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Cardiology MCQ 322: Plastic bronchitis
Plastic bronchitis is seen after: a) ASD repair b) Arterial switch c) Fontan repair d) VSD closure ["Click here for the answer with explanation", "Correct Answer:"] c) Fontan repair Plastic bronchitis is characterised by marked obstruction of the large airways due to the formation of bronchial casts with rubber like consistency. Elevated central venous pressure leading to endobronchial lymph leakage is thought to be the mechanism of plastic bronchitis in post Fontan state. The post Cardiology MCQ 322: Plastic bronchitis appeared first on Cardiophile MD.
Source: Cardiophile MD - September 26, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiac Surgery Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 327: Change in systolic pressure with IABP
Change in systolic pressure with IABP in augmented beats: a) Falls by 10% b) Rises by 15% c) No change d) Biphasic response ["Click here for the answer with explanation", "Correct Answer:"] a) Falls by 10% During intra aortic balloon pump (counterpulsation) or IABP, a balloon kept in the descending aorta is inflated during diastole and deflated during systole. Hence the augmented diastolic pressure is above the systolic pressure generated by the failing left ventricle. Systolic pressure of augmented beats is lower than that of non augmented beats because the left ventricle pumps into an aorta that is virtually empty ...
Source: Cardiophile MD - September 28, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Angiography and Interventions Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 326: Omapatrilat
Omapatrilat inhibits: a) Angiotensin converting enzyme b) Neprilysin c) Aminopeptidase P d) All of the above ["Click here for the answer with explanation", "Correct Answer:"] d) All of the above Omapatrilat inhibits angiotensin converting enzyme, neprilysin, and aminopeptidase P [McMurray JJ et al. Angiotensin-neprilysin inhibition versus enalapril in heart failure. N Engl J Med. 2014 Sep 11;371(11):993-1004]. Neprilysin is a neutral endopeptidase which degrades vasoactive peptides like natriuretic peptides, bradykinin and adrenomedullin. The post Cardiology MCQ 326: Omapatrilat appeared first on Cardiophile MD.
Source: Cardiophile MD - September 28, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 324: Add on therapy in heart failure
Preferred add on therapy to ACE inhibitors and beta blockers in heart failure: a) Angiotensin receptor blocker b) Mineralocorticoid antagonist c) Renin antagonist d) None of the above ["Click here for the answer with explanation", "Correct Answer:"] b) Mineralocorticoid antagonist Mineralocorticoid antagonist like spironolactone or eplerenone is the preferred add on therapy to angiotensin converting enzyme inhibitors (ACEI) and beta blockers in the management of heart failure. Angiotensin receptor blockers (ARB) and renin antagonist (e.g. aliskiren) are not preferred. ARBs with proven benefit in heart failure like va...
Source: Cardiophile MD - September 28, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 323: Beta blocker in heart failure
Which of the following beta blockers had no benefit demonstrated in treatment of heart failure? a) Carvedilol b) Metoprolol extended release c) Nebivolol d) Bucindolol ["Click here for the answer with explanation", "Correct Answer:"] d) Bucindolol Bucindolol is a beta blocker with partial agonist activity, which did not reduce mortality in heart failure trial. The post Cardiology MCQ 323: Beta blocker in heart failure appeared first on Cardiophile MD.
Source: Cardiophile MD - September 28, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 327: Neurocardiogenic syncope
Asystole is not seen in type … of neurocardiogenic syncope: a) Type I b) Type IIa c) Type IIb d) None of the above ["Click here for the answer with explanation", "Correct Answer:"] b) Type IIa In type IIA, there is no asystole, but heart falls below 40 per minute and remains so for over 10 seconds without any asystole of more than 3 seconds. In cardioinhibitory type IIB, there is asystole of more than 3 seconds. Read more… The post Cardiology MCQ 327: Neurocardiogenic syncope appeared first on Cardiophile MD.
Source: Cardiophile MD - September 30, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance ECG / Electrophysiology Source Type: blogs

Cardiology MCQ 328: Giant coronary aneurysms
Giant coronary aneurysms are seen in: a) Kartagener Syndrome b) Ellis-van Creveld Syndrome c) Kawasaki disease d) None of the above ["Click here for the answer with explanation", "Correct Answer:"] c) Kawasaki disease The post Cardiology MCQ 328: Giant coronary aneurysms appeared first on Cardiophile MD.
Source: Cardiophile MD - October 1, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: General Cardiology Source Type: blogs

Cardiology MCQ 328: Neurocardiogenic syncope
Asystole is not seen in type … of neurocardiogenic syncope: a) Type I b) Type IIa c) Type IIb d) None of the above ["Click here for the answer with explanation", "Correct Answer:"] b) Type IIa In type IIA, there is no asystole, but heart falls below 40 per minute and remains so for over 10 seconds without any asystole of more than 3 seconds. In cardioinhibitory type IIB, there is asystole of more than 3 seconds. Read more… The post Cardiology MCQ 328: Neurocardiogenic syncope appeared first on Cardiophile MD.
Source: Cardiophile MD - September 30, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance ECG / Electrophysiology Source Type: blogs

Cardiology MCQ 329: Giant coronary aneurysms
Giant coronary aneurysms are seen in: a) Kartagener Syndrome b) Ellis-van Creveld Syndrome c) Kawasaki disease d) None of the above ["Click here for the answer with explanation", "Correct Answer:"] c) Kawasaki disease Read more on long term follow up of giant coronary aneurysms in Kawasaki disease… The post Cardiology MCQ 329: Giant coronary aneurysms appeared first on Cardiophile MD.
Source: Cardiophile MD - October 1, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 331: Fallacies of assessing mitral valve area by planimetry
Fallacies of assessing mitral valve area by planimetry: a) Inability to get a cut at the tip of the funnel b) Inability to get a cut parallel to the orifice c) Inability to get a full circle at the right time of the cardiac cycle d) All of the above ["Click here for the answer with explanation", "Correct Answer:"] d) All of the above Read more… The post Cardiology MCQ 331: Fallacies of assessing mitral valve area by planimetry appeared first on Cardiophile MD.
Source: Cardiophile MD - October 2, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance Echocardiogram Library Source Type: blogs

Cardiology MCQ 330: Angiotensin II receptor
Angiotensin II receptor 1 (AT1) is involved in: a) Vasoconstriction b) Proliferation c) Aldosterone secretion d) All of the above ["Click here for the answer with explanation", "Correct Answer:"] d) All of the above Angiotensin II receptors are AT 1, 2, 3 and 4. AT 1 is the most important and is involved in vasoconstriction, proliferation, matrix formation and aldosterone secretion. The post Cardiology MCQ 330: Angiotensin II receptor appeared first on Cardiophile MD.
Source: Cardiophile MD - October 2, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 332: Catheter thrombosis with Fondaparinux
Catheter thrombosis with Fondaparinux is due to: a) High anti-Factor Xa activity b) Low anti-Factor IIa activity c) High factor VIIIa activity d) Low factor Xa activity ["Click here for the answer with explanation", "Correct Answer:"] b) Low anti-Factor IIa activity Catheter related thrombosis is higher with Fondaparinux unless supplemented with heparin. This is because of low anti-Factor IIa activity. Fondaparinux has relatively pure anti-Factor Xa activity. Unfractionated heparin has the best anti-Factor IIa activity. The post Cardiology MCQ 332: Catheter thrombosis with Fondaparinux appeared first on Cardiophile MD.
Source: Cardiophile MD - October 3, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Angiography and Interventions Cardiology MCQ Coronary Interventions DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 333: Tricuspid stenosis
Minimum gradient required for diagnosis of tricuspid stenosis: a) 2 mm Hg b) 4 mm Hg c) 6 mm Hg d) 8 mm Hg ["Click here for the answer with explanation", "Correct Answer:"] a) 2 mm Hg The minimum transvalvular gradient required for significant tricuspid stenosis is 2mm Hg. Cath derived pull back gradient may not be superior to Doppler echo derived gradient as cath gradient ideally requires simultaneous right atrial and right ventricular tracing. The post Cardiology MCQ 333: Tricuspid stenosis appeared first on Cardiophile MD.
Source: Cardiophile MD - October 3, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Cardiology MCQ DM / DNB Cardiology Entrance Source Type: blogs

Cardiology MCQ 332: Catheter thrombosis with Fondaparinux
Catheter thrombosis with Fondaparinux is due to: a) High anti-Factor Xa activity b) Low anti-Factor IIa activity c) High factor VIIIa activity d) Low factor Xa activity ["Click here for the answer with explanation", "Correct Answer:"] b) Low anti-Factor IIa activity Catheter related thrombosis is higher with Fondaparinux unless supplemented with heparin. This is because of low anti-Factor IIa activity. Fondaparinux has relatively pure anti-Factor Xa activity. Unfractionated heparin has the best anti-Factor IIa activity. The post Cardiology MCQ 332: Catheter thrombosis with Fondaparinux appeared first on Cardiophile MD.
Source: Cardiophile MD - October 3, 2014 Category: Cardiology Authors: Prof. Dr. Johnson Francis MD, DM, FACC, FRCP Edin, FRCP London Tags: Angiography and Interventions Cardiology MCQ Coronary Interventions DM / DNB Cardiology Entrance Source Type: blogs