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        <title>Clinical Neurophysiology via MedWorm.com</title>
        <description>MedWorm.com provides a medical RSS filtering service. Over 6000 RSS medical sources are combined and output via different filters. This feed contains the latest items from the 'Clinical Neurophysiology' source.</description>
        <link><![CDATA[http://www.medworm.com/rss/search.php?qu=Clinical+Neurophysiology&t=Clinical+Neurophysiology&s=Search&f=source]]></link>
        <lastBuildDate>Wed, 08 Feb 2012 20:43:02 +0100</lastBuildDate>
        <item>
            <title>Automatic detection and analysis of the EEG sharp wave-slow wave patterns evoked by fluorinated inhalation anesthetics.</title>
            <link>http://www.medworm.com/index.php?rid=5664899&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22300687%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Synchronization criterion and adequately selected morphological features of &quot;slow wave&quot; were sufficient to achieve the high sensitivity and specificity of the method. SIGNIFICANCE: The monitoring of SWSW patterns is important in view of possible side effects of volatile anesthetics. The analysis of SWSW patterns' recruitment and morphology could be helpful in the diagnosis of the anesthesia effects on the CNS.
    PMID: 22300687 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5664899</comments>
            <pubDate>Tue, 31 Jan 2012 05:00:00 +0100</pubDate>
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            <title>sLORETA-qm for interictal MEG epileptic spike analysis: Comparison of location and quantity with equivalent dipole estimation.</title>
            <link>http://www.medworm.com/index.php?rid=5664900&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22296839%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: sLORETA-qm was closely correlated with ECD concerning point source location and quantity in analysis of the interictal epileptic spike. SIGNIFICANCE: sLORETA-qm is a reliable quantifiable method without arbitrariness for analysis of the interictal epileptic spike.
    PMID: 22296839 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5664900</comments>
            <pubDate>Mon, 30 Jan 2012 05:00:00 +0100</pubDate>
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        <item>
            <title>24. Basal ganglia and cerebellum in the motor timing prediction task: Effective connectivity</title>
            <link>http://www.medworm.com/index.php?rid=5636756&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007826%2Fabstract%3Frss%3Dyes</link>
            <description>Objective: Over the years, the cerebellum, the basal ganglia (BG) and other cortical regions have emerged as important structures dealing with various aspects of timing. We investigated the effective connectivity (EC) and its modulation by behavioral outcome in a network comprised of cerebellum, BG and SMA in group of patients with Parkinson’s disease (PD) and healthy controls. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636756</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:19 +0100</pubDate>
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        <item>
            <title>23. Anti-CV2 associated cerebellar degeneration and polyneuropathy</title>
            <link>http://www.medworm.com/index.php?rid=5636755&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007814%2Fabstract%3Frss%3Dyes</link>
            <description>Introduction: Paraneoplastic damage of the nervous system frequently precedes the detection of the malignancy. Tumors cells indicate the production of anti-CV2 antibodies; that presence is associated with cerebellar degeneration, polyneuropathy and uveitis. The symptoms may occur acutely or subacutely. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636755</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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        <item>
            <title>22. Wavelet transform based features for analysis of newborn behavioral states</title>
            <link>http://www.medworm.com/index.php?rid=5636754&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007802%2Fabstract%3Frss%3Dyes</link>
            <description>In this study we addressed the application of the wavelet transform (WT) for analysis of newborn behavioral states. Selection of the most appropriate parameters of the transform was performed and potential of its features was tested. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636754</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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            <title>21. Analysis of newborn EEG recorded at 40th week of postconceptional age</title>
            <link>http://www.medworm.com/index.php?rid=5636753&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007796%2Fabstract%3Frss%3Dyes</link>
            <description>In this paper we present results of computer processing of newborn polysomnographic (PSG) recordings.  For experiments we used data from 22 newborns, recorded at 40th week of postconceptional age, consisting of 8×EEG, EOG, EMG, ECG and PNG. For each recording, 6min of quiet sleep (QS) and active sleep (AS) were selected. These segments do not include physiological artifacts and can be regarded as representative for listed sleep stages. Data were collected, classified and selected by Karel Paul, MD. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636753</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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        <item>
            <title>20. Neural correlates of traffic educational campaign content evaluation</title>
            <link>http://www.medworm.com/index.php?rid=5636752&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007784%2Fabstract%3Frss%3Dyes</link>
            <description>This study compare differences in brain activation during watching the video-clips from the national traffic educational campaign in contrast to neutral videos in order to identify which areas of the brain are involved in detailed perception of these videos and detection of differences between them. Functional magnetic resonance imaging data were acquired in 15 healths 19- to 28-year-old male volunteer drivers. During fMRI scanning, clips from the campaign with a catastrophic end were presented together with control videos without dramatic ending to participants. General linear model implemented in SPM5 software was used for analysis. Immediately after fMRI scanning, the verb description, valence and arousal (by scale from 1 to 10) were evaluated from participants. The maximum of significa...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636752</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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        <item>
            <title>19. Default mode network and extrastriate visual resting state network in patients with Parkinson’s disease dementia</title>
            <link>http://www.medworm.com/index.php?rid=5636751&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007772%2Fabstract%3Frss%3Dyes</link>
            <description>Aims: Our aim was to evaluate two resting state networks (RSNs): the default mode network (DMN) and an extra-striate visual RSN (ESV-RSN) using the seed-based functional connectivity analysis of resting state data or deactivations in patients with Parkinson’s disease dementia (PDD) as compared with Parkinson’s disease (PD) without dementia and healthy controls (HC). (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636751</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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        <item>
            <title>18. Treatment-induced change of cortical activation: fMRI evidence of the central effect of BoNT-A</title>
            <link>http://www.medworm.com/index.php?rid=5636750&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007760%2Fabstract%3Frss%3Dyes</link>
            <description>The central mechanism of idiopathic dystonia is presumably localized within the brain although there has been no direct evidence for involvement of specific brain structures. Similarly, the exact cerebral correlate of post-stroke spasticity is unknown. We have performed a series of investigation with aiming to localize the potential central mechanisms. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636750</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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        <item>
            <title>17. Oscillations in the basal ganglia – Cognitive aspects</title>
            <link>http://www.medworm.com/index.php?rid=5636749&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007759%2Fabstract%3Frss%3Dyes</link>
            <description>Background: The aim of this work was to study high- frequency oscillations in the subthalamic nucleus and internal pallidum linked to cognitive and motor activities using event-related de/synchronizations (ERD/S) methodology. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636749</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
            <guid isPermaLink="false">5636749</guid>        </item>
        <item>
            <title>16. Influence of water on the character of a surface EMG signal (WaS-EMG)</title>
            <link>http://www.medworm.com/index.php?rid=5636748&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007747%2Fabstract%3Frss%3Dyes</link>
            <description>We present our personal experience with Water Surface Electromyography (WaS-EMG), and we compare our results with sources in the literature.  Methodology: We started with a series of experiments conducted in the Swimming Sports Laboratory at FTVS, UK. We registered muscle activity with the help of a telemetric surface EMG instrument TelemyoMini 16 by Neurodata. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636748</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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            <title>15. Intraoperative monitoring of evoked potentials in scoliosis surgery at University Hospital Brno</title>
            <link>http://www.medworm.com/index.php?rid=5636747&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007735%2Fabstract%3Frss%3Dyes</link>
            <description>Aim: The aim of our study was the evaluation of sensitivity and specificity of intraoperative monitoring (IOM) of evoked potentials (EP) in scoliosis and developmental spinal deformities surgery in our hospital. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636747</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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        <item>
            <title>14. Electrophysiological intraoperative monitoring in vestibular schwannoma surgery</title>
            <link>http://www.medworm.com/index.php?rid=5636746&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007723%2Fabstract%3Frss%3Dyes</link>
            <description>We present the results of 48 patients, who underwent surgery of vestibular schwannomas (VS) from 2001 to 2011, with electrophysiological monitoring.  Material and methods: Fifty operations (two re-resection). Monitoring used: EMG in identification and monitoring of CN VII, auditory evoked potentials (BAEP, ABR), motor evoked potentials (MEP). House–Brackmann scale (HB) was used in evaluation of postoperative deficit of CN VII. To determine the hearing disability we used audiometry or AAO-HNS score. We detected preoperative anacusis (respectively D score in the AAO-HNS classification) by 82% of patiens. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636746</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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        <item>
            <title>13. Cutaneous silent periods in multiple system atrophy</title>
            <link>http://www.medworm.com/index.php?rid=5636745&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007711%2Fabstract%3Frss%3Dyes</link>
            <description>Objective: The cutaneous silent period (CSP) is a spinal inhibitory reflex mediated by A-delta fibers. Prolonged CSPs have previously been reported in patients with restless legs syndrome (RLS) and idiopathic Parkinson’s disease (IPD). Dopaminergic medication normalized the CSP alteration, concurring with dopaminergic influence on CSPs. To date, CSPs have not been extensively studied in patients with multiple system atrophy (MSA). (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636745</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:18 +0100</pubDate>
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            <title>12. Stance with head extension – Posturographic test</title>
            <link>http://www.medworm.com/index.php?rid=5636744&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS138824571100770X%2Fabstract%3Frss%3Dyes</link>
            <description>Stance with head extension represents significant stress on postural system. Posturographic studies on healthy young subjects indicate a reduction of stability during stance with head extension when compared to direct head position (Jaskson and Epstein, 1991). However, some patients with peripheral vestibular deficit improve postural stability during stance with head extension when comparing to stance with direct head position (Norris, 1995). Based on these data, we included stance with head extension to our posturographic examination. We compared measurements in stance with head extended (Ex) with parameters measured during the stance with direct head position (Pr). Both situations measured with eyes closed to eliminate the influence of visual information. For comparison of individual par...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636744</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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        <item>
            <title>11. Pilot attempt to monitor Graves’ ophthalmopathy with pattern-reversal and motion-onset VEPS</title>
            <link>http://www.medworm.com/index.php?rid=5636743&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007693%2Fabstract%3Frss%3Dyes</link>
            <description>Objective: The aim of this pilot study was to evaluate efficacy of visual evoked potentials (VEPs) in monitoring of visual functions during the high-dose intravenous steroid pulse therapy and apheresis treatment of severe Graves’ ophthalmopathy (GO). (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636743</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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        <item>
            <title>10. Electrophysiological correlate of auditory processing initiative on an unconscious level in the nucleus subthalamicus</title>
            <link>http://www.medworm.com/index.php?rid=5636742&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007681%2Fabstract%3Frss%3Dyes</link>
            <description>Introduction: In the protocol of mismatch negativity (MMN) the frequent and target auditory stimuli are presented to the patient while his attention is excluded. These evoked potentials (EP) are considered to correlate with automatic working memory and a primitive intelligence on an unconscious level. The aim of the study was to elucidate whether subthalamicus nucleus (STN) is involved in the processing of audio signals. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636742</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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        <item>
            <title>9. Origins of VEP response suppression during prolonged visual motion stimulation</title>
            <link>http://www.medworm.com/index.php?rid=5636741&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS138824571100767X%2Fabstract%3Frss%3Dyes</link>
            <description>Response suppression of motion-onset visual evoked potentials (M-VEPs) can be measured during repetitive stimulation (Kremlacek et al. Doc Ophthalmol 115: 95–103; 2007). To reveal the origins of this suppression we evaluated the latency jittering and the effect of its correction on the amplitudes of dominant peaks P1, N2 and P3. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636741</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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        <item>
            <title>8. Sulcus temporalis superior</title>
            <link>http://www.medworm.com/index.php?rid=5636740&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007668%2Fabstract%3Frss%3Dyes</link>
            <description>Superior temporal sulcus (STS) is a brain structure, which has been related to many cognitive functions recently. It can be presumed that it is a part of many cognitive functions and that in particular situation, its relationship with other brain areas is relevant. Our work aimed to its effective connectivity during processing of potentially relevant unusual events. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636740</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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            <title>7. The inter-individual differences and the test–retest reliability of a EEG signal</title>
            <link>http://www.medworm.com/index.php?rid=5636739&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007656%2Fabstract%3Frss%3Dyes</link>
            <description>The majority of EEG studies in the area of cognitive neuroscience are administered without any retest. The reliability of these studies should be questionable. The analysis of test–retest reliability in the Näpflin et al. study (2008) revealed high correlation between measurements. We did similar comparison and we also tested inter-individual differences and differences between scalp projections and source activity of independent components. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636739</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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            <title>6. Changes in strategy during the mental rotation task and its correlation with EEG</title>
            <link>http://www.medworm.com/index.php?rid=5636738&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007644%2Fabstract%3Frss%3Dyes</link>
            <description>Shepard and Metzler (1971) showed that the mental rotation is performed with approximately steady rate. Further studies were concerned whether there is a relation between reaction times and stimuli complexity (both “piecemeal” theory and propositional theory predict decrease in the reaction times due to the increasing stimulus difficulty). Relation was not approved. Folk and Luce (1987) discuss possible problems in the methodology. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636738</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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            <title>5. High prevalence of restless legs syndrome does not correlates with brain damage</title>
            <link>http://www.medworm.com/index.php?rid=5636737&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007632%2Fabstract%3Frss%3Dyes</link>
            <description>Introduction: Restless legs syndrome (RLS) has a prevalence of approx. 10% in the general population. Recently, multiple sclerosis (MS) was identified as a common cause for secondary RLS with prevalence ranging between 13.3% and 37.5%. The aim of our study was to evaluate the prevalence of RLS in MS and to compare the extent of brain damage between patients with and without RLS using magnetic resonance imaging (MRI). (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636737</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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            <title>4. Sleep apnoea syndrome in acute stroke patients – Results of the first phase prospective study</title>
            <link>http://www.medworm.com/index.php?rid=5636736&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007620%2Fabstract%3Frss%3Dyes</link>
            <description>We present in 1st phase demographic data type of OSA, time onset and type of stroke of the 54 patients. Polygraphic finding is being performed up to 72h and 7days after stroke. Negative results of an apnea/hypopnea index (AHI) (up to AHI=4) form control group. We use basic statistic methods for quantitative and qualitative data, statistic hypothesis measured by parametric tests at 5% level of significance. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636736</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:17 +0100</pubDate>
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            <title>REM sleep and immunogenetic research in narcolepsy</title>
            <link>http://www.medworm.com/index.php?rid=5636735&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007619%2Fabstract%3Frss%3Dyes</link>
            <description>The discovery of REM (Rapid Eye Movement) sleep laid the foundations for modern development of sleep medicine. Polysomnographic recording enabled to make distinction between different sleep stages, and on this basis, the criteria for different sleep disorders including narcolepsy came into being. The main feature of narcolepsy rests in increased REM sleep penetration verified by the multiple sleep latency test (MSLT). In clinical terms, the disease is marked by imperative attacks of daytime sleep and usually also by cataplexy – sudden loss of muscle tone evoked by emotive experience. The first indication of the immunogenetic basis of narcolepsy came with the discovery of its link to the HLA-DR2 (15) subgroup and with the subsequently revealed connection between narcolepsy–cataplexy and...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636735</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:16 +0100</pubDate>
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        <item>
            <title>2. The blink reflex: Long known, but still fascinating</title>
            <link>http://www.medworm.com/index.php?rid=5636734&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007607%2Fabstract%3Frss%3Dyes</link>
            <description>The blink reflex is routinely used in clinical neurophysiology to characterize the functional integrity of the trigemino-facial reflex pathway covering supraorbital nerve, pons, medulla oblongata, and facial nerve. Recent findings of unconditioned blink reflex alterations, as well as changes in blink reflex modulation by subthreshold prepulse stimuli or suprathreshold double pulse stimulation, in patients with spinal cord injury or chronic pain syndromes, e.g. fibromyalgia, suggest plastic changes at the brainstem level in these conditions. The blink reflex and its modulation also serve to document acute pharmacological effects, e.g. following intrathecal bolus application of the GABA-ergic drug baclofen, thus concurring with an antispastic action of baclofen at the brainstem level. Finall...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636734</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:16 +0100</pubDate>
            <guid isPermaLink="false">5636734</guid>        </item>
        <item>
            <title>1. Neuromodulation of motor learning in health and after stroke</title>
            <link>http://www.medworm.com/index.php?rid=5636733&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007590%2Fabstract%3Frss%3Dyes</link>
            <description>Ischemic cerebral stroke is the leading cause of long-term disability among adults in industrialized countries. A fundamental but still not sufficiently solved question is how to improve disability after stroke. One approach to this unmet medical need is to explore ways of modifying synaptic plasticity and learning along a translational pathway from basic neuroscience over healthy human subjects to stroke patients [Ziemann et al. 2006, Neurorehabil Neural Repair; 20: 243–51]. This presentation will review evidence for this translational pathway on how pharmacological treatment or brain stimulation modulates synaptic plasticity (long-term plasticity, LTP) and motor learning, which is thought to be an LTP-dependent process. The focus will be on drugs that are agonists or antagonists at the...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636733</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:16 +0100</pubDate>
            <guid isPermaLink="false">5636733</guid>        </item>
        <item>
            <title>Preliminary Announcement</title>
            <link>http://www.medworm.com/index.php?rid=5636708&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245712000351%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636708</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:10 +0100</pubDate>
            <guid isPermaLink="false">5636708</guid>        </item>
        <item>
            <title>Editorial Board</title>
            <link>http://www.medworm.com/index.php?rid=5636707&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245712000296%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636707</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:10 +0100</pubDate>
            <guid isPermaLink="false">5636707</guid>        </item>
        <item>
            <title>Contents</title>
            <link>http://www.medworm.com/index.php?rid=5636706&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245712000284%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636706</comments>
            <pubDate>Sat, 28 Jan 2012 16:42:10 +0100</pubDate>
            <guid isPermaLink="false">5636706</guid>        </item>
        <item>
            <title>Lateralized movement-related potential amplitudes differentiate between schizophrenia/schizoaffective disorder and major depression.</title>
            <link>http://www.medworm.com/index.php?rid=5645332&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22289671%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Deficits in focal motor cortex activation during movement execution may reflect rather schizophrenia-specific deficits in fronto-striatal circuits. A general lack of drive and depressed mood did not alter the degree of lateralization of motor activation during movement execution. SIGNIFICANCE: Lateralization of movement-related potentials could differentiate psychotic from non-psychotic disorders on the group level.
    PMID: 22289671 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5645332</comments>
            <pubDate>Sat, 28 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5645332</guid>        </item>
        <item>
            <title>Conductive neuromagnetic fields in the lumbar spinal canal.</title>
            <link>http://www.medworm.com/index.php?rid=5645333&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22285173%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: We successfully measured ascending neuromagnetic fields originating at the nerve root and the cauda equina with high spatial resolution. Future studies will determine whether neuromagnetic field measurement of the lumbar spine can be a useful diagnostic method for the identification of the disordered site in spinal nerves. SIGNIFICANCE: We successfully measured neuromagnetic fields in the lumbar spinal canal, which have previously been difficult to verify. Future studies will determine whether neuromagnetic field measurement of the lumbar spine can be a useful diagnostic method for identifying disorders of spinal nerves.
    PMID: 22285173 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5645333</comments>
            <pubDate>Fri, 27 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5645333</guid>        </item>
        <item>
            <title>Bidirectional modulation of sensory cortical excitability by quadripulse transcranial magnetic stimulation (QPS) in humans.</title>
            <link>http://www.medworm.com/index.php?rid=5645334&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22280937%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: QPSs over motor cortices modulated the S1 cortical excitability (heterotopic effects). Mutual connections between dPMC or M1 and S1 might be responsible for these modulations. SIGNIFICANCE: QPSs induced heterotopic LTP or LTD-like cortical excitability changes.
    PMID: 22280937 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5645334</comments>
            <pubDate>Tue, 24 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5645334</guid>        </item>
        <item>
            <title>Upper-extremity H-reflex measurement post-stroke: Reliability and inter-limb differences.</title>
            <link>http://www.medworm.com/index.php?rid=5645336&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22277759%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: These results establish reliability of FCR H-reflexes in stroke and non-disabled participants. SEM and SRD measurements can be used to interpret recovery patterns and longitudinal effects of therapeutic interventions. SIGNIFICANCE: FCR H-reflex amplitude and slope can be reliably measured and used to investigate neurophysiological mechanisms of motor recovery post-stroke.
    PMID: 22277759 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5645336</comments>
            <pubDate>Mon, 23 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5645336</guid>        </item>
        <item>
            <title>The utility of amplitude-integrated EEG and NIRS measurements as indices of hypoxic ischaemia in the newborn pig.</title>
            <link>http://www.medworm.com/index.php?rid=5645335&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22277760%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Parameters measured via aEEG and NIRS displayed different time profiles during and following the HI event. SIGNIFICANCE: These results highlight the potential advantage of using aEEG and NIRS in conjunction to monitor neonatal brain function, and provide an objective and rigorous method for the characterisation of cerebral function both during and following HI insults.
    PMID: 22277760 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5645335</comments>
            <pubDate>Mon, 23 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5645335</guid>        </item>
        <item>
            <title>Somatosensory evoked potentials are of additional prognostic value in certain patterns of brain injury in term birth asphyxia.</title>
            <link>http://www.medworm.com/index.php?rid=5627767&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22264394%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: SEPs are of additional prognostic value after term birth asphyxia. SIGNIFICANCE: In certain patterns of postasphyxial neonatal brain injury like asymmetrical watershed lesions and symmetrical white matter injury, EPs are complementary to information obtained from cEEG and MRI for prognostication.
    PMID: 22264394 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5627767</comments>
            <pubDate>Wed, 18 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5627767</guid>        </item>
        <item>
            <title>Selective autonomic failure: Ross syndrome.</title>
            <link>http://www.medworm.com/index.php?rid=5627766&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22264395%26dopt%3DAbstract</link>
            <description>Authors: Macefield VG
    PMID: 22264395 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5627766</comments>
            <pubDate>Wed, 18 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5627766</guid>        </item>
        <item>
            <title>Introducing a novel approach of network oriented analysis of ERPs, demonstrated on adult attention deficit hyperactivity disorder.</title>
            <link>http://www.medworm.com/index.php?rid=5627768&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22261156%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: These results suggest that the ADHD group was more characterized by the process of exerting attention in the early monitoring stages of the No-go signal while the controls were more characterized by the process of inhibiting the response to that signal. SIGNIFICANCE: The BNA method may provide both diagnostic and drug development tools for use in diverse neurological disorders.
    PMID: 22261156 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5627768</comments>
            <pubDate>Tue, 17 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5627768</guid>        </item>
        <item>
            <title>The bilateral movement condition facilitates maximal but not submaximal paretic-limb grip force in people with post-stroke hemiparesis.</title>
            <link>http://www.medworm.com/index.php?rid=5608196&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22248812%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The bilateral condition facilitates paretic limb grip force at maximal, but not submaximal levels. SIGNIFICANCE: In some people with post-stroke hemiparesis, the paretic limb may benefit from bilateral training with high force requirements.
    PMID: 22248812 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5608196</comments>
            <pubDate>Sat, 14 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5608196</guid>        </item>
        <item>
            <title>18. Where is my hand? The blink reflex evoked by hand stimulation is increased when the hand enters the peripersonal space surrounding the face</title>
            <link>http://www.medworm.com/index.php?rid=5579137&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004871%2Fabstract%3Frss%3Dyes</link>
            <description>Background: Electrical stimulation of the hand elicits a blink reflex that has the features of a startle response. In a Sherringtonian sense, this is a defensive reflex, probably mediated by a brainstem circuit. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579137</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579137</guid>        </item>
        <item>
            <title>17. Masseter reflex abnormalities with meso-diencephalic lesions</title>
            <link>http://www.medworm.com/index.php?rid=5579136&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS138824571100486X%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: According to current knowledge, unilateral masseter reflex (MassR) abnormalities indicate ipsilesional brainstem lesions between the levels of 5th and 3rd nerve nuclei, provided that 5th nerve functions are intact. We re-discuss possible suprasegmental influences on the MassR based on occasional observations of single patients with unilateral meso-diencephalic lesions and ipsi- or contralesional MassR abnormalities. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579136</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579136</guid>        </item>
        <item>
            <title>16. Increasing clinical variety of brainstem infarcts</title>
            <link>http://www.medworm.com/index.php?rid=5579135&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004858%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: Classical crossed brainstem syndromes and lacunar brainstem syndromes are well known consequences of brainstem infarcts. With the widespread use of magnetic resonance imaging (MRI), an increasing variety of clinical manifestations of brainstem infarcts were recognized. This prompted us to review this topic. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579135</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579135</guid>        </item>
        <item>
            <title>15. Functional mapping of monaural auditory brainstem responses</title>
            <link>http://www.medworm.com/index.php?rid=5579134&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004846%2Fabstract%3Frss%3Dyes</link>
            <description>Introduction: The auditory brainstem circuits on the basis of rat and cat experiments include the dorsal and ventral cochlear nuclei, the superior and medial olivary nuclei, the lateral lemniscal nucleus and the inferior colliculus. The aim of this fMRI study was to determine whether an auditory broadband stimulus which covers all frequencies of human hearing can induce significant ponto-medullary BOLD responses in humans to reveal the entire auditory pathway after monaural stimulation. If so could one also demonstrate a right ear preference for the processing of monaural signals in right-handers? (). (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579134</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579134</guid>        </item>
        <item>
            <title>14. Vestibular modulation of theta band oscillations in human pedunculopontine nucleus</title>
            <link>http://www.medworm.com/index.php?rid=5579133&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004834%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: The pedunculopontine nucleus (PPN) is a new deep brain stimulation (DBS) target, thought to be particularly for useful in ameliorating gait disturbance in Parkinson’s disease. Recent evidence shows a prominent theta (4–7Hz) rhythm in the PPN (Tsang et al., Neurology, 2010; Simon et al., J. Neurophysiol., 2010; Shimamoto et al., JNNP, 2010). Given that theta activity is modulated by vestibular signals elsewhere in the brain, e.g. hippocampus, (Shin, Synapse, 2010; Chen et al., Neuroimage, 2010), we assessed whether vestibular signals modulate PPN theta activity. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579133</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579133</guid>        </item>
        <item>
            <title>13. Trigemino-cervical reflex: Clinical and neuroradiological links</title>
            <link>http://www.medworm.com/index.php?rid=5579132&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004822%2Fabstract%3Frss%3Dyes</link>
            <description>Background: Trigemino-cervical reflex (TCR) is considered to be head protective reflex as well as head and neck postural responses. It is believed that TCR is mediated by polysynaptic brainstem neuronal pathways including trigeminal afferents, trigeminal nuclei and motor neurons of neck muscles. In an attempt to increase our knowledge about the mechanism and alteration of TCR, we studied the trigemino-cervical reflex and the conventional blink reflex (BR) in neurobehcet (NB), multiple sclerosis (MS), and ischemic stroke patients with brainstem involvement. We also recorded the blink reflex to study the association between two different trigeminal reflexes. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579132</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579132</guid>        </item>
        <item>
            <title>12. The importance of neurophysiological studies in the diagnosis of orthostatic tremor</title>
            <link>http://www.medworm.com/index.php?rid=5579131&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004810%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: Orthostatic tremor (OT) is a rare motor disorder characterized by unsteadiness on standing that characteristically decreases markedly on sitting or walking. The diagnosis depends on electromyographic confirmation of a typical 13–18Hz pattern. It has been assumed an OT generation in the bilaterally projecting brainstem centers regulating stance or tone. Our objective was to present two cases of orthostatic tremor and the neurophysiological studies performed for the diagnosis. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579131</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579131</guid>        </item>
        <item>
            <title>11. The effects of the StartReact paradigm in homonymous contralateral muscles</title>
            <link>http://www.medworm.com/index.php?rid=5579130&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004809%2Fabstract%3Frss%3Dyes</link>
            <description>Introduction: Ballistic movement execution is faster when the imperative signal (IS) is accompanied by a loud auditory stimulus (the StartReact effect). In focal tasks, excitation is increased in the muscles expected to react and decreased in others such as, for instance, the homonymous muscles of the contralateral side. The contrast between the increase and decrease of excitability in homonymous muscles is likely much more marked in SRT than in CRT. We reasoned that, if inhibition is not fully accomplished, some remaining activity would be seen in the muscles not to be responding in a StartReact paradigm using SRT and CRT. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579130</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579130</guid>        </item>
        <item>
            <title>10. Sensorimotor interaction and motor learning in facial muscles</title>
            <link>http://www.medworm.com/index.php?rid=5579129&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004792%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aim: Integration of sensory information with motor output is thought to be important in motor learning. In limb muscles, this is studied using the short afferent inhibition (SAI) paradigm, to assess sensorimotor interaction, and paired associative stimulation (PAS), to evaluate LTP-like plasticity. As far as we know, SAI and PAS paradigms have never been used in the territory of the cranial nerves. The present study was aimed at testing in normal subjects whether sensorimotor interaction and LTP-like plasticity can be observed in facial muscles as well as in limb muscles. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579129</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579129</guid>        </item>
        <item>
            <title>9. Atypical stimulus-sensitive myoclonus presenting abnormal sensory-motor integration in a case of congenital hemiatorophy of cerebellum</title>
            <link>http://www.medworm.com/index.php?rid=5579128&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004780%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: Some diseases with cerebellar pathology such as a celiac disease, are associated with stimulus-sensitive cortical reflex myoclonus. It has therefore been suggested that disordered output of the cerebellum may be one factor that increases the excitability of sensorimotor cortex leading to muscle jerks. Here we present a case of stimulus-sensitive myoclonus due to congenital cerebellar hemiatrophy. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579128</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579128</guid>        </item>
        <item>
            <title>8. The blink reflex to median nerve stimuli in brainstem disorders</title>
            <link>http://www.medworm.com/index.php?rid=5579127&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004779%2Fabstract%3Frss%3Dyes</link>
            <description>We examined the differences between TBR and SBR in relation to brainstem lesion topography (upper vs lower lesions and intra-axial vs extra-axial lesions). (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579127</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579127</guid>        </item>
        <item>
            <title>7. Deficient prepulse inhibition of blink reflex in narcolepsy-cataplexy: Evidence of pedunculopontine involvement</title>
            <link>http://www.medworm.com/index.php?rid=5579126&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004767%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: Hypocretin deficiency plays a major role in the pathophysiology of narcolepsy-cataplexy. In animal models, hypocretinergic projections to the pedunculopontine nucleus (PPN) are directly involved in muscle tone regulation mediating muscle atonia – a hallmark of cataplexy. We hypothesized that PPN function, tested with prepulse inhibition of the blink reflex, is altered in human narcolepsy-cataplexy. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579126</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579126</guid>        </item>
        <item>
            <title>6. Auditory startle reflex in cerebrovascular disease</title>
            <link>http://www.medworm.com/index.php?rid=5579125&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004755%2Fabstract%3Frss%3Dyes</link>
            <description>Objectives: Cerebrovascular diseases (CVD) are an important cause of motor disability and rehabilitation is only possible through a better understanding of the mechanisms underlying the recovery of motor functions after brain injury. In the presented study we aimed to investigate the properties and interactions of reticulospinal tract and corticospinal tract with the help of auditory startle reflex (ASR) which is an involuntary rapid contraction of muscles against a sudden sound and believed to play a role in conducting voluntary movements and transcranial magnetic stimulation (TMS) in CVD patients with different lesion sites and chronicity. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579125</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579125</guid>        </item>
        <item>
            <title>5. The effects of transcranial magnetic stimulation on vibratory-induced presynaptic inhibition of the soleus H reflex</title>
            <link>http://www.medworm.com/index.php?rid=5579124&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004743%2Fabstract%3Frss%3Dyes</link>
            <description>The soleus H-reflex can be used to assess the effect on the motoneuronal pool of conditioning volleys elicited by transcranial magnetic stimulation (TMS) in descending motor tracts. In healthy subjects, a single-pulse TMS induces two facilitatory phases of the soleus H-reflex: one at inter-stimulus intervals (ISI) between 5 and 30ms and the other at ISIs between 80 and 110ms. The first facilitatory phase is supposed to be due to summation of the excitatory input from the descending excitatory postsynaptic potentials (EPSP) generated by TMS and the Ia-afferents, while the mechanisms responsible for the second phase are less clear. To further investigate these mechanisms, we evaluated in 13 healthy subjects how the soleus H-reflex was modulated by conditioning single pulse TMS at ISIs from 0...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579124</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579124</guid>        </item>
        <item>
            <title>4. Trigemino cervical reflexes in spinal cord injury</title>
            <link>http://www.medworm.com/index.php?rid=5579123&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004731%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: Abnormal enhancement of polysynaptic brainstem reflexes including ASR, has been previously reported in patients with spinal cord injury (SCI) (Kumru, 2009). We aimed to investigate trigeminocervical reflex (TCR) in SCI since it may reflect alterations of the brainstem neuronal networks specifically trigeminocervical motoneurons in patients with SCI. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579123</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579123</guid>        </item>
        <item>
            <title>3. Brainstem reflex studies in patients with progressive supranuclear palsy and primary progressive freezing of gait</title>
            <link>http://www.medworm.com/index.php?rid=5579122&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS138824571100472X%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aim: Freezing of gait (FOG) is a common feature of Parkinsonian syndromes. Primary progressive freezing of gait (PPFOG) manifests predominantly as progressive freezing gait and difficulties of writing or speaking without accompanying any other movement disorders. Although FOG is mostly associated with PSP, some authors proposed that PPFOG is a distinct clinical and pathophysiological entity. Previous studies showed that startle response is absent or TCR is abnormal in most PSP patients, and its measurement has been proposed as a method of distinguishing PSP from other Parkinsonian disorders. On the other hand, startle response was found to be exaggerated in PPFOG. Based on those previous observations we aimed to investigate the characteristics of brainstem reflexes in PPFOG ...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579122</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579122</guid>        </item>
        <item>
            <title>2. The StartReact effect in tasks requiring a precise endpoint</title>
            <link>http://www.medworm.com/index.php?rid=5579121&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004718%2Fabstract%3Frss%3Dyes</link>
            <description>Background and aims: Many daily precision grip tasks demand a high level of dexterity with a precise endpoint. For proficiency subjects may use programmed movements to be done following an external command. Voluntary reactions can be speeded up by a startling auditory stimulus (SAS) delivered at the same time as the imperative signal, a phenomenon termed StartReact (Valls-Solé et al., 1995). It is unknown if tasks requiring a precise endpoint can also be affected by SAS. The aim of the present study is to examine how a task that requires a precise endpoint responds to a SAS delivered at different timings during the task. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579121</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579121</guid>        </item>
        <item>
            <title>1. The physiology of the brainstem respiratory centres: Lessons from a concurrent multithreaded computational model of Rett syndrome</title>
            <link>http://www.medworm.com/index.php?rid=5579120&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004706%2Fabstract%3Frss%3Dyes</link>
            <description>Background: The brain is primarily a massively parallel computer and the brainstem is no exception. Doubtless of vital importance to life, the brainstem centres controlling respiration continue to evade precise identification. Rett syndrome (RTT) is an interesting neurodevelopmental disorder that produces characteristic neurological and behavioural features. Clinical features include autistic traits, loss of purposeful hand movements, stereotyped hand movements and life threatening respiratory rhythm disturbances. The respiratory rhythm abnormalities of RTT include hyperventilation, breath holding, apneustic breathing, etc. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579120</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:57 +0100</pubDate>
            <guid isPermaLink="false">5579120</guid>        </item>
        <item>
            <title>Preliminary Announcement</title>
            <link>http://www.medworm.com/index.php?rid=5579087&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711011369%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579087</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:56 +0100</pubDate>
            <guid isPermaLink="false">5579087</guid>        </item>
        <item>
            <title>Editorial Board</title>
            <link>http://www.medworm.com/index.php?rid=5579086&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711011308%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579086</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:56 +0100</pubDate>
            <guid isPermaLink="false">5579086</guid>        </item>
        <item>
            <title>Contents</title>
            <link>http://www.medworm.com/index.php?rid=5579085&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711011291%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579085</comments>
            <pubDate>Thu, 12 Jan 2012 11:11:56 +0100</pubDate>
            <guid isPermaLink="false">5579085</guid>        </item>
        <item>
            <title>Muscle ultrasonography: A diagnostic tool for amyotrophic lateral sclerosis.</title>
            <link>http://www.medworm.com/index.php?rid=5608198&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22244867%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Muscle ultrasound can differentiate between amyotrophic lateral sclerosis and mimics with high sensitivity and specificity, and is a sensitive tool to screen for regional lower motor neuron involvement. SIGNIFICANCE: Muscle ultrasonography is a promising tool in the diagnostic work up of ALS.
    PMID: 22244867 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5608198</comments>
            <pubDate>Thu, 12 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5608198</guid>        </item>
        <item>
            <title>Comparison of a row-column speller vs. a novel lateral single-character speller: Assessment of BCI for severe motor disabled patients.</title>
            <link>http://www.medworm.com/index.php?rid=5608197&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22244868%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The results suggest that LSC is an effective alternative to RC, and that LSC still has a margin for potential improvement in bit rate and accuracy. SIGNIFICANCE: The high bit rates and accuracy of LSC are a step forward for the effective use of BCI in clinical applications.
    PMID: 22244868 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5608197</comments>
            <pubDate>Thu, 12 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5608197</guid>        </item>
        <item>
            <title>Online use of error-related potentials in healthy users and people with severe motor impairment increases performance of a P300-BCI.</title>
            <link>http://www.medworm.com/index.php?rid=5608199&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22244309%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Error-related potentials as a secondary source of information can be used to increase overall bit rate in a P3 BCI. SIGNIFICANCE: The method should be made available to any patient using the P3 BCI for communication.
    PMID: 22244309 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5608199</comments>
            <pubDate>Wed, 11 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5608199</guid>        </item>
        <item>
            <title>Muscle and skin sympathetic activities in Ross syndrome.</title>
            <link>http://www.medworm.com/index.php?rid=5608201&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22240417%26dopt%3DAbstract</link>
            <description>CONCLUSION: Microneurographic study of sympathetic activity from affected skin confirmed the selective involvement of skin sympathetic activity with spared muscle sympathetic activity and it may represent the neurophysiological hallmark of the disease. SIGNIFICANCE: Microneurography together with clinical and skin biopsy findings may contribute to RS diagnosis. Our data also suggest that autonomic damage in RS does not involve cardiovascular activity.
    PMID: 22240417 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5608201</comments>
            <pubDate>Tue, 10 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5608201</guid>        </item>
        <item>
            <title>Muscle artifacts in multichannel EEG: Characteristics and reduction.</title>
            <link>http://www.medworm.com/index.php?rid=5608200&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22240418%26dopt%3DAbstract</link>
            <description>CONCLUSION: The characteristics of unintentional muscle activities align with the reported characteristics of controlled muscle activities. Artifact spatial characteristics can be EEG equipment dependent. The ICA-SR method can effectively and efficiently process clinical EEG. SIGNIFICANCE: Armed with advanced signal processing algorithms, this study expands our knowledge of muscle activities in EEG from muscle-controlled experiments to general clinical trials. The ICA-SR method provides an urgently needed solution with validated performance for efficiently processing large volumes of clinical EEG.
    PMID: 22240418 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5608200</comments>
            <pubDate>Tue, 10 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5608200</guid>        </item>
        <item>
            <title>Pattern-specific changes and discordant prognostic values of individual leg-muscle motor evoked potentials during spinal surgery.</title>
            <link>http://www.medworm.com/index.php?rid=5582745&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22227063%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Although mMEPs were generated at a high rate in the AH, mMEP in the TA can play an important complementary role in intra-operative mMEP monitoring, because mMEP in the TA can be more sensitive to potential neural damage. SIGNIFICANCE: Using a combination of muscles with individual sensitivities and clinical significances will improve intra-operative mMEP monitoring strategies.
    PMID: 22227063 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5582745</comments>
            <pubDate>Fri, 06 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5582745</guid>        </item>
        <item>
            <title>Electromyography and muscle ultrasound in ALS diagnosis, complementary or competitive?</title>
            <link>http://www.medworm.com/index.php?rid=5582748&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22226794%26dopt%3DAbstract</link>
            <description>Authors: Dengler R
    PMID: 22226794 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5582748</comments>
            <pubDate>Thu, 05 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5582748</guid>        </item>
        <item>
            <title>Sleep cyclic alternating pattern analysis in infants with apparent life-threatening events: A daytime polysomnographic study.</title>
            <link>http://www.medworm.com/index.php?rid=5582747&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22226795%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Our results show a higher level of arousal and an increased non-REM sleep discontinuity in babies with apparent life-threatening events, compared to controls. SIGNIFICANCE: The enhanced mechanism of arousal might counteract life-threatening events and represent an important neurophysiologic distinction from future victims of sudden infant death syndrome who also experience similar events.
    PMID: 22226795 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5582747</comments>
            <pubDate>Thu, 05 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5582747</guid>        </item>
        <item>
            <title>Reply to &quot;Unmasking of presynaptic cutaneous HFOs burst by DBS lead recordings&quot;</title>
            <link>http://www.medworm.com/index.php?rid=5582746&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22226796%26dopt%3DAbstract</link>
            <description>Reply to &quot;Unmasking of presynaptic cutaneous HFOs burst by DBS lead recordings&quot;
    Clin Neurophysiol. 2012 Jan 5;
    Authors: Ozaki I, Hashimoto I
    PMID: 22226796 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5582746</comments>
            <pubDate>Thu, 05 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5582746</guid>        </item>
        <item>
            <title>Unmasking of presynaptic cutaneous HFOs burst by DBS lead recordings.</title>
            <link>http://www.medworm.com/index.php?rid=5582750&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22225650%26dopt%3DAbstract</link>
            <description>Authors: Insola A, Padua L, Mazzone P, Restuccia D
    PMID: 22225650 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5582750</comments>
            <pubDate>Wed, 04 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5582750</guid>        </item>
        <item>
            <title>Semiautomatic quantification of spiking in patients with continuous spikes and waves in sleep: Sensitivity to settings and correspondence to visual assessment.</title>
            <link>http://www.medworm.com/index.php?rid=5582749&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22225651%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Semiautomatic quantification of SI functions well with maxISI of 3s, and the first hour of sleep represents the whole night SI with a clinically relevant accuracy. SIGNIFICANCE: This method opens a possibility for objective quantification of near-continuous epileptiform spiking during sleep, and it supports the use of shorter epochs for quantitative assessment of CSWS.
    PMID: 22225651 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5582749</comments>
            <pubDate>Wed, 04 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5582749</guid>        </item>
        <item>
            <title>Cross-frequency decomposition: A novel technique for studying interactions between neuronal oscillations with different frequencies.</title>
            <link>http://www.medworm.com/index.php?rid=5570309&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22217959%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: CFD allows a compact representation of the sets of interacting components. The application of CFD to EEG data allows differentiating cross-frequency synchronization arising due to genuine neurophysiological interactions from interactions occurring due to quasi-sinusoidal waveform of neuronal oscillations. SIGNIFICANCE: CFD is a method capable of extracting cross-frequency coupled neuronal oscillations even in the presence of strong noise.
    PMID: 22217959 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5570309</comments>
            <pubDate>Mon, 02 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5570309</guid>        </item>
        <item>
            <title>Emotional anticipation rather than processing is altered in patients with vasovagal syncope.</title>
            <link>http://www.medworm.com/index.php?rid=5570308&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22217960%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: These results suggest that the anticipation, rather than the processing, of aversive stimuli is altered in syncopal patients. SIGNIFICANCE: The reduced cortical anticipation in fainters might reflect the use of non-adaptive emotion regulation strategies for reducing the impact of upcoming highly arousing (and, particularly, of unpleasant) events.
    PMID: 22217960 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5570308</comments>
            <pubDate>Mon, 02 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5570308</guid>        </item>
        <item>
            <title>Quantitative assessment of nerve echogenicity: Comparison of methods for evaluating nerve echogenicity in ulnar neuropathy at the elbow.</title>
            <link>http://www.medworm.com/index.php?rid=5570307&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22217961%26dopt%3DAbstract</link>
            <description>CONCLUSION: Quantitative nerve echogenicity assessment can be successfully used to distinguish between a group of patients with UNE and a healthy control group, preferably by using the MaxEntropy, RenyiEntropy or Yen methods. SIGNIFICANCE: Automatic thresholding techniques using the MaxEntropy, RenyiEntropy or Yen methods are the best quantitative tests, and these quantitative measures can probably be used in further studies evaluating echogenicity in mono- and polyneuropathies.
    PMID: 22217961 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5570307</comments>
            <pubDate>Mon, 02 Jan 2012 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5570307</guid>        </item>
        <item>
            <title>Bilateral N20 absence in post-anoxic coma: Do you pay attention?</title>
            <link>http://www.medworm.com/index.php?rid=5570311&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22209660%26dopt%3DAbstract</link>
            <description>Authors: Machado C, Estévez M, Rodríguez R, Carrick FR, Melillo R, Leisman G
    PMID: 22209660 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5570311</comments>
            <pubDate>Thu, 29 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5570311</guid>        </item>
        <item>
            <title>Origin of the low-level EMG during the silent period following transcranial magnetic stimulation.</title>
            <link>http://www.medworm.com/index.php?rid=5570310&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22209661%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Muscle lengthening contributes to low-level EMG activity in the silent period, through spinal reflex facilitation by muscle spindle afferents. SIGNIFICANCE: The silent period depth is not only dependent on cortical output but also reflex effects evoked by muscle lengthening.
    PMID: 22209661 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5570310</comments>
            <pubDate>Thu, 29 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5570310</guid>        </item>
        <item>
            <title>Chronic trauma-induced neck pain impairs the neural control of the deep semispinalis cervicis muscle.</title>
            <link>http://www.medworm.com/index.php?rid=5554124&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22206690%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The activity of the semispinalis cervicis muscle is reduced and less defined in patients with neck pain confirming a disturbance in the neural control of this muscle. SIGNIFICANCE: This finding suggests that exercises that target the deep semispinalis cervicis muscle may be relevant to include in the management of patients with neck pain.
    PMID: 22206690 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5554124</comments>
            <pubDate>Tue, 27 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5554124</guid>        </item>
        <item>
            <title>Impact of aging on visual reweighting during locomotion.</title>
            <link>http://www.medworm.com/index.php?rid=5554126&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22204920%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Old age affects the ability to re-weight visual information and make postural or locomotor adjustments in real time. The lower errors of the young adults in the fast conditions suggest decreased cortical control of locomotion with increasing speeds. SIGNIFICANCE: Visual information presented in real time can impact on balance and mobility in older adults, and thus should be given serious consideration for the purpose of evaluation and intervention.
    PMID: 22204920 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5554126</comments>
            <pubDate>Mon, 26 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5554126</guid>        </item>
        <item>
            <title>Differences in excitability between median and superficial radial sensory axons.</title>
            <link>http://www.medworm.com/index.php?rid=5554125&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22204921%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: These findings suggest that membrane potential in human median sensory axons is more negative than in superficial radial axons, possibly due to greater activity of electrogenic Na(+)/K(+) pump. These results may reflect adaptation to impulses load carried by median axons that would be far greater with a higher frequency. SIGNIFICANCE: Biophysical properties are not identical in different human sensory axons, and therefore their responses to disease may differ.
    PMID: 22204921 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5554125</comments>
            <pubDate>Mon, 26 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5554125</guid>        </item>
        <item>
            <title>Eliminating muscle artifacts from EEG recordings: A necessary imperative.</title>
            <link>http://www.medworm.com/index.php?rid=5554127&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22200415%26dopt%3DAbstract</link>
            <description>Authors: Bautista RE
    PMID: 22200415 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5554127</comments>
            <pubDate>Sat, 24 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5554127</guid>        </item>
        <item>
            <title>The auditory P200 is both increased and reduced in schizophrenia? A meta-analytic dissociation of the effect for standard and target stimuli in the oddball task.</title>
            <link>http://www.medworm.com/index.php?rid=5554128&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22197447%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The results obtained suggest that claims of reduced or augmented P200 in schizophrenia based on the sole examination of standard or target stimuli fail to consider the stimulus effect. SIGNIFICANCE: Quantification of effects for standard and target stimuli is a required first step to understand the nature of P200 deficits in schizophrenia.
    PMID: 22197447 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5554128</comments>
            <pubDate>Thu, 22 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5554128</guid>        </item>
        <item>
            <title>Preliminary Announcement</title>
            <link>http://www.medworm.com/index.php?rid=5521745&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711009047%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521745</comments>
            <pubDate>Tue, 20 Dec 2011 23:48:50 +0100</pubDate>
            <guid isPermaLink="false">5521745</guid>        </item>
        <item>
            <title>Editorial Board</title>
            <link>http://www.medworm.com/index.php?rid=5521744&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711008972%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521744</comments>
            <pubDate>Tue, 20 Dec 2011 23:48:50 +0100</pubDate>
            <guid isPermaLink="false">5521744</guid>        </item>
        <item>
            <title>Contents</title>
            <link>http://www.medworm.com/index.php?rid=5521743&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711008960%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521743</comments>
            <pubDate>Tue, 20 Dec 2011 23:48:50 +0100</pubDate>
            <guid isPermaLink="false">5521743</guid>        </item>
        <item>
            <title>Peripheral neuropathy in hereditary spastic paraplegia due to spastin (SPG4) mutation - A neurophysiological study using excitability techniques.</title>
            <link>http://www.medworm.com/index.php?rid=5541512&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22192498%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Peripheral neuropathy occurs in HSP patients with SPG4 mutations, but axonal excitability studies provide limited additional evidence for subclinical peripheral nerve dysfunction, and add little further to standard nerve conduction studies. SIGNIFICANCE: The features of HSP due to SPG4 mutations include sensorimotor polyneuropathy. The value of excitability studies is limited in individual patients.
    PMID: 22192498 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5541512</comments>
            <pubDate>Tue, 20 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5541512</guid>        </item>
        <item>
            <title>Auditory change detection in fragile X syndrome males: A brain potential study.</title>
            <link>http://www.medworm.com/index.php?rid=5541511&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22192499%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The current study demonstrates that auditory stimulus discrimination in the FXS brain is already compromised during the pre-attentive stages of information processing. Furthermore, the apparent pre-attentive information processing deficiencies in FXS coincide with a weakness in the involuntary engagement of attentional resources. SIGNIFICANCE: The stimulus-driven information processing deficiencies in FXS might compromise information processing in several domains and, thus, present a key-deficit in FXS neurocognition.
    PMID: 22192499 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5541511</comments>
            <pubDate>Tue, 20 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5541511</guid>        </item>
        <item>
            <title>Increased contact heat pain and shortened latencies of contact heat evoked potentials following capsaicin-induced heat hyperalgesia.</title>
            <link>http://www.medworm.com/index.php?rid=5541513&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22188747%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Our study showed a decrease in late CHEPs latencies and appearance of ultralate potentials compatible with sensitization of Aδ fibers and C fibers. SIGNIFICANCE: Contact heat may be a useful tool to assess sensitization of the pain system.
    PMID: 22188747 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5541513</comments>
            <pubDate>Mon, 19 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5541513</guid>        </item>
        <item>
            <title>Quantitative assessment of peripheral nerve ultrasound echogenicity. A step forward.</title>
            <link>http://www.medworm.com/index.php?rid=5541514&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22182966%26dopt%3DAbstract</link>
            <description>Authors: Wilder-Smith EP
    PMID: 22182966 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5541514</comments>
            <pubDate>Sat, 17 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5541514</guid>        </item>
        <item>
            <title>The mismatch negativity (MMN) – A unique window to disturbed central auditory processing in ageing and different clinical conditions</title>
            <link>http://www.medworm.com/index.php?rid=5636710&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006882%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► The mismatch negativity (MMN) indexes different types of central auditory abnormalities in different neuropsychiatric, neurological, and neurodevelopmental disorders. ► The diminished amplitude/prolonged peak latency observed in patients usually indexes decreased auditory discrimination. ► An MMN deficit may also index cognitive and functional decline shared by different disorders irrespective of their specific aetiology and symptomatology. ► MMN deficits index deficient N-methyl-d-aspartate (NMDA) receptor function affecting memory-trace formation and hence cognition in different disorders.Abstract: In this article, we review clinical research using the mismatch negativity (MMN), a change-detection response of the brain elicited even in the absence of attention or be...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636710</comments>
            <pubDate>Wed, 14 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5636710</guid>        </item>
        <item>
            <title>The N20 in post-anoxic coma: Are you listening?</title>
            <link>http://www.medworm.com/index.php?rid=5519681&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22172767%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Somatosensory neurons have both &quot;receiving&quot; and &quot;sending&quot; properties, where the latter mainly reflects cortical synaptic function. After hypoxic insults, these two modes may be selectively damaged. Preservation of the N20 reflects an intact &quot;receiving mode&quot;, while the EEG mainly reflects the &quot;sending mode&quot;. SIGNIFICANCE: Insight into the physiological processes underlying the generation of the SSEP and the EEG is relevant in clinical decision making and may guide novel monitoring strategies.
    PMID: 22172767 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519681</comments>
            <pubDate>Tue, 13 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519681</guid>        </item>
        <item>
            <title>Seizure source imaging by means of FINE spatio-temporal dipole localization and directed transfer function in partial epilepsy patients.</title>
            <link>http://www.medworm.com/index.php?rid=5519680&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22172768%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: FINE together with DTF was able to localize seizure onset zones of partial epilepsy patients. High-density EEG recording can help achieve improved seizure source imaging. SIGNIFICANCE: The present results suggest the promise of high-density EEG and electrical source imaging for noninvasively localizing seizure onset zones.
    PMID: 22172768 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519680</comments>
            <pubDate>Tue, 13 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519680</guid>        </item>
        <item>
            <title>Effects of afferent stimulation rate on inhibitory spinal pathways in hemiplegic spastic patients.</title>
            <link>http://www.medworm.com/index.php?rid=5519679&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22172769%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The reinforcement of inhibitory spinal networks induced by repetitive stimulation of afferent fibres is preserved in spastic patients, whereas the mechanisms underlying this phenomena might be altered. SIGNIFICANCE: The results of these experiments open up a number of possibilities for novel spasticity therapies based on non-invasive techniques.
    PMID: 22172769 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519679</comments>
            <pubDate>Tue, 13 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519679</guid>        </item>
        <item>
            <title>The mismatch negativity (MMN) - A unique window to disturbed central auditory processing in ageing and different clinical conditions.</title>
            <link>http://www.medworm.com/index.php?rid=5519682&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22169062%26dopt%3DAbstract</link>
            <description>Authors: Näätänen R, Kujala T, Escera C, Baldeweg T, Kreegipuu K, Carlson S, Ponton C
    Abstract
    In this article, we review clinical research using the mismatch negativity (MMN), a change-detection response of the brain elicited even in the absence of attention or behavioural task. In these studies, the MMN was usually elicited by employing occasional frequency, duration or speech-sound changes in repetitive background stimulation while the patient was reading or watching videos. It was found that in a large number of different neuropsychiatric, neurological and neurodevelopmental disorders, as well as in normal ageing, the MMN amplitude was attenuated and peak latency prolonged. Besides indexing decreased discrimination accuracy, these effects may also reflect, depending on the s...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519682</comments>
            <pubDate>Mon, 12 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519682</guid>        </item>
        <item>
            <title>Long-term and within-day variability of working memory performance and EEG in individuals.</title>
            <link>http://www.medworm.com/index.php?rid=5519683&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22154302%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Objective working memory measures incorporating performance and EEG are stable over time and sensitive at the level of individual subjects to interventions that affect neurocognitive function. SIGNIFICANCE: With further research these measures may be suitable for use in individualized medical care by providing a sensitive assessment of incipient illness and response to treatment.
    PMID: 22154302 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519683</comments>
            <pubDate>Fri, 09 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519683</guid>        </item>
        <item>
            <title>Motor cortical functional geometry in cerebral palsy and its relationship to disability.</title>
            <link>http://www.medworm.com/index.php?rid=5519685&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22153667%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Abnormalities in TMS-derived motor maps cut across the clinical classifications of hemiplegic and diplegic CP. The lateralization of the upper and lower extremity motor representation demonstrates reorganization after insults to the affected hemispheres of both diplegic and hemiplegic children. SIGNIFICANCE: The current study is a step towards defining the relationship between changes in motor maps and functional impairments in CP. These results suggest the need for further work to develop improved classification schemes that integrate clinical, radiologic, and neurophysiologic measures in CP.
    PMID: 22153667 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519685</comments>
            <pubDate>Tue, 06 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519685</guid>        </item>
        <item>
            <title>Performance of the frequency domain indices with respect to sleep staging.</title>
            <link>http://www.medworm.com/index.php?rid=5519684&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22153785%26dopt%3DAbstract</link>
            <description>CONCLUSION: The frequency components of electroencephalogram perform well in identifying sleep, sleep with spindles, and slow wave sleep. Electromyogram, heart rate, and body acceleration offer high agreement only when differentiating between wakefulness and sleep. SIGNIFICANCE: The human-machine agreement is acceptable with spectral parameters, but heart rate and body acceleration still cannot substitute for electroencephalogram.
    PMID: 22153785 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519684</comments>
            <pubDate>Mon, 05 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519684</guid>        </item>
        <item>
            <title>Intra-operative neurophysiology during microvascular decompression for hemifacial spasm</title>
            <link>http://www.medworm.com/index.php?rid=5521756&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007474%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► This work contribute to the better understanding pathophysiology of the hemifacial spasm (HFS). ► During MVD changes in: facial corticobulbar motor evoked potentials (FCoMEPs), blink reflex, F wave and lateral spread responses (LSRs) were presented.. ► The data support the theory that hyperexcitability of facial nerve in HFS is not caused only by pulsating vessels at the REZ but also by antidromic activation of the facial motor nucleus.Abstract: There is evidence that primary hemifacial spasm (HFS) in the majority of patients is related to a vascular compression of the facial nerve at its root exit zone (REZ). As a consequence, the hyperexcitability of facial nerve generates spasms of the facial muscles. Microvascular decompression (MVD) of the facial nerve near its REZ...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521756</comments>
            <pubDate>Fri, 02 Dec 2011 05:00:00 +0100</pubDate>
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        <item>
            <title>Movement preparation and cortical processing of afferent inputs in cortical tremor: An event-related (de)synchronization (ERD/ERS) study.</title>
            <link>http://www.medworm.com/index.php?rid=5519686&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22138352%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The present results suggest a hyperexcitability of the cortico-subcortical loops responsible for movement preparation and execution. Post-movement inhibition related to cortical processing of afferent input is unaffected in isolated cortical myoclonus. SIGNIFICANCE: Intracortical abnormalities can differ in patients suffering from cortical myoclonus, according to whether or not the individuals have associated epileptic symptoms.
    PMID: 22138352 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519686</comments>
            <pubDate>Fri, 02 Dec 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519686</guid>        </item>
        <item>
            <title>Intra-operative neurophysiology during microvascular decompression for hemifacial spasm.</title>
            <link>http://www.medworm.com/index.php?rid=5519688&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22136736%26dopt%3DAbstract</link>
            <description>Authors: Fernández-Conejero I, Ulkatan S, Sen C, Deletis V
    Abstract
    There is evidence that primary hemifacial spasm (HFS) in the majority of patients is related to a vascular compression of the facial nerve at its root exit zone (REZ). As a consequence, the hyperexcitability of facial nerve generates spasms on the facial muscles. Microvascular decompression (MVD) of the facial nerve near its REZ has been established as an effective treatment of HFS. Intra-operative disappearance of abnormal muscle responses (lateral spread) elicited by stimulating one of the facial nerve branches has been used as a method to predict MVD effectiveness. Other neurophysiologic techniques, such as facial F-wave, blink reflex and facial corticobulbar motor evoked potentials (FCoMEP), are feasible to in...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519688</comments>
            <pubDate>Wed, 30 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519688</guid>        </item>
        <item>
            <title>Contribution of anti-ryanodine receptor antibody to impairment of excitation-contraction coupling in myasthenia gravis.</title>
            <link>http://www.medworm.com/index.php?rid=5519687&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22136737%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Presence of anti-RyR antibodies is associated with significantly prolonged masseteric ECCT compared to absence of the antibodies in MG. SIGNIFICANCE: Anti-RyR antibody contributes to E-C coupling impairment in the masseter muscle in patients with MG.
    PMID: 22136737 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519687</comments>
            <pubDate>Wed, 30 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519687</guid>        </item>
        <item>
            <title>Clinical utility of brain tissue oxygen tension in treatment of brain injury more complicated than it appears.</title>
            <link>http://www.medworm.com/index.php?rid=5519698&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119796%26dopt%3DAbstract</link>
            <description>Authors: Michael Schmidt J, Claassen J
    PMID: 22119796 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519698</comments>
            <pubDate>Fri, 25 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519698</guid>        </item>
        <item>
            <title>Value of scalp delayed rhythmic ictal transformation (DRIT) in presurgical assessment of temporal lobe epilepsy.</title>
            <link>http://www.medworm.com/index.php?rid=5519694&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119797%26dopt%3DAbstract</link>
            <description>CONCLUSION: Two thirds of seizures showed unilateral scalp DRIT, which had a lateralising value of up to 79%. SIGNIFICANCE: Scalp DRIT is a reliable marker to lateralise seizure focus in patients without a focal seizure onset on the scalp EEG.
    PMID: 22119797 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519694</comments>
            <pubDate>Fri, 25 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519694</guid>        </item>
        <item>
            <title>Auditory evoked magnetic fields in patients with absent brainstem responses due to auditory neuropathy with optic atrophy.</title>
            <link>http://www.medworm.com/index.php?rid=5519692&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119798%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Presence and abnormality of auditory cortical responses can be evaluated by AEFs in patients with auditory neuropathy even under null responses in ABRs. SIGNIFICANCE: AEFs are useful to evaluate residual hearing in patients with auditory neuropathy.
    PMID: 22119798 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519692</comments>
            <pubDate>Fri, 25 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519692</guid>        </item>
        <item>
            <title>What if you are not sure? Electroencephalographic correlates of subjective confidence level about a decision.</title>
            <link>http://www.medworm.com/index.php?rid=5519690&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119799%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Subjective confidence levels of decision making can be discriminated by time and time-frequency analyses of ERPs and EEG. SIGNIFICANCE: Time-frequency parameters of EEG can be useful in the detection of subjective confidence in single-trials, which might be efficiently used for faster human-computer interaction.
    PMID: 22119799 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519690</comments>
            <pubDate>Fri, 25 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519690</guid>        </item>
        <item>
            <title>Minimal heating of aneurysm clips during repetitive transcranial magnetic stimulation.</title>
            <link>http://www.medworm.com/index.php?rid=5519689&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119800%26dopt%3DAbstract</link>
            <description>Authors: Hsieh TH, Dhamne SC, Chen JJ, Carpenter LL, Anastasio EM, Pascual-Leone A, Rotenberg A
    PMID: 22119800 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519689</comments>
            <pubDate>Fri, 25 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519689</guid>        </item>
        <item>
            <title>A comparison between disposable and reusable single fiber needle electrodes in relation to stimulated single fiber studies.</title>
            <link>http://www.medworm.com/index.php?rid=5519699&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119663%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Disposable single fibre needle electrodes obtained MCD values were comparable to those obtained with reusable single fibre needle electrodes but the amplitudes of the potentials were higher. The greater number of potentials obtained per acquisition run means that the examination can be performed quicker. SIGNIFICANCE: Disposable single fiber needle use avoids the risk of prion infections and performs better than reusable needles.
    PMID: 22119663 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519699</comments>
            <pubDate>Thu, 24 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519699</guid>        </item>
        <item>
            <title>Effects of attentional dispersion on sensory-motor processing of anticipatory postural control during unilateral arm abduction.</title>
            <link>http://www.medworm.com/index.php?rid=5519708&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119174%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: With attentional dispersion, brain activation decreased in the area related to the sensory processing and especially in the stimulus discrimination area. With increasing attentional dispersion, the delay in motor preparation or anticipatory attention to target stimuli was related to the delay in stimulus discrimination and onset time of postural muscle activation. SIGNIFICANCE: Effects of attentional dispersion on sensory-motor processing of anticipatory postural control were experimentally demonstrated.
    PMID: 22119174 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519708</comments>
            <pubDate>Wed, 23 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519708</guid>        </item>
        <item>
            <title>Assessment of a portable device for the quantitative measurement of ankle joint stiffness in spastic individuals.</title>
            <link>http://www.medworm.com/index.php?rid=5519705&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119175%26dopt%3DAbstract</link>
            <description>CONCLUSION: The portable device correlated well with measures obtained by a torque motor in both controls and MS participants, showed high intra- and inter-rater reliability for the SCI participants, and could easily distinguish between stiff and control ankle joints. However, the device, in its current form, may be less accurate during rapid movements when inertia contributes to stiffness and the shape of the air-filled pads did not provide a good interface with the foot. SIGNIFICANCE: This study demonstrates that a portable device can potentially be a useful diagnostic tool to obtain reliable information of stiffness for the ankle joint.
    PMID: 22119175 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519705</comments>
            <pubDate>Wed, 23 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519705</guid>        </item>
        <item>
            <title>Greater sensitivity of the P300 component to bimodal stimulation in an event-related potentials oddball task.</title>
            <link>http://www.medworm.com/index.php?rid=5519701&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119176%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Although the two groups differed in their levels of anxiety and depression, only the bimodal tasks were able to identify these differences. SIGNIFICANCES: These results suggest that a bimodal oddball design should be used in future studies to increase the sensitivity of P300 differences for differentiating between healthy participants and those with clinical symptoms.
    PMID: 22119176 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519701</comments>
            <pubDate>Wed, 23 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519701</guid>        </item>
        <item>
            <title>Early attentional modulation by alcohol-related cues in young binge drinkers: An event-related potentials study.</title>
            <link>http://www.medworm.com/index.php?rid=5519700&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22119177%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The present study provides evidence for an early processing enhancement, indexed by increased P100 amplitude, in binge drinkers when confronted with alcohol cues. SIGNIFICANCE: These findings suggest that higher reactivity to alcohol cues is not a phenomenon limited to adult alcoholics, but that young binge drinkers exhibit signs of prioritizing processing related to alcohol. Prevention intervention for alcohol misuse in young people should consider approaches that address this automatic cue reactivity.
    PMID: 22119177 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519700</comments>
            <pubDate>Wed, 23 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519700</guid>        </item>
        <item>
            <title>Neurophysiology of the brainstem-structure and function of brainstem circuits</title>
            <link>http://www.medworm.com/index.php?rid=5521746&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007486%2Fabstract%3Frss%3Dyes</link>
            <description>The brainstem is a small but most important center in the brain. It serves as a central relay station between spinal cord, cerebellum and cerebrum and is intricately involved in functions ranging from motor control, sensorimotor integration and regulation of autonomic functions to consciousness and attention. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521746</comments>
            <pubDate>Fri, 18 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521746</guid>        </item>
        <item>
            <title>Cerebral perfusion pressure and intracranial pressure are not surrogates for brain tissue oxygenation in traumatic brain injury.</title>
            <link>http://www.medworm.com/index.php?rid=5519710&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22104471%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: CPP and ICP should not be used as surrogates for pBtO(2) since cerebral oxygenation varies independently of cerebral hemodynamics and pressures. Brain tissue oxygen monitoring in patients with TBI provides unique information regarding cerebral oxygenation the utility of which remains to be fully described. SIGNIFICANCE: CPP and ICP are not surrogates for pBtO(2). Brain tissue oxygenation monitoring provides unique information for the treatment of traumatically injured patients.
    PMID: 22104471 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519710</comments>
            <pubDate>Fri, 18 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519710</guid>        </item>
        <item>
            <title>Intra-operative monitoring of lower extremity motor-evoked potentials by direct cortical stimulation.</title>
            <link>http://www.medworm.com/index.php?rid=5519709&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22104472%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: We accomplished LE-MEP recording during supratentorial surgery using monopolar DCS with a subdural electrode placed on the convex side of the motor cortex. SIGNIFICANCE: A useful method of intra-operative LE-MEP recording was described.
    PMID: 22104472 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519709</comments>
            <pubDate>Fri, 18 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519709</guid>        </item>
        <item>
            <title>Continuous theta-burst rTMS over primary somatosensory cortex modulates tactile perception on the hand.</title>
            <link>http://www.medworm.com/index.php?rid=5519713&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22100859%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: CTBS over SI impairs both temporal and spatial domains of tactile acuity for a similar duration. SIGNIFICANCE: CTBS over SI appears to decrease neural activity within targeted cortex and has potential utility in reducing excessive sensory processing.
    PMID: 22100859 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519713</comments>
            <pubDate>Thu, 17 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519713</guid>        </item>
        <item>
            <title>Density spectral array analysis of SREDA during EEG-video monitoring.</title>
            <link>http://www.medworm.com/index.php?rid=5519712&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22100860%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The temporal pattern of SREDA varies during the sleep-wake cycle. Lorazepam appears to suppress SREDA. SIGNIFICANCE: Response to lorazepam cannot distinguish SREDA from electrographic seizure activity.
    PMID: 22100860 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519712</comments>
            <pubDate>Thu, 17 Nov 2011 05:00:00 +0100</pubDate>
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        <item>
            <title>Clinical neurophysiology and immunology of E-C coupling of muscle.</title>
            <link>http://www.medworm.com/index.php?rid=5519711&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22100861%26dopt%3DAbstract</link>
            <description>Authors: Kuwabara S
    PMID: 22100861 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519711</comments>
            <pubDate>Thu, 17 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519711</guid>        </item>
        <item>
            <title>Neurophysiology of the brainstem-structure and function of brainstem circuits Editorial overview to the special section on the 5th International Meeting of the BrainStem Society, London, UK, December 09-10, 2010.</title>
            <link>http://www.medworm.com/index.php?rid=5519715&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22099259%26dopt%3DAbstract</link>
            <description>Authors: Rothwell J, Kofler M
    PMID: 22099259 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519715</comments>
            <pubDate>Wed, 16 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519715</guid>        </item>
        <item>
            <title>Choosing to improve or to impair.</title>
            <link>http://www.medworm.com/index.php?rid=5519714&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22099260%26dopt%3DAbstract</link>
            <description>Authors: Dinse HR
    PMID: 22099260 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5519714</comments>
            <pubDate>Wed, 16 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5519714</guid>        </item>
        <item>
            <title>Pain and attention - friends or foes?</title>
            <link>http://www.medworm.com/index.php?rid=5429437&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22093687%26dopt%3DAbstract</link>
            <description>Authors: Hauck M, Lorenz J
    PMID: 22093687 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5429437</comments>
            <pubDate>Tue, 15 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5429437</guid>        </item>
        <item>
            <title>Clinically-oriented monitoring of acute effects of methylphenidate on cerebral hemodynamics in ADHD children using fNIRS.</title>
            <link>http://www.medworm.com/index.php?rid=5429438&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22088661%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The improved cognitive performance was associated with activation in the right LPFC, which might serve as a biological marker to monitor the effect of MPH in ADHD children. SIGNIFICANCE: MPH-effect assessment in ADHD children using fNIRS can be performed within a 3h stay at a hospital during a single visit, and thus may be integrated into clinical practice.
    PMID: 22088661 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5429438</comments>
            <pubDate>Mon, 14 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5429438</guid>        </item>
        <item>
            <title>The cerebellum in dystonia – Help or hindrance?</title>
            <link>http://www.medworm.com/index.php?rid=5521754&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006936%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► There are intimate structural and functional connections between cerebellum and basal ganglia that appear to be involved in patients with dystonia. ► The data summarised in this review suggest that in most forms of dystonia the cerebellum has abnormal, probably compensatory activity, secondary to pathology elsewhere within the sensori-motor network which is yet to be fully characterised. ► It is likely that in some types of dystonia cerebellar dysfunction plays a primary role in the pathophysiology.Abstract: Dystonia has historically been considered a disorder of the basal ganglia. This review aims to critically examine the evidence for a role of the cerebellum in the pathophysiology of dystonia. We compare and attempt to link the information available from both clinica...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521754</comments>
            <pubDate>Fri, 11 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521754</guid>        </item>
        <item>
            <title>The cerebellum in dystonia - Help or hindrance?</title>
            <link>http://www.medworm.com/index.php?rid=5429439&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22078259%26dopt%3DAbstract</link>
            <description>Authors: Sadnicka A, Hoffland BS, Bhatia KP, van de Warrenburg BP, Edwards MJ
    Abstract
    Dystonia has historically been considered a disorder of the basal ganglia. This review aims to critically examine the evidence for a role of the cerebellum in the pathophysiology of dystonia. We compare and attempt to link the information available from both clinical and experimental studies; work detailing cerebellar connectivity in primates; data that suggests a role for the cerebellum in the genesis of dystonia in murine models; clinical observation in humans with structural lesions and heredodegenerative disorders of the cerebellum; and imaging studies of patients with dystonia. The typical electrophysiological findings in dystonia are the converse to those found in cerebellar lesions. Howeve...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5429439</comments>
            <pubDate>Wed, 09 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5429439</guid>        </item>
        <item>
            <title>The re-emergence of evoked potentials in multiple sclerosis; from diagnosis to prognostication</title>
            <link>http://www.medworm.com/index.php?rid=5579091&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711004470%2Fabstract%3Frss%3Dyes</link>
            <description>While the majority of multiple sclerosis (MS) patients exhibit a relapsing-remitting course (RRMS), approximately 40% of patients develop progressive disease 10–15years after symptom onset, termed secondary progressive MS (SPMS) (). The ability to predict which MS patients will develop disability remains elusive, and development of techniques that reliably predict disability development would be of immense therapeutic benefit for MS patients. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5579091</comments>
            <pubDate>Mon, 07 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5579091</guid>        </item>
        <item>
            <title>The oscillating central network of Essential tremor</title>
            <link>http://www.medworm.com/index.php?rid=5521753&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006997%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► Essential tremor emerges from a number of cortical and subcortical motor centres, that is an oscillating network in which the cerebellum and the inferior olive are key structures. ► Each of the network components may act as an oscillator on its own and its contribution changes dynamically. ► Paradoxically the same network governs voluntary movements and pathological tremor but there is emerging evidence that the interaction within the network determines the emerging type of movement, and this may be a basis of the selective effect of deep brain stimulation on tremor.Abstract: Essential tremor (ET) is a centrally driven tremor. It is meanwhile well established that it does not emerge from one single oscillator but an oscillatory network comprising most parts of the physi...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521753</comments>
            <pubDate>Mon, 07 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521753</guid>        </item>
        <item>
            <title>Functional anatomy of subcortical circuits issuing from or integrating at the human brainstem</title>
            <link>http://www.medworm.com/index.php?rid=5521747&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006900%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► High-resolution MRI allows for the analysis of human brainstem microstructure. ► Diffusion tensor imaging at 7T improves the characterization of the brainstem white matter organization. ► These techniques can be useful in research, diagnostic and therapeutic studies on the human brainstem.Abstract: The functional organization of the main human brainstem centers and circuits are described as revealed in post-mortem material with high-resolution structural magnetic resonance imaging (MRI), diffusion tensor imaging (DTI) and diffusion tensor tractography (DTT) acquired at ultra-high magnetic field 7T. The description is complemented with a conventional in vivo fiber tracking study of the descending motor pathways. This type of neuroanatomic depiction of nuclei and nerve tr...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521747</comments>
            <pubDate>Mon, 07 Nov 2011 05:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521747</guid>        </item>
        <item>
            <title>Table to Contents to Volume 122</title>
            <link>http://www.medworm.com/index.php?rid=5378212&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007449%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5378212</comments>
            <pubDate>Sat, 05 Nov 2011 20:36:05 +0100</pubDate>
            <guid isPermaLink="false">5378212</guid>        </item>
        <item>
            <title>Index of Subjects to Volume 122</title>
            <link>http://www.medworm.com/index.php?rid=5378211&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007437%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5378211</comments>
            <pubDate>Sat, 05 Nov 2011 20:36:05 +0100</pubDate>
            <guid isPermaLink="false">5378211</guid>        </item>
        <item>
            <title>Index of Authors to Volume 122</title>
            <link>http://www.medworm.com/index.php?rid=5378210&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007425%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5378210</comments>
            <pubDate>Sat, 05 Nov 2011 20:36:05 +0100</pubDate>
            <guid isPermaLink="false">5378210</guid>        </item>
        <item>
            <title>Acknowledgement to Reviewers</title>
            <link>http://www.medworm.com/index.php?rid=5378209&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007413%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5378209</comments>
            <pubDate>Sat, 05 Nov 2011 20:36:05 +0100</pubDate>
            <guid isPermaLink="false">5378209</guid>        </item>
        <item>
            <title>Preliminary Announcement</title>
            <link>http://www.medworm.com/index.php?rid=5378177&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007450%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5378177</comments>
            <pubDate>Sat, 05 Nov 2011 20:36:00 +0100</pubDate>
            <guid isPermaLink="false">5378177</guid>        </item>
        <item>
            <title>Editorial Board</title>
            <link>http://www.medworm.com/index.php?rid=5378176&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007358%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5378176</comments>
            <pubDate>Sat, 05 Nov 2011 20:36:00 +0100</pubDate>
            <guid isPermaLink="false">5378176</guid>        </item>
        <item>
            <title>Contents</title>
            <link>http://www.medworm.com/index.php?rid=5378175&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711007346%2Fabstract%3Frss%3Dyes</link>
            <description>(Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5378175</comments>
            <pubDate>Sat, 05 Nov 2011 20:36:00 +0100</pubDate>
            <guid isPermaLink="false">5378175</guid>        </item>
        <item>
            <title>Neurophysiological changes after intramuscular injection of botulinum toxin</title>
            <link>http://www.medworm.com/index.php?rid=5521752&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006948%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► This review is focussed on neurophysiological changes after botulinum toxin (BT) injection. ► Will help readers to understand better what are the distant effects of BT treatment. ► Will help readers to know about the different neurophysiological techniques to study functional changes after BT treatment.Abstract: Botulinum toxin (BT) acts peripherally by inhibiting acetylcholine release from the presynaptic neuromuscular terminals and by weakening muscle contraction. Therefore, its clinical benefit is primarily due to its peripheral action. As a result, local injection of BT has become a successful and safe tool in the treatment of several neurological and non-neurological disorders. Studies in animals have also shown that the toxin can be retrogradely transported and ev...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521752</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521752</guid>        </item>
        <item>
            <title>Exploring semantic and phonological picture-word priming in adults who stutter using event-related potentials.</title>
            <link>http://www.medworm.com/index.php?rid=5384801&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055837%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Results point to atypical processing of semantic and phonological information in AWS. Discussion considers whether AWS ERP effects reflect unstable activation of target label semantic and phonological representations, strategic inhibition of target label phonological neighbors, and/or phonological label-probe competition. SIGNIFICANCE: Results raise questions about how mechanisms that regulate activation spreading operate in AWS.
    PMID: 22055837 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384801</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384801</guid>        </item>
        <item>
            <title>Functional anatomy of subcortical circuits issuing from or integrating at the human brainstem.</title>
            <link>http://www.medworm.com/index.php?rid=5384800&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055838%26dopt%3DAbstract</link>
            <description>Authors: Prats-Galino A, Soria G, Notaris MD, Puig J, Pedraza S
    Abstract
    The functional organization of the main human brainstem centers and circuits are described as revealed in post-mortem material with high-resolution structural magnetic resonance imaging (MRI), diffusion tensor imaging (DTI) and diffusion tensor tractography (DTT) acquired at ultra-high magnetic field 7T. The description is complemented with a conventional in vivo fiber tracking study of the descending motor pathways. This type of neuroanatomic depiction of nuclei and nerve tracts at very high spatial resolution opens new possibilities to analyze the fine structure and circuits of the human brainstem, at least in post-mortem material.
    PMID: 22055838 [PubMed - as supplied by publisher] (Source: Clinical Neur...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384800</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384800</guid>        </item>
        <item>
            <title>Adiposity measures predict olfactory processing speed in older adult carriers of the apolipoprotein E ε4 allele.</title>
            <link>http://www.medworm.com/index.php?rid=5384799&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055839%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: This study indicates that adiposity predicts olfactory processing speed in older adults, specifically in ε4 carriers. SIGNIFICANCE: The results suggest that olfactory processing speed may be a useful measure for detecting and following the effects of adiposity on brain integrity and cognitive function in those at genetic risk for AD.
    PMID: 22055839 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384799</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384799</guid>        </item>
        <item>
            <title>A-waves in Guillain-Barré syndrome: Correlation with electrophysiological subtypes and antiganglioside antibodies.</title>
            <link>http://www.medworm.com/index.php?rid=5384798&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055840%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Abundant A-waves in GBS after the acute phase were strongly associated with demyelination that was not mediated by antiganglioside antibodies, possibly through the mechanism of proximal re-excitation induced by electrical inhomogeneities due to segmental demyelination. SIGNIFICANCE: Abundant A-waves are promising as a novel reliable marker of demyelination.
    PMID: 22055840 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384798</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384798</guid>        </item>
        <item>
            <title>Cerebral effects of binge drinking: Respective influences of global alcohol intake and consumption pattern.</title>
            <link>http://www.medworm.com/index.php?rid=5384797&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055841%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Binge drinkers presented early and global ERP deficits, affecting basic and high-level cognitive stages. Moreover, we showed that binge drinking is deleterious for the brain because of alcohol consumption per se, and also because of its specific consumption pattern. SIGNIFICANCE: The present results show that binge-drinking habits lead to striking brain consequences, particularly because of the repeated alternation between intense intoxications and withdrawal episodes.
    PMID: 22055841 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384797</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384797</guid>        </item>
        <item>
            <title>The oscillating central network of Essential tremor.</title>
            <link>http://www.medworm.com/index.php?rid=5384796&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055842%26dopt%3DAbstract</link>
            <description>Authors: Raethjen J, Deuschl G
    Abstract
    Essential tremor (ET) is a centrally driven tremor. It is meanwhile well established that it does not emerge from one single oscillator but an oscillatory network comprising most parts of the physiological central motor network. Several lines of evidence hint at the olivocerebellar system and the thalamus as key structures within this network whereas the cortical motor regions are only intermittently entrained in the tremor rhythm in thalamocortical loops. Dynamic changes in network composition and the interaction in symmetric loops seem to be specific to the generation of tremor. The same network in voluntary motor control is more fixed and subcortico-cortical interactions are preferentially via thalamocortical relays. Thus it is not primari...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384796</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384796</guid>        </item>
        <item>
            <title>The re-emergence of evoked potentials in multiple sclerosis; from diagnosis to prognostication.</title>
            <link>http://www.medworm.com/index.php?rid=5384795&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055843%26dopt%3DAbstract</link>
            <description>Authors: Vucic S
    PMID: 22055843 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384795</comments>
            <pubDate>Thu, 03 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384795</guid>        </item>
        <item>
            <title>Isokinetic strength and power deficits in the hand following stroke.</title>
            <link>http://www.medworm.com/index.php?rid=5384802&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22055766%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: The fact that movement velocity produces larger deficits in torque and power production may explain why patients who have limited strength impairments still experience functional deficits. SIGNIFICANCE: This study demonstrates that strength testing of stroke survivors under isometric conditions may underestimate the overall level of impairment.
    PMID: 22055766 [PubMed - as supplied by publisher] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384802</comments>
            <pubDate>Wed, 02 Nov 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5384802</guid>        </item>
        <item>
            <title>Neurophysiological changes after intramuscular injection of botulinum toxin.</title>
            <link>http://www.medworm.com/index.php?rid=5384803&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D22051548%26dopt%3DAbstract</link>
            <description>Authors: Palomar FJ, Mir P
    Abstract
    Botulinum toxin (BT) acts peripherally by inhibiting acetylcholine release from the presynaptic neuromuscular terminals and by weakening muscle contraction. Therefore, its clinical benefit is primarily due to its peripheral action. As a result, local injection of BT has become a successful and safe tool in the treatment of several neurological and non-neurological disorders. Studies in animals have also shown that the toxin can be retrogradely transported and even transcytosed to neurons in the central nervous system (CNS). Further human studies have suggested that BT could alter the functional organisation of the CNS indirectly through peripheral mechanisms. BT can interfere with and modify spinal, brainstem and cortical circuits, including cort...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5384803</comments>
            <pubDate>Tue, 01 Nov 2011 04:00:00 +0100</pubDate>
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        <item>
            <title>Exaggerated startle reactions</title>
            <link>http://www.medworm.com/index.php?rid=5521750&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006912%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► The startle reflex is a physiological phenomenon, originating in the caudal brainstem, which can be elicited by an unexpected stimulus. It includes two subsequent responses: the initial motor response and a second orienting response. ► Three categories of syndromes with an exaggerated startle reflex can be distinguished: hyperekplexia, stimulus-induced disorders and neuropsychiatric syndromes. ► Polymyographic EMG startle recordings can help demonstrate the different features of the startle reflex in these categories.Abstract: The origin of the startle reflex lies in the caudal brainstem; it can be elicited by an unexpected stimulus resulting in a bilateral activation of many muscles. Two subsequent responses can be measured during EMG recordings; after the initial moto...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521750</comments>
            <pubDate>Fri, 28 Oct 2011 04:00:00 +0100</pubDate>
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        <item>
            <title>Preparation for voluntary movement in healthy and clinical populations: Evidence from startle</title>
            <link>http://www.medworm.com/index.php?rid=5521749&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006961%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► Presenting a startling acoustic stimulus (SAS) during simple RT tasks results in substantial RT facilitation suggesting a subcortical mechanism for response triggering. ► Using a SAS during voluntary motor tasks can reveal when and if advance motor preparation occurs and whether this process changes with practice. ► An updated model of motor system preparation and response initiation is outlined based on results from studies employing a SAS.Abstract: In this review we provide a summary of the observations made regarding advance preparation of the motor system when presenting a startling acoustic stimulus (SAS) during various movement tasks. The predominant finding from these studies is that if the participant is prepared to make a particular movement a SAS can act to di...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521749</comments>
            <pubDate>Fri, 28 Oct 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521749</guid>        </item>
        <item>
            <title>Pathophysiology of primary burning mouth syndrome</title>
            <link>http://www.medworm.com/index.php?rid=5521755&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006924%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► Most patients with primary burning mouth sdr (BMS) suffer from subclinical neuropathic pain. ► Lesions at several levels of neuraxis can give rise to clinically similar BMS symptoms. ► Three distinct subclasses of BMS have been neurophysiologically characterized: (i) Peripheral small fibre neuropathy (ii) Subclinical major trigeminal neuropathy (iii) Central pain that maybe related to deficient dopaminergic top-down inhibition. ► Accurate diagnosis can only be done with neurophysiologic, psychophysical and neuropathological tests.Abstract: Primary burning mouth syndrome (BMS) is severe, disabling and chronic intraoral pain condition for which no local or systemic cause can be found and clinical examination is normal. It mostly affects elderly citizens, especially post...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521755</comments>
            <pubDate>Wed, 26 Oct 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521755</guid>        </item>
        <item>
            <title>Effect of spinal cord injury and of intrathecal baclofen on brainstem reflexes</title>
            <link>http://www.medworm.com/index.php?rid=5521751&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS138824571100695X%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► Neurophysiological testing can demonstrate neuronal reorganization in patients with spinal cord injury. ► Studies on neuronal reorganization following spinal cord injury have so far focused on changes below the lesion and at the cerebral cortex, while only few studies to date have addressed functional changes at the brainstem level. ► Studies of altered brainstem reflexes in patients with spinal cord injury may contribute to the understanding of mechanisms underlying functional changes following injury to the central nervous system, and can help to identify and quantify pharmacological actions upon altered reflexes.Abstract: Reorganization of neural circuits within the central nervous system following injury appears to be a means of compensatory mechanism for loss of fu...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521751</comments>
            <pubDate>Wed, 26 Oct 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521751</guid>        </item>
        <item>
            <title>Assessment of excitability in brainstem circuits mediating the blink reflex and the startle reaction</title>
            <link>http://www.medworm.com/index.php?rid=5521748&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711006973%2Fabstract%3Frss%3Dyes</link>
            <description>Highlights: ► One of the most important domains in neurophysiology studies is testing excitability of neural circuits. ► Excitability of the blink reflex and the startle reaction reflect basal ganglia modulation of brainstem interneuronal circuits. ► Prepulse inhibition is a common mechanism for control of excitability changes in the blink reflex and the startle reaction.Abstract: Excitability is probably the concept that fits better with the definition of the role of neurophysiology in the study of brainstem functions and circuits. Neurophysiological techniques are likely the best suited of all paraclinical tests for documenting the eventual excitability changes that may occur in certain physiological states and in many neurological disorders. The best known test of brainstem excita...</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5521748</comments>
            <pubDate>Wed, 26 Oct 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5521748</guid>        </item>
        <item>
            <title>Increased baseline temperature improves the acquisition of contact heat evoked potentials after spinal cord injury</title>
            <link>http://www.medworm.com/index.php?rid=5636725&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711005621%2Fabstract%3Frss%3Dyes</link>
            <description>Conclusions: Enhancing the acquisition of CHEPs can be achieved by increasing the baseline temperature. This effect can be attributed, in part, to shortening the stimulation duration.Significance: After SCI, increasing the baseline temperature for CHEPs in dermatomes with absent or diminished sensation improved the neurophysiological resolution of afferent sparing. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636725</comments>
            <pubDate>Mon, 03 Oct 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5636725</guid>        </item>
        <item>
            <title>Limb segment vibration modulates spinal reflex excitability and muscle mRNA expression after spinal cord injury</title>
            <link>http://www.medworm.com/index.php?rid=5636722&amp;cid=s_35404_168_f&amp;fid=38452&amp;url=http%3A%2F%2Fwww.clinph-journal.com%2Farticle%2FPIIS1388245711005451%2Fabstract%3Frss%3Dyes</link>
            <description>Conclusions: Vibration of the lower leg inhibits the H-reflex amplitude, influences post-activation depression, and alters skeletal muscle mRNA expression of genes associated with synaptic plasticity.Significance: Limb segment vibration may offer a long term method to reduce spinal reflex excitability after SCI. (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5636722</comments>
            <pubDate>Mon, 03 Oct 2011 04:00:00 +0100</pubDate>
            <guid isPermaLink="false">5636722</guid>        </item>
        <item>
            <title>F wave to height or limb length ratios as rational alternatives for F wave latency in clinical electrodiagnostic medicine.</title>
            <link>http://www.medworm.com/index.php?rid=5269286&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D21530387%26dopt%3DAbstract</link>
            <description>CONCLUSIONS: Using the Fmin/He or Fmin/L ratios are more accurate than reporting only the F wave minimum latency, since these ratios incorporate corrections for height and/or limb length. This method also eliminates the false positive and negative reports caused by using a single reference table for all heights.
    SIGNIFICANCE: A unique reference table can be produced using the Fmin/He or Fmin/L for all populations and races, so that the comparison between different population samples can be done easily. Also data of different investigations can be pooled together for meta-analysis purposes when a single reference table is used for different samples.
    PMID: 21530387 [PubMed - in process] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5269286</comments>
            <pubDate>Sat, 01 Oct 2011 00:04:46 +0100</pubDate>
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        <item>
            <title>A cautionary note on the interpretation of phase-locking estimates with concurrent changes in power.</title>
            <link>http://www.medworm.com/index.php?rid=5269285&amp;cid=s_35404_25_f&amp;fid=35404&amp;url=http%3A%2F%2Fwww.ncbi.nlm.nih.gov%2Fentrez%2Fquery.fcgi%3Ftmpl%3DNoSidebarfile%26db%3DPubMed%26cmd%3DRetrieve%26list_uids%3D21543253%26dopt%3DAbstract</link>
            <description>Authors: Muthukumaraswamy SD, Singh KD
    PMID: 21543253 [PubMed - in process] (Source: Clinical Neurophysiology)</description>
            <author>Clinical Neurophysiology</author>
            <type>journals</type>
        <comments>http://www.medworm.com/rss/comments.php?id=5269285</comments>
            <pubDate>Sat, 01 Oct 2011 00:04:37 +0100</pubDate>
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