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Condition: Obesity
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Evaluation of cardiac autonomic function and low-grade inflammation in children with obesity living in the Northeast Brazilian region
CONCLUSIONS: The findings suggest that children with obesity have impaired autonomic function and systemic low-grade inflammation compared to children within the normal weight range, the inflammatory biomarkers were correlated with HRV parameters in schoolchildren living in the northeastern region of Brazil.PMID:37597533 | DOI:10.1016/j.jped.2023.07.003
Source: Jornal de Pediatria - August 19, 2023 Category: Pediatrics Authors: Paulo C ésar Trindade Costa Joelma Rodrigues de Souza Poliana Correia Lima Davyson Barbosa Duarte Thallyta Alanna Ferreira Viana das Neves Joicy Karla Grangeiro Pereira Cristiane Cosmo Silva-Luis R úbia Cartaxo Squizato de Moraes Valdir de Andrade Braga Source Type: research
Imperatorin alleviates metabolic and vascular alterations in high-fat/high-fructose diet-fed rats by modulating adiponectin receptor 1, eNOS, and p47 < sup > phox < /sup > expression
Eur J Pharmacol. 2021 Mar 9:174010. doi: 10.1016/j.ejphar.2021.174010. Online ahead of print.ABSTRACTIn the present study, the therapeutic effects of imperatorin on metabolic and vascular alterations and possible underlying mechanisms were investigated in high-fat/high-fructose diet (HFFD)-fed rats. Male Sprague-Dawley rats were fed a high-fat diet plus 15% fructose in drinking water for 16 weeks. HFFD-fed rats were treated with imperatorin (15 or 30 mg/kg/day) for the last 4 weeks. In HFFD-fed rats, imperatorin significantly reduced obesity, hypertension, dyslipidemia, and insulin resistance. Imperatorin markedly improved...
Source: European Journal of Pharmacology - March 12, 2021 Category: Drugs & Pharmacology Authors: Sarawoot Bunbupha Patoomporn Prasarttong Anuson Poasakate Putcharawipa Maneesai Poungrat Pakdeechote Source Type: research
Vascular Effects of Disrupting Endothelial mTORC1 (Mechanistic Target of Rapamycin Complex 1) Signaling in Obesity
In this study, we utilized animal models and human tissues to examine the role of vascular mTORC1 signaling in the endothelial dysfunction associated with obesity. In mice, obesity induced by high fat/high sucrose diet feeding for ~2 months resulted in aortic endothelial dysfunction without appreciable changes in vascular mTORC1 signaling. On the other hand, chronic high fat diet feeding (45% or 60% kcal: ~9 months) in mice resulted in endothelial dysfunction associated with elevated vascular mTORC1 signaling. Endothelial cells and visceral adipose vessels isolated from obese humans display a trend toward elevated mTORC1 s...
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology - June 30, 2021 Category: Physiology Authors: John J Reho Deng-Fu Guo Andreas M Beyer Lauren Wegman-Points Gary L Pierce Kamal Rahmouni Source Type: research
Metformin alleviates monoamine oxidase-related vascular oxidative stress and endothelial dysfunction in rats with diet-induced obesity
In conclusion, METF elicited vascular protective effects via the mitigation of MAO-related oxidative stress in the rat model of diet-induced obesity.PMID:34216348 | DOI:10.1007/s11010-021-04194-2
Source: Molecular and Cellular Biochemistry - July 3, 2021 Category: Biochemistry Authors: Loredana N Ionic ă Laura Gai ță Anca M B înă Raluca So șdean Rodica Lighezan Alexandra Sima Daniel Mali ța Octavian M Cre țu Ovidiu Burlacu Danina M Muntean Adrian Sturza Source Type: research
Vascular Effects of Disrupting Endothelial mTORC1 (Mechanistic Target of Rapamycin Complex 1) Signaling in Obesity
In this study, we utilized animal models and human tissues to examine the role of vascular mTORC1 signaling in the endothelial dysfunction associated with obesity. In mice, obesity induced by high fat/high sucrose diet feeding for ~2 months resulted in aortic endothelial dysfunction without appreciable changes in vascular mTORC1 signaling. On the other hand, chronic high fat diet feeding (45% or 60% kcal: ~9 months) in mice resulted in endothelial dysfunction associated with elevated vascular mTORC1 signaling. Endothelial cells and visceral adipose vessels isolated from obese humans display a trend toward elevated mTORC1 s...
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology - June 30, 2021 Category: Physiology Authors: John J Reho Deng-Fu Guo Andreas M Beyer Lauren Wegman-Points Gary L Pierce Kamal Rahmouni Source Type: research
Metformin alleviates monoamine oxidase-related vascular oxidative stress and endothelial dysfunction in rats with diet-induced obesity
In conclusion, METF elicited vascular protective effects via the mitigation of MAO-related oxidative stress in the rat model of diet-induced obesity.PMID:34216348 | DOI:10.1007/s11010-021-04194-2
Source: Molecular and Cellular Biochemistry - July 3, 2021 Category: Biochemistry Authors: Loredana N Ionic ă Laura Gai ță Anca M B înă Raluca So șdean Rodica Lighezan Alexandra Sima Daniel Mali ța Octavian M Cre țu Ovidiu Burlacu Danina M Muntean Adrian Sturza Source Type: research
Vascular Effects of Disrupting Endothelial mTORC1 (Mechanistic Target of Rapamycin Complex 1) Signaling in Obesity
In this study, we utilized animal models and human tissues to examine the role of vascular mTORC1 signaling in the endothelial dysfunction associated with obesity. In mice, obesity induced by high fat/high sucrose diet feeding for ~2 months resulted in aortic endothelial dysfunction without appreciable changes in vascular mTORC1 signaling. On the other hand, chronic high fat diet feeding (45% or 60% kcal: ~9 months) in mice resulted in endothelial dysfunction associated with elevated vascular mTORC1 signaling. Endothelial cells and visceral adipose vessels isolated from obese humans display a trend toward elevated mTORC1 s...
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology - June 30, 2021 Category: Physiology Authors: John J Reho Deng-Fu Guo Andreas M Beyer Lauren Wegman-Points Gary L Pierce Kamal Rahmouni Source Type: research
Metformin alleviates monoamine oxidase-related vascular oxidative stress and endothelial dysfunction in rats with diet-induced obesity
In conclusion, METF elicited vascular protective effects via the mitigation of MAO-related oxidative stress in the rat model of diet-induced obesity.PMID:34216348 | DOI:10.1007/s11010-021-04194-2
Source: Molecular and Cellular Biochemistry - July 3, 2021 Category: Biochemistry Authors: Loredana N Ionic ă Laura Gai ță Anca M B înă Raluca So șdean Rodica Lighezan Alexandra Sima Daniel Mali ța Octavian M Cre țu Ovidiu Burlacu Danina M Muntean Adrian Sturza Source Type: research
Vascular Effects of Disrupting Endothelial mTORC1 (Mechanistic Target of Rapamycin Complex 1) Signaling in Obesity
In this study, we utilized animal models and human tissues to examine the role of vascular mTORC1 signaling in the endothelial dysfunction associated with obesity. In mice, obesity induced by high fat/high sucrose diet feeding for ~2 months resulted in aortic endothelial dysfunction without appreciable changes in vascular mTORC1 signaling. On the other hand, chronic high fat diet feeding (45% or 60% kcal: ~9 months) in mice resulted in endothelial dysfunction associated with elevated vascular mTORC1 signaling. Endothelial cells and visceral adipose vessels isolated from obese humans display a trend toward elevated mTORC1 s...
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology - June 30, 2021 Category: Physiology Authors: John J Reho Deng-Fu Guo Andreas M Beyer Lauren Wegman-Points Gary L Pierce Kamal Rahmouni Source Type: research
Metformin alleviates monoamine oxidase-related vascular oxidative stress and endothelial dysfunction in rats with diet-induced obesity
In conclusion, METF elicited vascular protective effects via the mitigation of MAO-related oxidative stress in the rat model of diet-induced obesity.PMID:34216348 | DOI:10.1007/s11010-021-04194-2
Source: Molecular and Cellular Biochemistry - July 3, 2021 Category: Biochemistry Authors: Loredana N Ionic ă Laura Gai ță Anca M B înă Raluca So șdean Rodica Lighezan Alexandra Sima Daniel Mali ța Octavian M Cre țu Ovidiu Burlacu Danina M Muntean Adrian Sturza Source Type: research
