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Nutrition: Calcium

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Total 39934 results found since Jan 2013.

NADPH Oxidase 1 Mediates Acute Blood Pressure Response to Angiotensin II by Contributing to Calcium Influx in Vascular Smooth Muscle Cells
CONCLUSIONS: NOX1 mediates Ca2+ signal generation and thereby contributes to vascular contraction and blood pressure elevation by Ang II.PMID:35354309 | DOI:10.1161/ATVBAHA.121.317239
Source: Arteriosclerosis, Thrombosis and Vascular Biology - March 31, 2022 Category: Cardiology Authors: Jung-Min Park Van Quan Do Yoon-Seok Seo Hyun Jong Kim Joo Hyun Nam Ming Zhe Yin Hae Jin Kim Sung Joon Kim Kathy K Griendling Moo-Yeol Lee Source Type: research

Trimethylamine-N-Oxide Promotes Vascular Calcification Through Activation of NLRP3 (Nucleotide-Binding Domain, Leucine-Rich-Containing Family, Pyrin Domain-Containing-3) Inflammasome and NF- κB (Nuclear Factor κB) Signals.
CONCLUSIONS: This study for the first time demonstrates that TMAO promotes vascular calcification through activation of NLRP3 inflammasome and NF-κB signals, suggesting the potential link between gut microbial metabolism and vascular calcification. Reducing the levels of TMAO could become a potential treatment strategy for vascular calcification in chronic kidney disease. PMID: 31941382 [PubMed - as supplied by publisher]
Source: Arteriosclerosis, Thrombosis and Vascular Biology - January 15, 2020 Category: Cardiology Authors: Zhang X, Li Y, Yang P, Liu X, Lu L, Chen Y, Zhong X, Li Z, Liu H, Ou C, Yan J, Chen M Tags: Arterioscler Thromb Vasc Biol Source Type: research

Arterial Stiffness: A Focus on Vascular Calcification and Its Link to Bone Mineralization.
Abstract This review focuses on the association between vascular calcification and arterial stiffness, highlighting the important genetic factors, systemic and local microenvironmental signals, and underlying signaling pathways and molecular regulators of vascular calcification. Elevated oxidative stress appears to be a common procalcification factor that induces osteogenic differentiation and calcification of vascular cells in a variety of disease conditions such as atherosclerosis, diabetes mellitus, and chronic kidney disease. Thus, the role of oxidative stress and oxidative stress-regulated signals in vascular...
Source: Arteriosclerosis, Thrombosis and Vascular Biology - April 1, 2020 Category: Cardiology Authors: Chen Y, Zhao X, Wu H Tags: Arterioscler Thromb Vasc Biol Source Type: research

Vitamin D Receptor From VSMCs Regulates Vascular Calcification During CKD: A Potential Role for miR-145a
CONCLUSIONS: Our study provides evidence proving that inhibition of local VDR signaling in VSMCs could prevent VC in CKD and indicates a possible role for miR-145a in this process.PMID:37381989 | DOI:10.1161/ATVBAHA.122.318834
Source: Arteriosclerosis, Thrombosis and Vascular Biology - June 29, 2023 Category: Cardiology Authors: Maite Caus Cristina Alonso-Montes Jose Luis Fernandez-Martin Manuel Marti-Antonio Milica Bozic Jose M Valdivielso Source Type: research

Aberrant Splicing Induced by Dysregulated Rbfox2 Produces Enhanced Function of CaV1.2 Calcium Channel and Vascular Myogenic Tone in HypertensionNovelty and Significance Vascular Myogenic Tone
Calcium influx from activated voltage-gated calcium channel CaV1.2 in vascular smooth muscle cells is indispensable for maintaining myogenic tone and blood pressure. The function of CaV1.2 channel can be optimized by alternative splicing, one of post-transcriptional modification mechanisms. The splicing factor Rbfox2 is known to regulate the CaV1.2 pre-mRNA alternative splicing events during neuronal development. However, Rbfox2’s roles in modulating the key function of vascular CaV1.2 channel and in the pathogenesis of hypertension remain elusive. Here, we report that the proportion of CaV1.2 channels with alternative e...
Source: Hypertension - November 8, 2017 Category: Cardiology Authors: Yingying Zhou, Jia Fan, Huayuan Zhu, Li Ji, Wenyong Fan, Isha Kapoor, Yue Wang, Yuan Wang, Guoqing Zhu, Juejin Wang Tags: Ion Channels/Membrane Transport, Vascular Biology, Hypertension Original Articles Source Type: research