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Nutrition: Calcium

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Total 39934 results found since Jan 2013.

Non-Endoplasmic Reticulum-Based Calr (Calreticulin) Can Coordinate Heterocellular Calcium Signaling and Vascular Function.
CONCLUSIONS: Our data suggest that Calr specifically at HIEL may act in a non-ER dependent manner to regulate arteriolar heterocellular communication and blood pressure. PMID: 29122814 [PubMed - as supplied by publisher]
Source: Arteriosclerosis, Thrombosis and Vascular Biology - November 9, 2017 Category: Cardiology Authors: Biwer LA, Good ME, Hong K, Patel RK, Agrawal N, Looft-Wilson R, Sonkusare SK, Isakson BE Tags: Arterioscler Thromb Vasc Biol Source Type: research

Synthesis of Extracellular Pyrophosphate Increases in Vascular Smooth Muscle Cells During Phosphate-Induced Calcification.
CONCLUSIONS: Pyrophosphate synthesis increases in VSMCs during phosphate-induced calcification because of compensatory regulation of extracellular pyrophosphate metabolism. PMID: 30002059 [PubMed - as supplied by publisher]
Source: Arteriosclerosis, Thrombosis and Vascular Biology - July 12, 2018 Category: Cardiology Authors: Villa-Bellosta R Tags: Arterioscler Thromb Vasc Biol Source Type: research

Fibroblast growth factor 21; review on its participation in vascular calcification pathology.
Abstract Vascular calcification (VC) is an independent cardiovascular event and also a complication commonly found in chronic kidney disease (CKD) and diabetic patients. The mechanisms underpinning pathophysiology of VC is yet to be fully understood. Nevertheless, certain processes are generally believed to participate in its onset and progression. VC pathology is characterized by disequilibrium in the amount of natural inhibitors and active inducers of VC process. The imbalance may favor ectopic deposition of calcium-phosphate in form of hydroxyapatite in media or intima tunica compartments of blood vessels. This...
Source: Vascular Pharmacology - December 23, 2019 Category: Drugs & Pharmacology Authors: Olapoju Samuel O, Isaiah AO, Xoan LT Tags: Vascul Pharmacol Source Type: research

HuR (Human Antigen R) Regulates the Contraction of Vascular Smooth Muscle and Maintains Blood Pressure.
CONCLUSIONS: Our findings indicate that HuR regulates vascular smooth muscle contraction and maintains blood pressure by modulating RGS expression. PMID: 32075416 [PubMed - as supplied by publisher]
Source: Arteriosclerosis, Thrombosis and Vascular Biology - February 19, 2020 Category: Cardiology Authors: Liu S, Jiang X, Lu H, Xing M, Qiao Y, Zhang C, Zhang W Tags: Arterioscler Thromb Vasc Biol Source Type: research

Inhibition of endoplasmic reticulum stress protected DOCA-salt hypertension-induced vascular dysfunction.
Abstract Hypertension has complex vascular pathogenesis and therefore the molecular etiology remains poorly elucidated. Endoplasmic reticulum stress (ERS), which is a condition of the unfolded/misfolded protein accumulation in the endoplasmic reticulum, has been defined as a potential target for cardiovascular disease. In the present study, the effects of ERS inhibition on hypertension-induced alterations in the vessels were investigated. In male Wistar albino rats, hypertension was induced through unilateral nephrectomy, deoxycorticosterone-acetate (DOCA) injection (20 mg/kg, twice a week) and 1% NaCl with 0.2%...
Source: Vascular Pharmacology - November 17, 2018 Category: Drugs & Pharmacology Authors: Han S, Bal NB, Sadi G, Usanmaz SE, Tuglu MM, Uludag MO, Demirel-Yilmaz E Tags: Vascul Pharmacol Source Type: research

Differential Control of Calcium Homeostasis and Vascular Reactivity by Ca2+/Calmodulin-Dependent Kinase II Calcium Channels and Vasoconstriction
The multifunctional Ca2+/calmodulin-dependent kinase II (CaMKII) is activated by vasoconstrictors in vascular smooth muscle cells (VSMC), but its impact on vasoconstriction remains unknown. We hypothesized that CaMKII inhibition in VSMC decreases vasoconstriction. Using novel transgenic mice that express the inhibitor peptide CaMKIIN in smooth muscle (TG SM-CaMKIIN), we investigated the effect of CaMKII inhibition on L-type Ca2+ channel current (ICa), cytoplasmic and sarcoplasmic reticulum Ca2+, and vasoconstriction in mesenteric arteries. In mesenteric VSMC, CaMKII inhibition significantly reduced action potential duratio...
Source: Hypertension - July 17, 2013 Category: Cardiology Authors: Prasad, A. M., Nuno, D. W., Koval, O. M., Ketsawatsomkron, P., Li, W., Li, H., Shen, F. Y., Joiner, M.-l. A., Kutschke, W., Weiss, R. M., Sigmund, C. D., Anderson, M. E., Lamping, K. G., Grumbach, I. M. Tags: ACE/Angiotension receptors, Calcium cycling/excitation-contraction coupling, Hypertension - basic studies, Ion channels/membrane transport, Other Vascular biology Calcium Channels and Vasoconstriction Source Type: research

The effects of anti-hypertensive drugs and the mechanism of hypertension in vascular smooth muscle cell-specific ATP2B1 knockout mice
; Kouichi Tamura
Source: Hypertension Research - February 5, 2018 Category: Cardiology Authors: Yuki Okuyama Nobuhito Hirawa Megumi Fujita Akira Fujiwara Yosuke Ehara Keisuke Yatsu Koichiro Sumida Minako Kagimoto Mari Katsumata Yusuke Kobayashi Sanae Saka Satoshi Umemura Kouichi Tamura Tags: ATP2B1 calcium channel blocker vascular smooth muscle cell Source Type: research

Calcium phosphate product level as a predictor for arteriovenous fistula re-operations in patients with chronic renal failure.
CONCLUSIONS: High calcium phosphate product level leads to increased risk of arteriovenous fistula re-operation by causing arterial stiffness in this patient group. Additionally, these re-operations place additional burden on morbidity and cost efficacy. Thus, we recommend keeping the calcium phosphate product level at the optimal level in these patients to avoid both the risk of arteriovenous fistula re-operation and the other cardiovascular problems. PMID: 30463499 [PubMed - as supplied by publisher]
Source: Vascular - November 21, 2018 Category: Surgery Authors: Tüysüz ME, Dedemoğlu M Tags: Vascular Source Type: research

Skeletal and cardiovascular consequences of a positive calcium balance during hemodialysis.
Authors: Elias RM, Moe S, Moysés RMA Abstract Patients on hemodialysis are exposed to calcium via the dialysate at least three times a week. Changes in serum calcium vary according to calcium mass transfer during dialysis, which is dependent on the gradient between serum and dialysate calcium concentration (d[Ca]) and the skeleton turnover status that alters the ability of bone to incorporate calcium. Although underappreciated, the d[Ca] can potentially cause positive calcium balance that leads to systemic organ damage, including associations with mortality, myocardial dysfunction, hemodynamic tolerability, vascul...
Source: Jornal Brasileiro de Nefrologia - October 28, 2020 Category: Urology & Nephrology Tags: J Bras Nefrol Source Type: research

Performance of fecal S100A12 as a novel non-invasive diagnostic biomarker for pediatric inflammatory bowel disease: a systematic review and meta-analysis
CONCLUSION: Fecal S100A12 may serve as an accurate and non-invasive tool for diagnosing pediatric IBD.PMID:37094752 | DOI:10.1016/j.jped.2023.03.002
Source: Jornal de Pediatria - April 21, 2023 Category: Pediatrics Authors: Bendix Samarta Witarto Visuddho Visuddho Andro Pramana Witarto Mahendra Tri Arif Sampurna Abyan Irzaldy Source Type: research