null and no Ad as controls). In vitro cytoxicity, colony formation and apoptosis assays were performed ...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2563473</comments>
            <pubDate>Sun, 05 Jul 2009 09:08:21 +0100</pubDate>
            <guid isPermaLink="false">2563473</guid>        </item>
        <item>
            <title>Anti-inflammatory, anti-proliferative and antioxidant profiles of selective cyclooxygenase-2 inhibition as chemoprevention for rat bladder carcinogenesis.</title>
            <link>http://www.medworm.com/index.php?rid=2563461&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19571668%26dopt%3DAbstract</link>
            <description>Conclusions: Celecoxib has demonstrated an outstanding inhibitory effect on bladder cancer chemoprevention, which might be due to its expected anti-inflammatory actions, as well as by anti-proliferatory and antioxidant actions. This data supports a pivotal role of cancer chemoprevention strategies based on COX-2 inhibition.
    PMID: 19571668 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2563461</comments>
            <pubDate>Sun, 05 Jul 2009 09:04:06 +0100</pubDate>
            <guid isPermaLink="false">2563461</guid>        </item>
        <item>
            <title>Parameters of protection against ultraviolet radiation-induced skin cell damage.</title>
            <link>http://www.medworm.com/index.php?rid=2558878&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19360745%26dopt%3DAbstract</link>
            <description>Authors: Cao C, Wan Y
    Epidemiological and experimental evidence has supported the notion that solar ultraviolet (UV) radiation is the leading cause of skin cell damage and skin cancer. Non-melanoma skin cancer, one of the malignancies with the most rapidly increasing incidence, is suggested to be directly related to the total exposure to solar UV light. Over the past few years, the mechanisms of cellular responses to UV radiation have received unprecedented attention. Understanding how skin cells respond to UV radiation will undoubtedly help decipher what goes wrong in a variety of clinical skin disorders including skin cancer and will facilitate the development of novel therapeutic strategies. In the past decade, studies have established that UV radiation induces multifarious signal t...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558878</comments>
            <pubDate>Wed, 01 Jul 2009 16:44:02 +0100</pubDate>
            <guid isPermaLink="false">2558878</guid>        </item>
        <item>
            <title>Dietary energy density in relation to subsequent changes of weight and waist circumference in European men and women.</title>
            <link>http://www.medworm.com/index.php?rid=2558810&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19396357%26dopt%3DAbstract</link>
            <description>CONCLUSION: Our results suggest that lower ED diets do not prevent weight gain but have a weak yet potentially beneficial effect on the prevention of abdominal obesity as measured by waist circumference.
    PMID: 19396357 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558810</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:34 +0100</pubDate>
            <guid isPermaLink="false">2558810</guid>        </item>
        <item>
            <title>Epidemiology of the complications of gastroesophageal reflux disease.</title>
            <link>http://www.medworm.com/index.php?rid=2558808&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19439954%26dopt%3DAbstract</link>
            <description>Authors: Belhocine K, Galmiche JP
    The complications of gastroesophageal reflux disease (GERD) include ulcers, strictures, Barrett's esophagus and carcinoma. Although the prevalence of GERD is very high, the development of complications remains quite rare and usually occurs in association with factors generally observed in more severe disease such as hiatal hernia, bile reflux or severely disturbed motility. Recent studies have emphasized the role of obesity and genetic factors as aggravating factors in the development of GERD complications. Barrett's esophagus is the most prevalent complication of GERD and seems to be associated with an increased mortality rate. However, cancer incidence is low and most patients die from other causes, especially cardiovascular disease.
    PMID: 194399...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558808</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:34 +0100</pubDate>
            <guid isPermaLink="false">2558808</guid>        </item>
        <item>
            <title>Chemoprevention of adenocarcinoma associated with Barrett's esophagus: potential options.</title>
            <link>http://www.medworm.com/index.php?rid=2558803&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19439956%26dopt%3DAbstract</link>
            <description>This article focuses on known and postulated targets for prevention of distal esophageal cancer.
    PMID: 19439956 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558803</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:34 +0100</pubDate>
            <guid isPermaLink="false">2558803</guid>        </item>
        <item>
            <title>Screening and surveillance of Barrett's esophagus.</title>
            <link>http://www.medworm.com/index.php?rid=2558799&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19439961%26dopt%3DAbstract</link>
            <description>Authors: Regula J
    Screening and surveillance of Barrett's esophagus is a matter of discussion in the current world literature due to uncertainties concerning its cost-effectiveness. Important and valid arguments for and against it are available. Despite the fact that this issue is still to be resolved, screening using standard gastroscopy once in a lifetime and surveillance using repeated endoscopies with multiple blind biopsies are widely practiced by individual gastroenterologists. Further studies that mainly focus on finding better predictors of progression to cancer are needed to be able to prepare clear and useful guidelines. The ultimate aim of screening and surveillance should be decrease in mortality from esophageal adenocarcinoma resulting from Barrett's esophagus.
    PMID: 1...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558799</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:34 +0100</pubDate>
            <guid isPermaLink="false">2558799</guid>        </item>
        <item>
            <title>Acupuncture for nonpalliative radiation therapy-related fatigue: feasibility study.</title>
            <link>http://www.medworm.com/index.php?rid=2558795&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19476739%26dopt%3DAbstract</link>
            <description>Authors: Mao JJ, Styles T, Cheville A, Wolf J, Fernandes S, Farrar JT
    The purpose of this study was to determine the feasibility of an acupuncture clinical trial to prevent radiation therapy (RT)-induced fatigue. We conducted a cross-sectional survey study and a single-arm acupuncture clinical trial among patients undergoing RT. Patients with a Karnofsky score of less than 60, severe anemia, or substantial psychological diagnoses were excluded. Patients received up to 12 treatments of acupuncture over the entire course of their RT. The Lee Fatigue Scale (LFS) was administered at baseline, in the middle of RT, and at the end of RT, along with the Patient Global Impression of Change (PGIC). Among the 48 of 53 (91% response rate) survey participants, 20 (42%) reported that they would part...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558795</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:34 +0100</pubDate>
            <guid isPermaLink="false">2558795</guid>        </item>
        <item>
            <title>New double-stapling technique for esophagojejunostomy and esophagogastrostomy in gastric cancer surgery, using a peroral intraluminal approach with a digital stapling system.</title>
            <link>http://www.medworm.com/index.php?rid=2558771&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19562464%26dopt%3DAbstract</link>
            <description>We describe a new double-stapling technique for esophagojejunostomy and esophagogastrostomy, using a peroral intraluminal approach with a digital stapling system, a flexible shaft remote-control stapler - the Surg-ASSIST and Power Circular Stapler 21 mm (PCS). The overtube of the flexible shaft of the PCS is prepared with a nylon tie and secured to a nasogastric (NG) tube. The flexible shaft is manually advanced down the esophagus with guidance by pulling the NG tube from the abdominal cavity side. The trocar of the flexible shaft is removed from the stump of the abdominal esophagus and connected to the anvil and they are approximated; the stapler device is then fired to form a double-stapled esophagojejunostomy and esophagogastrostomy. Our peroral intraluminal approach does not require a ...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558771</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:27 +0100</pubDate>
            <guid isPermaLink="false">2558771</guid>        </item>
        <item>
            <title>Duodenal stump cancer after Billroth-II distal gastrectomy for gastric cancer.</title>
            <link>http://www.medworm.com/index.php?rid=2558764&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19562467%26dopt%3DAbstract</link>
            <description>Authors: Lee J, Lee K, Kim W
    Duodenal cancer is an uncommon neoplasm and it mostly arises from the periampullary area. However, metachronous or even recurrent cancer at the duodenal stump following Billroth II type distal gastrec tomy for gastric cancer is extremely rare and, to our knowledge, has not yet been reported. A 68-year-old man underwent Billroth II distal gastrectomy with D2 lymph node dissection for an advanced gastric cancer. At that time the tumor stage was T2bN3M0, with 44 of 78 retrieved lymph nodes showing metastasis. He was well without recurrence for 3 years; however, he visited our hospital because of the abrupt onset of dizziness and tarry stool. A polypoid tumor that bled easily when touched was found at the end of the afferent loop of the duodenal stump by gastro...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558764</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:27 +0100</pubDate>
            <guid isPermaLink="false">2558764</guid>        </item>
        <item>
            <title>Use of DNA combing to study DNA replication in xenopus and human cell-free systems.</title>
            <link>http://www.medworm.com/index.php?rid=2558759&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19563130%26dopt%3DAbstract</link>
            <description>We describe (a) the preparation and use of egg extracts and demembranated sperm chromatin templates; (b) a simple method for preparing silanized glass coverslips suitable for DNA combing and fluorescence detection; (c) two alternative replicative DNA labelling schemes and their respective advantages; and (d) a protocol for combining replicative labelling with detection of specific DNA sequences by fluorescent in situ hybridization (FISH). Although most observations made in Xenopus egg extracts are applicable to other eukaryotes, there are differences in cell-cycle regulation between mammalian somatic cells and embryonic amphibian cells, which led to the development of human cell-free systems that can initiate semi-conservative chromosomal DNA replication under cell-cycle control. We have e...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558759</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:21 +0100</pubDate>
            <guid isPermaLink="false">2558759</guid>        </item>
        <item>
            <title>Molecular mechanisms of endothelial hyperpermeability: implications in inflammation.</title>
            <link>http://www.medworm.com/index.php?rid=2558738&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19563700%26dopt%3DAbstract</link>
            <description>Authors: Kumar P, Shen Q, Pivetti CD, Lee ES, Wu MH, Yuan SY
    Endothelial hyperpermeability is a significant problem in vascular inflammation associated with trauma, ischaemia-reperfusion injury, sepsis, adult respiratory distress syndrome, diabetes, thrombosis and cancer. An important mechanism underlying this process is increased paracellular leakage of plasma fluid and protein. Inflammatory stimuli such as histamine, thrombin, vascular endothelial growth factor and activated neutrophils can cause dissociation of cell-cell junctions between endothelial cells as well as cytoskeleton contraction, leading to a widened intercellular space that facilitates transendothelial flux. Such structural changes initiate with agonist-receptor binding, followed by activation of intracellular signalli...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558738</comments>
            <pubDate>Wed, 01 Jul 2009 16:18:11 +0100</pubDate>
            <guid isPermaLink="false">2558738</guid>        </item>
        <item>
            <title>Active transcription of rRNA operons condenses the nucleoid in Escherichia coli: examining the effect of transcription on nucleoid structure in the absence of transertion.</title>
            <link>http://www.medworm.com/index.php?rid=2558826&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19395497%26dopt%3DAbstract</link>
            <description>Authors: Cabrera JE, Cagliero C, Quan S, Squires CL, Jin DJ
    In Escherichia coli the genome must be compacted approximately 1,000-fold to be contained in a cellular structure termed the nucleoid. It is proposed that the structure of the nucleoid is determined by a balance of multiple compaction forces and one major expansion force. The latter is mediated by transertion, a coupling of transcription, translation, and translocation of nascent membrane proteins and/or exported proteins. In supporting this notion, it has been shown consistently that inhibition of transertion by the translation inhibitor chloramphenicol results in nucleoid condensation due to the compaction forces that remain active in the cell. Our previous study showed that during optimal growth, RNA polymerase is concentra...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558826</comments>
            <pubDate>Tue, 30 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558826</guid>        </item>
        <item>
            <title>[Results and participation factors to the European Randomized study of Screening for Prostate Cancer (ERSPC) with Prostate Specific Antigen: French departments of Tarn and Hérault.]</title>
            <link>http://www.medworm.com/index.php?rid=2558788&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19559380%26dopt%3DAbstract</link>
            <description>CONCLUSION: Participation rate at first round was modest. Profile of men who participated compared to men who did not were different. The control group was probably contaminated by PSA testing outside study protocol. Consequences at ERSPC level of this low participation rate on final analysis remain to be determined.
    PMID: 19559380 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558788</comments>
            <pubDate>Tue, 30 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558788</guid>        </item>
        <item>
            <title>Molecular epidemiology of primary brain tumors.</title>
            <link>http://www.medworm.com/index.php?rid=2558785&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19560733%26dopt%3DAbstract</link>
            <description>Authors: Gu J, Liu Y, Kyritsis AP, Bondy ML
    Although primary brain tumors (PBTs) are generally considered to be a multifactorial disorder, understanding the genetic basis and etiology of the disease is essential for PBT risk assessment. Understanding of the genetic susceptibility for PBT has come from studies of rare genetic syndromes, linkage analysis, family aggregation, early-onset pediatric cases, and mutagen sensitivity. There are currently no effective markers to assess biological dose of exposures and genetic heterogeneity. The priorities recently recommended by the Brain Tumor Epidemiology Consortium emphasized the need for expanding research in genetics and molecular epidemiology. In this article, we review the literature to identify molecular epidemiologic case-control studie...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558785</comments>
            <pubDate>Tue, 30 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558785</guid>        </item>
        <item>
            <title>Triple negative breast cancer: a study from the point of view of basal CK5/6 and HER-1.</title>
            <link>http://www.medworm.com/index.php?rid=2558782&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19561231%26dopt%3DAbstract</link>
            <description>CONCLUSION: Expression of CK5/6 and HER-1 is frequent in ER-negative breast cancers, in triple negative and in non-triple negative tumours. In a minority of cases, HER-1 overexpression may be caused by HER-1 gene amplification. Further studies are needed to investigate whether such cases might benefit from anti-HER-1 therapy.
    PMID: 19561231 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558782</comments>
            <pubDate>Tue, 30 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558782</guid>        </item>
        <item>
            <title>Fumagillin inhibits colorectal cancer growth and metastasis in mice: in vivo and in vitro study of anti-angiogenesis.</title>
            <link>http://www.medworm.com/index.php?rid=2558757&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19563408%26dopt%3DAbstract</link>
            <description>In conclusion, Fumagillin was found to suppress colorectal cancer growth and metastasis by suppressing angiogenesis.
    PMID: 19563408 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558757</comments>
            <pubDate>Tue, 30 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558757</guid>        </item>
        <item>
            <title>Cancer risk among children with very low birth weights.</title>
            <link>http://www.medworm.com/index.php?rid=2558732&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19564288%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: These data suggested no association between most cancers and very low birth weight, with the exception of the known association of hepatoblastoma and possibly moderately increased risks of other gliomas and retinoblastoma, which may warrant confirmation.
    PMID: 19564288 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558732</comments>
            <pubDate>Tue, 30 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558732</guid>        </item>
        <item>
            <title>Primary lung cancer and TNF-alpha gene polymorphisms: A case-control study in a Croatian population.</title>
            <link>http://www.medworm.com/index.php?rid=2558712&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19564826%26dopt%3DAbstract</link>
            <description>Conclusions: This study indicates that the TNF-alpha-308 and TNF-alpha-238 polymorphisms do not influence susceptibility to or severity of lung cancer in a Croatian population.&amp;lt;br /&amp;gt;
    PMID: 19564826 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558712</comments>
            <pubDate>Tue, 30 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558712</guid>        </item>
        <item>
            <title>FAS and FAS Ligand Polymorphisms in the Promoter Regions and Risk of Gastric Cancer in Southern China.</title>
            <link>http://www.medworm.com/index.php?rid=2558710&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19565204%26dopt%3DAbstract</link>
            <description>Authors: Wang M, Wu D, Tan M, Gong W, Xue H, Shen H, Zhang Z
    The FAS and FAS ligand (FASLG) system plays a key role in regulating apoptotic cell death, and corruption of this signaling pathway has been shown to participate in tumorigenesis. Functional promoter polymorphisms of the FAS and FASLG genes can alter transcriptional activities and thus alter risk of cancer. We hypothesized that the FAS -1377G&amp;gt;A, FAS -670A&amp;gt;G, and FASLG -844T&amp;gt;C polymorphisms in the promoter regions are associated with risk of gastric cancer. In a population-based case-control study of 332 gastric cancer cases and 324 controls, we genotyped these three polymorphisms and evaluated their association with risk of gastric cancer. We found that the FAS and FASL genotypes and the FAS haplotypes had no signifi...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558710</comments>
            <pubDate>Mon, 29 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558710</guid>        </item>
        <item>
            <title>The Role and Extent of Pelvic Lymphadenectomy in the Management of Patients with Invasive Urothelial Carcinoma.</title>
            <link>http://www.medworm.com/index.php?rid=2558708&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19565335%26dopt%3DAbstract</link>
            <description>Authors: Lerner SP
    OPINION STATEMENT: Lymph node metastases are the most important prognostic variable in determining outcome following radical cystectomy. An anatomic bilateral node dissection includes at a minimum the external and internal iliac and obturator lymph nodes. An extended node dissection may include the distal aortic and vena caval nodes, bilateral common iliac, and pre-sacral nodes, which receive direct lymphatic drainage from the posterior bladder and trigone. This approach sets up the cystectomy, maximizes sensitivity for detection of nodal metastasis, assures optimum local pelvic cancer control, and accurately identifies those high-risk patients with node metastases who may benefit from adjuvant chemotherapy. Lymph node retrieval is affected by several variables of no...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558708</comments>
            <pubDate>Mon, 29 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558708</guid>        </item>
        <item>
            <title>Accelerated mammary maturation and differentiation, and delayed MMTVneu-induced tumorigenesis of K303R mutant ERalpha transgenic mice.</title>
            <link>http://www.medworm.com/index.php?rid=2558774&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19561644%26dopt%3DAbstract</link>
            <description>Authors: Herynk MH, Lewis MT, Hopp TA, Medina D, Corona-Rodriguez A, Cui Y, Beyer AR, Fuqua SA
    We identified a somatic mutation in estrogen receptor-alpha (ERalpha) in breast cancer causing a lysine to arginine transition (K303R) resulting in hypersensitivity to estrogen, altered associations with coactivators and corepressors and altered posttranslational modifications of ERalpha. We have developed a transgenic mouse expressing the K303R mutant ERalpha under control of the mouse mammary tumor virus (MMTV) promoter. At 4 months of age, K303R ERalpha transgenic animals demonstrate precocious alveolar budding compared with wild-type ERalpha transgenic mice or nontransgenic littermates. Despite these morphologic differences, K303R ERalpha transgenic mice displayed no differences in levels...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558774</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558774</guid>        </item>
        <item>
            <title>Infections in non-myeloablative hematopoietic stem cell transplantation patients with lymphoid malignancies: spectrum of infections, predictors of outcome and proposed guidelines for fungal infection prevention.</title>
            <link>http://www.medworm.com/index.php?rid=2558772&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19561648%26dopt%3DAbstract</link>
            <description>Authors: Safdar A, Rodriguez GH, Mihu CN, Mora-Ramos L, Mulanovich V, Chemaly RF, Champlin RE, Khouri I
    The overall risk of infections is lower in patients undergoing non-myeloablative allogeneic stem cell transplantation (NST) than in conventional stem cell transplant recipients. We sought to evaluate conditions associated with increased risk of infections after NST. In 81 patients, 187 infection episodes were noted; chronic lymphocytic leukemia (138 episodes/100 person-years) and recipients of matched unrelated donor graft (128 episodes/100 person-years) had higher risk of infection. Only half of the cytomegalovirus (CMV) infections occurred 31-100 days after transplantation. Most patients with CMV infection were non-neutropenic (100%), had lymphoma (76%), were younger (&amp;lt;55 years;...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558772</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558772</guid>        </item>
        <item>
            <title>Serum soluble CD44v6 levels in patients with oral and maxillofacial malignancy.</title>
            <link>http://www.medworm.com/index.php?rid=2558752&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19563418%26dopt%3DAbstract</link>
            <description>Conclusion: The possible roles of CD44v6 in the diagnosis of oral and maxillofacial malignancy deserve further elucidation and evaluation. Serum sCD44v6 may be a valuable marker in monitoring treatment response in patients with OSCC.
    PMID: 19563418 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558752</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558752</guid>        </item>
        <item>
            <title>A phase III clinical trial of exercise modalities on treatment side-effects in men receiving therapy for prostate cancer.</title>
            <link>http://www.medworm.com/index.php?rid=2558750&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19563641%26dopt%3DAbstract</link>
            <description>DISCUSSION: The principal outcome of this project will be the determination of the strength of effect of exercise on the well established musculoskeletal, cardiovascular and insulin metabolism side effects of androgen deprivation in prostate cancer patients. As this project is much longer term than previous investigations in the area of exercise and cancer, we will gain knowledge as to the continuing effects of exercise in this patient population specifically targeting bone density, cardiovascular function, lean and fat mass, physical function and falls risk as primary study endpoints. In terms of advancement of prostate cancer care, we expect dissemination of the knowledge gained from this project to reduce fracture risk, improve physical and functional ability, quality of life and ultima...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558750</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558750</guid>        </item>
        <item>
            <title>NMD inhibition fails to identify tumour suppressor genes in microsatellite stable gastric cancer cell lines.</title>
            <link>http://www.medworm.com/index.php?rid=2558742&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19563644%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Although UPF1 was substantially repressed, thus resulting in the inhibition of the NMD system, we did not find genes inactivated by nonsense mutations. Our results show that the GINI strategy leads to a high number of false positives.
    PMID: 19563644 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558742</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558742</guid>        </item>
        <item>
            <title>CXCR4 expression on circulating pan-cytokeratin positive cells is associated with survival in patients with advanced non-small cell lung cancer.</title>
            <link>http://www.medworm.com/index.php?rid=2558739&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19563666%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: This study suggests that CXCR4 may be a prognostic marker in NSCLC, and provides hypothesis-generating results, which may be important in determining metastatic potential. In future studies, we will prospectively evaluate the prognostic significance of pan-cytokeratin/CXCR4+ cells, and determine the mechanisms involved in the regulation of CXCR4 expression on tumor cells in a larger patient population.
    PMID: 19563666 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558739</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558739</guid>        </item>
        <item>
            <title>Cholesterol sensor ORP1L contacts the ER protein VAP to control Rab7-RILP-p150Glued and late endosome positioning.</title>
            <link>http://www.medworm.com/index.php?rid=2558720&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19564404%26dopt%3DAbstract</link>
            <description>Authors: Rocha N, Kuijl C, van der Kant R, Janssen L, Houben D, Janssen H, Zwart W, Neefjes J
    Late endosomes (LEs) have characteristic intracellular distributions determined by their interactions with various motor proteins. Motor proteins associated to the dynactin subunit p150(Glued) bind to LEs via the Rab7 effector Rab7-interacting lysosomal protein (RILP) in association with the oxysterol-binding protein ORP1L. We found that cholesterol levels in LEs are sensed by ORP1L and are lower in peripheral vesicles. Under low cholesterol conditions, ORP1L conformation induces the formation of endoplasmic reticulum (ER)-LE membrane contact sites. At these sites, the ER protein VAP (VAMP [vesicle-associated membrane protein]-associated ER protein) can interact in trans with the Rab7-RILP com...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558720</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558720</guid>        </item>
        <item>
            <title>Homodimerization Controls the FGF9 Subfamily's Receptor Binding and Heparan Sulfate Dependent Diffusion in the Extracellular Matrix.</title>
            <link>http://www.medworm.com/index.php?rid=2558716&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19564416%26dopt%3DAbstract</link>
            <description>Authors: Kalinina J, Byron SA, Makarenkova HP, Olsen SK, Eliseenkova AV, Larochelle WJ, Dhanabal M, Blais S, Ornitz DM, Day LA, Neubert TA, Pollock PM, Mohammadi M
    Uncontrolled fibroblast growth factor (FGF) signaling can lead to human diseases necessitating multiple layers of self-regulatory control mechanisms to keep its activity in check. Herein we demonstrate that FGF9 and FGF20 ligands undergo a reversible homodimerization, occluding their key receptor binding sites. To test the role of dimerization in ligand autoinhibition we introduced structure-based mutations into the dimer interfaces of FGF9 and FGF20. The mutations weakened the ability of the ligands to dimerize, effectively increasing the concentrations of monomeric ligands capable of binding and activating their cognate FG...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558716</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558716</guid>        </item>
        <item>
            <title>Building a Protocol Expressway: The Case of Mayo Clinic Cancer Center.</title>
            <link>http://www.medworm.com/index.php?rid=2558714&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19564529%26dopt%3DAbstract</link>
            <description>CONCLUSION: DMAIC framework combined with Lean methodologies is an effective tool to structure the definition, planning, analysis, and implementation of significant process changes.
    PMID: 19564529 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558714</comments>
            <pubDate>Sun, 28 Jun 2009 23:00:00 +0100</pubDate>
            <guid isPermaLink="false">2558714</guid>        </item>
        <item>
            <title>Impact of Graphic and Text Warnings on Cigarette Packs: Findings from Four Countries over Five Years.</title>
            <link>http://www.medworm.com/index.php?rid=2558777&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19561362%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Australia's new health warnings increased reactions that are prospectively predictive of cessation activity. Warning size increases warning effectiveness and graphic warnings may be superior to text-based warnings. While there is partial wear-out in the initial impact associated with all warnings, stronger warnings tend to sustain their effects for longer. These findings support arguments for governments to exceed minimum FCTC requirements on warnings.
    PMID: 19561362 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2558777</comments>
            <pubDate>Sat, 27 Jun 2009 23:00:00 +0100</pubDate>
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        <item>
            <title>Cancer Control; +2969 new citations</title>
            <link>http://www.medworm.com/index.php?rid=2530543&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fsites%2Fentrez%3Fcmd%3DSearch%26db%3Dpubmed%26term%3D%28%28%28Cancer%2520Control%29%2520AND%2520%25222009%252F04%252F07%252003.43%2522%255BMHDA%255D%253A%25222009%252F06%252F27%252010.35%2522%255BMHDA%255D%29%29%2520NOT%2520%28%28%2520%28%28%28%2522Cancer%2520Control%2522%255BTIAB%255D%2520OR%2520%28%2522cancer%2522%255BTIAB%255D%2520AND%2520%2522control%2522%255BTIAB%255D%29%2520OR%2520%2522cancer%2520control%2522%255BTIAB%255D%29%29%29%2520AND%2520%25220001%2522%255BEDAT%255D%253A%25222009%252F04%252F07%252003.43%2522%255BEDAT%255D%29%29</link>
            <description>2969 new pubmed citations were retrieved for your search.
Click on the search hyperlink below to display the complete search results:

Cancer Control
These pubmed results were generated on 2009/06/27PubMed, a service of the National Library of Medicine, includes over 15 million 
citations for biomedical articles back to the 1950's.
These citations are from MEDLINE and additional life science journals. 
PubMed includes links to many sites providing full text articles and other related resources. (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2530543</comments>
            <pubDate>Sat, 27 Jun 2009 14:35:03 +0100</pubDate>
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        <item>
            <title>Safety of human papillomavirus (HPV)-16/18 AS04-adjuvanted vaccine for cervical cancer prevention: A pooled analysis of 11 clinical trials.</title>
            <link>http://www.medworm.com/index.php?rid=2198593&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19221517%26dopt%3DAbstract</link>
            <description>In conclusion, analysis of this large database shows the HPV-16/18 AS04-adjuvanted cervical cancer vaccine to have a favorable safety profile in women of all ages.
    PMID: 19221517 [PubMed - as supplied by publisher] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2198593</comments>
            <pubDate>Fri, 20 Feb 2009 13:57:19 +0100</pubDate>
            <guid isPermaLink="false">2198593</guid>        </item>
        <item>
            <title>Effects of potentised substances on growth rate of the water plant Lemna gibba L.</title>
            <link>http://www.medworm.com/index.php?rid=2155491&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19185263%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Lemna gibba may be considered as a suitable test organism for further studies on the efficacy of homeopathic potencies. Evidence accumulates, that adjacent potency levels may strongly differ in their biological activity. Potential consequences for therapeutical application might be worth investigating.
    PMID: 19185263 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2155491</comments>
            <pubDate>Wed, 04 Feb 2009 05:02:37 +0100</pubDate>
            <guid isPermaLink="false">2155491</guid>        </item>
        <item>
            <title>National audit of provision of MRI services 2006/07.</title>
            <link>http://www.medworm.com/index.php?rid=2155490&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19185658%26dopt%3DAbstract</link>
            <description>Authors: Barter S, Drinkwater K, Remedios D
    In 2003 the Royal College of Radiologists Clinical Radiology Audit Sub-Committee began an audit process evaluating the standards of provision of magnetic resonance imaging (MRI) services. This was prompted by the publication of the 2002 Audit Commission Report, which had identified that lack of MRI provision was responsible for more than half of the total waiting times for diagnostic imaging investigations. The audit found that the time from request to report did not meet the standard for cancer staging examinations, but nationally, was within the target set for routine orthopaedic examinations. However, national mean waiting times were longer than recommended for both cancer and orthopaedic MRI. Since then, there has been massive investment ...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2155490</comments>
            <pubDate>Wed, 04 Feb 2009 05:02:33 +0100</pubDate>
            <guid isPermaLink="false">2155490</guid>        </item>
        <item>
            <title>Assessment of selection bias in clinic-based populations of childhood cancer survivors: a report from the childhood cancer survivor study.</title>
            <link>http://www.medworm.com/index.php?rid=2149034&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D18989878%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Applying chronic condition prevalence estimates from a clinical population to the general population of childhood cancer survivors must be undertaken with caution. Survivorship research must maintain a high level of scientific rigor to ensure that results reported in the literature are interpreted within the appropriate context.
    PMID: 18989878 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2149034</comments>
            <pubDate>Sun, 01 Feb 2009 04:23:21 +0100</pubDate>
            <guid isPermaLink="false">2149034</guid>        </item>
        <item>
            <title>Targeting 11q23 positive acute leukemia cells with high molecular weight-melanoma associated antigen-specific monoclonal antibodies.</title>
            <link>http://www.medworm.com/index.php?rid=2143853&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D18677475%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: HMW-MAA-specific mAb down-regulated P-Pyk2 expression and enhanced the anti-proliferative effect of cytarabine in vitro, but had no detectable effect on survival or growth of leukemia cells in vivo. Whether the HMW-MAA-specific mAb can be used as carriers of toxins or chemotherapeutic agents against 11q23-acute leukemia remains to be determined.
    PMID: 18677475 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2143853</comments>
            <pubDate>Fri, 30 Jan 2009 04:26:13 +0100</pubDate>
            <guid isPermaLink="false">2143853</guid>        </item>
        <item>
            <title>LCA5, a Rare Genetic Cause of Leber Congenital Amaurosis in Koreans.</title>
            <link>http://www.medworm.com/index.php?rid=2140498&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19172513%26dopt%3DAbstract</link>
            <description>Conclusions: This result shows that mutation in LCA5 is likely to be a rare genetic cause in Koreans and suggests that further investigation to identify other causative genes is necessary in Koreans.
    PMID: 19172513 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2140498</comments>
            <pubDate>Thu, 29 Jan 2009 05:40:33 +0100</pubDate>
            <guid isPermaLink="false">2140498</guid>        </item>
        <item>
            <title>A nursing quality program driven by evidence-based practice.</title>
            <link>http://www.medworm.com/index.php?rid=2136800&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19167551%26dopt%3DAbstract</link>
            <description>This article reviews the activities of the Best Practice Council and the projects of teams assigned to study best practice in (1) preventing bloodstream infection (related to central lines), (2) preventing patient falls, (3) assessing and preventing pressure ulcers, and (4) ensuring good hand-off communication.
    PMID: 19167551 [PubMed - in process] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2136800</comments>
            <pubDate>Wed, 28 Jan 2009 04:31:34 +0100</pubDate>
            <guid isPermaLink="false">2136800</guid>        </item>
        <item>
            <title>Novel genetic variants in microRNA genes and familial breast cancer.</title>
            <link>http://www.medworm.com/index.php?rid=2130646&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19048628%26dopt%3DAbstract</link>
            <description>Authors: Shen J, Ambrosone CB, Zhao H
    MicroRNA (miRNA) plays an important role in tumorigenesis, but whether miRNA is a cancer predisposition factor or not is still unknown. Considering the fact that miRNA regulates a number of tumor suppressor genes (TSGs) and oncogenes, genetic variations in miRNA genes could affect the levels of expression of TSGs or oncogenes and, thereby, cancer risk. To test this hypothesis, we screened genetic variants in 17 selected miRNA genes, which are predicted to regulate key breast cancer genes, in 42 patients with familial breast cancer. Seven novel genetic variants were observed in 7 primary or precursor miRNA genes. Among them, 1 rare variant in the precursor of miR-30c-1 and 1 rare variant in the primary precursor of miR-17 were only observed in nonca...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2130646</comments>
            <pubDate>Sun, 25 Jan 2009 04:16:28 +0100</pubDate>
            <guid isPermaLink="false">2130646</guid>        </item>
        <item>
            <title>A simple and effective method for cancer immunotherapy by inactivated allogeneic leukocytes infusion.</title>
            <link>http://www.medworm.com/index.php?rid=2130645&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19048630%26dopt%3DAbstract</link>
            <description>Authors: Su X, Guo S, Zhou C, Wang D, Ma W, Zhang S
    Allogeneic mixed leukocytes reaction has been reported to activate vast numbers of T lymphocytes and produce large amounts of type 1 cytokines that are linked to an initiation of antitumor immunity. Using poor immunogeneic B16-F10 and Lewis lung carcinoma (LLC) tumor model, we evaluated the effects of inactivated allogeneic leukocytes infusion (ALI) on the generation of antitumor immune response, as well as its effect on the primary and metastatic tumor. Allogeneic response promoted the generation of both specific and nonspecific antitumor immunity in an in vitro mixed lymphocytes-tumor cell culture system. Introveinous infusion of mitotically inactivated allogeneic leukocytes resulted in increased type-1 cytokines (including IL-2 and...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2130645</comments>
            <pubDate>Sun, 25 Jan 2009 04:16:25 +0100</pubDate>
            <guid isPermaLink="false">2130645</guid>        </item>
        <item>
            <title>Increased frequency of hematopoietic malignancies in relatives of patients with lymphoid neoplasms: a French case-control study.</title>
            <link>http://www.medworm.com/index.php?rid=2130644&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19058175%26dopt%3DAbstract</link>
            <description>Authors: Villeneuve S, Orsi L, Monnereau A, Berthou C, Fenaux P, Marit G, Soubeyran P, Huguet F, Milpied N, Leporrier M, Hemon D, Troussard X, Clavel J
    Lymphoid neoplasms (LNs), including non-Hodgkin's lymphoma (NHL), Hodgkin's lymphoma (HL), lymphoproliferative syndrome (LPS) and multiple myeloma (MM), are among the most frequent cancers ( approximately 17,000 new cases per year in France), after those related to smoking. LNs were investigated using the data from the ENGELA study. ENGELA is a multicenter hospital-based case-control study that was carried out in France over the period September 2000-December 2004. In all, 822 cases (397 NHL, 149 LH, 168 SLP and 108 MM) and 752 controls were included and described 5,481 and 5,188 first-degree relatives, respectively. A positive associat...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2130644</comments>
            <pubDate>Sun, 25 Jan 2009 04:16:22 +0100</pubDate>
            <guid isPermaLink="false">2130644</guid>        </item>
        <item>
            <title>Global expression profiling of sex cord stromal tumors from Men1 heterozygous mice identifies altered TGF-beta signaling, decreased Gata6 and increased Csf1r expression.</title>
            <link>http://www.medworm.com/index.php?rid=2130643&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19058182%26dopt%3DAbstract</link>
            <description>Authors: Mould AW, Duncan R, Serewko-Auret M, Loffler KA, Biondi C, Gartside M, Kay GF, Hayward NK
    Heterozygous disruption of the Men1 gene predisposes mice to the development of multiple endocrine tumors, accurately mimicking the human MEN1 cancer predisposition syndrome. Additionally, Men1(+/-) mice frequently develop sex cord adenomas. The mechanism underlying the susceptibility of these mice to sex cord tumor development has not been fully determined, but data suggest it may involve transcriptional regulation of key growth promoting/repressing genes. To identify potential menin-regulated genes that may be important for tumor suppression in sex cord cells, we compared the global gene expression profiles of testis and ovary adenomas with other endocrine tumors of the pancreas and pit...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2130643</comments>
            <pubDate>Sun, 25 Jan 2009 04:16:18 +0100</pubDate>
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        <item>
            <title>Hypertension is an independent predictor of survival disparity between African-American and white breast cancer patients.</title>
            <link>http://www.medworm.com/index.php?rid=2130642&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19058216%26dopt%3DAbstract</link>
            <description>In conclusion, hypertension has prognostic significance in relation to survival disparity between African-American and white breast cancer patients. If our findings are replicated in contemporary cohorts, it may be necessary to include hypertension in the Charlson Comorbidity Index and other comorbidity measures.
    PMID: 19058216 [PubMed - indexed for MEDLINE] (Source: Cancer Control)</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2130642</comments>
            <pubDate>Sun, 25 Jan 2009 04:16:14 +0100</pubDate>
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        <item>
            <title>Chromosomes 6 and 18 induce neoplastic suppression in epithelial ovarian cancer cells.</title>
            <link>http://www.medworm.com/index.php?rid=2130641&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19058220%26dopt%3DAbstract</link>
            <description>Authors: Dafou D, Ramus SJ, Choi K, Grun B, Trott DA, Newbold RF, Jacobs IJ, Jones C, Gayther SA
    Metaphase comparative genomic hybridisation (CGH) studies indicate that chromosomes 4, 5, 6, 13, 14, 15 and 18 are frequently deleted in primary ovarian cancers (OCs). Therefore we used microcell-mediated chromosome transfer (MMCT) to establish the functional effects of transferring normal copies of these chromosomes into 2 epithelial OC cell lines (TOV112D and TOV21G). The in vitro neoplastic phenotype (measured as anchorage dependent and independent growth and invasion) was compared between recipient OC cell lines and multiple MMCT hybrids. Chromosomes 6 and 18 showed strong evidence of functional, neoplastic suppression for multiple hybrids in both cell lines. We also found evidence in 1...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2130641</comments>
            <pubDate>Sun, 25 Jan 2009 04:16:10 +0100</pubDate>
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        <item>
            <title>Dietary administration of the licorice flavonoid isoliquiritigenin deters the growth of MCF-7 cells overexpressing aromatase.</title>
            <link>http://www.medworm.com/index.php?rid=2130640&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19065667%26dopt%3DAbstract</link>
            <description>Authors: Ye L, Gho WM, Chan FL, Chen S, Leung LK
    Licorice is the sweet-tasting rhizomes of a bean plant and is quite commonly used in Western countries for culinary purposes, while it is a medicinal herb in China. Many flavonoids have been isolated from licorice, and their pharmacological properties may be applicable in preventive medicine. Overexposure to estrogen has been implicated in the etiology of breast cancer, and cytochrome P450 (CYP) 19 enzyme, or aromatase, catalyzes the rate-limiting reaction. Phytocompounds that are able to inhibit this enzyme may potentially suppress breast cancer development. In the present study the licorice flavonoid isoliquiritigenin (ILN) was shown to be an aromatase inhibitor in recombinant protein and MCF-7 cells stably transfected with CYP19 (MCF-...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2130640</comments>
            <pubDate>Sun, 25 Jan 2009 04:16:06 +0100</pubDate>
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        <item>
            <title>The epidemiology of neuroblastoma: a review.</title>
            <link>http://www.medworm.com/index.php?rid=2127811&amp;cid=s_31096_6_f&amp;fid=31096&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D19159399%26dopt%3DAbstract</link>
            <description>Authors: Heck JE, Ritz B, Hung RJ, Hashibe M, Boffetta P
    Neuroblastoma is the most common tumour in children less than 1 year of age. The goal of this review was to summarise the existing epidemiological research on risk factors for neuroblastoma. A comprehensive search of the literature was undertaken using PubMed for epidemiological studies on neuroblastoma risk factors. We ascertained 47 articles which examined the risk factors. Ten studies employed population-based case-control designs; six were hospital-based case-control studies; two were cohort studies; and five employed ecological designs. Studies ranged in size from 42 to 538 cases. Three studies showed evidence of an increased risk of disease with use of alcohol during pregnancy (OR range 1.1, 12.0). Protective effects were s...</description>
            <author>Cancer Control</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=2127811</comments>
            <pubDate>Sat, 24 Jan 2009 05:21:25 +0100</pubDate>
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